Serums, Vaccines and toxines IN TREATMENT AND DIAGNOSIS WM. CECIL BOSANQUET M.A., M.D. OXON., f.t^.C.P. LOND. Physician to Out-Patients, Victoria Hospi'al for Ohildrcn Assistant Physician (late Pathologist) to Charing Cross Hospital Formerly Fellow of New College, Oxford CHICAGO: W:=^=Ff--"KEENER & CO. LONDON: CASSELL & COMPANY, LIMITED MCMIV. All Rights Reserved (XSt. PREFACE. Our knowledge of the nature of infective agents, and of the mode in which we are able to resist them, is of com- paratively recent development, and the theories now in vogue to explain the observed facts are still more novel ; so that the whole subject forms almost unknown ground to the majority of medical practitioners. Yet the importance of this branch of study is very great : it is in this direction that our insight into the nature of disease has advanced most rapidly, and that the prospects of successful treatment are most encouraging. In the present volume I have endeavoured to give a short account of the principal facts which have been ascertained, and of the general trend of speculation, on the subject of immunity— one which shall be intelligible to readers who have not had opportunities of making them selves acquainted with the vast and daily-increasing litera- ture on this branch of pathology — as well as to elucidate the practical value of the many remedial agents which have been introduced in the form of serums, vaccines and toxines. I have not attempted to discuss in detail the mode of preparation of these substances, since this is a matter which concerns expert bacteriologists and laboratory workers rather than practitioners of medicine, and is one of which I have no personal knowledge. Those who are interested in this side of the subject may refer to such vi PREFACE. books as Prof. Hewlett's " Serum Therapy," or the German work of Deutsch and Feistmantel, " Die Impfstoffe imd Sera," both of which I have consulted. I must also acknowledge my indebtedness to Marx' " Diagnostik, Serumtherapie und Prophylaxe," and to Dieudonn^'s " Immunitat, Schutzimpfung und Serumtherapie." I have as far as possible quoted original authorities throughout for statements on subjects which have not passed beyond the sphere of controversy. If I am accused of setting forth my conclusions as to the value of the different remedies with more dogmatism than is justifiable in the present state of our ignorance, I must plead that for practical purposes definite assertions are necessary. I have endeavoured to give due weight to the difficulties and disappointments met with in applying these preparations, as well as to the advantages to be expected from the use of them. Finally, 1 have included the study of diagnosis, as well as that of treatment, by methods involving the use of serums and toxines, since the connection between the subjects is so close that they can scarcely l)e separated, depending as they do upon the same general principles, and involving the action of bodies formed in response to almost identical stimuli. My very sincere thanks are due to my friend Dr. J. W. Eyre, Lecturer on Bacteriology at Guy's Hospital, for his kindness in criticising the proof-sheets : without the confidence derived from his supervision I should scarcely have ventured to undertake the duties of a pilot amid the shifting and ill-charted shoals of recent bacterio- logical speculation and research. London, May, 1904. W. C. B. CONTENTS. CHAPTER I. PAGE Historical Ketrospect 1 CHAPTER II. Recovery and liiMuxiTT . . • 11 CHAPTER III. Preparation and Administration of Serums and Vaccines . 44 CHAPTER IV. Serums and Toxines in the Diagnosis ofDisease . . .58 CHAPTER V. Diphtheria G3 CHAPTER VI. Tetanus ..... 101 CHAPTER VII. Snake-Bite 119 CHAPTER VIII. Small-Pox and Vaccinia 128 CHAPTER IX. Hydrophobia (Rabies) 148 CHAPTER X. Plague 163 viii CONTENTS. CHAPTER XI. PAOE ExTEEic Fever . . 175 CHAPTEE XII. Cholera 200 CHAPTER XIIL Affections ]iue to Streptococci (Including Diplococci) . . 208 CHAPTER XIV. Tuberculosis 235 CHAPTER XV. Malignant Tumours , . . 283 CHAPTER XVI. Other Conditions Treated by Antibacterial Methods . . 296 CHAPTER XVII. Epizootic Diseases 322 Appendix 332 ndex . 341 or THE ^ SERUMS, VACCINES, AND TOXINES IN TREATMENT AND DIAGNOSIS. CHAPTER L HISTORICAL RETROSPECT. Evolution of Therapeutics.— It is evident that a scientific method of treatment for any disease can only exist when the exact nature of the morbid process — its causation and the alterations in the functions of the body which underlie its manifestations — is thoroughly understood. All therapeutic measures which are not founded on this basis are merely empirical. Even at the present day very many of our remedies come under the latter heading ; they are found by long experience to do good in certain conditions, but of their mode of action we are still entirely ignorant. Of this nature is the use of mercury in syphilis, a disease of which we do not know the cause. Equally empirical was the discovery of the value of quinine in malaria, although the parasite which produces the malady has now been identified, and we assume that quinine pre- vents its development. In the earliest condition of the healing art of which we have records almost all remedies were of this empirical nature. A scientific pathology was impossible until a rational physiology existed, as it is impossible to distinguish and describe with accuracy morbid disturbances of health, until we know for certain what is the normal course of B 2- SEKUMS, VACCINES, AND TOXINES. fiifil p}^jiQtDeA£i. : ^imilfj rly, accurate morbid anatomy was Impossible until tlie liGrmal structure of the body was ascertained. Modern anatomy may perhaps be said to have been founded by Yesalius (1543) ; modern physiology, as far as the coarser phenomena of life are concerned, may be said to own for its parent Harvey, who discovered the circulation of the blood in 1628; but any knowledge of the finer vital actions was necessarily impossible before the microscope was brought to some degree cf perfection. Cellular physiology and cellular pathology were the fruits of the first half of the nineteenth century. Since Rudolph Yirchow set pathology on the firm basis of observed facts and rescued it from the mists of the humoral theories, advance has been steady and sure. In no department, however, has such progress been made in eluci- dating the processes of disease as in the sphere of infectious disorders. Not only has it been definitely ascertained that these are due to the invasion of the body by minute living organisms — generally belonging to the vegetable kingdom, occasionally to the animal — but the method by which these parasites produce their effects has been discovered, their toxines have been prepared artificially, and the means by which the body resists the invaders has been to some extent elucidated. On the basis of these discoveries a scientific system of treatment of the diseases caused by parasitic organisms has been worked out, and in some instances actually introduced into practice. Still fuither develop- ments of this mode of treatment are in prospect, and by proceeding along these lines it seems legitimate to hope that in the course of time we may possess remedies which will seriously reduce the mortality from infectious disease, if we cannot actually abolish such conditions altogether. The method of treatment by serums has been pushed into regions far apart from its original field of action. Serums are ofiered which profess to be curative of a number of conditions not definitely infective. A serum treatment for the morphine habit has been suggested ; another serum HISTORICAL IIETIIOSPECT. 3 is supposed to cure liay-fever ; a third has been vaunted as a remedy for Graves' disease. Most of tliese are yet in tlie experimental stage, and no certain verdict as to their efficacy can as yet be given. In the following pages we shall endeavour, as far as possible, to sift the wheat from the chatf, and to ascertain to what extent the man}'' serums announced as remedial are in reality of practical value. Since the method of treatment is still so comparatively new, many practitioners of medicine are not yet familiar with the details of the technique employed ; hence some consideration of the procedure generally adopted will also ba useful, along with criticism of the results obtained. The study of the serum of the blood has brought to light a peculiar effect (agglutination) which it produces in many diseases upon the micro-organisms which cause the condition. This reaction is useful in a number of cases for the purpose of diagnosis, and some account will therefore be given of this property ; for, although it is not actually a part of the treatment of disease, yet it is of great use in the practice of medicine, and is very closely allied to those other proDe^jiu^ of serum by which resistance to disease is brough|^Tjout. E^liest Knowledge of Immunity. — It has long been known that, in the case of many of the infectious diseases, persons who have su tiered from one attack are to some extent protected from any recurrence of the malady. In the case of small-pox, which was one of the greatest scourges of the world in ancient and mediajval times, use was made of this fact at a comj)aratively early date, as a means of conferring immunity on individuals against the disease. Inoculation from a mild case of small-pox was practised in the East long before it w^as introduced into Europe. From Turkey it was brought to England, about the year 1721, mainly by the instrumentality of Lady Mary Wortley Montagu, who had become acquainted with the method in Constantinople. Considering the almost universal 4 SERUMS, VACCINES, AND TOXINES. prevalence of the malady in those days, there can be little doubt that this method of inoculation was on the whole beneficial, for it was then exceptional for anyone to go through life without suffering from small-pox, and a face not scarred by pitting was a beauty in itself. But the practice had obvious dangers. Although the attack of the disease produced by inoculation was generally a mild one, there was no means of ensuring that such would be the case. Sometimes a severe and even fatal attack was the result. Hence, when an equally certain and much safer method of prevention was introduced, the practice of inoculation was made illegal. VaOCination. — In the year 1796 Jenner performed his first vaccination, and shortly afterwards he announced his discovery to the world — the first and, to this day, the most successful attempt at artificial immunisation against any disease. Common observation had led to the discovery that milkmaids and others who had to do with cattle were liable to contract a mild disorder (cow-pox), communicated by handling the animals when they were suffering from a peculiar vesicular eruption of the udders ; and that those who had been thus infected were in future protected to a great extent against the incidence of small-pox. Jenner investigated the subject, and ascertained that the jDopular view was well founded. He considered that the milder disease, vaccinia, was a comparatively harmless form of small-pox occurring in cattle ; and he held that, while the dangers inherent in the actual inoculation of small-pox were avoided, the protection afforded was scarcely, if at all, inferior to that conferred by an attack of the human form of the malady. Hence he advised that human beings should be vaccinated, i.e. inoculated with cow-pox, in order to protect them against the more dangerous malady. Nature of Infection. — It was, of course, impossible to give any explanation of the exact mode in which an individual who had suffered from one attack of small-pox or other infectious disease was protected against its INFECTION AND FERMENTATION. 5 recurrence, so long as the true nature of infection was unknown. This knowledge has been the fruit of the modern science of Bacteriology. Before the actual discovery of these minute fungi [Bacteria) was made, it had been sug- gested that infectious diseases were spread by conveyance of living particles from one person to another. The proba- bility of such a " contagium vivimi " rested on the fact that only an infinitesimal quantity of infectious material coidd in most cases pass from the sick person to the hitherto healthy one ; hence it seemed necessary that a multipli- cation of the infective matter must take place within the body of the latter. Such multiplication is the distin- guishing property of living matter as contrasted with ordinary poisons. Analogy with Fermentation.— The discovery by Pasteur of the cause of fermentation, which is produced by the yeast-plant (Saccharomyces cerevisice), brought the question a step nearer to solution. The resemblance between an infective disease and the process of fermentation was seen to be very close. Thus, the act of infection corresponds with the addition of yeast to a saccharine solution ; the incubation-period with the time which elapses before ferment- ative changes are seen in the liquid ; the onset of the disease with the rise of temperature and evolution of gas seen when the solution begins to ferment j and the decline of the disease with the spontaneous cessation of the saccha- rolytic process. When once Pasteur had identified the yeast-fungus as the cause of fermentation, the probability, suggested by Henle (1840), that vegetable organisms are the causal agents in infectious diseases was rendered still stronger ; and Trousseau definitely asserted that such would prove to be the case, before any pathogenic organisms were actually discovered. Bacteria. — The discovery of the first bacterium was made in 18.50 by Davaine, who identified the bacillus of anthrax or splenic fever, and showed that the disease could be conveyed by its means (18G3). At that time, however, 6 SERUMS, YACCINES, AND TOXINES. no means existed for obtaining pure cultures of micro- organisms, and some doubt consequently remained as to the truth of Davaine's conclusions. The discovery of this organism, and of the possibility of transmitting the disease by inoculation with it, opened up a vast field of research, and showed the way to our present knowledge of infective processes. The spread of discovery in this direction was very rapid. The invention by Koch of solid media on which to grow the organisms, and the possibility of thus obtaining pure cultures of the different kinds, put the new science on a sure basis. Bacteria have been identified as the causes of diphtheria, tetanus, glanders, tuberculosis, pneumonia, enteric fever, Malta fever, septicaemia and suppurative conditions, gonorrhoea, plague, relapsing fever, influenza, erysipelas, leprosy, actinomycosis, botulism, and cerebro-spinal meningitis. Organisms have also been described which have some claims to be considered the excitants of dysentery and infantile diarrhoea, of rheumatic fever, whooping cough, yellow fever, scarlatina, syphilis, and rhinoscleroma. Fresh additions are being made almost daily to the number of vegetable organisms which are pathogenic to man or the lower animals. Microscopical animal parasites are recognised as the causes of the different forms of malaria, and other organisms belonging to the group are now said to be connected with small-pox, hydro- phobia, dengue, and yellow fever. Amceba; are probably resjDonsible for one form of dysentery, and another protozoan parasite produces the disease called trypanosomiasis, and most probably causes African lethargy (sleeping sickness). Theories of their Pathogenic Action. — When the bacterial causation of infective diseases was firmly estab- lished, it was natural that various hypotheses should be put forward to account for the manner in which these minute fungi produce their pathogenic effects. The bacillus of anthrax, as it was one of the first to be discovered, was also one of the earliest to be studied in this connection. In an animal which had died of this malady immense numbers of MODE OF ACTION OF BACTERIA. 1 these comparatively large, rod-shaped organisms can be seen lying in the capillaries of most of the organs, appear- ing in places actually to block up the blood-channels. The suggestion was therefore made that a process analogous to embolism — a blocking of the smaller blood-vessels by the masses of organisms — was the cause of the morbid symptoms. This view could not^ however, be maintained in the presence of the fact, recognised later, that in other infective conditions the bacteria are comparatively few and far between, no such masses being found as occur in anthrax. In certain diseases it is even the case that the organisms remain localised in some one spot, and do not enter the general blood-stream at all (diphtheria, tetanus). Another suggestion made was that the parasites seized on the nourishment circulating in the blood, which was necessary for the life of the tissue-cells, and that so they practically starved the body, and produced their injurious effects. This was obviously no more tenable than the former theory, for the same reason. Toxines. — Finally the true facts of the matter were discovered, viz. that the micro-organisms secrete, as the products of their vital activity, certain poisonous sub- stances, which kill or injure the cells of their host and so produce disease. Much valuable work has been done towards elucidating the nature of these poisons or toxines;^ and a very long list of substances of various kinds could be drawn up, to which the actual toxic powers have been attributed. Brieger isolated poisonous alkaloids from decomposing meat, &c., and considered that it was to this class of chemical substances that bacterial toxines belonged. 1 In the following pages the word " toxine " is used in its original sense, viz. a poisonous substance formed in the growth of bacteria or other parasitic micro-organisms. It has been proposed to limit the use of the term to those bodies formed by Vacteria to w'hich antitoxines (see p. 18) are produced in infected animals, but in the present state of our knowdedge of the.>e bodies this restriction seems scarcely feasible. The term "antigen" has been coined by Marx to bear the latter meaning. 