trigram

This is a table of type trigram and their frequencies. Use it to search & browse the list to learn more about your study carrel.

trigram	frequency
of ang ii	478
in patients with	324
angiotensin converting enzyme	247
of the renin	202
as well as	199
of ace in	186
of the ras	181
the development of	176
of angiotensin ii	158
of the ace	149
the role of	146
ang ii and	145
angiotensin ii type	141
the expression of	138
acute respiratory syndrome	132
the effects of	130
the presence of	126
activation of the	121
the at receptor	121
severe acute respiratory	120
ii type receptor	118
is associated with	111
in the brain	110
ace and ace	106
in the heart	106
the treatment of	106
of ace and	105
been shown to	104
expression of ace	103
the use of	103
angiotensin ii receptor	102
role of the	100
role in the	98
to ang ii	98
ang ii levels	95
the pathogenesis of	94
in addition to	91
in order to	91
the ace ang	90
the effect of	90
respiratory distress syndrome	89
it has been	87
acute respiratory distress	87
ang ii to	86
and ang ii	85
converting enzyme in	85
in the kidney	85
effects of ang	85
ace in the	83
ang ii in	83
patients with covid	82
the synthesis of	82
the regulation of	82
levels of ang	81
due to the	80
of angiotensin converting	80
involved in the	78
smooth muscle cells	78
acute lung injury	77
the formation of	77
one of the	76
the ang ii	76
the production of	76
components of the	75
respiratory syndrome coronavirus	75
of the angiotensin	74
has been shown	73
angiotensin system in	73
by ang ii	73
the mas receptor	73
the activation of	73
an increase in	71
of ang i	70
actions of ang	68
an important role	66
role of ace	66
converting enzyme inhibition	65
effect of angiotensin	65
in response to	65
in the pathogenesis	64
ang ii at	64
of ace inhibitors	63
was associated with	63
ace expression in	62
a role in	62
role of angiotensin	62
and angiotensin ii	61
that ang ii	61
converting enzyme inhibitors	60
in ang ii	59
ii in the	59
a variety of	59
have shown that	58
effects of the	58
of the raas	58
in heart failure	57
spontaneously hypertensive rats	57
the progression of	57
on the other	56
of at r	56
in the rat	56
the at r	56
blood pressure and	56
expression in the	55
in the treatment	54
in the regulation	54
of patients with	54
based on the	54
ang i and	53
the angiotensin ii	53
ii to ang	53
induced lung injury	53
functional receptor for	53
formation of ang	53
for the treatment	53
converting enzyme and	52
in the lung	52
vascular smooth muscle	52
renin angiotensin system	52
ang i to	51
of the at	51
effects of ace	51
of the peptide	51
the other hand	51
the discovery of	51
the present study	51
conversion of ang	51
angiotensin ii in	50
in the presence	50
converting enzyme is	50
in this study	50
of the ang	50
ang ii is	50
from ang ii	50
apelin ko mice	49
levels of ace	49
ii at r	49
the actions of	49
important role in	49
the activity of	49
ace and ang	48
the release of	48
shown to be	48
the levels of	47
the importance of	47
after myocardial infarction	47
of blood pressure	47
in the lungs	47
a crucial role	47
blood pressure in	47
expressed in the	46
structure of the	46
in blood pressure	46
ace is a	45
ace ang ii	45
i to ang	45
central nervous system	45
ace inhibitors and	44
angiotensin i to	44
chronic kidney disease	44
angiotensin receptor blockers	44
is involved in	44
found to be	44
of heart failure	44
with ang ii	44
in the human	44
i to angiotensin	44
a number of	43
the ace gene	43
in the development	43
contribute to the	43
expression of the	43
as a result	43
the existence of	42
tumor necrosis factor	42
at and at	42
receptor for sars	42
beneficial effects of	41
in animal models	41
has been reported	41
patients with hypertension	41
the conversion of	40
production of ang	40
in vitro and	40
in the present	40
the renin angiotensin	40
of ace inhibition	40
effects of angiotensin	40
receptor for the	40
the action of	39
a novel angiotensin	39
be involved in	39
and at r	39
ace inhibitor therapy	39
loss of ace	39
a role for	38
at r and	38
activity of the	38
ang ii by	38
a series of	38
at r axis	38
reactive oxygen species	38
was shown to	37
of angiotensin i	37
associated with the	37
the generation of	37
blockade of the	37
actions of angiotensin	37
we found that	37
studies have shown	36
from ang i	36
of angiotensin peptides	36
concentrations of ang	36
a reduction in	35
are associated with	35
contributes to the	35
mas receptor axis	35
to be a	35
the absence of	35
related to the	35
the active site	35
plasma levels of	35
the central nervous	35
angiotensin ii and	35
angiotensin type receptor	35
i and ang	35
responsible for the	34
the pathophysiology of	34
ace activity in	34
axis of the	34
the cardiovascular system	34
is expressed in	34
converting enzyme activity	34
analysis of the	34
converting enzyme gene	34
heart failure and	34
has been found	34
part of the	34
an ace inhibitor	34
and in vivo	34
distribution of ace	33
associated with increased	33
with heart failure	33
of ace is	33
in endothelial cells	33
well as the	33
and blood pressure	33
was found to	33
the increase in	33
converts angiotensin i	33
the level of	33
according to the	32
of the sars	32
angiotensin system and	32
inhibition of the	32
converting enzyme inhibitor	32
such as the	32
clinical characteristics of	32
downregulation of ace	32
a result of	32
ii and ang	32
enzyme in the	32
and at receptors	32
a systematic review	32
the g protein	32
have been identified	32
of the n	32
ang ii infusion	32
and activation of	31
is an essential	31
also known as	31
be associated with	31
vitro and in	31
there is a	31
to be the	31
in spontaneously hypertensive	31
in contrast to	31
and ace in	31
inhibitors and angiotensin	31
has also been	31
may contribute to	31
regulation of the	31
addition to the	31
has not been	30
associated with a	30
understanding of the	30
for ang ii	30
a decrease in	30
transforming growth factor	30
endothelial growth factor	30
coupled receptor mas	30
some of the	30
at receptors in	30
the loss of	30
vascular endothelial growth	30
increased ang ii	30
acute lung failure	30
of ace protein	30
the ras in	30
activity in the	29
changes in the	29
that ace is	29
peptides by human	29
in rats with	29
receptors in the	29
ace and the	29
and heart failure	29
of the mas	29
human homolog of	29
cells in the	29
ang ii receptor	29
is an endogenous	29
compared with the	29
the nlrp inflammasome	29
increased levels of	29