8 SERUMS, VACCINES, AND TOXINES. Sidney INIartin and others, working chiefly with the bacillus of diphtlieria, attributed a prominent role to albiunoses, and it is to this group of substances that snake-venom has been assigned. The tubercle-bacillus is said to form an acid substance which so acts upon the tissues as to produce the peculiar change known as caseation ; while in the case of yet other bacteria the poisons produced by them have been assigned to the mysterious class of substances known as ferments, none of which has ever been isolated in pure condition, but which are known only by their action in bringing about chemical changes in other matter. To this class belong the digestive ferments, pepsin and trypsin, and very similar bodies are probably formed by bacteria. Thus in the growth of the anthrax-bacillus, albumoses and pep- tone are formed, just as in digestion ; and the micrococci which give rise to suppuration {Staphylococci and Strepto- cocci) exert a peptonising action on the surrounding tissues, killing and dissolving the cells, by which means the spread of an acute abscess is brought about. Certain facts with regard to bacterial toxines point very strongly to their being of the nature of ferments. Thus when attempts are made to isolate them by means of chemical reagents, precipitation, (fee, it is possible, indeed, to separate solid matters of a poisonous nature, but these substances, of whatever chemical composition, are never so powerfully toxic as the original solution of the poison, formed by the growth of the bacteria in the culture- medium. Something always seems to be lost in the process of separation ; instead of a more concentrated poison being obtained, only a weaker variety is produced. The actual toxine eludes capture and identification. When, however, we have said that toxines are of the nature of ferments, we have done little more than confess our ignorance, for of the constitution of ferments nothing certain is known. Preparation of Toxines. — The usual method adopted for the preparation of the toxine of any micro-organism is to grow the bacterium in a suitable fluid medium (broth) PREPARATION OF TOXINES. 9 under favourable circumstances, and then either to kill the actual germs by the addition of cldoroform or some other volatile antiseptic, or, better, to strain them off by passing the fluid through a porcelain filter, by which means the poison is obtained free from the dead bodies of the bacteria. It may be easily understood that such a fluid is very com- plex in character, containing not only the poisons for which search is to be made, but also much unaltered nutrient medium, along with by-products of the gro^^'th of germs — excretory, ikc. — which are possibly harmless substances. Further, it is not possible in the case of all organisms to obtain soluble toxines in ordinary nutrient media. Some bacteria refuse to part with their toxines under these circumstances, whereas the dead bodies of the germs them- selves may have a poisonous action if injected into animals (B. typhosus, &c.). Such species are said to contain " intracellular toxines." It does not seem possible that under circumstances of disease these poisons can remain entirely within the bodies of the parasites, as it is necessary that they should be brought into contact with the cells forming the tissues of the host in order that their poisonous effects may be manifested. Hence it must be concluded that within living animals these toxines, like the extra- cellular variety, pass out of the bacteria into the surround- ing vital fluids, whereas in artificial culture-media they remain inside the organisms; in other words, the toxines are needed to enable the germs to carry on their war with the living tissues when they are parasitic, but are unnecessary when suitable food of a non-resistant nature is provided, and therefore under the latter conditions they are not formed in any abundance. It has, however, been suggested that the escape of the toxines from the body of the bacteria is effected by the destruction and disintegration of many of the germs by the tissues of the infected animal. To this it might be objected that infection must be produced by the entrance of a very small number of germs ; and it seems unlikely that any large amount of poison would be 10 SERUMS, VACCINES, AND TOXINES. forthcoming by destruction of individual bacteria, if sufficient germs are to be left to multiply and cause infection. Soluble toxines have been prepared in suitable media from the organisms of tetanus, diphtheria, and botulism, as well as from some less-distinctly pathogenic organisms, such as Proteus vulgaris, tfec. In the case of Bacillus typhosus and B, tuberculosis poisonous substances can be prepared by triturating the bodies of the bacteria themselves and extracting the residue. It will be seen in the next chapter that the power of extracting the toxines of bacteria is of considerable import- ance as enabling us to prepare specific antidotes (anti- toxines) to counteract their effects. 11 CHAPTER II. RECOVERY AXD IM:\rrNlTY. Spontaneous Recovery. — When bacteria were thus definitely established as the causal agents in a number of infectious diseases, it was natural that speculation and enquiry should be directed to the explanation of the facts observed with regard to these diseases, on lines suggested by a study of the life-history and peculiarities of the organisms. One well-known phenomenon to be explained was the tendency of the majority of infective diseases to be self-limited — to run a definite course ending in recovery. Thus, acute pneumonia tends to end suddenly about the seventh day, and typhus on the fourteenth ; other infective diseases come to a natural termination gradually in an indefinite number of days or weeks (enteric fever, broncho- pneumonia, ifec). It was necessary to enquire how this tendency of infective diseases to die out spontaneously was explicable on the parasitic theory of their origin — why the micro-organism which caused the condition should not live indefinitely in a soil which at first showed itself so suitable for its growth and development. Exhaustion-Theory. — In 1880 Pasteur put forward the hypothesis that, just as a plant cannot live for more than a certain time in a given portion of soil, dying as soon as it has exhausted the nourishment available in it, so bacteria use up all the existing material, of the special kind necessary for their growth, which is present in a given human or other animal body ; when this point is reached, they necessarily die out spontaneously. This so-called "exhaustion-theory" was, however, soon abandoned, as it was found possible to cultivate micro-organisms on the 12 SERUMS, VACCINES, AND TOXINES. juices derived from an animal which had just recovered from the very disease which was due to their acti\'ity, and in which, therefore, all available nourishment for them should theoretically have been exhausted. It was evident that, as a matter of fact, plenty of suitable food was still left. Retention-Theory. — A second theory was propounded by Chauveau, who pointed out that the excretory products of living organisms are generally poisonous to the organisms themselves, just as carbonic acid is poisonous to man and the higher animals, if respired. Chauveau therefore sug- gested that a large quantity of the excretory products of the bacteria gradually accumulated in the body of the host — the diseased animal — and that, when this amount reached a certain point, further bacterial life and development were arrested, and the disease pame to a spontaneous end. There is probably a large amount of truth in this ''retention- theory," as applied to the life of bacteria in non-living media. The life of the yeast-plant [Saccharomyces cerevisice), which gives rise to fermentation, is arrested by the presence of more than a certain percentage of the alcohol which is formed by its activity, although there may be plenty of nutrient material still left, on which it can continue to thrive if the alcohol be removed. But in living animals the excretory products of the bacteria are rapidly passed out of the system of the host, so that no such accumulation takes place as is found in artificial cultures or a fermenting- vat. Varieties of Immunity. — After the failure of these two chemical theories to explain the spontaneous cessation of bacterial activity which takes place in recovery from infective diseases, attention was turned to the possibility of the existence of some vital reaction on the part of the animal attacked, whereby it was enabled to resist the invading parasites. There were also other facts to be explained. Thus it was known that bacteria of certain species could flourish in the bodies of particular animals, PHENOMENA OF IMMUNITY. 13 but not in those of other kinds, sometimes not even in species nearly allied to the susceptible races. The animals which are not liable to suffer from a given organism are said to be " immune " to it. A few examples may Ije quoted. White rats, adult dogs, many kinds of birds, and frogs are naturally immune to the bacillus of anthrax, which is very fatal to cattle, common rats, field-mice, and man. Algerian sheep resist the organism, whereas other breeds of sheep readily succumb. Dogs are })ractically immune to tuberculosis, while guinea-pigs are killed by the most minute dose of the bacillus. Rats and mice are not susceptible to diphtheria. In none of these cases, however, is the immunity absolute. By altering the circumstances of the animal it is generally possible to render it susce[)tible to the disease. Thus, by keeping frogs at a raised tem- perature, it is possible to infect them with anthrax, and by overtiring animals by excessive work they may be made susceptible to infection with organisms to which they other- wise possess almost complete immunity. Racial and Individual Immunity.— It is not only in different species of animals that varying powers of resist- ance to particular organisms may be found. It is well known that different races of mankind possess very varying powers of resistance to diseases. Thus measles, which is a mild disorder among Europeans, took the form of a disastrously - fatal epidemic when introduced among the Sandwich Islanders, and killed enormous numbers of these natives. Negroes are little subject to yellow fever and malaria, while to small-pox they are very susceptible. Further, different individuals of the same race possess varying degrees of resistance. Many persons never suffer from scarlet fever, though they may be constantly brought into contact with the disease ; others equally resist influenza when it is epidemic all around them, whereas less fortunate individuals may suffer from it year after year. As in the case of the immunity peculiar to certain species of animals, this personal immunity may be lost owing to temporary 14 SERUMS, VACCINES, AND TOXINES. conditions of healthy as for instance fatigue or depression. Thus the case is quoted of two medical students who for a space of two months regularly visited wards containing patients suffering from scarlet fever, but did not contract the disease. One day they visited the hospital when they were over-tired with severe exercise, and had been fasting for five hours. Their resistance was lowered by these circum- stances, and both of them suffered from scarlet fever, one actually dying from the disease. i Acquired Immunity. — We thus see that immunity may be peculiar to species, races, or individuals, and that it may be modified by special circumstances. Allusion has already been made (page 3) to the form of protection conferred by an attack of an infective disease, viz. that in the case of many maladies a person is not attacked twice by the same disorder. This is not the case, indeed, in all infective diseases, for there are some in which no immunity is conferred, or in which there may even seem to be produced a special proclivity to future attacks rare in complaint. Thus, whereas second attacks are rare in small-pox, measles, scarlet fever, typhus, mumps, &c., in the case of erysipelas an increased liability to the disease seems to be brought about, and the same is perhaps true of pneumonia and influenza. Diphtheria, again, does not seem to protect against subsequent attacks, nor does se})ti- csemia ; while recent experience in South Africa has proved that second attacks of enteric fever are by no means rare. There is, however, reason to suppose that even in the latter group of diseases some temporary immunity is produced by an attack, so that the convalescent is not liable to become infected again within a certain number of weeks or months ; but the duration of this protection is £0 short that it is easily overlooked. Nature of Recovery from Disease.— It is evident that if a disease is due to the action of parasitic organisms, 1 Green's "Pathology and Morbid Anatomy," 9th Edition; Ed. by H. Montague Murray; p, 286. RESISTANCE TO DISEASE. 15 it cannot oe said to be cured till all of these are destroyed or driven out of the body. A certain number of the germs are pa^.sed out of the system in the urine, and a larger number in some diseases by means of other discharges, as in the expectoration in phthisis, or in the discharges from an ulcerated surface. These methods of elimination, how- ever, only account for a very small proportion of all the bacteria present in any disease. The greater number are in some way destroyed within the body, and they may l)e seen sometimes undergoing a process of degeneration, becoming granular, and finally breaking up and vanishing. It is easy to see an analogy here to the process of digestion, and we have to enquire how the destruction of the organisms is brought about. If we study the phenomena taking place at the site of a focus of local infection, we see the different stages of what is known as "inflammation." Around the spot at which the organisms have established themselves there takes place first a dilatation of the blood-vessels, leading to an increase in the amount of blood flowing through the part. This is followed by the escape of a quantity of blood-serum into the interstices of the tissues, and soon afterwards by the active migration of leucocytes through the walls of the vessels towards the neighbourhood of the invaders. These leucocytes seem to constitute a defensive force, which has come out to do battle with the microbes. The bacteria, if they are virulent (poisonous), cause a destruction of some of the cells of the body at the point where they settle, and they also succeed in killing many of the first leucocytes which come to attack them. The destroyed tissue may remain entire, constituting a slough, or it may be dissolved and practically disappear. The mass of dead leucocytes lying in the cavity formed by the de- struction of the tissue forms what is known as an " abscess." If the bacteria are too strong for the tissues, they continue to increase and multiply ; more and more tissue is destroyed, and increasing nuiabers of leucocytes gather 16 SERUMS, VACCINES, AND TOXINES. to the spot and are killed. This constitutes the sjDreading of the abscess. It may go on till a surface is reached, and the dead leucocytes (pus) and the bacteria are together discharged from the body. If, however, the bacteria are not sufficiently virulent to be able to establish themselves firmly in the tissues, we see that the first phenomena of inflammation are followed by the degeneration and disintegration of the germs. Only a few leucocytes may exude from the vessels to combat them, and these may be successful in their task. They are not killed in the attempt, and no mass of dead cells is therefore formed ; in other words, no suppuration occurs. The red- ness and swelling characteristic of inflammation quickly disappear, and the morbid process is at an end. In cases of general infection it is not so easy to observe what goes on as it is in such a local infection as has just been described. The bacteria, instead of remaining in one place, get into the general circulation, and there multiply. There is, however, in many cases the same development of a defensive force of leucocytes, which in this case results in an increase of the white corpuscles found in the circulat- ing blood. This is known as " leucocytosis." This is well marked in some diseases, as in acute pneumonia or septi- caemia : in others, as in enteric fever, it does not occur. In the latter class of cases some other protective measure must take the place of the leucocytes. In most infective diseases, even in those which give rise to only a localised focus of disease, the temperature of the body rises considerably, constituting a condition of '' fever." It is probable that this rise of temperature is in many cases of a protective nature, as we know that most kinds of pathogenic bacteria grow best at the ordinary temperature of the healthy body (98-4° F. or about 37° C). In fever the heat of their surroundings may rise to 104*^ or 105° F., at which point the vital acti\dties of some forms of bacteria are much retarded ; hence it is possible that the febrile reaction is of a defensive nature. PHAGOCYTOSIS. 17 All these i^lienomena of immunity to, and recovery from, infective diseases have to be explained on any theory that claims to explain the method of its production. Phagocytosis. — The first theory of resistance to disease on lines of vital action, if it may be so called, was that of Metchnikoff, who studied the behaviour of the white blood- corpuscles (leucocytes) in many of the lower animals, and attributed the destruction of bacteria in the body to the activity of these cells. This was the well-known theory of '"'■ pluujocytosis^'' (^ayeiv, to eat; kvtoq, a cell). According to Metchnikoff, the leucocytes attack and devour any invad- ing organisms which they may meet, and thus rid the body of these parasites, just as they may be seen to take into their substance particles of any foreign matter which comes in their way. When they have swallowed and thus destroyed all the bacteria which have gained a footing in the body, the disease necessarily comes to an end. The careful and minute study carried out by the French observer cannot be too much admired, and there can be no doubt that it contains a large proportion of truth. Thus, the assemblage of leucocytes which takes place at any focus of irritation is almost certainly protective in character ; and it has been shown by Kanthack and others that the granules contained in the protoplasm of the leucocytes consist of substances which tend to combat the bacteria and stop their growth. But in man, at all events, this phagocytic action is not the most important factor in the struggle with the invading germs. Leucocytes, by means of their power of swallowing foreign bodies, may play a subordinate role, carrying away the products of inflammation (broken- down cells, etc.), and at times they may actually engulf the germs themselves. Indeed, in this latter case it is possible that, they may not unfrequently do actual harm instead of good, as they may thus carry living bacteria into the tissues, and spread, instead of preventing, the disease. On the whole, they may be said to act while alive rather as scavengers than as a defensive garrison in the human c 18 SERUMS, VACCINES, AND TOXINES. economy. The part which the substances contained within them phiy in producing immunity will be referred to again later on. Humoral Theory.