expression of angiotensin	29
a functional receptor	29
mouse model of	29
effect of ace	28
levels in the	28
hydrolysis of biological	28
ace activity and	28
of cardiac hypertrophy	28
been reported to	28
biological peptides by	28
cardiovascular and renal	28
of the two	28
congestive heart failure	28
showed that the	28
the brain ras	28
the spike protein	28
the involvement of	28
the degradation of	28
in the central	28
li et al	28
for the sars	28
converting enzyme protects	28
severe acute lung	28
of the enzyme	28
effect of the	28
a human homolog	28
increase in ang	28
of heart function	28
of biological peptides	28
plays an important	28
the severity of	27
cloning and functional	27
functional expression as	27
leading to the	27
patients with heart	27
enzyme protects from	27
the fact that	27
was used to	27
system in the	27
patients with severe	27
in the ace	27
in women with	27
and ace activity	27
ras in the	27
ii receptor blockers	27
to determine the	27
expression as a	27
cells of the	27
and functional expression	27
the risk of	27
crucial role of	27
binding to the	27
in angiotensin ii	27
in which the	27
by angiotensin ii	27
cardiac hypertrophy and	27
compared to the	27
have been shown	27
of at receptors	26
an essential regulator	26
the ang tie	26
and in the	26
high levels of	26
in combination with	26
may lead to	26
the inhibition of	26
ang tie system	26
evidence for a	26
regulator of the	26
this article is	26
essential regulator of	26
amino acid residues	26
activity of ace	26
increased expression of	26
regulator of heart	26
of the brain	26
recombinant human ace	26
of the disease	26
of the classical	26
effect of ang	26
the aim of	26
role of ang	26
ligand for the	26
the gastrointestinal tract	25
in a rat	25
animal models of	25
a rat model	25
have been reported	25
to evaluate the	25
found in the	25
review and meta	25
which can be	25
the sars coronavirus	25
chronic heart failure	25
at r in	25
play a role	25
the surface of	25
enzyme inhibitors and	25
blood pressure regulation	25
the cell surface	25
effect on the	25
nitric oxide synthase	25
induced cardiac hypertrophy	25
of diabetic nephropathy	25
and at receptor	25
from severe acute	25
systematic review and	25
angiotensin ii receptors	25
is a functional	25
in the ras	25
a mouse model	25
stimulation of the	25
the ratio of	25
of the human	25
by human angiotensin	25
and angiotensin receptor	25
ang ii type	25
is known to	25
generation of ang	25
of cyp b	25
found that the	25
homolog of angiotensin	25
the ability of	24
morbidity and mortality	24
the reninangiotensin system	24
have demonstrated that	24
at room temperature	24
deletion of the	24
of ace expression	24
is mediated by	24
in the absence	24
in cardiovascular disease	24
in sars coronavirus	24
enzyme and angiotensin	24
due to its	24
first step in	24
for sars coronavirus	24
of amino acids	24
the balance between	24
as shown in	24
ace knockout mice	24
and treatment of	24
inflammation and fibrosis	24
ang ii or	24
the ras is	24
differences in the	24
pulmonary arterial hypertension	24
the structure of	24
of severe acute	24
of a novel	24
the cytokine storm	24
sodium and water	24
in the cardiovascular	24
angiotensin ii at	24
observed in the	23
patients with chronic	23
tissue distribution of	23
ace as a	23
enzyme is a	23
an angiotensin ii	23
the functional receptor	23
is one of	23
it is not	23
the heart and	23
highly expressed in	23
santos et al	23
a recent study	23
of the reninangiotensin	23
there is no	23
on blood pressure	23
most of the	23
increase in the	23
ace is also	23
coronary artery disease	23
an endogenous ligand	23
treatment of covid	23
with coronavirus disease	23
enzyme is an	23
acts as a	23
the beneficial effects	23
protects from severe	23
the control of	23
to the development	23
the function of	23
conflict of interest	23
the design of	23
all rights reserved	23
of the kidney	23
the prevention of	23
protective role of	23
both ace and	22
high blood pressure	22
and function of	22
converting enzyme angiotensin	22
step in understanding	22
in vitro studies	22
in the circulation	22
to study the	22
in the pathophysiology	22
is an important	22
protein of sars	22
a first step	22
the interaction of	22
ang ii formation	22
the classical ras	22
lung injury in	22
of the most	22
endogenous ligand for	22
as a potential	22
known to be	22
the lack of	22
rat model of	22
the upregulation of	22
structure and function	22
of nitric oxide	22
results showed that	22
increase in ace	22
as a captopril	22
which in turn	22
the case of	22
in this work	22
the ace angiotensin	22
the mechanism of	22
ace is the	22
amyotrophic lateral sclerosis	22
the b receptor	22
of cardiovascular disease	22
caused by the	22
within the kidney	22
binds to the	22
in individuals with	22
for the g	22
in understanding sars	22
the concentrations of	22
understanding sars pathogenesis	22
this study was	22
retrospective cohort study	21
in the case	21
the plasma membrane	21
the concentration of	21
of recombinant human	21
the metabolism of	21
ace and tmprss	21
patients with coronavirus	21
of the c	21
hek t cells	21
of acute lung	21
in acute respiratory	21
arm of the	21
of myocardial infarction	21
protected by copyright	21
h c cells	21
and mortality in	21
thought to be	21
the ras system	21
of ace inhibitor	21
article is protected	21
ang i is	21
oxidative stress and	21
with normal pregnancies	21
and the renin	21
mrna and protein	21
may be a	21
as a therapeutic	21
of human angiotensin	21
it is possible	21
suggest that the	21
be due to	21
is protected by	21
as a receptor	21
ii and na	21
in the early	21
in a mouse	21
the number of	21
ace activity was	21
r and at	21
of the severe	21
amino acids and	21
the reduction in	20
campbell et al	20
play an important	20
present in the	20
in this review	20
at receptor antagonists	20
the administration of	20
crucial role in	20
is thought to	20
inhibition and angiotensin	20
are presented as	20
mediated by the	20
h n virus	20
administration of ang	20
has been described	20
of coronavirus disease	20
in experimental models	20
is increased in	20
the novel coronavirus	20
expression of ang	20
were found to	20
was carried out	20
for the development	20
is responsible for	20
epithelial cells and	20
expression and activity	20
of the first	20
was able to	20
benefits of ace	20
that can be	20
ang ii was	20
enzyme inhibition and	20
the cell membrane	20
and characterization of	20
the ras and	20
of inflammatory cytokines	20
synthesis of the	20
form of ace	20
its role in	20
well as in	20
the ace inhibitor	20
effects on the	20
of the complement	20
therapeutic target for	20
converted to ang	20
is required for	20
ectodomain shedding of	20
has been demonstrated	20
are involved in	20
of ace ang	20
the majority of	20
many of the	20
of at receptor	20
mas axis in	20
ace inhibition in	20
for the synthesis	19
inflammatory effects of	19
is reduced in	19
expression of at	19
been found to	19
shedding of the	19
as compared to	19
will be presented	19
those of ang	19
a retrospective cohort	19
alveolar epithelial cells	19
endothelial cells and	19
for the first	19
results suggest that	19
a range of	19
the immune system	19
the reduction of	19
at r expression	19
at the c	19
to those of	19
acei or arb	19
the binding of	19
data suggest that	19
sympathetic nervous system	19
reduction in the	19
the context of	19
several studies have	19
and oxidative stress	19
been identified in	19
enzyme activity in	19
in diabetic nephropathy	19
a receptor for	19
by ace inhibition	19
ace gene expression	19
protective effect of	19
between ace and	19
is the main	19
i and ii	19
shown that the	19
bradykinin b receptor	19
regulation of ace	19
myocardial infarction in	19
a key role	19
the most important	19
was supported by	19
to angiotensin ii	19
have also been	19
the severe acute	19
the first time	19
the s protein	19
on ang ii	19
with ace inhibitors	19
revealed that the	19
may also be	19
acute myocardial infarction	19
the ace receptor	19
infected with sars	19
on the cell	19
ace inhibitors or	19
the amino acid	19
a protective role	18
at least in	18
serve as a	18
implicated in the	18
by ace inhibitors	18
in the mouse	18
patients infected with	18
function of the	18
effects in the	18
view of the	18
to that of	18
on the surface	18
role for the	18
in this regard	18
ang ii effects	18
transgenic mice with	18
in the renal	18
to generate ang	18
and lung injury	18
our understanding of	18
the degree of	18
mrna expression of	18
to investigate the	18
women with pe	18
increased in the	18
is a key	18
ace levels in	18
by means of	18
is the most	18
appears to be	18
receptor blockers on	18
with ace inhibitor	18
has been suggested	18
ace has been	18
was observed in	18
and expression of	18
and progression of	18
led to the	18
women with normal	18
is characterized by	18
the stimulation of	18
to have a	18
converting enzyme type	18
which is the	18
of this study	18
the result of	18
in failing human	17
levels and activity	17
protective effects of	17
cleavage of the	17
shown in fig	17
is a potent	17
the angiotensin converting	17
the cleavage of	17
ang ii into	17
leads to the	17
the authors declare	17
in the context	17
and the at	17
decrease in the	17
at receptor antagonist	17
of the renal	17
ace expression is	17
is present in	17
urinary levels of	17
consistent with the	17
role for ace	17
lung tumor tissue	17
component of the	17
important role of	17
experimental models of	17
in the renin	17
were observed in	17
a critical role	17
use of ace	17
been associated with	17
vsels and hscs	17
as previously described	17
plasma renin activity	17
this work was	17
with novel coronavirus	17
functions as a	17
of novel coronavirus	17
via the at	17
levels of the	17
the distribution of	17
ace levels and	17
regulation of blood	17
resulting in the	17
diabetic kidney disease	17
response to ang	17
identified in the	17
binding of the	17
vascular endothelial cells	17
expression in human	17
of the protein	17
the combination of	17
in vascular smooth	17
ang ii production	17
in the kidneys	17
the spontaneously hypertensive	17
supplementary figure s	17
classical raas axis	17
be related to	17
action of ang	17
through activation of	17
a central role	17
used for the	17
interaction with the	17
aldosterone system inhibitors	17
is the first	17
p r o	17
hospitalized patients with	17
amino acid transporter	17
zhang et al	17
inhibitors in patients	17
ii levels in	17
hypertrophy and fibrosis	16
wide variety of	16
the healthy individuals	16
circulating levels of	16
the cells were	16
target for the	16
to understand the	16
inhibitors and arbs	16
the induction of	16
associated with covid	16
ace activity is	16
the immune response	16
receptor in the	16
it was found	16
the blood pressure	16
coronavirus spike protein	16
it is known	16
studies on the	16
the raas is	16
can be used	16
the identification of	16
ace levels were	16
when compared with	16
induced by ang	16
overexpression of ace	16
failing human heart	16
be responsible for	16
domain of ace	16
been reported in	16
as a consequence	16
db db mice	16
is located on	16
in hypertensive patients	16
results of the	16
in the proximal	16
but not in	16
by the action	16
normal lung tissue	16
ace is an	16
acute kidney injury	16
and neprilysin in	16
in terms of	16
of the novel	16
to reduce the	16
the proximal tubule	16
involvement of the	16
activated protein kinase	16
are mediated by	16
mas receptor in	16
basis for the	16
the course of	16
ace protein expression	16
patients with diabetes	16
left ventricular dysfunction	16
gene expression in	16
to produce ang	16
heart and kidney	16
from patients with	16
heart failure patients	16
in a variety	16
the structure and	16
ace expression and	16
production of angiotensin	16
roles of angiotensin	16
as compared with	16
donoghue et al	16
mrna expression in	16
no conflict of	16
to play a	16
cell proliferation and	16
suppressed ang ii	16
been detected in	16
and protein expression	16
ace and neprilysin	16
for ace in	16
in cardiovascular and	16
approved by the	16
function of ace	16
left ventricular hypertrophy	16
systolic blood pressure	16
identified as a	16
ang ii ang	16
evidence suggests that	15
with type diabetes	15
the complement system	15
mortality in patients	15
critically ill patients	15
in ace expression	15
upregulation of the	15
of podocyte injury	15
ang ii can	15
in relation to	15
in the shr	15
to inhibit the	15
j o u	15
roles of the	15
been found in	15
l p r	15
r n a	15
expression levels of	15
ang i into	15
n a l	15
blood pressure elevation	15
in association with	15
oxidative stress in	15
for heart failure	15
in the plasma	15
and regulation of	15
in the angiotensin	15
and septic shock	15
we have shown	15
p r e	15
studies have demonstrated	15
o u r	15
and risk of	15
the in vivo	15
associated with an	15
in the same	15
ii and angiotensin	15