— The alternative theory, brought forward by the op[)onents of the phagocytic hypothesis, has now established itself firmly as the true explanation of the phenomena of immunity and resistance. It is known as the '• humoral theory." According to this the fluids of the body, and not the cellular elements, constitute the important factor in opposing the invasion of bacteria. Early experi- ments showed that the serum of the blood, even when all formed elements such as the corpuscles had been removed, still exerted in many instances an inhibitory action on the growth of micro-organisms (Nuttall, Biichner.) There must, therefore, be present in the plasma some substance of a protective nature upon which immunity depends. To these hypothetical substances Biichner gave the name "alexines." It is by means of such alexines, as will shortly be shown, that destruction of bacteria is brought about ; and it is by other chemical substances circulating in the blood that the poisonous products of the organisms are neutralised. AntitOXineS. — In the controversies, thus briefly sketched, between the upholders of the phagocytic and of the humoral theories respectively, attention was directed entirely to the resistance of the bod}^ against the bacteria and to the fate of these latter in the body. As, however, the importance of the toxines formed by the germs became thoroughly appreciated, it was natural that the question should be studied from the chemical, as well as from the biological point of view. Research on these chemical lines has proved increasingly fruitful, both from its purely scientific interest and from its practical importance. In the year 1890 Behring and Kitasato published the result of their important researches on the poison of tetanus and the possibility of rendering animals immune to it. Their investigations laid the foundations of our present knowledge of immunity and susceptibility. These DISCOVERY OF x\NTITOXINES. 19 observers proved that it was [)ossible by injecting animals first with infinitesimal quantities, later with increasing doses, of the toxines of tetanus to render them immune to the disease. The animals thus treated were able to support with impunity doses of the tetanus-poison many times as great as would suffice to kill an ordinary non-immunised animal of the same species. If the serum of an immunised animal were mixed with an equivalent amount of the poison and injected into a non-immune animal, no ill effects were produced, while the injection of the immune serum itself into a non-immunised animal rendered the latter also resistant to the toxine. If a dose of immune serum was administered within a short period of time to an animal previously inoculated with the tetanus-bacillus, the disease did not develop. The same observers, and also Wernicke, shortly after- wards showed that similar possibilities existed with regard to the bacillus of diphtJieria — that by treating animals with the toxines of this organism a serum could be obtained which was capable of neutralising the poison, and which also possessed a curative effect on the disease. To the unknown substance in the serum which had the property of neutralising the toxine they gave the name of "antitoxine." The antitoxic bodies formed in the two cases were not the same ; the tetanus-antitoxine did not act as an antidote to the poison of diphtheria, nor vice versa. Each serum was "specific," neutralising only the poison of the corresponding disease; and this peculiarity has been found to exist in all subsequently-prepared antitoxines. In the light of these discoveries as to the reaction oi living animals to bacterial toxines, attention was turned to the effects produced by other organic poisons, and it was found that it was possible to immunise animals to the vegetable poisons, abrin (from jequirity), ricin (castor oil), and crotin (croton oil), which are probably of complex proteid nature, and resemble ferments in their action. In the case of each of these substances it was possible to obtain 20 SERUMS, VACCINES, AND TOXINES. a specific antitoxic serum, protecting only against its appro- priate toxine.^ Similarly, in the case of snake-venom an antitoxic serum was prepared, of which use has been made therapeutically with some degree of success. Antibacterial Seram. — It may here be pointed out that in order to prepare an antitoxic serum it is necessary to obtain the toxine of the bacterium in question for the purpose of injection into animals. In the case of diphtheria and tetanus this was easily done. In the case of many other organisms, as previously mentioned, the poison is not secreted into culture-media, but remains in the bodies of the bacteria themselves. If the actual germs are injected into animals, beginning with minute doses of attenuated cultures and gradually increasing until large quantities of virulent bacteria can Ije tolerated, in most cases a serum is produced which is not antitoxic in the sense of neutralising the poisons of the micro-organism, but which destroys the bacteria themselves when they are sub- mitted to its action. Such a serum is said to be " anti- bacterial" or "bactericidal," instead of antitoxic. Thus, if an animal is injected with cholera- vibrios until it is very resistant to these germs, and then a little of its blood-serum is added to a culture of these organisms, the latter are found to undergo degeneration, and finally to be completely dis- integrated ; but no quantity of this serum will neutralise a lethal-dose of the poison of the chole/a-germ. On the other hand, an antitoxic serum has no eSect in preventing the growth of the appropriate organism ; for instance, diph- therial antitoxine is a very favourable culture-medium for the Klebs-Loefiler bacillus. The process of destruction of bacteria by an antibacterial serum is called "bacteriolysis," and the property resides not only in the blood-serum, but also in other vital fluids, such as the peritoneal exudate. It is ^ In the case of ricin and the allied vegetable substances, feeding animals on small, but graduallj^ increasing, doses will produce im- munity ; but this is not the case with bacterial toxines, which are destroyed by digestion. ( UNIVERSITY i 21 evident that some special substance is produced in the bodies of the immunised animals which acts as a solvent of the bacterial protoplasm. Haemolysis. — Further research showed that it is not only bacteria which, by injection into living animals, give Fowl's Corpuscles. Normal Rabbit's Serum Fowl's Corpuscles No SoluCion of Corpuscles. Solution of Corpuscles Fio-. 1- — Action of hremolytic serum. ^ rise to the production of substances destructive to them- selves. In 1891 Bordet, to whom much of our knowledge of the phenomena of bapteriolysis is due, discovered that, ^ This and the following figures are founded on diagrams used by Dr. F. W. Mott., F.R.S., to illustrate his Lectures on Pathology at Charing Cross Hospital. 22 SERUMS, VACCINES, AND TOXINES. if the blood of one species of animal were injected into an individual of another kind, the serum of the latter developed the property of dissolving the corpuscles of animals of the former species. Thus, if the blood of a fowl is injected into a rabbit, the serum of the rabbit gains the power of dis- FowPa Corpuscles. Immune Serum heated to 60°C. No Solution of Corpuscles. Fig. 2. — Effect of heat on liEemol^-tic serum (destruction of alexine). solving the corpuscles of fowl's blood when added to it in a test-tube. This phenomenon is called " haemolysis," and the hremolytic power is exactly analogous to the bacterio- lytic property in the cases previously alluded to. Two Distinct Substances Necessary.— Now, if the hsemolytic serum of the rabbit in this experiment is lieated, it is found to have lost its solvent power ; but if a little SUBSTANCES CONCERNED IN HEMOLYSIS. 23 serum from a normal rabbit is added to the heated serum, the property of dissolving the fowl's corpuscles returns to it. The same occurs in the case of bacteriolytic serum. Thus Pfeifier showed that in the peritoneal cavity of an Immune Serum heated to 60°C. Normal Rabbit's Serum Solution of Corpuscles. Fig. 2a. — Addition of alexine from normal serum to heated immune serum. immunised guinea-pig cholera-vibrios undergo a process of destruction, and that the same occurs in a test-tube con- taining serum derived from such an animal. If this serum is heated it loses its potency. If, however, a little of this heated serum is injected along with cholera-germs into the peritoneal cavity of a non-immune guinea-pig, solution takes place, the peritoneal fluid of the normal guinea-pig supplying the substance which was destroyed by the heat. 24 SERUMS, VACCINES, AND TOXINES. The accompanying diagrams (Figs. 1, 2, 2a) will perhaps make these statements more clear. From this it is apparent that in the process of haemo- lysis the interaction of two bodies is necessary to bring about the result. One of these— the alexine or complement («)^is present in normal serum, whereas the second — the copula or immune-body (6)— is only developed in immunised animals. The former («) is destroyed by heat, whereas the latter (h) is more stable, and is resistant to it. The alexine is probably of ferment-like nature, and the process of destruction has been supposed to be one of hydrolysis (Turro).i The multiplicity of names applied to these two bodies by different writers is liable to lead to some confusion. We may tabulate them thus : — {a) Alexine (always present), [b] Immune sul)stance (deve- caHed also — loped in immunised ani- Addiment. mals), called also— Cytase. Amboceptor. Complement. Fixative. Sensitising substance. Mediator. Desmon. Intermediary body. Copula. Perhaps the words alexine (aXe^te, help or protection) and copula (a bond of union) are as convenient as any to denote these substances respectively. The foregoing experiments do not succeed if the serum and corpuscles be kept at a low temperature (0° C). If, however, a mixture of hsemolytic serum and corpuscles is made and kept at this degree of temperature^ and then the corpuscles are separated from the serum and washed clean by saline solution, it is found that they are now destroyed by the addition of a normal (non-haemolytic) serum. This shews that the copula or intermediary body has become 1 £erUn. Idin. JFoch., Sept. 7, 1903. CYTOLYSIS. 25 fixed to the corpuscles in some way, so that these are now " sensitive " to tlic action of the alexine contained in normal serum. For this reason the coi)ula is sometimes called the " sensibiliser " or " preparator " (substance sensibilitrice ; preparatrice). Use is made of corpuscles— or of bacteria, for the same occurs in their case also — thus sensitised, for the purpose of experiments, to which allusion will be made on a subsequent page (see p. 41). The serum of some animals is found to be actively- destructive of that of other species, without any preliminary treatment by injection of blood. Thus the serum of the eel produces rapid haemolysis if injected into mammalian animals, and is thus highly poisonous in its action. It is probable that in this case the intermediary body or copula necessary for the action upon blood-corpuscles of a ferment already existing in the blood, is supplied by the serum of the eel. In other cases minor degrees of the same toxicity may be observed, the serum of many animals exerting, without preliminary treatment, a hsemolytic action on the corpuscles of other species. In some diseases haemolytic substances may develop in human blood, capable of acting on the blood of another human individual (isolysins). The possibility of the exist- ence of such substances in states characterised by great anaemia — the patient thus destroying his own blood- corpuscles — opens up an interesting field of speculation ; but there is as yet no definite evidence of the existence of such substances (autolysins). Cytolysis. — It is found that similar " anti-])odies " are produced by the injection, not only of bacteria and blood- corpuscles, but of many other kinds of cells, as, for instance, spermatozoa, nerve-cells, leucocytes, liver-cells, epithelial cells, (fee. Serum from an animal thus injected with sper- matozoa derived from another species is capable of causing the destruction of the spermatozoa existing within the living body of an individual of the latter species {cytolysis). The question of the possibility of preparing a serum which 26 SERUMS, VACCINES, AND TOXINES. should be capaljle of destroying the cells of a tumour, e.g. a cancer, without affecting tlie normal epithelium, opens up an interesting field of speculation (see p. 292). Precipitins.— Very closely allied to this formation of " cytolysins," or substances which are capable of dissolving cells, is the appearance of materials which act in a peculiar way on the soluble albuminous substances contained in serum itself. These are called " precipitins," and are formed when the serum of one kind of animal is injected into the body of another species.^ Thus, if the serum of, say, a horse is injected into a goat, the serum of the latter acquires the property of forming a precipitate with normal horse's serum. It has been suggested that use might be made of this fact to constitute a test for different kinds of blood. The possession of such a test for human blood would be of considerable medico-legal value. But unfortunately this particular test is not so absolutely specific as might be wished, as the serum of the injected animal is found to give a precipitate not only with the serum of the exact species of animal used to inject it, but also with the blood of allied kinds {e.g. apes and man). Some authorities ~ deny that even this limited degree of specificity exists, finding that a precipitating substance, formed by injecting human blood into an animal — which therefore should act solely on human or anthropoid blood — - will react also with that of oxen, horses, sheep, pigs, kc. The most pronounced action, is, however, on human blood, and according to these authors error may be avoided by diluting the serum. Thus a precipitating serum diluted to 1 in 1000 will react only with the blood of the animal with 1 The serum of animals treated by injection of the Wood of another species possesses in addition to its destructive action a power of agglutinating the blood-corpuscles of the latter, i.e. causing them to adhere together. This property is not lost on heating the serum to bb^ C. H. Marx and Ehrnrooth find that human corpuscles are agglutinated by the serum of any other animal, and suggest the use of this property as a medico-legal test [Milnch. mecl. JFoch., Feb. IG, 1904, p. 293). - Linessier and Lemoine, Gaz. des Hop., March 27, 1902. AGGLUTINATION. 27 whicli it was prepared. It is possible, therefore, that with this modification the test may still prove to possess a lield of usefulness. 1 The age of the blood used — stains on linen, ^c. — does not afllect the reaction. Agglutinins. — Finally we have to mention another peculiar reaction produced by the serum of animals which have suffered from a disease or been inoculated with a micro-organism. When such a serum is added to a culture of the specific bacteria of the disease it causes them to stick together m clumps or masses, instead of floating separately Fig. 3. — Diagianimatic representation of agglutination. A, Free bacill in the culture-fluid (Fig. 3). The best-known instance of this is the reaction produced by the serum of a patient suffering, or who has recovered, from enteric fever, when added to a culture of the Bacillus typhosus. It is found that if we take a young and vigorous culture of typhoid bacilli in broth, and add to it a small quantity of the serum of a patient with enteric fever, the bacilli almost immediately cease their normal active movement, and soon become collected together into clumps. The unknown bodies on which this reaction def)ends are called " agglutinins." The discovery of this phenomenon is due to Gruber and Durham,' ' A full account of the literature bearing upon this question is given by Layton, American Medicine, June 6, 1903, p. 913. Compare the work of Posselt and Sagasser on *' Agglutinins" (p. 28). 28 SERUMS, VACCINES, AND TOXINES. and experiments were made with regard to its clinical possibilities by Griinbaum ; but Widal first published his results, showing the possible use of the phenomenon as a test for the existence of enteric fever, and the reaction is generally associated with his name. Not only are typhoid bacilli agglutinated by their appropriate serum, but the same occurs with other organ- isms, such as the Micrococcus melitensis of Malta fever, the bacillus of dysentery (Shiga), streptococci, tubercle bacilli, &c. The reaction has been used as a means of classifying bacteria into distinct genera by Zupnik,i who shows that all the organisms of a particular group are clumped by the serum derived from an animal inoculated with any one of them. Thus all the acid-resisting - group of bacilli are agglutinated by serum prepared by injection of B. tuberculosis. The value of this classification has yet to be determined. It was at first thought that the property of agglutinating bacteria was a "specific" one, i.e. that serum would only clump the particular kind of organism which had been injected into the animal, or which had caused disease in the patient. This appears to be only generally true. On the one hand, it is found that a certain degree of agglutinating power towards many different kinds of bacteria may exist in normal blood, so that inoculation or disease only increases an already-existing projDerty ; on the other hand, it appears that in some cases, at all events, treatment with a particular organism may increase the agglutinating power as affecting other varieties of germs. Thus, in a case quoted by Posselt and Sagasser, it was ^ found that the serum of a rabbit before treatment possessed an agglutinating power against typhoid bacilli in a dilution of 1 : 10 ; against colon-bacilli, 1:8; against cholera-vibrios, 1:10; and against Shiga's 1 Deut. Arch. f. Jclin. lied. (Festschr. Herrn Hofr., Dr. Alfred Pribram), Band 76 (p. 290). - Those bacilli which retain staining with an aniline dj-e when treated with dilute .mineral acids. 3 Wien. Mi)). JFoch., 1903, June 11 ; No. 24, p. 691. PHENOMENA OF AGGLUTINATION. 29 bacilli (dysentery), 1 : 5. After inoculation with colon- bacilli, the figures rose to : — B. typhosus, 1 : 1 50 ; B. coli, 1 : 650 ; Y. choler?e, 1 : 50 ; and B. dysenteria^ 1 : 80. Thus treatment with one organism may apparently increase the agglutinating power against a number of others, and hence the property cannot be looked on as quite specific. It is noticeable, however, that the clumping power towards the bacilli injected rose much more rapidly and to a vastly higher point than that towards other organisms. That the agglutinating power depends on a definite substance present in the serum is shown by the fact that it is possible to exhaust the agglutinin in a specimen of serum by adding a sufficient amount of the bacteria on which it acts. Thus if we continually add fresh cultures of typhoid bacilli to the serum derived from a patient suffering from enteric fever, there at length comes a time when no further aggregation of the organisms takes place. But such a specimen of serum may still agglutinate other organisms, as, for instance, B. dysenteria?. This proves that different substances serve as agglutinins for different species of organisms. The agglutinins are in all probability not the same as the antibacterial bodies by which immunity is brought about, but in the majority of cases they seem to be developed in the serum pari passu with the latter. It has been suggested that the agglutinating power might be made use of as a criterion of the strength of an immune serum. Koch considers that in tuberculosis the agglutinative power possessed by the serum is an index of the patient's power of resistance. It is held by Buffer and Crendiropoulo i that the agglutinating substances are formed by the leucocytes, especially the multinuclear variety generally associated with inflammation. Nature of the Agglutinative Process.