in the liver	15
characterization of a	15
upregulation of ace	15
at receptor stimulation	15
indicate that the	15
of neutral endopeptidase	15
with the ace	15
physiological and pathological	15
did not affect	15
presence of ace	15
expression of mir	15
concentration of ang	15
r o o	15
the results of	15
the rate of	15
to increase the	15
and nitric oxide	15
cd t cells	15
effects of losartan	15
was performed in	15
regulated ectodomain shedding	15
spontaneously hypertensive rat	15
in critically ill	15
hypertensive rats angiotensin	15
phase peptide synthesis	15
of ace by	15
are needed to	15
mediates regulated ectodomain	15
negative regulator of	15
of ace on	15
u r n	15
and cardiovascular disease	15
decreased expression of	15
and found that	15
may be involved	15
treated with ace	15
presence of the	15
considered to be	15
and smooth muscle	15
of ace to	15
airway epithelial cells	15
activation of ace	15
previous studies have	15
is a major	15
cardiovascular effects of	15
the onset of	15
interaction of the	15
a l p	15
at receptor blockade	15
the mineralocorticoid receptor	15
ace is expressed	15
a therapeutic target	15
genetic deletion of	15
was found that	15
o o f	15
insights into the	15
actions of the	15
ang ii were	15
heart failure in	15
reduction of the	15
to the at	15
the significance of	15
a model of	15
risk factors for	15
of these peptides	15
at receptor activation	15
in the ang	15
of angiotensinconverting enzyme	15
it is well	15
action of angiotensin	15
expression in rat	15
results in the	15
which is a	15
aim of this	15
of angiotensin receptor	15
can also be	15
of cell volume	15
ang ii has	15
increased ace expression	15
of oxidative stress	15
rostral ventrolateral medulla	14
active site of	14
the mechanisms of	14
upregulation of mir	14
have not been	14
in some cases	14
individuals with diabetes	14
of the protective	14
in the gut	14
in a study	14
vickers et al	14
key role in	14
ang ii signaling	14
may play a	14
at receptor and	14
degradation of ang	14
ace inhibitors in	14
of resveratrol on	14
ischaemic heart disease	14
or ang ii	14
by the ace	14
the possibility of	14
angiotensin ii induces	14
and risk factors	14
ace in human	14
the decrease in	14
protein expression of	14
patients treated with	14
plasminogen activator inhibitor	14
a wide variety	14
is found in	14
axis in the	14
the hypothesis that	14
metabolism in the	14
patients with type	14
of ace activity	14
to the formation	14
was reported to	14
there were no	14
may be an	14
spike protein of	14
blocked by a	14
found that ace	14
by binding to	14
i converting enzyme	14
fluid and electrolyte	14
ferrario et al	14
for ace and	14
the recognition of	14
by inhibiting the	14
risk factor for	14
activation of nf	14
cleaved by ace	14
may be due	14
metabolism of angiotensin	14
corresponding to the	14
wide range of	14
risk of covid	14
a study of	14
in acute lung	14
stimulation of at	14
syndrome coronavirus infection	14
of hypertension and	14
used as a	14
and in vitro	14
and fibrosis in	14
for multiple comparisons	14
be used in	14
it is also	14
that the expression	14
have been found	14
were purchased from	14
demonstrated that the	14
of action of	14
increased blood pressure	14
treatment of hypertension	14
mechanism of action	14
crystal structure of	14
nucleus of the	14
of the active	14
blood pressure by	14
and can be	14
plasma ang ii	14
development of new	14
metabolism of ang	14
release of vasopressin	14
have been developed	14
the components of	14
in the spontaneously	14
is known that	14
the downregulation of	14
addition to its	14
of the study	14
diseases such as	14
in subjects with	14
lung injury and	14
central role in	14
has recently been	14
p and mir	14
were able to	14
of ras in	14
than that of	14
similar to those	14
essential role in	14
reported that the	14
and clinical studies	14
a combination of	14
ace is not	14
demonstrated that ang	14
severity of covid	14
be used as	14
mice per group	14
suggests that the	14
the benefits of	14
ratio of ace	14
there was no	14
of the alternative	14
leading to a	14
of hospitalized patients	14
n mice per	14
inhibition of ace	14
decrease in ace	14
the extent of	13
these data suggest	13
significantly higher than	13
critical role in	13
c bl mice	13
in comparison with	13
the balance of	13
clinical course and	13
peptide in the	13
characterization of the	13
significantly increased in	13
inhibitors of angiotensin	13
identification of a	13
at receptor blockers	13
is a novel	13
intensive care unit	13
ace inhibitor or	13
the ability to	13
east respiratory syndrome	13
and ace levels	13
effects of captopril	13
may be the	13
that activation of	13
vascular inflammation and	13
cyp b and	13
the sympathetic nervous	13
a role of	13
and its receptor	13
and is associated	13
of reactive oxygen	13
importance of ace	13
is critical for	13
with regard to	13
and h c	13
analysis revealed that	13
in the expression	13
as a key	13
remodeling and pah	13
brosnihan et al	13
cov and sars	13
to ace inhibitors	13
of a new	13
no effect on	13
was identified as	13
blockers on cardiac	13
loss of angiotensin	13
balance between the	13
use of angiotensin	13
lung injury angiotensin	13
on ace and	13
antihypertensive effects of	13
angiotensin receptor blocker	13
experimental and clinical	13
has been proposed	13
discovery of ace	13
found that ang	13
characterized by a	13
i into ang	13
with an increase	13
in serum of	13
been identified as	13
have been proposed	13
middle east respiratory	13
contrast to the	13
is blocked by	13
been suggested that	13
an at receptor	13
bind to the	13
of the rat	13
that has been	13
cytokines such as	13
of the effects	13
the biological activity	13
in the pulmonary	13
for treatment of	13
system inhibitors in	13
a potential therapeutic	13
responses to ang	13
and angiotensin converting	13
the association of	13
development of cardiac	13
the classical raas	13
profiling of ace	13
in the s	13
cytokines and chemokines	13
the preparation of	13
that the ace	13
ang ii accumulation	13
insertion deletion polymorphism	13
of these drugs	13
evidence for the	13
and tmprss and	13
in vivo and	13
ii to the	13
the same time	13
enzyme inhibition in	13
ii into ang	13
been implicated in	13
plays a role	13
at r is	13
the rostral ventrolateral	13
in the nucleus	13
the amount of	13
studies are needed	13
lung injury by	13
a reduction of	13
is cleaved by	13
in the metabolism	13
an essential role	13
from the cell	13
depends on ace	13
and safety of	13
of the heart	13
in our study	13
in transgenic rats	13
cardiac function and	13
expressed on the	13
ras has been	13
expression profiling of	13
shedding of ace	13
or at receptor	13
in ace levels	13
but did not	13
ace ace ratio	13
and chronic kidney	13
has been identified	13
and has been	13
is highly expressed	13
from the rat	13
ace inhibitors are	13
gs and bs	13
of the nlrp	13
of ace has	13
for disease control	13
presented as the	13
in a model	13
ang ii could	13
acei and arb	13
remains to be	13
mechanism by which	13
proven to be	13
conversion of angiotensin	13
gene expression of	13
of the receptor	13
with severe covid	13
regulation of cardiovascular	13
at the n	13
to form ang	13
endothelial dysfunction and	13
also associated with	13
at the same	12
step in the	12
for min at	12
higher risk of	12
there is an	12
normotensive and hypertensive	12
against ang ii	12
released into the	12
was confirmed by	12
serum levels of	12
may have a	12
mortality of adult	12
shown that ace	12
the association between	12
such as hypertension	12
ii receptor antagonist	12
to assess the	12
was assessed by	12
has been observed	12
been proposed to	12
homolog of ace	12
inhibitors and at	12
of this system	12
angiotensin and bradykinin	12
ang ii are	12
the receptor for	12
prevention and treatment	12
in human airway	12
specificity of the	12
of cardiovascular function	12
the study was	12
cell entry depends	12
in ace knockout	12
located on the	12
shown to have	12
functions of the	12
impact on the	12
factors for mortality	12
in kidney disease	12
the impact of	12
as a new	12
or arb use	12
together with the	12
are shown in	12
the management of	12
were analyzed using	12
and ace inhibitors	12
to interact with	12
at receptor in	12
by a clinically	12
role for ang	12
removal of the	12
synthesis of a	12
such as il	12
potential therapeutic target	12
inhibition of sars	12
members of the	12
levels of angiotensin	12
patients with acute	12
of the glomerular	12
of this peptide	12
for patients with	12
risk of cancer	12
in the cardiac	12
recombinant human angiotensin	12
concentrations of resveratrol	12
of the cytokine	12
structural basis for	12
de ang ii	12
induced cellular hypertrophy	12
there was a	12
we have developed	12
leads to a	12
which could be	12
the peptide in	12
new class of	12
by the use	12
cleavage of ang	12
in plasma and	12
human ace in	12
inhibition of ang	12
between the two	12
work was supported	12
in severe covid	12
biological activity of	12
the current study	12
of sars coronavirus	12
formation of the	12
viral entry into	12
that of the	12
with essential hypertension	12
at or at	12
angiotensin at receptor	12
were incubated with	12
ace plays a	12
by the at	12
as an important	12
in the gastrointestinal	12
reported to be	12
at receptor gene	12
is also a	12
and the ace	12
and activity of	12
is important to	12
higher than that	12
to be associated	12
is due to	12
effects of at	12
ii at receptor	12
ko mice showed	12
in ace and	12
the interaction between	12
induced acute lung	12
like stem cells	12
endothelial dysfunction in	12
dysregulation of the	12
upregulation of angiotensin	12
chen et al	12
not able to	12
compared to ang	12
as the mean	12
dependent on the	12
therapy in patients	12
ang ii may	12
there are no	12
of the b	12
at a receptor	12
inpatients with covid	12
type ii pneumocytes	12
and patients with	12
it is a	12
the maintenance of	12
control and prevention	12
is possible that	12
and na in	12
of the peptides	12
a clinically proven	12
are thought to	12
adult inpatients with	12
evidence that the	12
been shown that	12
molecular mechanisms of	12
of the central	12
homologue of ace	12
the angiotensin type	12
has shown that	12
angiotensin aldosterone system	12
the introduction of	12
activation of prorenin	12
this suggests that	12
of ace was	12
to be involved	12
data not shown	12
showed that ang	12
very small embryonic	12
to improve the	12
the influence of	12
proven protease inhibitor	12
in the generation	12
a wide range	12
clinically proven protease	12
and is blocked	12
by blockade of	12
level of ang	12
course and risk	12
of type diabetes	12
kidney disease and	12
of somatic ace	12
reduction in ace	12
regulation of cell	12
entry depends on	12
by ace to	12
the most common	12
tmprss and is	12
in the progression	12
amino acid transporters	12
mediated by at	12
of adult inpatients	12
mutations in the	12
for mortality of	12
ii is a	11
have been described	11
the detection of	11
the renin receptor	11
based on a	11
the development and	11
endothelial nitric oxide	11
ii production and	11
of the role	11
complexity of the	11
the complexity of	11
fetal bovine serum	11
in rat brain	11
disease in china	11
avian influenza virus	11
higher levels of	11
binding to ace	11
is able to	11
in the current	11
is induced by	11
activation of at	11
peptide levels in	11
due to their	11
were synthesized and	11
roles in the	11
increased production of	11
of an angiotensin	11
polymerase chain reaction	11
a cytokine storm	11
enzyme in human	11
pathways of the	11
ii by ace	11
in these patients	11
may serve as	11
health and disease	11
smooth muscle cell	11
into ang ii	11
on the x	11
based on these	11
glomerular basement membrane	11
shown that ang	11
in apelin ko	11
increase in blood	11
study of the	11
ace transgenic mice	11
human angiotensinconverting enzyme	11
which has been	11
enzyme inhibition on	11
which leads to	11
binding of ang	11
can lead to	11
we showed that	11
converting enzymerelated carboxypeptidase	11
progression of diabetic	11
to explore the	11
to the ace	11
contributed to the	11
circulating ang ii	11
further studies are	11
angiotensin i converting	11
converting enzyme homologue	11
in rat and	11
the expression and	11
ang i by	11
within the brain	11
regions of the	11
its interaction with	11
to prevent the	11
the basis of	11
blood pressure control	11
ace inhibition is	11
and inflammation in	11
of the activation	11
localized to the	11
cardiac and renal	11
the adrenal gland	11
cytokine storm and	11
findings suggest that	11
effects of these	11
ang ii binds	11
angiotensin peptides in	11
gs bs patients	11
ace null mice	11
the synthesis and	11
development of a	11
the x chromosome	11
left ventricular remodeling	11
the metabolite formation	11
ace mrna expression	11
with respect to	11
the antihypertensive effects	11
as ang ii	11
the incidence of	11
innate immune response	11