— The exact method by which the agglutination of bacteria by their ^ Brit. Med. Journ., April 5, 1902, p. 821. 30 SERUMS, VACCINES, AND TOXINES. appropriate serum is brought about is not understood. It lias been suggested that it is owing to some alteration of their covering membrane, so that th.ey are rendered liable to be wetted by the fluid in which they are floating (Defalle^). Bodies which are wetted by a liquid in which they are suspended tend to adhere to one another, while those which are not so wetted tend to repel each other, A more probable explanation is that some proteid sub- stance is precipitated by the action of the serum and binds the Ijacteria together in its meshes. It is noteworthy that in old cultures of typhoid bacilli an agglutinative substance passes out into the culture-fluid, so that the addition of a portion of such fluid freed from organisms confers on a normal serum the power of agglutinating the bacilli. The relation between precipitins and agglutinins is probably very close. It would appear that two separate bodies take part in the process of agglutination, analogous to the two required for bacteriolysis, haemolysis, itc. Thus Bail ~ finds that the serum of a patient sufieriug from enteric fever loses its agglutinative power if heated to 70° C, but that if the bacilli (B. typhosus) are kept for some time in this heated serum the addition of normal serum will cause them to be clumped There is some evidence that althousfh ao^ojlutination and bacteriolysis are se]3arate processes, yet bacilli are rendered less virulent by agglutination. Besredke ^ found this to be the case with typhoid bacilli, animals being able to withstand larger doses of the clumped organisms than of the normal variety. Bright and Temple* agree that the bacilli are maimed, but not killed, by agglutination. There is some conflict of opinion as to whether the agglutinative power can be transmitted from mother to 1 Ann. de VInst. Pasteur, 1902, p. 595. 2 Frager med. Jl'och., 1901, Nos. 32 and 33. 3 Ann. de VInst. Pasteur, 1901, p. 207. ^ Brit. Med. Journ., 1897, I., p. 206, INHERITANCE OF IMMUNITY. 31 offspring. Eemlinger ^ considers that the niotlier transmits some passive immunity and some agghitinativc power to her offspring ; the father, as might be expected, none of either. Agglutinative power is certainly not alwa3^s thus inherited by the human offspring. Courmont and Cade ~ found that it was transmitted by the milk of a wet-nurse to the infant she was suckling : others deny this possibility.-^ Pre- cipitins are acquired by the foetus from the mother, and antitoxines are similarly handed on.* In all these cases the properties are merely " passive," and are soon lost by the offspring. We have thus seen that the serum of an "immunised" animal may contain, not only antitoxic substances capable of neutralising the poisons of bacteria, and antibacterial bodies (alexine and copula) fitted to destroy the organisms, but also substances which agglutinate bacilli or corpuscles (p. 26, note), others which destroy living cells (cytolysins), and others still which cause a precipitate with the albumens of the serum of other species (precipitins). It would unduly complicate matters to consider here the further developments which have been made in the study of the formation of anti-bodies, such as the possibility of producing anti-cytolysins by injecting small quantities of cytolytic serum into animals, or the discovery made by Ainley Walker that by adding to the culture-fluid in which a bacterium grows small quantities of its appropriate bacteriolytic serum, it is possible to immunise the germ itself to some extent against this, thereby rendering it more virulent. Welsh has suggested that in cases of infection a con- flict may be supposed to occur between a bacterium and the body-cells, each side replying to the destructive substances brought against it by its opponent, with anti-bodies capable 1 Ann. de VInsf. Pasteur, 1899, p. 129. 2 Compi. If end. de la Soc. de Biol., Nov. 25, 1899. 3 Miinch. med. Woch., ]\Iay 2, 1889. ■^ For irEerature, see Mcrkel, Jfunch. med. Woch., Feb. 23, 1901, p. 329. 32 SERUMS, VACCINES, AND TOXINES. of neutralising them — toxine being met by antitoxine, bacteriolysin by antibacteriolysin, and so on. Enough has been said, at any rate, to show the immense complexity of tlie serum, and the capacity possessed by animal bodies of i^rotecting themselves in many ways against injurious influences. Ehrlich's Theory of Immunity. — Having thus briefly sketched the peculiar properties which are conferred on the blood-serum by treatment with bacteria and other CH COH HC HC CH CH HC CNO, CH CH Benzene CNO2 Tri n it ro- benzene Fig. 4. bodies, it remains to consider the theory of the j^roduction of immunity and allied phenomena which at present holds the field. This is due to Ehrlich, and is known as his " side-chain " hypothesis. The name is taken from organic chemistry, in which complex molecules have the property of picking up, and combining with, other atom-growths. Thus in the example given in Fig. 4 we see that a benzene nucleus has joined to itself three NOo groups and one OH group, becoming trinitro-benzene or picric acid. The chemical processes which occur in living proto- plasm are, of course, much more complicated than those of inorganic matter. Instead of a comparatively simple change brought about once and for all, as in the interaction of two simple salts, or the rather more complex phenomena of organic chemistry, we have a continual series of changes taking place between a mass of protoplasm and the sur- rounding lymph. The molecule of living matter is itself EHRLICH'S SIDE-CHAIN THEORY. 33 vastly complex. We know that it can break down into a number of simpler substances, such as albumen, globulin, lecithin, ikc, each of which is in reality a complex body, yet all of which are loosely or tightly bound together into a huge molecule of protoplasm. Of the true nature of this last we have no real knowledge. For the purpose, how- ever, of forming a mental picture of the chemical processes taking place in living matter, we may imagine the cell as consisting of a central mass — corresponding with the ring of a benzene molecule — to which are united outlying groups of molecules which have the power of entering into com- bination with other substances circulating in the lymph, such as particles of food, (fee. These outlying groups are the "side-chains" of Ehrlich's theory. Thus a side-chain attached to a cell may join to itself a particle of oxygen, of carbohydrate, of fat, (fee, and thereby take part in the nourishment of the cell ; or it may become united with a molecule of poison, such as the toxine of the diphtheria- bacillus. In the latter case, either of two things may conceivably happen : the toxine inay, through the medium of the side-chain, become part of the whole cell and may thus poison it, producing actual death (necrosis) or degeneration {e.g. cloudy swelling) ; or it may cause the death only of the individual side-chain to which it has attached itself, in which case the latter is thrown off and a new one is formed by the cell. This reproductive j^rocess is supposed to represent what takes place in the presence of only a small quantity of poison, such as first reaches the cell in a case of disease. The side chains of living cells, in virtue of their pro- perties of taking up food and other materials — useful and harmful — from the lymph, are known as " receptors." By way of illustration of the working of this hypothesis we may take the process of haemolysis, as it affords perhaps the easiest example. Here, as we saw above, two sub- stances, [a) and (6), are necessary to effect destruction of the corpuscle. Ehrlich's theory supposes that the proto- D 34 SERUMS, VACCINES, AND TOXINES. plasm of the corpuscle has not the power of combining directly with the dissolving substance (alexine) which is always present in serum, but that it can attach to itself a second body (produced in immunised animals), which in its turn can grapple to itself the solvent; and that thus the destructive matter is enabled to combine with the corpuscle and dissolve it. This process is illustrated in Fig. 5. An exactly similar jDrocess is at woi'k in bacteriolysis ; only here, owing to the minuteness of the organisms, it is a little more difficult to realise the existence of side-chains. Fig. 5. — Diagrammatic representation of haemolysis (or cytolysis). A, Corpuscle or cell, consisting of molecules of different sizes, with special groups (side-chains, receptors) at the ]jeriphery. B, Molecvdes of alexine. C, Copula or intermediary (immune) body. D, C-orpuscle with alexine-molecules attached to side chains by copula. The process by which a bacterial toxine acts on a cell, though at first sight more direct, is found to be very similar to the action of a hsemolytic substance on a corpuscle ; only the toxine consists of both destructive substance and uniting substance joined together in one molecule. The two parts in this case are called respectively the toxophore and the haptophore (to^ikov, poison ; (j)ipu), I carry; and utttu), I join; (pipio, I carry). The combined toxic molecule seizes on an appropriate side-chain of a cell, and if a number of side-chains thus take up poisonous groups the cell itself dies. If only one or two side-chains are thus attacked, they are themselves killed and drop off, but the cell escapes. It then jDroceeds to put out a fresh supply of the particular kind of side-chains, of which some EHRLICH'S SIDE-CHAIX THEORY. 35 have been killed. As frequently happens in living bodies, the repair goes beyond the original supply, and the cell thus becomes furnished with an increasing number of the side-chains capable of fixing the particular toxine. It is thus able to deal with a larger and larger amount of the poison in the blood around it. In this way animals gradu- ally become able to tolerate much larger doses of a toxine, if it is injected in gradually-increasing doses. But as this process goes on, the cell may form so many side-chains that it cannot keep them all attached to itself, and some of them are cast oft' into the lymph around the --yh ^ Fig. 6. — Diagram of cell with numerous side-chains (receptors) produced b}- stimulation with toxine. Some of these have been cast off as antitoxine, and are combining with molecules of toxine. On the left are seen molecules of toxine, showing t, toxophore, and h, hap- tophore, elements. cell and ultimately get into the blood. These free side- chains constitute the antitoxme. They are capable of uniting with the molecules of the toxine before it reaches the cells, and in this way they prevent any poisonous action resulting. Further, if the serum containing these free side-chains is injected into another animal, they will still perform the same otiice under their new conditions, and will confer on the second animal the same immvmity as was possessed by the original immunised one. The curative and prophylactic action of antitoxine is thus explained. (See Fig. 6.) If we add to some toxine an equivalent 36 SERUMS, VACCINES, AND TOXINES. quantity of antitoxine,! the molecules of poison present become combined with the free side-chains in the anti- toxine, and can no longer attack the tissue-cells. Hence the injection of a mixture of the two is innocuous. If the bacteria have already gained a footing in the patient and are pouring out constantly a stream of toxine, the injection of a dose of antitoxine neutralises the poison ; but it is necessary to give a very large dose of it in order to meet the continuous inflow of toxine. If, on the other hand, a person has not yet got, say, diphtheria, but is exposed to the chance of infection, then a protective dose of antitoxine may be given to anticipate infection, so as to be lying in wait, as it were, for any poison that may be formed. To this last application it might be objected that, as previously stated, the antitoxine is not antibacterial, and that, therefore, it does nothing to stop infection. But as the bacilli conduct their conflict with the body by means of their poisons, injuring the cells and so preventing them from forming antibacterial matter, the neutralisation of the flrst doses of poison enables the organism (the animal attacked) to gain time to form its defensive weapons. To use a military simile, we may do much to resist the first assault if we can damp the powder of our antagonists, and so we may enable reinforcements to come into action. It is important to remember that by administering antitoxine we only neutralise the Jree toxine present. That which has already entered into combination with the side- chains of the tissue-cells is practically beyond the reach of the remedy. It is possible that, if there is a very large quantity of antitoxine present, some kind of " mass " action may take place, whereby the toxine may be withdrawn from its combination with the cells and caused to combine with the antitoxine instead ; but this is not absolutely 1 Properly speaking, antitoxine is the actual substance which combines with the toxine and neutralises it. In ordiiary parlance the word is used for the serum containing- the antitoxic body. For further consideration of the interaction of toxine and antitoxine, see p. 70. TOXINE AND ANTITOXINE. 37 certain. Hence arises the urgent need, in giving antitoxine, to give it as early as possible in the disease. For example, in the case of tetanus, it may be already too late to make use of antitoxic serum when the symptoms of the disease have api)eared, the poison being already closely attached to the cells. Hence many failures and much disappointment in the treatment of this disease. It may be pointed out that so long as the side-chains are attached to the cells they are a source of weakness, as enabling the toxines to attack them, whereas, when they are cast off into the serum as antitoxine, they become a protection. A toxine can only act if it finds appropriate side-chains to which it can attach itself ; otherwise it would circulate harmlessly in the lymph. Since it would be better for the cell, from the point of view merely of its relation with toxines, if it had no side-chains, it is supposed that these exist originally for some other purpose ; and this is considered to be the assimilation of food-materials, as already suggested. These presumably are taken up by the cell for purposes of nutrition, in the same manner that toxines are attached, by means of the affinities possessed by the side-chains. A distinction is held to exist between the poisons formed by bacteria on the one hand — and with them must be grouped the toxines elaborated by poisonous snakes, and those resident in certain plants, such as ricin, abrin, &c. — and the ordinary mineral and vegetable poisons— mercury, arsenic, strychnine, morphine, and the like. It is supposed that the substances which form the first group are proteid in character (globulins, &c.), and that it is towards them especially that the side-chain activity of the cell is directed. The poisons of the second group appear to act directly on the whole cell, as in the case of these substances no antitoxic serum can be prepared. ^ The cell as a whole is either killed or recovers from the effects of the poison, but it cannot protect itself by throwing oft side-chains to neutralise ^ But see Appendix C. 38 SERUMS, VACCINES, AND TOXINES. the iioison. This property is distinct from the power which the body undoubtedly possesses of accustoming itself to increasing doses of poison such as morphine or arsenic. Incubation-Period. — A peculiar feature in the action of bacterial toxines is the occurrence of an incubation- period between the administration of the dose and the onset of symptoms. Thus, by giving increasing doses of tetanus- toxine, the rapidity of the onset of spasm may be increased up to a certain point ; but after this is reached, no further addition of poison will accelerate the event. It may be suggested that time is needed, not only for the combination of the toxine with the side-chains, which is probably a somewhat slow process, but also for the absorption of the poisoned receptor into the general body of the cell, which must precede symptoms of intoxication. Forms of Immunity. — We are now in a position to explain, on the theories just set forth, the various forms of immunity already alluded to. In the case of species, races, or individuals who are naturally immune to certain infec- tions, we must suppose either that they possess no side- chains capable of uniting with the toxines of the bacterium causing the disease, the latter thus becoming harmless,^ or that they normally contain in their systems the two sub- stances necessary to repel the bacteria, viz. the alexine and the copula. In those who are artificially immunised or who have recovered from a disease {acquired immunity) the copula has been produced, as has been described, by gradual education of the cells to throw it off.-. In all these cases the immunity is said to be " active." When an animal has received into its system a dose of antibacterial serum and is thereby enabled to resist a disease, it is said to possess 1 An instance of this condition may be seen in the tortoise, which is immune to the toxines of tetanus ; if, however, the blood of a toi'- toise which has received a dose of tetanus-toxine be injected into another animal, it will cause death, showing that the poison is not neutralised in any way, but merely has no power of affecting the cells of the tortoise. - The copula maybe of the nature of a special kind of side-chains. VARIOUS FORMS OF IMMUNITY. :i9 " passive " immunity. This lasts only as long as the injected serum remains in the body ; and this is not, as a rule, for any long period of time, since the foreign serum is more or less rapidly excreted. i The animal in this latter case has not gained for itself any power of forming protective substances ; whereas in active immunity its cells have been educated to i)erform this duty, and this acquirement seems to be retained either permanently or for a considerable period of time. It is found that, if an individual has gained this active immunity to one disease and then becomes infected with a second distinct malady, the former protec- tive power is often lost. We may repeat that it is the possession of antibacterial serum that constitutes true immunity. Injection of anti- toxic serum acts only indirectly in this sense. Modification of Phenomena of Bacteriolysis, &c., in the presence of living tissues.— It might seem from a consideration of the foregoing facts that the explanation of immunity was a fairly simple one — that defence against the invasion of any micro-organism depended on the existence in the blood of the individual of two special bodies, a ferment and an intermediary body capable of joining the ferment on to the bacterium. The absence of either of these would constitute susceptibility ; their simultaneous presence would confer protection. But other ascertained facts render the question more complicated. Thus, taking the case of anthrax-bacilli, we find that the blood of the rabbit, a highly susceptible animal, acts destructively upon the organisms in a test-tube : within the body it manifestly does not do so. Similarly, all white rats possess serum which destroys anthrax-bacilli, but they are not all immune. On the other hand, the serum of animals immune to the dis- ease, as that of the hen, forms a good culture-medium for the bacillus. It thus becomes clear that we have to take ^ The antitoxine or copula injected may perhaps be neutralised bj' the formation of an anti-antitoxine or anti-copula, and not merely passed out of the body. 40 SERUMS, VACCINES, AND TOXINES. into account not only the blood, but also the living tissues by which it is surrounded in the body. In the one case the tissues seem to exercise some inhibitory influence over the bacteriolytic action of the blood ; in the other, they supply some factor necessary for the defence of the animal against the bacilli. Further experiments show that while the serum of the rabbit is destructive for anthrax-bacilli, an ex- tract of its organs prepared by triturating them with salt- solution has no such power. Indeed, if crushed organs are added to the serum and the bacilli exposed to the action of the mixture, no bacteriolysis takes place. The tissue-cells have in some way deprived the serum of its bacteriolytic property. The explanation^ given of these phenomena is as follows : — We have already seen that a copula, or immune body, is needed to fasten the ferment to the bacteria and so produce their destruction. This copula has affinity for the tissue-cells — in this case an even greater affinity than it has for the bacilli. Hence it unites with the cells, and is no longer available for the process of bacteriolysis. Source of Aiexines. — With regard to the source of the alexine or complement, an experiment has been per- formed which points to the leucocytes as at least one source of these bodies. Thus, dog's serum alone is found not to act bacteriolytically on anthrax-bacilli, but if some leuco- cytes from the same animal are added to the serum, then destruction of the bacilli takes place. From other data it is believed that the serum of dogs contains the immune body, so that it appears that in this case the leucocytes are the source of the alexine. ^ In other cases it has been possible to supply the complement by the addition of an extract of the tissues, so that these also must be regarded as forming ferments capable of acting bacteriolytically in the presence of a suitable copula. 1 Bail, Prager meil. TFoch., Nov. 25, 1903, p. 307. '- Professor Orth's "Festschrift," Berlin, 1903 (Aug. Hirschwald), p. 396, et seq. ; see £rit. 3£ed. Jour. Epit., June 27, 1903, p. 104. NATURE OF ALEXINES. 41 Plurality of Alexines.— Considerable controversy has centred round the question whether the alexines or com- plements, by which, for example, bacteriolysis is brought about, are the same for all micro organisms, and the same in all species of animals. Thus, it might be possible to prepare an immune serum (one containing a copula or intermediary body) which should be capable of immunising a certain species of animal (a) against a particular bacillus, but this intermediary body might only be capable of uniting to tlie bacilli a special form of alexine, such as exists in the kind of animal (a) used. We cannot be certain without making the actual experiment that it will act in the same manner within the body of a second species of animal (b) : it may be incapable of uniting with the form of alexine which is here present. For example, we might prepare an anti- typhoid serum capable of protecting an animal (say, horse) against typhoid-bacilli, i.e. of causing destruction of B. typhosus when it is injected into this animal ; but it does not necessarily follow that it will act as a cure in cases of enteric fever in man, since human beings may not possess the kind of alexine with which horse-copula can unite so as to attach the micro-organisms. It has also been a moot point whether in a single species of animal there is present only one alexine, which is ready to act upon all bacteria alike, and upon blood-corpuscles, cells, ifcc, if it is only supplied with the requisite inter- mediary body ; or whether more than one alexine is present in the serum — one, perhaps, capable of producing haemo- lysis, another of causing bacteriolysis. The mode of experi- menting is by ''preparing " (p. 24) bacteria and corpuscles with a certain immune body, and then adding these prepared bacteria or corpuscles to a specimen of serum till no more lysis takes place. When the serum has thus been saturated w^th one kind of organism or corpuscles till it can dissolve no more, a second variety of prepared body (corpuscle or bacteria) is added, and it is seen whether destruction of any of this occurs. Results appear to be contradictory. Bordet 42 SERUMS, VACCINES, AND TOXINES. and Biichner hold to the unity of the alexine ; MetchnikofF, Neisser, Ehrlich, and Morgenroth support a plurality. ^ It is quite possible that the same answer to the question may not hold good in all species of animals. There is con- siderable evidence, however, pointing to the fact that — at all events, in some instances — the alexine which causes haemolysis may be different from that causing bacteriolysis in the same animal. Neisser 2 has shown that rabbit's serum can be deprived of its bacteriolytic alexine by addi- tion of anthrax-bacilli, but that it still remains capable of haemolysis. Gengou and Tarassevitch ^ have adduced ex- periments tending to show that dijfferent kinds of leucocytes are the sources of h?emolytic and bacteriolytic alexines respectively — the " microphages " producing bacteriolysins, the *' macrophages " hremolysins. Dangers of Excess of Copula or Alexine.— It has recently been pointed out ^ that the presence of excess of antitoxic serum may have an ill effect, since the antitoxine which is not employed in neutralising toxine gives rise to the formation of anti-antitoxine, which may prevent the action of antitoxine in the future stages of the disease. It seems doubtfid, however, if this constitutes a practical danger in therapeutics ; and considering the entire ignorance which we are in with regard to the exact quantity of toxine present in any given case, we must continue to be guided by purely empirical rules for administration of antitoxines. A danger similar to that just mentioned is said to exist in the case of antibacterial serums. If an excess of such a serum be administered, there is produced an excess of copula in the absence of sufficient alexine. When the former unites with the bacteria, its affinity for the alexine appears to be reduced ; at all events, it is not increased. The free cojDula (the excess) then appears to attach itself to the 1 See A>m. de flnsf. Pasteur, 1899, 1900; Berl. hl'm, Wocli., 1899, 1900, 1901. - Beuisch. med. TFoch., 1900. 3 A>in. de flnsf. Pasteur, 1900, 1901. 4 See Aialey Walker, Cli7i, Journ., June 17, 1903, p. 144. DEFICIENCY OF ALEXINE. 43 available alexine, and we have bacteria with copula attached to them, and alexine -molecules with copula attached to them. This double combination seems to prevent bacterio- lysis, as it would be necessary, in order that this should occur, for copula to unite with copula, which is not possible. Deficiency of Alexine. — So far we have paid perhaps more attention to the copula than to the alexine in the production of immunity. But susceptibility to disease may depend on lack of sufficient alexine as mucli as on defect of the intermediar3^ Some individuals may be naturally ill- supplied with alexines. In others pre-existing disease may exhaust the supply. Thus it has been shown that in chronic maladies (carcinoma, Briglit's disease, etc.) the quantity of alexine present in the serum tends to fall, and in this way we may explain the tendency to terminal acute infections in these conditions. Excessive exertion may j)erhaps cause destruction of alexine, and thus predispose to infectious diseases. The use of such remedies as yeast and cinnamic acid may lie in their power of supplying alexines, the former perhaps directly, the latter by stimulating leuco- cytosis — the leucocytes being the main source of alexine. Before leaving this subject it may be well to emphasise the fact that the explanation given by Ehrlich of the phenomena of bacteriolysis, the action of toxines, &c., is pure hypothesis. The hypothesis has been fruitful, suggesting new lines of research ; and so far the results obtained are consistent with the theory. But care must be taken not to confuse the fascinating diagrams, by which w^e are enabled to form a mental picture of the events that take place, with realities. An antitoxine is probably analogous to a secre- tion, and the process of bacteriolysis is a chemical reaction into which three bodies enter — very similar to the inter- action of the fibrin-ferment, fibrinogen, and calcium-salts to form actual fibrin. In the present account the illustra- tions have been kept as diagrammatic as possible, at the expense of verisimilitude and artistic merit, in order to avoid any undue pretence of reality. 44 CHAPTER III. PREPARATIOX AND ADMIXI8TRATI0X OF SEPtUMS AND VACCINES. SERUMS. Preparation of Antitoxic Serum. — Some account of the prejDaration of individual serums will be found under their separate headings ; only a general outline of the processes adopted will here be given. It is necessary, first of all, to make sure that the animal (generally a horse) to be inoculated is itself free from disease of any kind which might be transmitted to human beings. For this purpose it is submitted to a preliminary test by being injected with tuberculin to eliminate the presence of tuberculosis, and with mallein to ensure the absence of glanders. If it prove sound, it is in some cases first inoculated with a dose of attenuated (weakened) toxine, prepared by heating the virulent poison, or by treating it with some chemical agent which reduces its strength. After this the animal is inoculated with increasing doses of the virulent poison at stated intervals of time. Each dose is generally sufiicient to produce some febrile reaction, from which the horse recovers in the intervals. The doses are given, as a rule, siibcutaneously, but they may finally be administered intra- venously when a high degree of immunity has been attained. Sometimes, as a finishing touch, the bodies of the actual dead organisms are injected. The horses used flourish under the treatment, and grow sleek and fat. The toxines used for injection are prepared by growing the organisms in suitable fluid media ; the cultures are then passed through a Pasteur-Chambeiland or other similar filter of porcelain to remove the bodies of the bacteria. PREPARATION OF SERUMS. 45 It is important that tlie toxines should be as potent as possil)le, and special methods are adopted to secure the highest possible degree of virulence. As the site for the injections administered to the horse the root of the neck is generally selected, the hair being first shaved, and the skin thoroughly scrubbed with lysol or some other antiseptic. When a sufficient degree of immunity has been reached, the blood is withdrawn from the jugular vein by means of a simple incision, made with all antiseptic precautions, a sterile cannula being thrust into the vein, and the blood received into sterilised vessels. In these it remains till coagulation has taken place, and the free serum is then decanted off and mixed with a small quantity of some antiseptic. It is transferred to bottles of convenient size, and is ready for use. A large quantity of blood can be obtained at a single operation from a horse (16 to 20 pints from a good-sized animal) without ill effects. It is important to wait for a few days after the last injection of toxine before withdrawing the blood, as other- wise the serum may contain poisonous material. Antibacterial Serum. — -Antibacterial serums are pro- duced by very similar means, but the actual bacteria are injected instead of their toxines. Sometimes the dead bodies of the organisms are first used, or an attenuated culture, the virulent bacteria being withheld till some degree of tolerance is established. A dose of antitoxic serum may be used to mitigate the effects of the first dose, if it be available. In the case of antibacterial serums it is most important that the preparation used for treatment of disease should be freshly made, since it has been shown that the value of such serums rapidly falls. Two special factors enter into the question of the manufacture of antibacterial serums which do not apply to antitoxic preparations. In the first place it is found that some species of bacteria comprise different strains or varieties, which reiict differently towards protective serums. Thus, a serum may be prepared which will be fatal to a certain 46 SERUMS, VACCINES, AND TOXINES. strain of streptococci — the variety used for its preparation — but which will have no similar effect on another race of the same organisms, derived perhaps from a different patient or from a slightly different form of pyogenic disease. Now, in any individual case of illness w^e cannot tell what strain of bacteria may be present, and therefore in preparing a serum for practical therapeutic use it is advisable to use several strains for the purpose of immunising the animal, in order that the chances of combating the causal organism in any human patient may be increased. A serum thus prepared with several strains of an organism is said to be "polyvalent." Again, as has already been pointed out, it does not follow that the alexine existing in the body of one animal will be capable of uniting with the copula supplied by another animal so as to destroy a given bacterium. An antibacterial serum originally, no doubt, possesses in itself ooth alexine and intermediary body. But the former is an unstable substance : it rapidly vanishes from the serum when it is kept,i and it is not improbable that it is destroyed when the serum is injected into another kind of animal. Henc3 the copula contained in the immune serum may have to depend on an alexine found in the animal or man to which it is administered, in order to have a bactericidal effect. If the two bodies do not fit one another, no curative result will ensue. It has been recommended, therefore, that serums for human use should be prepared from some animal nearly allied to man, such as the ape. Such a Berum is said to be " homologous." Testing Serum. — Before a specimen of serum is issued for use it ought to be tested, to ensure that it is free from contamination. It must not contain living bacteria or toxines. In order to test it a portion of it must be mixed with some culture-medium and incubated, to see if any bacteria develop. Some of it must also be injected into an animal to make sure that it is not toxic. Cases have 1 See Ainlej Walker, Lancet, 1901, i., p. 18. STAXDAUDISATION OF SERUMS. 47 occurred in which death has been caused by the use of a serum containing the toxines of tetanus. Standardisation of Serums.— Since it has bitlierto been found impossible to isolate the actual toxines of bacteria, so that no process of weighing or measuring can be applied to them, it is necessary to devise some other way of calculating their strength. A physiological test of some sort is the only available means of measuring their effects. For this purpose it is necessary to find some animal which reacts in a constant manner to the poison, dying regularly within a certain time as the result of a given dose of toxine. In the case of the diphtherial toxines it is found that guineapigs are suitable test-animals. It is possible to ascertain accurately the quantity of a particular specimen of poison which will invariably cause the death of a guinea- pig of a certain w^eight (250 grammes) on the fourth day. This is taken as the standard dose of poison, or "minimal lethal dose." It is then necessary to find what amount of antitoxine is required to neutralise this dose exactly, and we find that equal quantities of a given antitoxine will perform this duty. A standard is thus set up. AY hat is known as a " unit " of antitoxine is that quantity of antitoxic serum which will exactly neutralise 100 minimum lethal doses as above described, i.e. of which one hundredth part will prevent the appearance of any morbid symptoms in a guineapig, if injected along with the minimal dose of toxine which would otherwise kill it within four days. In the case of an antibacterial serum the matter is rather more complicated. We may take as an example a serum which jDroduces destruction of cholera- vibrios. Such serum, if injected into the peritoneal cavity of a normal animal along with a loopful of virulent cholera-organisms, will rapidly cause their disintegration. On the other hand, the bacteria, if injected into the abdomen of an animal (not immune) without any protective serum, rapidly multiply and kill the animal. In testing the strength of a serum it is diluted for the sake of accuracy, different portions 48 SERUMS, VACCINES, AND TOXINES. being diluted to (say) 1 : 100 and 1 : 1000. Two guinea- pigs are taken, one of which receives 1 cc. of the first dilution along with a loopful of a virulent culture of cholera-vibrios, while the other receives the same quantity of the second dilution with the loopful of the bacteria- Within forty minutes search is made in the peritoneal cavities of the animals to see whether the vibrios therein are flourishing — multiplying and moving actively about — or whether they are in process of disintegration. If the smaller dose of serum has failed to kill them, while the larger one has done so, further experiments are necessary to determine the exact quantity of serum which just suffices for the purpose ; if the lower dose has proved sufficient, then smaller quantities still are tried, and so on. VACCINES. Method of Vaccination. — To confer active immunity, it is necessary to inject either the actual bacteria or their toxines into the individual to be immunised. For the purpose of vaccination it is necessary to inject a dose of the bacteria which the individual is capable of overcoming by means of his natural powers of resistance, the result being to raise his protective properties against future infection by stimulating the tissue-cells to manufacture antibacterial bodies. It is evident that to inoculate a person or animal with virulent bacteria by the same channel by which infection is produced in disease would merely induce the very condition from which it is sought to gain protection. Some other method must be selected. Several different ways of inoculation without conveying an actual attack of the disease are available : — 1. — It is possible to inoculate attenuated organisms, i.e. bacteria which have lost some of their power of produc- ing disease. Attenuation^ may be brought about (a) by ^ The employment of organisms attenuated by passage through animals has been called " Jennerisation " ; the use of chemical and other methods of weakening their virulence, " Pasteurisation." ATTENUATION OF BACTERIA. 