in the formation	11
in engineered human	11
effect of losartan	11
referred to as	11
ii binds to	11
infusion of ang	11
contributing to the	11
was significantly reduced	11
similar to that	11
the attachment of	11
ace mrna and	11
in angiotensin peptide	11
weeks of gestation	11
ace may be	11
gene expression and	11
type receptor blockade	11
suggested that the	11
been linked to	11
at receptor expression	11
in the rostral	11
on cardiac angiotensin	11
blood pressure responses	11
human angiotensin i	11
located on chromosome	11
these findings suggest	11
disease control and	11
administration of recombinant	11
to the antihypertensive	11
of the s	11
angiotensin peptides and	11
the growth of	11
at least two	11
effects of ras	11
seem to be	11
high level of	11
modulation of the	11
effect of resveratrol	11
myocardial infarction and	11
the setting of	11
blood flow and	11
in vascular endothelial	11
also been shown	11
ii levels were	11
genetic ace deficiency	11
is converted to	11
the ras has	11
seems to be	11
and sudden death	11
an upregulation of	11
study showed that	11
the observation that	11
by activation of	11
first time that	11
of left ventricular	11
the kidney of	11
the complement cascade	11
region of the	11
ang ii also	11
are responsible for	11
efficacy and safety	11
glomerular injury in	11
ii and the	11
blockade of angiotensin	11
mechanism of the	11
was determined by	11
in the aorta	11
salt and water	11
demonstrated that ace	11
blood pressure was	11
pathogenesis of covid	11
american heart association	11
for the prevention	11
et al b	11
in accordance with	11
forming activity in	11
resulted in the	11
i or ang	11
role in maintaining	11
a significant role	11
the proximal tubules	11
elevated blood pressure	11
to form the	11
been demonstrated to	11
with acute respiratory	11
two active sites	11
the brain and	11
with chronic heart	11
ang ii concentration	11
amino acid sequence	11
in the production	11
of h n	11
a and b	11
were significantly higher	11
heart failure with	11
homologue of angiotensin	11
proximal tubular cells	11
of normal and	11
protein kinase c	11
surface of the	11
data indicate that	11
activity in failing	11
xu et al	11
lead to a	11
ace and its	11
months after ot	11
of the patients	11
ang ii treatment	11
to ang i	11
suggesting that the	11
it is important	11
type receptor in	11
the application of	11
properties of the	11
localization of the	11
of bradykinin by	11
cigarette smoke exposure	11
the results showed	11
were treated with	11
ang i or	11
monocyte chemoattractant protein	11
in comparison to	11
has been associated	11
we demonstrated that	11
ii receptor subtypes	11
due to a	11
avian influenza a	11
of human ace	10
in the setting	10
the changes in	10
in human plasma	10
be used to	10
be an important	10
the nucleus tractus	10
a consequence of	10
effects of feb	10
in diabetic rats	10
axes of the	10
a review of	10
of ace as	10
that in the	10
ang ii as	10
we investigated the	10
evidence for upregulation	10
of ace are	10
solid phase synthesis	10
in essential hypertension	10
generation of angiotensin	10
and vascular smooth	10
it was shown	10
been observed in	10
mortality and morbidity	10
formation rate of	10
ii receptor blocker	10
et al a	10
insight into the	10
of these two	10
the kidney and	10
isolated perfused rat	10
amino acids from	10
inhibits growth of	10
respiratory syncytial virus	10
receptor blockers and	10
data are presented	10
related to increased	10
for upregulation of	10
in normal and	10
the subfornical organ	10
ang ii stimulation	10
of renin and	10
has been detected	10
belongs to the	10
the protein expression	10
in human and	10
potential target for	10
cancer cell lines	10
wang et al	10
h n influenza	10
probably due to	10
to the c	10
through inhibition of	10
by the angiotensin	10
result in the	10
driven by the	10
plays a crucial	10
podocyte injury in	10
to ace expression	10
in the prevention	10
in the cell	10
least in part	10
following myocardial infarction	10
in gs bs	10
outcomes in patients	10
coronavirus disease in	10
tissue levels of	10
to examine the	10
large number of	10
with the development	10
the circulating ras	10
the proliferation of	10
vascular remodeling and	10
significant reduction of	10
and development of	10
prevention of angiotensin	10
action of ace	10
cells and the	10
no significant difference	10
mechanisms of the	10
in the cns	10
expression of cyp	10
the process of	10
and its expression	10
also showed that	10
the brain of	10
in line with	10
actions of ace	10
its ability to	10
mice treated with	10
and endothelial dysfunction	10
rats with myocardial	10
those of the	10
was found in	10
active sites of	10
is capable of	10
severe and critical	10
type diabetes and	10
on the structure	10
the angiotensin receptor	10
of severe covid	10
pivotal role of	10
protein in the	10
studies have reported	10
terminal catalytic domain	10
sudden death in	10
be mediated by	10
derived from the	10
that there is	10
a previous study	10
to the renin	10
a potential target	10
receptor is the	10
with at r	10
the lungs of	10
beneficial effect of	10
amino acid from	10
results from the	10
angiotensin system angiotensin	10
acei and arbs	10
with hypertension and	10
in clinical trials	10
is based on	10
of cardiac myocytes	10
a significant reduction	10
the site of	10
in the hearts	10
at receptor axis	10
of natriuretic peptides	10
are present in	10
depending on the	10
to an increase	10
proteolytic cleavage of	10
in this context	10
was performed using	10
in transgenic mice	10
is regulated by	10
compared with non	10
followed by the	10
of endogenous ang	10
angiotensin ii on	10
world health organization	10
from the c	10
the protective role	10
based on this	10
was significantly higher	10
and inhibition of	10
infusion of angiotensin	10
regulation of angiotensin	10
with high affinity	10
metabolite formation rate	10
crackower et al	10
angiotensin system blockers	10
been found that	10
of the lungs	10
human heart ventricles	10
in the control	10
characterization of angiotensin	10
that ace may	10
the united states	10
of patients infected	10
advanced glycation end	10
and water retention	10
severe heart failure	10
authors declare that	10
is well known	10
did not have	10
by treatment with	10
significantly different from	10
of mas receptor	10
were synthesized by	10
injury in mice	10
their ability to	10
the isolated perfused	10
type diabetes mellitus	10
of ace may	10
the brain renin	10
zhou et al	10
catalytic activity of	10
significantly higher in	10
and arbs in	10
cell growth and	10
the respiratory system	10
this effect was	10
of hypertension in	10
the innate immune	10
was used for	10
carried out in	10
member of the	10
r in the	10
pi k akt	10
have found that	10
and mas receptor	10
that patients with	10
as a novel	10
the secretion of	10
results indicated that	10
ang ii receptors	10
to be an	10
the specificity of	10
i d polymorphism	10
with myocardial infarction	10
in ace transgenic	10
rats angiotensin ii	10
of expression of	10
syndrome coronavirus spike	10
be explained by	10
aortic smooth muscle	10
internalization and degradation	10
infections in engineered	10
in part by	10
was increased in	10
protective role in	10
patients with primary	10
of the virus	10
high concentrations of	10
department of chemistry	10
soluble form of	10
are related to	10
hypertension and diabetes	10
preserved ejection fraction	10
plays a key	10
as evidenced by	10
and mas receptors	10
of pulmonary hypertension	10
for the recognition	10
human tissues using	10
isolated from the	10
because of its	10
has been implicated	10
the occurrence of	10
in h n	10
was significantly increased	10
research ethics committee	10
the stability of	10
system in cardiovascular	10
a major role	10
are known to	10
when compared to	10
the analysis of	10
existence of a	10
pulmonary vascular remodeling	10
the study of	10
induced by the	10
in the acute	10
glycosylation of ace	10
downregulation of the	10
inhibitors of the	10
ace can be	10
in the adult	10
the accumulation of	10
the lungs and	10
designed and synthesized	10
was blocked by	10
that the use	10
on the basis	10
characteristics of coronavirus	10
focused on the	10
in health and	10
angiotensin ii signaling	10
engineered human tissues	10
renal ang ii	10
effect of captopril	9
are required to	9
release of the	9
c cells were	9
at doses of	9
plasma and tissue	9
enzyme as a	9
in myocardial ischemia	9
the left ventricle	9
tissue ang ii	9
renal injury in	9
and other organs	9
beneficial effects in	9
induced diabetic mice	9
and activity in	9
pneumonia in wuhan	9
novel coronavirus in	9
in diabetic mice	9
as part of	9
significant role in	9
and vascular endothelial	9
at tissue sites	9
prevention of pulmonary	9
converting enzyme by	9
urinary excretion of	9
and increase in	9
of renin in	9
studies suggest that	9
role of ras	9
in cardiac hypertrophy	9
in cardiovascular diseases	9
in the late	9
which results in	9
shown to inhibit	9
death in ace	9
in lung tumor	9
there have been	9
after ang ii	9
on angiotensin and	9
hypertension in the	9
cardiac fibrosis in	9
could lead to	9
patients with left	9
of inflammatory cells	9
form of the	9
learning and memory	9
of the gut	9
ang ii with	9
well known that	9
influence on the	9
a member of	9
an animal model	9
induced cardiac fibrosis	9
in normal lung	9
is linked to	9
biologically active peptides	9
activated protein kinases	9
of the cell	9
receptor binding domain	9
increase of endocan	9
of brain ras	9
depends on the	9
ang ii from	9
were associated with	9
in the failing	9
amino acids are	9
system blockers and	9
glomerular filtration rate	9
renal function in	9
of ace gene	9
the university of	9
of the inflammatory	9
under the control	9
and water transport	9
statistical significance was	9
detected in the	9
signaling in the	9
decompensated heart failure	9
ace to ace	9
as a carboxypeptidase	9
significant increase in	9
and ulcerative colitis	9
in a number	9
overexpression of angiotensin	9
patients in the	9
was approved by	9
and may be	9
in the intestine	9
therapeutic target in	9
soluble human ace	9
conformation of the	9
with chronic kidney	9
of ace with	9
diabetes mellitus and	9
of the membrane	9
were found in	9
human coronavirus nl	9
patients with coronary	9
at r by	9
and cardiovascular diseases	9
seen in the	9
on the heart	9
compared with normal	9
in view of	9
human recombinant ace	9
in ace activity	9
most of these	9
the physiological and	9
angiotensin ii stimulates	9
the last decade	9
nitric oxide and	9
degradation of angiotensin	9
of ras inhibitors	9
severity of disease	9
correlated inversely with	9
ferreira et al	9
ii has been	9
the conformation of	9
the design and	9
to the apical	9
ace and mas	9
in keeping with	9
did not significantly	9
increases ace expression	9
b and its	9
in the serum	9
of local renin	9
there is evidence	9
cases of covid	9
this study is	9
be used for	9
infarction in rats	9
ii and its	9
in regulation of	9
angiotensin system inhibitors	9
appeared to be	9
a negative regulator	9
with a reduction	9
analysis of randomized	9
suggesting that ace	9
reduction of ace	9
control of the	9
receptor for ang	9
inhibitors of ace	9
overexpression of human	9
its homologue ace	9
of the three	9
induced cardiac dysfunction	9
of the reaction	9
the addition of	9
normal cardiac phenotype	9
serum ace and	9
cell entry and	9
results are presented	9
site in the	9
neurons in the	9
system by angiotensin	9
with left ventricular	9
cells were incubated	9
attenuates angiotensin ii	9
de la eca	9
to at r	9
ang ii synthesis	9
in the synthesis	9
cellular responses to	9
significance of the	9
release of cytokines	9
in cardiac myocytes	9
ii or ang	9
in the pancreas	9
center for disease	9
for h at	9
accumulation of ang	9
as shown by	9
in case of	9
plasma membrane of	9
design of the	9
cytochrome p b	9
ii as a	9
ace inhibitor arb	9
the incorporation of	9
and as a	9
peptides and proteins	9
for the ace	9
no significant differences	9
participate in the	9
ace inhibitor and	9
glycation end products	9
the contribution of	9
mainly in the	9
for h with	9
consistent with our	9
single amino acid	9
linked to the	9
chain hete metabolites	9
structural and functional	9
importance of the	9
development of heart	9
physiological roles of	9
society of cardiology	9
there has been	9
the angiotensin iv	9
act as a	9
treatment with the	9
deletion of angiotensin	9
produced by the	9
mouse and rat	9
is a highly	9
of spontaneously hypertensive	9
system in heart	9
reach statistical significance	9
with the aim	9
acts as an	9
as a potent	9
of angiotensin type	9
of the system	9
in rats and	9
ang ii on	9
of proinflammatory cytokines	9
to clarify the	9
aldosterone system in	9
of vascular endothelial	9
the crystal structure	9
encoded by the	9
ang ii stimulates	9
the extracellular matrix	9
of the lung	9
care and use	9