49 passage of the organisms through an animal which has a greater power of resistance to them than man, as in the case of small-pox, which in the cow takes the form of the mild disorder, vaccinia; f 6^ by passage through an animal which, although equally or even more sensitive than man, yet alters in some way the properties of the bacteria, so that tliey are less adapted to cause human infection ; thus it is said that the virus of hydrophobia, after passage through a series of rabbits, although its virulence for these animals is innnensely increased, is yet rendered less potent for mankind ; (c) by growing the germs under conditions unfavourable to their development : anthrax-bacilli grown at a temperature of 40° C. lose much of their virulence ; and Pasteur made use of this method for preparing a vaccine for the protection of animals against this disease ; (d) by addition, to cultures of the organisms, of chemical substances inimical to their growth : thus tetanus-bacilli may be attenuated by means of iodine trichloride for the purpose of the first inoculations made in a horse in the preparation of an antitoxine ; (e) by heating the cultures of the organisms, as in the case of the vaccines for black-leg in cattle ; (/) ^y t^O'i^&> ^s i^ ^^® ^^^® ^^ ^^^® spinal cords of rabbits used in inoculation against hydrophobia (but see below, 5). 2. — The dead bodies of the bacteria may be injected instead of the living germs. This method is adopted in Wright's antityphoid vaccination. 3. — The bacteria may be inoculated in some special way, different from that by which they normally enter tlie body to cause infection. Thus cholera-germs may be injected hypodermically, instead of reaching the alimentary tract by the mouth, as they do in cases of infection. The bacillus of black-leg {rauschhrand) may be inoculated subcutaneously or intravenously for purposes of protection, its natural seat being the muscles. The tail is sometimes chosen as a site of inoculation in animals, as being colder than the rest of the body and poorly supplied with blood. 4. — An injection of antitoxic serum may be administered E 50 SERUMS, VACCINES, AND TOXINES. along with the ^drulent organisms, so as to neutralise part of their toxines at first, until the animal has gained for itself the power of manufacturing antagonistic bodies. This method has been used by Sobernheim in inoculation against anthrax, and by others against cattle-plague and swine-erysipelas. 5. — A very small number of the germs may be inoculated, so that the patient can overcome them naturally, wdiereas grave infection would be induced by a larger number of organisms. This is the principle of Hogyes' vaccination against rabies, in which a diluted virus is employed. It is not improbable that this is practically the basis of Pasteur's method in this disease, a certain number of the infective agents — those present in the external portion of the spinal cord — being killed by the desiccation, rather than all those present being reduced in virulence. 6. — The bacteria may be weakened, before injection, by treatment with the copula or immune body of their specific bactericidal serum, the alexine being previously destroyed by heat. Besredka^ has made use of this method in vaccinating animals against the organisms of plague, cholera, and enteric fever ; and Barie ~ has employed a similar method in the case of rabies. It is claimed that by this procedure the primary disagreeable effects of vaccination are avoided. In many cases (not small-pox) there a23pears to be a risk in undertaking protective injections in the presence of an actual epidemic of the disease, since it seems that before the body-cells have " learnt " to produce the antibodies there exists a preliminary period in which the susceptibility to the disease may be actually increased. Some of the existing side-chains are occupied by the toxines of the bacilli injected, wdiile the cells have not had time to reproduce others 'in excess. The word "vaccine" is used on the analogy of the 1 Ann. de VInst. Pastern; xvi., 1902, p. 918. 2 Comp. rend, de la Soc, de Biologie, Dec. 5, 1902. TOXINES AS CURATIVE AGENTS. 51 original discovery announced by Jenner, the principle l)eing the same in the modern procedures. In diseases which have a comparatively short incubative period it is necessary to administer the vaccine before infection has occurred. In small-pox vaccination at the time of infection may probably exercise an effect in modifying the disease, since the incubative period of vaccinia (about four days) is shorter than that of small-pox (twelve days). In hydrophobia the latent period is so long that it has been found possible to produce the immunity after the patient has been infected with the disease, but before the symptoms have appeared. This is the principle of Pasteur's antirabic inoculation, of the protective value of which there can now be no reason- able doubt. (See p. 153.) Toxines as Curative Agents.— The toxines of the tubercle-bacillus have been used by Koch as a curative agent, under the name of " tuberculin." It was found that the injection of this substance caused a distinct reaction at the seat of tubercular lesions, such as lupus, and that the inflammation thus produced seemed to act beneficially on the course of the disease. In the case of the " new tuber- culin " the toxic bodies of the bacteria are dissolved and injected, with the view of strengthening the patient's resistance to the disease. A slightly different method, for the treatment of enteric fever, has been introduced by Jez, who employs extracts of the spleen and other organs derived from rabbits which have been inoculated with B. typhosus. It is supposed that bodies antagonistic to the bacilli are formed in these organs, and that they may be of use if administered to human patients suffering from the disease. (See p. 185.) This method is rather an instance of treatment by anti- toxine than by a vaccine. THE ADMINISTRATIOX OF SERU^I. Early Administration. — It has already been explained that the symptoms of an infective disease are due to the 52 SERUMS, VACCINES, AND TOXINES. effects produced on the cells and tissues of the body by the toxines of its specific micro-organism, and consist in the resulting perversions of function ; also that tlie action of an antitoxic serum is to neutralise the poison circulating free in the blood and lymph, whereas it does not prev^ent the growth of the bacteria or exercise any restraining effect on them. Now, as the bacteria pour out a constant stream of toxine, and this is continually entering into combination with the side-chains (receptors) of the cells, it is most important to introduce the antidote as soon as possible, before any great amount of mischief is done. If the disease has too long a start, the antitoxine may come too late to be of any service. The great principle, therefore, in giving antitoxine of any kind is to give it at the beginning of the disease, at the earliest possible moment. In the case of diphtheria, statistics show conclusively that the power of the remedy over the disease varies directly with the promptitude of its administration, while in the case of tetanus there seems reason to doubt whether it is not already too late, in man at all events, to use the antitoxine when the malady has declared itself. (See pp. 83 and 108.) Large Dose. — A second principle is to administer a large initial dose, since we do not in any case know the amount of toxine which has to be counteracted, and the supply of the latter is constantly increasing, whereas the remedy is given all at once in a single dose, and is not in any case rejDeated for some hours afterwards. There is also the possibility that the presence of a very large quantity of the antitoxine may tend to withdraw from the cells any poison which has already united with them. Similarly, we ought not to hesitate to repeat the dose, if it seem in the least necessary. It is better to err on the side of giving too much than too little. The danger of producing anti-antitoxine or anti-immune bodies, previously alluded to (p. 42), does not seem to exist in practical therapeutics, though it might suggest the advisability of giving quite small doses of serum for prophylactic purposes. ADMINISTRATION OF ANTITOXINES. 53 Use Fresh AntitOXine.— Thirdly, it is important that the antitoxine used slioukl be as fresh as possible. There is evidence that the remedy tends to deteriorate in course of time. How long the different serums may retain their antitoxic powers is not yet definitely settled, and in the case of diphtherial antitoxine it seems probable that it may remain effective for at least two years. But there has been shown to be a slow process of deterioration at work in all cases, so that, in order to be on the safe side, it is well to use only quite fresh serum. If this is not to be obtained, or only after some delay, an older brand should be used rather than none at all, in preference to delaying unduly the administration of the initial dose. The same rule also applies to vaccines, which should be used fresh. Subcutaneous Injection.— Antitoxic serum is in general administered subcutaneously. It is immateri:d vrhat spot is selected for the injection ; the sides of the abdomen are the favourite localities as a rule, especially the skin near the groin. The back, between the shoulders, is equally convenient, but in the (unlikely) event of an abscess forming at the point of injection, as the result of some failure in antisepsis, the lesion would add more to the discomfort of the patient in this situation than if it were on the front of the body, where it would not interfere with the ordinary dorsal position of rest. The skin should be first washed with soap and water, and then with some antiseptic, such as 1 : 20 carbolic-acid lotion. The needle is passed quickly through the skin into the subcutaneous tissue, and the fiuid is injected fairly slowly. The puncture is then sealed with a " scab " of collodion. In severe cases of diphtheria and in plague it has been recommended to inject the serum directly into a vein, and good results have been claimed for this method (see pp. 85 and 170). The Syringe. — The syringe should be of a capacity of not less than 10 c.c, as this is about the largest dose of serum usually given. It should preferably have a glass piston (Fig. 7), as this can be more readily sterilised than 54 SERUMS, VACCINES, AND TOXINES. those provided with an ordinary leather washer. The latter may, however, be made of asbestos or of indiarubber. The needle should be longer than that of a common hypo- dermic syringe ; 2-2J inches is an adequate length. The bore of the needle need not be large, as the serum is perfectly fluid, and will pass readily through any hollow needle. It is unnecessary cruelty to employ the large- bored instruments often supplied, as they cause considerably more pain, and it is an advantage rather than otherwise to give the injection slowly. The serum at first causes a slight swelling at the point where it is injected, but this soon Fig. 7. — All-glass serum-s3Tiuge ; readily sterilised by boiling, after separation of the component parts. ^ subsides. Its diffusion may be aided by a little massage of the part, but this is quite needless in the majority of instances. If a second injection is required, it may be (^iven at the point corresponding with the first, but on the opposite side of the body. If a series of doses is necessary, rows of punctures may be made in lines up the two sides of the abdomen. In young infants the small size of all the parts must be borne in mind, so that the needle may not be inserted unduly far. Cases are recorded in which the pleura has been punctured in the process of injection in the flanks, and o-ano-rene of the lung has ensued with fatal result.^ With ordinary care no fear of injury to the youngest baby need be entertained. After use the syringe and needle should be washed 1 Made by Messrs, Burroughs, Wellcome & Co. 2 R. Gr. H. Tate, Lublin Joiirn. of Med. Science, April 1900, p. 271. ILL EFFECTS OF SERUM. 55 through first with cold water and then with some antiseptic to ensure sterility. They should be boiled i just before use in all cases. The syringe should be allowed to cool some- what before the serum is drawn into it for use, to ensure that the temperature of the instrument is not such as to cause any coagulation of the albuminous fluid, thereby blocking the needle. Local Effects of Serums. — In some diseases which are characterised by distinct local lesions it has been advised to inject the serum in the neighbourhood of these, in order to procure a local effect. Thus in erysipelas good results have been obtained by injections of antistreptococcic serum into the periphery of the inflamed area, and in plague it has been recommended to make the injection into some part of the skin which is drained by the lymphatics leading to the bubo. OCCASIOXAL ILL EFFECTS OF SERUM. Effects on Man of the Serum of the Lower Animals. — It is probable that the blood-serum is not identical in composition in any two species of animals ; indeed, there are variations even in that of individuals of the same species, as shown in their different degrees of immunity to diseases. We have already alluded to the poisonous eflfects produced in mammalian animals by injection of eel's serum, by which an actual haemolysis is brought about. Other serums possess varying degrees of toxicity. Antitoxic and immune serums are necessarily prepared from the blood of the lower animals, and the horse is usually chosen for the purpose on account of its size, which enables a considerable quantity of blood to be drawn at a time, as well as owing to the comparatively innocuous nature of the serum of this animal in its action on man. The injection of normal horse's serum into man may, however, be followed by certain results of an un- ^ In the all-glass syringe the parts should be separated before boiling, to obviate danger of cracking. 56 SERUMS, VACCINES, AND TOXINES. pleasant, and at times even dangerous, character. It is found that the serum of some horses possesses these qualities in larger measure than that of others. The fact that a horse has been immunised to a certain toxine or organism does not seem to have anything to do with the production of the symptoms referred to ; the peculiarity resides in the serum of the animal, and is uninfluenced by the matters used for inoculation. It is also possible that an idiosyncrasy on the part of the patient injected may be the origin of some of the ill effects noticed. Nature of Symptoms. — As the most frequently used serum is the diphtherial antitoxine, it is chiefly in the case of this remedy tliat ill efiects have been observed. They consist in cutaneous eruptions of various kinds, pains with some swelling and tenderness in the joints, and occasionally rise of temperature and feeling of illness. A good deal of itching is frequently met with at the site of injection. The rashes appear, for the most part, some time after the administration of the serum (second week), and are of the type known as erythema multiforme, i.e. they present many diff'erent appearances, erythematous, urticarial, scarlatini- form, morbilliform, (fee, but all are essentially conditions of hyper?emia and escape of serum into the tissue-spaces in varying proportions. Sometimes the hyperaemia predomin- ates (erythema, &c.); sometimes the escape of serum (urticaria). In a very few instances more serious effects have ensued. Thus Rauschenbusch^ records the case of a child who received a prophylactic injection of antitoxine, and who was almost immediately seized with "giddiness, faintness, vomiting, and cutaneous irritation, with urticarial wheals, within a few minutes of the injection." She remained ill for some hours, but was nearly well on the following day. Actual death may occur. A few instances have been recorded after the use of diiDhtherial anti- toxine, and the present writer has seen a case in w^hich ^ Quoted by Durham. Art. "Antitoxins" in Quain's " Dictionary of Medicine," 1902. ILL EFFECTS OF SERUM. 57 an injection of antistreptococcic serum in a patient suffering from pernicious ant^mia was quickly followed by coma and death. When, however, we consider the enormous number of injections of serum of all kinds that are given, the number of fatal cases reported — and it is probable that scarcely a single one of such fatalities escapes record from its very rarity — becomes almost infinitesimal. The risk is much less than that of the smallest surgical operation, and can be entirely neglected in the presence of any real illness or even danger of infection. Mode of Obviating 111 Effects. — 111 effects appear to be associated to some extent with the amount of serum used for an injection, a large dose being more likely to be followed by rashes, &c., than a small one. There is thus reason to hope that the occurrence of these symptoms will become less and less frequent with the course of time, since, as it becomes possible to prepare serums of increasing anti- toxic strength, smaller doses will be required to produce the desired effects. Thus, diphtherial antitoxine has been prepared containing as much as 1,500 units to the cubic centimetre, a very minute quantity of such a potent remedy being necessary for any one injection. Certain horses whose serum exhibits specially toxic qualities should not be used for the preparation of serum. 58 CHAPTER IV. SERUMS AND TOXINES IN THE DIAGNOSIS OF DISEASE. Agglutination-Test. — Allusion has already been made to the diagnostic use of the agglutinating power which the serum derived from patients suffering from certain infective diseases exerts upon the bacteria causing the condition. The first observation of this property was made by Gruber and Durham with regard to the reaction as it affects the bacilli of enteric fever. It was afterwards found that very many kinds of micro-organisms were similarly affected by the serum of animals immunised against them. Widal first suggested the use of the reaction as a test of the existence of enteric fever, and the test is commonly known as " Widal's reaction " in consequence. At first it was thought that the mere fact of a serum from a patient possessing the clumping property was con- clusive evidence that the disease from which he was suffer- ing was enteric fever ; but it was soon found that the serum of many normal persons was capable of producing the same effect. That derived from typhoid patients, however, is much more strongly agglutinative than normal serum, and will produce the reaction even if considerably diluted. The test was therefore modified, a diluted serum being employed. A one-in-ten dilution was at first considered sufficient, the method adopted being to mix on a glass slide one loopful of the serum to be tested with nine loopfuls of a fresh and vigorously-moving culture of Bacillus typhosus. It is necessary that the culture should be a young one, as some agglutination of the bacilli takes place in older cultures without any addition of serum, while the bacteria move AGGLUTINATION-TEST. 59 more vigcrously in young cultures. It is recommended, therefore, to inoculate a brotli-tube twelve to twenty-four hours before use in order to have the organisms at their best for the test. A drop of the mixed fluid — serum and culture — is brought into the field of the microscope, and the condition of the bacilli is observed. At first they can be seen moving actively about in all directions, but their movements gradually become more sluggish and finally cease, while the organisms may be seen to become aggre- gated into clumps or masses. A time-limit is necessary for this test, and half-an-hour is that usually taken. If within this time the bacilli have all, or nearly all, ceased to move and become massed together, then the test is said to be positive. It is now recognised that a dilution of 1 : 10 is not sufficient to exclude a number of cases in which the individual normally possesses a somewhat high agglutina- tive power without any infection with enteric fever. A dilution of 1 : 50 is now taken as the proper strength from which to judge of the reaction of a serum in suspected enteric fever ; if such a diluted serum causes agglutination within half-an-hour, the reaction is called positive. It is also advisable to dilute the serum itself before mixing it with the culture of bacilli, and not merely to use the latter for purposes of dilution ; since the pure serum may produce some clumping on first coming into contact with the bacilli, before the whole is properly mixed, and errors may thus arise. For dilution of the serum an in- different fluid, such as sterile salt-solution (0'6 per cent.), may be employed. It was at first hoped that in Widal's reaction we possessed a certain test for the existence of enteric fever, but we now know that this is not the case. On the one hand, a certain number of undoubted cases of enteric fever fail to give the reaction at all. Mention has already been made of fatal cases which never showed any power of agglutination, but which presented post mortem the charac- 60 SERUMS, VACCINES, AND TOXINES. teristic lesions of the disease. On the other hand, cases which are not enteric may exhibit a comparatively high agglutinative power. (See also p. 191.) The serum of patients suffering from other diseases may possess towards the corresponding bacteria as high an agglutinative power as that found in enteric fever, or even hiofher degrees. Thus in Mediterranean or Malta fever it is quite usual for the serum of patients to clump the micro- coccus in dilutions of 1 : 250, though here, too, a dilution of 1 : 50 is recommended as a good practical w^orking strength for diagnosis. The serum of dysenteric patients may clump Shiga's bacilli in a dilution of even 1 : 1000 in some instances. Posselt and Sagasser^ consider that a dilution of 1 : 50, recommended by Shiga, is here not sufficient to secure an accurate diagnosis. The serum of a guinea-pig artificially immunised against colon-bacilli may react with these organisms in a dilution of 1 : 25,000, while that of a typhoid-immunised horse may possess nearly equal strength. The observers just quoted show that while a serum may normally possess a powder of agglutinating several kinds of bacteria, the process of immunising the animal against one kind of organism wnll raise the agglutinative power agamst the others, though not in equal degree. Thus the serum of a patient suffering from dysentery may possess an aggluti- native power for B. dysenterise of 1 : 300, while it may react with B. typhosus at 1 : 75, with B. coli at 1 : 30, and "V. cholerse at 1 : 35. If examination were only made for its reaction with typhoid bacilli, an error of diagnosis might easily result. They therefore hold that it is necessary, before accepting a reaction as positive, to test the aggluti- nating power against several organisms. It need hardly be pointed out that, if such be the case, it adds considerably to the difficulty of making the test, and thereby detracts greatly from its value for every-day use. Another way of making use of the agglutinative reaction for diagnosis is to add a measured volume of serum to a 1 Op. cit. (see p. 28). DIAGNOSIS BY TOXINES. 61 known quantity of culture in a test-tube. If the former possesses agglutinative proi)erties, a precipitate forms in the tube, visible to the naked eye, owing to the subsidence of the clumped bacteria to the bottom of the glass. This is known as the " precipitation test." It is also possible to cultivate organisms in the serum and to compare the appear- ances which they present with those of cultures in ordinary serum. In some cases the growth in the specific serum is characterised by clumping or chain-formation. The agglutinative power remains present in the serum long after the infection which led to its appearance has subsided. Hence, not only do convalescents from, for ex- ample, enteric fever react to Widal's test, but also persons who have suffered from the disease in previous years. How long the property remains is not known for certain. Probably it varies in different individuals, and perhaps according to the severity of the attack. In the case of dysentery it has been found to last for at least a year in some cases (Kruse). It is suggested that the dui^ation of agglutinative power corresponds with that of immunity to the disease, but this cannot be considered proved as yet. A drawback to the use of the test as a means of diagnosis lies in the fact that it does not appear quite at the beginning of the illness, at which time it is most needed as an aid to diagnosis. Thus, in enteric fever, it cannot be relied upon to appear before the second week of the disease ; in plague it may be absent until convalescence. In dysentery the reaction is often wanting in mild cases, according to Shiga. This author holds that the agglutinative power in any case bears a direct proportion to the severity of the infection. Toxines as means of Diagnosis.— It is found that in some diseases the injection into the affected animal or patient of the toxines of the bacillus causing the condition produces a febrile reaction, and use has been made of this as a means of diagnosis in the case of glanders and tuber- culosis. The preparation used for the diagnosis of the former disease is known as " mallein," and is much used in 62 SERUMS, VACCINES, AND TOXINES. veterinary practice to discover the existence of the disease in horses, in which it is often very latent. Tuberculin is similarly used on cattle to reveal the existence of tubercu- losis. It has also been employed in human patients, though it has not come into general use, partly owing to the dis- agreeable nature of the effects produced, and a real or supposed risk of doing harm to the sufferer ; partly from the existence of other means of diagnosis;, such as physical examination and the search for bacilli in the expectoration (see pp. 238 and 327). 63 CHAPTER V. DIPHTHERIA. Nature of Diphtheria. — Diphtheria, derived from tlie Greek word ci(f)Oipa, a skin or piece of leather, was a term originally applied to cases of sore-throat characterised by the presence of "false membrane." When the condition came to be examined bacteriologically it was found that the great majority of these cases are associated with the growth of a particular bacillus (B. diphtlterice). It was therefore assumed that all cases of the disease were due to this organism, and it became the fashion to diagnose diphtheria solely on bacteriological findings. A case of sore-throat in which the bacillus is found is now called " diphtheria," apart from the presence or absence of the characteristic clinical symptoms (membrane-formation), while there is a tendency to refuse to recognise cases of membranous sore-throat in which no such bacilli are found, as instances of the disease. The practical result is to change the connotation of the term diphtheria from that of " membranous sore-throat " to that of "sore-throat due to B. diphtherias," A recog- nition of these facts will be seen to be of importance when the evidence for the value of antitoxic serum is discussed. Causal Organism.— The Bacillus diphthericH was first discovered by Klebs in the year 1883, and was cultivated Ijy Loeffler in the following year ; hence it is generally known as the Klebs-Loeffler bacillus. It is a non-motile, rod-shaped organism, of about the same length as the tubercle-bacillus, but thicker. It is not known to form spores. The bacillus stains readily by all ordinary methods — the dye usually adopted being Loeffler's methylene blue. The B. diphtheincE belongs to a group of organisms, the exact relations between the members of which are not 64 SERUMS, VACCINES, AND TOXINES. definitely decided. The most closely-allied form is the pseudo-diphtheria bacillus, which verj^ nearly resembles the pathogenic organism, but is not usually virulent for animals. This organism seems to occur in the throats of individuals who have been exposed to diphtherial affection, especially among school-children. At the Victoria Hospital for Sick Children, Chelsea, owing to frequent outbreaks of diphtheria, "swabs" were for a period of time taken from the throats of all children admitted to the wards. Diphtheria-bacilli of a virulent kind were found in a considerable j^roportion of the throats examined. In others pseudo-diphtheria bacilli alone were found, but it is noteworthy that among the latter cases at least two deaths subsequently occurred. This is suggestive that either the "pseudo" bacilli sub- sequently became virulent, or that they were in some way associated with the true organisms. But the matter must be considered to be still siih juclice. The pseudo-diphtheria bacillus is said not to be agglutinated by the serum of animals rendered immune against the Klebs-Loeffler bacillus. If this fact be verified, it will be strong evidence of the diversity of the two organisms. Another closely-allied, if not identical organism is the Bacillus xerosis, which is met with in the affection of the eye known as xerosis conjunctivae. Among the lower animals cats seem to suffer sponta- neously from true diphtheria, and infection of human beings may occur from them. Horses are also said to contract the disease. Most laboratory animals can be artificially infected, guinea-pigs and rabbits being especially sensitive. The disease called " fowl-diphtheria " is due to a different organism. (See p. 331.) Mice and rats seem to be immune to diphtheria. As test-animals for the virulence of a culture of the bacilli, guinea-pigs are generally used. Occurrence in the Body. — The Klebs-Loeffler bacillus is met with not only in cases of membranous sore-throat or diphtheria, but also in disease of the middle ear and in chronic nasal discharges. It may also be found in a virulent DIPHTHERIA-BACILLUS. 65 condition in the throats of healthy persons. It is supposed that such individuals have been exposed to infection from cases of di2:)htheria, but it does not seem to be proved that such contact need have occurred. It is equally possible that the organism is not unfrequently present in the fauces, as a casual inhabitant, and only becomes pathogenic owing to some lowering of the resistance of the tissues, due to other causes. In cases of membranous sore-throat in which the diphtheria-bacillus is found, it may occur either in almost pure culture or mixed with other organisms, especially streptococci. These mixed cases are generally more severe, and the prognosis is worse than in simple diphtherial infection. Diphtheria-bacilli are also found in the disease called Noma or Cancrum oris, and they may gain a footing on any open wound and there give rise to the formation of false membrane. As in the throat, so also on wounded surfaces, it is probable that other bacteria, such as strepto- cocci, may form false membrane, so that every such formation is not diphtheritic in the strict sense of the word, i.e. caused by the B. diphtheriie. In cases of diphtheria the bacilli remain for the most part confined to the false membrane in the fauces ; no general infection of the blood takes place as a rule, though in- severe cases a diphtherial septicaemia may perhaps occur. As the disease progresses, the bacilli multiply. The membrane spreads superficially by continuity, and may extend to the larynx and bronchi, the nose, the eye, the mouth, and rarely to the stomach. In their growth in the- false membrane — which consists of necrotic epithelial cells and fibrinous exudation — the bacilli manufacture a sub- stance, probably of the nature of a ferment, which is absorbed by the blood-vessels and carried all over the body. This ferment, by its action on the tissues, gives rise to other poisonous materials or secondary toxines. The bodies of the bacilli themselves are not so poisonous as their soluble products ; thus Kossel ^ showed that if the actual bacteria 1 Centralblatt f. Bakt., Bd. 19, 1896. 66 SERUMS, VACCINES, AND TOXINES. were washed free from the poison and then killed, the dead bodies had very little toxic influence when injected into animals. Toxines of Diphtheria — The nature of the poisons manufactured by the diphtheria-bacillus was studied very early in the history of modern bacteriology, since the organisms form soluble toxines which can be readily obtained in culture-rued ia. Roux and Yersin ^ were the first who discovered the presence of diphtherial-toxines in broth-cultures of the bacilli (1888, 1889). Solutions of the poisons may be prepared by growing the organisms in broth for periods of two to four weeks, and then either passing the fluid through a porcelain filter so as to strain oS" the bacilli, or adding a germicide of some sort to it so as to kill them. Toluol has been used for the latter purpose by Ehrlich and Wassermann.2 The fluid is shaken well up with this substance, which separates, on standing, into a layer floating on the surface of the broth. In this condition the preparation can be kept indefinitely, as the toluol prevents any decomposition taking place. The bodies of the bacilli sink to the bottom of the flask. A special method of growing the bacteria was devised by Aronson,^ in which they are induced to form a scum or coat on the surface of the broth ; by this means they produce a much stronger toxine than when they are cultivated in the ordinary way, diffused through the fluid medium. The effects of the poison are seen equally well whether the living bacilli or the prepared toxines are used for experimental injection. A guinea-pig which has received a dose of the organisms subcutaneously presents first at the site of injection an oedematous swelling, which is followed by enlargement of the neighbouring lymphatic glands. The animal appears to become weaker and weaker, and dies, if a 1 A)/n. de VInst. Pasteur, 1888-9. - Zeitschr.f. Hygiene, Bd. 19, 1893. 3 ;serL klin. JJ^och., 1894, p. 426. EFFECTS OF DIPHTHERIAL TOXINES. 67 moderately strong dose has been given, in the space of about four days. An examination of the body then shows the existence of oedema and liiemorrhage at the site of injection, and serous effusion into the cavities of the pleurae, peri- cardium, and peritoneum. The bacilli are not found to have become generalised throughout the body. Very large doses of toxines or very virulent bacilli may produce death in twenty-four hours. The hsemorrhagic nature of the rcdema, and the occurrence sometimes of haemorrhages elsewhere in the body of the animal, are of interest in view of the malignant or ha^morrhagic form of diphtheria which is seen at times clinically : this would appear to be due to the B. diphtheri?e^ not to secondary infection with pyogenic or similar organisms. If weaker doses of poison are administered, insufficient to cause death, the most marked phenomena may be the local swelling and the subsequent appearance of paralysis. TJiere is, indeed, reason to believe that at least three separate poisons are manufactured by the Klebs Loeffler l)acillus : one, which is the most important, causes death b}^ a general intoxication ; a second produces the local oedema at the point of inoculation, which may actually go on to necrosis of the superficial tissues ; and the third is responsible for the paralysis which sometimes occurs as a sequel. The oedema is more pronounced in those cases which do not die ; and the animal loses hair over the oedematous area, which may slough. Apparently the oedema has not time to form completely in the instances in which rapid death occurs. If life is prolonged for as much as a fortnight, paralytic symptoms may supervene, and the guinea-pigs die of asthenia. As to the exact chemical composition of the toxines little is definitely known. Roux and Yersin considered that the main poison was of the nature of a ferment ; they found that the toxic substance which they succeeded in isolating did not act in the presence of acid. S. Martin was also led to believe that the primary poison is a ferment. He G8 SERUMS, VACCINES, AND TOXINES. isolated from tlie tissues of animals dead of the disease, as well as from the culture-media in which the organisms had been grown, a series of albumoses (proto-, deutero-, and hetero-albumose), as well as an organic acid. He considered that the albumoses Avere formed in the body- tissues, especially in the spleen, not in the false membrane. In this latter the ferment was generated, and thence it was absorbed by the blood-vessels. Brieger and Bor i grew the bacilli in dialysed urine, a non-albuminous fluid, and precipi- tated the toxine by means of zinc chloride. The material thus prepared was non-albuminous ; it was very sensitive to oxidising agents, but resistant to reducing substances. It was highly toxic to animals, and the injection of it in" small quantities produced immunising substances in their serum. These observers found that the bodies of the bacteria contained a substance which was capable of caus- ing necrosis of living tissues, and which did not give rise to antitoxine in the serum. In this respect they are at issue with Kossel,- who found the bodies of the bacteria only slightly toxic. DIPHTHERIAL ANTITOXINE. Manufacture of Antitoxine.— For the production of antitoxine it is necessary to prej^are a toxine of the highest possible virulence. Certain strains of the bacillus appear to be specially adapted to form toxines in artificial media, taking on the peculiar form of growth alread}' described (formation of a pellicle on the surface of the nutrient fluid), which is found to be most advantageous for this purpose. When the organisms have grown for about a fortnight on the culture-fluid, the latter is passed through a porcelain filter ; the bacilli are thus removed, and the filtrate is ready for use. The horse selected for the production of antitoxic serum is submitted to a preliminary examination with mallein and tuberculin to ensiire that it is free from glanders and tuber- culosis. If it fails to react to these tests, it receives an 1 Deut. mcd. JFoc/t., 1896, p. 784. - loc. cit. (p. 65). PREPARATION OF ANTITOXINS. 