of diabetes and	9
the removal of	9
we report the	9
angiotensin ii production	9
hypotensive effect of	9
of ace mrna	9
deshotels et al	9
and pulmonary hypertension	9
a protective effect	9
the human heart	9
to result in	9
normal pregnant women	9
patients with novel	9
formation of a	9
with a new	9
mineralocorticoid receptor antagonist	9
increased risk of	9
mouse models of	9
neurons of the	9
the transcription factor	9
has led to	9
blockers and the	9
in recent years	9
early stages of	9
enzyme homologue ace	9
na in serum	9
independent of the	9
system and the	9
and kidney disease	9
in human heart	9
ace was identified	9
been attributed to	9
and severity of	9
podocyte injury and	9
is a critical	9
on the regulation	9
to increased levels	9
these results suggest	9
murine model of	9
therapeutic potential of	9
of host cells	9
of biologically active	9
growth factor receptor	9
nature of the	9
gene expression is	9
in the course	9
ii receptor type	9
renin prorenin receptor	9
and ang i	9
angiotensin peptide metabolism	9
have indicated that	9
gene is located	9
length human ace	9
development of glomerulosclerosis	9
coronary heart disease	9
physiological and pathophysiological	9
mice with downregulated	9
ace functions as	9
not reach statistical	9
showed that ace	9
could be a	9
of blockade of	9
ang ii degradation	9
peptides in the	9
at receptor is	9
renin angiotensin aldosterone	9
did not reach	9
to activation of	9
to a reduction	9
enhanced expression of	9
in the last	9
to the mas	9
interactions with the	9
of this article	9
in the hypothalamus	9
enzyme and its	9
mas r axis	9
of cardiovascular and	9
feb suppressed ang	9
cells were treated	9
treated apelin ko	9
human endothelial cells	9
the hearts of	9
of the major	9
recent studies have	9
blood flow in	9
in agreement with	9
at r has	9
was added to	9
deterioration of the	9
by activating the	9
and h o	9
is considered to	9
not associated with	9
significantly reduced in	9
p map kinase	9
the c domain	9
cardiac hypertrophy in	9
and ace are	9
discovery of the	9
of the immune	9
and it is	9
shown to reduce	9
formation in the	9
enzymatic activity of	9
a disintegrin and	9
with downregulated connexins	9
pathological cardiac hypertrophy	9
was detected in	9
western blot analysis	9
with increased ace	9
a murine model	9
is independent of	9
volume and water	9
change in the	9
the solitary tract	9
n virus infection	9
captopril on mortality	9
ii and at	9
human airway epithelia	9
a marker of	9
european society of	9
enzyme overexpression in	9
were stained with	9
potential role in	9
and that the	9
decreased ang ii	9
by the spike	9
have reported that	9
epicardial adipose tissue	9
is expected to	9
renal blood flow	9
treatment of cardiovascular	9
cardiac hypertrophy is	9
total rna was	9
physiology and pathophysiology	9
infection in the	9
ang ii generation	9
elucidation of the	9
and healthy controls	9
of the components	9
ii levels and	9
to regulate the	9
the ras are	9
against angiotensin ii	9
into the circulation	9
the hypotensive effect	9
focus on the	9
to be present	9
of lung injury	9
has emerged as	9
is a zinc	9
fold higher than	9
atrial natriuretic peptide	9
clinical features of	9
the classical axis	9
we have designed	8
and renal disease	8
systemic and local	8
higher concentrations of	8
studies have indicated	8
of cardiovascular diseases	8
diagnosis and treatment	8
mrna expression profiling	8
risk for covid	8
such as ang	8
s and s	8
of the covid	8
that administration of	8
to determine whether	8
type a receptor	8
of circulating ace	8
is important for	8
of vasopressin from	8
as bs type	8
of the amino	8
and enzymatic activity	8
chinese center for	8
infarction by blockade	8
to the plasma	8
the in vitro	8
angiotensin system blockade	8
is dependent on	8
sex differences in	8
in the covid	8
studies in mice	8
is the only	8
reninangiotensin system in	8
of pulmonary vascular	8
we have previously	8
axis has been	8
ang iv and	8
this study we	8
function as a	8
can induce the	8
in the p	8
added to the	8
in normotensive and	8
inhibition on angiotensin	8
to be effective	8
on mortality in	8
and viral replication	8
discovery of a	8
cardiac phenotype in	8
ace in cardiovascular	8
and stored at	8
hydrolysis of ang	8
than those of	8
associated with severe	8
in lung diseases	8
integrity of the	8
linked to viral	8
the concept that	8
treated with the	8
been demonstrated that	8
epidermal growth factor	8
effect of bradykinin	8
and it was	8
enzymes and receptors	8
aceis and arbs	8
randomized controlled trials	8
but not by	8
infected with novel	8
nlrp inflammasome in	8
may explain the	8
samples were collected	8
the potential of	8
use of these	8
in many tissues	8
it should be	8
into host cells	8
novel coronavirus pneumonia	8
kidney and testis	8
known that the	8
ace mrna levels	8
the acute phase	8
injury induced by	8
of ras blockade	8
in ace gene	8
at receptor blocker	8
the responses to	8
reduced in the	8
through binding to	8
were obtained from	8
cardiac dysfunction and	8
deficiency is associated	8
nitric oxide release	8
a new target	8
endoplasmic reticulum stress	8
of severe cases	8
of components of	8
the two peptides	8
myocardial infarction by	8
levels were measured	8
action of the	8
the apical surface	8
and cardiac dysfunction	8
in the process	8
renin and angiotensinogen	8
the search for	8
inhibitors or arbs	8
heart function angiotensin	8
because of the	8
a potent vasoconstrictor	8
with angiotensin ii	8
altered blood pressure	8
pathogenic avian influenza	8
of the cells	8
and endothelial cells	8
reduction of ang	8
angiotensin i and	8
in support of	8
of this receptor	8
than ang ii	8
were compared with	8
model of hypertension	8
the alternative raas	8
show that the	8
type i receptor	8
in adipose tissue	8
infected patients linked	8
explained by the	8
silva et al	8
of both ace	8
neutral amino acids	8
is inhibited by	8
not have any	8
been described in	8
the protective arm	8
to the synthesis	8
and its metabolites	8
appear to be	8
peptides and their	8
with h n	8
homologue ace and	8
into the extracellular	8
functional significance of	8
studies of the	8
approval of the	8
with our previous	8
the inflammatory response	8
the lung tumor	8
yet to be	8
suggest that ang	8
clinical trial of	8
is a type	8
with severe acute	8
role in cardiovascular	8
mas axis of	8
tubular epithelial cells	8
for cardiovascular diseases	8
signal transduction mechanisms	8
be induced by	8