69 injection of a small quantity of the toxine {}, to 1 cc.) sub- cutaneously in the loose tissue at the root of the neck. The injection is followed by considerable local reaction, causing the appearance of a large swelling, while the horse exhibits signs of fever and constitutional disturbance. It is necessary to wait till these symptoms have subsided before administering a second injection, which may be given on the opposite side of the neck. The antitoxic power of the blood rises gradually, reaching its highest in about six months. The doses are gradually increased till as much as 1 litre of the toxine may be injected for a single dose. The febrile disturbance produced by the poison becomes less and less as the treatment continues. It appears to be a good sign that the horse should react strongly at first, as such animals seem to produce in the end a more highly antitoxic serum. Some horses fail altogether to form antitoxine ; probably the receptors of their cells have not sufficient affinity for the toxine, and so a sufficient number of them are not killed to stimulate reproduction in excess. The injection of each dose of poison is followed by an immediate fall in the antitoxic value of the serum of the horse, but this rises again in the course of a day or two to a point higher each time than that at which it previously stood. It is important not to give a fresh dose of toxine till this rise in antitoxic power has taken place ; otherwise the antitoxine present may actually be diminished instead of increasing. As a rule the injections are given about once in three days. A horse will not go on in- definitely producing antitoxine ; its power in this direction appears to become exhausted after a time. Standardisation of Toxine and Antitoxine.— As has already been pointed out, it is not possible to weigh or measure toxines and antitoxines as we do ordinary drugs, and therefore their strength can only be measured by means of physiological tests, that is to say, by determining experi- mentally the efiects produced on living animals. For the 70 SERUMS, VACCINES, AND TOXINES. purpose of standardising the toxin es of diphtheria, guinea- pigs are the animals generally used, as it is found that they react in a very constant manner to the poison, those of the same weight being killed in approximately the same period of time by equal doses of a given toxine. A unit-dose of toxine is that amount of any preparation of diphtlierial poison which will just suffice to kill a guinea-pig w^eighing 250 grammes in a period of four days. This is also known as the "minimal lethal dose." A unit of antitoxine is the smallest quantity which, being mixed with 100 minimum lethal doses ^ of toxine injected into a guinea-pig, prevents the appearance of any toxic symptoms. This method of standardisation is that inaugurated by Ehrlich. It was necessary in the first instance to establish a toxic unit, and then to calculate the antitoxic unit from this. When this had once been done, however, it became easier in future to calculate backwards from antitoxine to toxine, since the former is more easily preserved^ not varying in strength even when kept for considerable periods of time. A standard antitoxine can now be procured from the " Serumprufungs Institut," at Frankfort-on-Maine, and is everj^where used as a standard. Interaction of Toxine and Antitoxine.— From the facts just recorded it has been assumed that the relation between given specimens of toxine and antitoxine is a con- stant one, the same quantity of the latter being always required to neutralise exactly a certain amount of the foi'mer. This is practically true within limits. The interaction between the two substances appears, therefore, to be a simple chemical combination, similar to that which takes place between an acid and an alkali. But in the case of the substances which we are considering certain curious phenomena have been observed, showing that we are not ^ This quantity of toxine, sufficient to kill 100 guinea-pigs, and exactly neutralised by one unit of antitoxine, is called by Ehrlich the L,^ dose. NEUTRALISATION OF TOXINE. 71 dealing with a case of simple chemical combination. If we take a certain quantity of a sim^jle acid and add to it the amount of the alkali which exactly neutralises it, we have a mixture corresponding with the mixture of one unit of anti- toxine with 100 minimal lethal doses of toxine. If to the former mixture we add any fresh quantity of the acid, it will remain uncombined and capable of producing its normal eifects (combining with more alkali, etc.). If, however, we take the mixture of toxine and antitoxine, and add to it one minimal fatal dose of toxine, we do not find that this addi- tional toxine has still its usual effect, vh. to kill a guinea- pig of 250 grammes in four days. On the contrary, if the mixture (unit of antitoxine + 1 00 minimal fatal doses of toxine + 1 extra minimal fatal dose) is injected into a guinea- pig, the animal recovers from the injection, only exhibiting a certain amount of oedema at the point of injection. If still further quantities of toxine are added, it will be found that quite a large number of toxic units ^ must be added before a point is reached at which the animal dies in four days. We may make the same experiment in another manner. If we take the amount of toxine which is exactly neutralised by one unit of antitoxine, viz. 100 lethal doses, add to it ^^ of a unit of antitoxine, and inject the mixture into a guinea pig, the animal does not die, but only suffers from some local oedema. This might, indeed, have been foretold, as there should theoretically be set free only one-half of a minimal lethal dose of poison. If, however, we proceed 1 This additional quantity is called by Ehrlich the L+ dose. A method of standardising antitoxine, founded on the determina- tion of this L + dose, has recently been introduced in place of the one described above. A standard antitoxine being available, unit-doses of it are taken ; varj'ing quantities of (any) toxine are added to these, and the mixtures are injected into guinea-pigs, until the exact mix- ture (1 unit antitoxine and x toxine) necessary to produce death on the fourth day is discovered. This amount {x) of the toxine is then mixed with varying quantities of the antitoxine under examination, and the quantity of this latter which must be added to the above {x) amount of toxine in order that the animal may be killed in the given time, is proved to contain exactly one unit of antitoxine, its action being precisely equivalent to that of the original standard unit. 72 SERUMS, VACCINES, AND TOXINES. further in the same way, and add to the same quantity of toxine ^^ of a unit of antitoxine, we should expect death to occur on the fourth day, as one lethal dose should now be available. But again only local oedema results. Pro- ceeding in this way, it is found that, even if i^§ of a unit is added, the mixture is still incapable of killing the animal in the stated time. When, however, the 100 lethal doses of toxine are mixed with only ^^ of a unit of antitoxine, then one minimal lethal dose is set free and the usual effect is produced. It is found at this point that for each -^^-^ of a unit of antitoxine that is subtracted, one lethal dose is set free. This continues till a point is reached at which we have arrived at a mixture of 100 m. 1. d. of toxine with /^"^ of a unit of antitoxine ; this is capable of killing 100 guinea-pigs. Any further diminution of antitoxine is with- out effect. Put in other words, it appears that it is possible to add to 100 lethal doses of poison as much as one-quarter of the total amount of antitoxine, which will exactly neutralise them without decreasing the available toxic capacity. If a further |-unit of antitoxine is added the whole of the poison is neutralised. The explanation of these phenomena given by Ehrlich is that the crude poison, if it may so be called — the culture- medium in which the bacteria have grown — contains several different substances, all of which have the power of com- bining with antitoxine. They have, however, different degrees of alhnity for the latter. The body which has the greatest avidity for antitoxine is called " prototoxoid." i The main toxine, which causes the death of the guinea-j)ig, occupies an intermediate place in point of afhnity, while a third substance, called " toxone," has the least afhnity of all. This last appears to be the body which is responsible for the local oedema seen at the point of injection of diphtherial toxines. 1 It has been suggested that toxoids consist of free " haptophore " molecules of toxine which have lost their " toxophore " element (see p. 34). TOXINE, TOXONE, AND TOXOID. 73 The accompanying diagram (Fig. 8) will perhaps serve to make a little clearer what happens on gradually adding anti- toxine to toxine. The tube on the left shows the relative proportions of each substance present in a specimen of crude poison. If now antitoxine be added, filling up, as it were, the tube from the bottom, it will first of all neu- tralise the proto toxoid, one-quarter of the whole antitoxine being thus occupied. The next two quarters will be taken up by the toxine, and the last quarter of all by the toxone. The first addition of anti- toxine does not reduce the toxicity of the mixed poisons, because it merely neutralises the prototoxoid which has no poisonous properties. The second addition counteracts the most active poison, the true toxine ; while the last addition prevents the local effects which are caused by the toxone. Again, if we take a mixture in which toxine and antitoxine are exactly neutralised (neglecting for the sake of simplicity the prototoxoid), the addition of a further unit of toxine will tend to set free an equivalent quantity of toxone, which has less affinity for the antitoxine ; and on adding further quantities of toxine a fatal amount will not be reached till all the toxone has been set free and its proportion of the antitoxine annexed by the toxine. The following illustration of the interaction of antitoxine Toxone ■ (5) t 1 4 Toxine (2) .1 1 2 Prototoxoid ■ (1) 1 4 Toxines. Antitoxine. Fig. 8. — Diagram illustrating the process of saturation of diiihtherial toxine with antitoxine. 74 SERUMS, VACCINES, AND TOXINES. and toxine in diphtheria, by means of an analogous process in ordinary chemistry, is given by Emery. ^ " You remember that in estimating chlorides by titration with silver nitrate you add a little chromate of potash to the solution to be tested. The silver has a greater affinity for the chloride than for the chromate, and you get a white precipitate of silver chloride until all the soluble chlorides have been decomposed, and then you begin to get a chocolate- coloured precipitate of silver chromate. In precisely the same way, when you add antitoxine to diphtheria poison, the first portion added goes to combine with the proto- toxoids, and these must be completely saturated before any toxine is neutralised." In addition to the above facts, there are certain peculiarities about the mixture resulting from addition of antitoxine to toxine, which throw doubt on the explanation of their interaction as a simple chemical com- bination. Thus, a mixture of toxine and antitoxine may be made which is neutral for a mouse, but which when injected into a guinea-pig may cause toxic symptoms. Again, if the mixture be heated to 100' C, the antitoxine is destroyed and the toxine remains unneutralised. Simi- larly, if the mixture be passed through a porcelain filter, the toxine passes through in the filtrate, while the anti- toxine is retained behind ; while if the same mixture be injected into an animal, the toxine may be eliminated in an active condition in the urine. It seems difficult to explain these phenomena on the basis of a simple chemical combination. Some authorities have maintained that anti- toxine does not act directly on the toxine, but indirectly through the medium of the living cell, which it stimulates in some way to resist the poison (Roux, Biichner). Danysz ~ considers that toxine and antitoxine may combine in ^ Sf. Bartholomew' s Hasp. Journ., Dec. 1902, p. 37. I am nuich indehted to this article for the clear account therein given of the reai-tion of toxine and antitoxine in diphtheria. - A7in, de Vinst. Fasteur, 1902, p. 345. RAPIDITY OF INTERACTION. 75 different proportions to form a series of "compounds," somewhat analogous to the series of oxides of nitrogen, N3O, NoOo, NoO.,, &c. ; and Bordet,^ who also holds this view, considers tliat toxone is in reality a molecule of toxine incompletely saturated with antitoxine. The question is a very difficult one, and cannot be decided on the data at present available. It seems probable that a definite chemical combination occurs, but that the affinity between the two substances is comparatively slight, so that the combination only takes place slowly, and is readily undone. In the case of the toxine and antitoxine of tetanus, Behring ^ considers that a third body, which he calls the " conductor," is necessary to bring about combination, this body acting in the same manner as the copula in haemolysis. If this is proved to be the case in tetanus, it is almost certain that the interaction of other toxines and antitoxines will be found to be produced in the same manner. Some interesting experiments by Donitz -^ throw light on the action of toxine and antitoxine within the body of a living animal. This observer found that if a dose of toxine was injected into a rabbit, it would be neutralised by the same amount of antitoxine which would neutralise it in vitro, provided that the latter were given within a space of nine minutes. If more than this time had elapsed, it was necessary to administer a considerably larger dose of the antitoxine ; but if this larger quantity were given, it was still possible to prevent the appearance of any serious ill effects. If, however, a longer space of time than about two hours were allowed to pass after the toxine had been given and before the antitoxine was injected, no amount of the latter would suffice to avert a fatal issue. It would appear from this that we can distinguish three separate periods, corresponding with distinct stages in the process of intoxi- cation :— (1) At first the poison is circulating in the blood, 1 Ann. de VInst. Past., 1903, p. 185. - BeuUch. med. Woch., Aug. 27, 1903. ^ Arch. Internat. de Tharmacodyn., Bd. 5, 1899. 76 SERUMS, VACCINES, AND TOXINES. and has not yet attacked the cells. (2) Later on it has entered into some sort of combination with them, but this is so loose that the presence of a large quantity of anti- toxine is capable of withdrawing the toxine from them again. (3) The toxine has become so firmly fixed to the cells that no amount of the remedy is capable of undoing the combination. What constitutes the difference between the two last stages is unknown. Perhaps in the former of the two the toxine has only attached itself to the side- chains, whereas in the latter it has entered into combina- tion with the whole body of the cell-protoplasm. The experiments just recorded point to the necessity for the early administration of antitoxine when used as a remedy for diphtheria. It is important to give it before the poison has gained so firm a hold upon the cells that it can no longer be withdrawn. It is fortunate that diphtheria is a disease in which it is possible to recognise the existence of infection at a comparatively early stage, since the false membrane appears on the fauces some time before any pro- found intoxication of the entire system has taken place. To this fact is due, no doubt, the infinitely greater success that has attended the use of diphtherial antitoxine as com- pared with that prepared for tetanus. In the latter disease the existence of the infection is only recognised by the appear- ance of the symptoms of general intoxication. It is then, in many cases, already too late to hope for good results from the administration of antitoxine. Statistics are given later which afford incontrovertible clinical evidence of the value of early injection of diphtherial antitoxine. Strength of Antitoxic Serum. — For human use it is important to possess a serum containing a large number of antitoxic units in a small volume, since it is not desirable to inject a larger quantity of the fluid than is absolutely necessary. Not only does the injection of a large dose cause a considerable local sw;elling, which is only slowly absorbed, but the various unpleasant effects which at times follow an injection are dependent to some extent VALUE OF ANTITOXINS. 77 on the actual volume of the serum which is administered. The majority of serums on tlie market contain 300 to 500 units in each cubic centimetre. Hence it is not often necessary to give more than 5 cc. for a dose. Stronger specimens still can be obtained at a higher price. Diph- therial antitoxine is generally supplied in liquid form, made up with a little antiseptic as a preservative. It can also be obtained in the desiccated form. According to Chiadini i it appears to keep well for a period of at least eighteen months. After two years it begins to deteriorate a little, but still possesses considerable antitoxic power ; after four years it is valueless. Ordinary degrees of light and heat do not affect its potency, nor does the addition of anti- septic agents. Value of Antitoxine. — It is extremely difficult to obtain definite proof of the curative value of any drug, .since the course of almost every disease is variable, and sudden improvements and relapses are liable to occur from natural causes, apart from the action of any remedy. The fluctuations are often ascribed to any drug which is being tried in the case, and there is no means by which the question, Post hoc or Propter hoc? can be decided. In the case of diphtheria the natural variations of the disease are even more marked than in many other disorders, and it is impossible to judge of the efficacy of antitoxine with any approach to accuracy in individual cases. Dependence must therefore be placed to a great extent on collected statistics. Even here a manifest source of fallacy is introduced by the undoubted fact that infective diseases exhibit great fluctuations in virulence when viewed over considerable periods of time, the mortality from them rising and falling in accordance with obscure periodic laws which are not well understood. Hence a fall in the mor- tality of an infective disease may occur apart from any new remedy w hich has come into vogue during the period of time under consideration. In the case of diphtheria there is i Gazzelta degll Ospedali, 1902. No. 60. 78 SERUMS, VACCINES, AND TOXINES. reason to believe that the disease has become more common in recent years, and also that the type of case seen is, on the whole, less virulent — apart from the use of antitoxine — than used to be the case. It does not seem, therefore, to be logical to ascribe all the reduction which has un- doubtedly taken place in the mortality from diphtheria to this remedy. We have to remember also that, as previously stated, there is a tendency to class as diphtheria, owing to the mere presence of B. dii:>htheri?e in the throat, cases which in earlier days would not have been considered to be suffering from this disease {e.g. cases of mild sore-throat without any formation of membrane, which would in- fallibly recover without any treatment) ; such instances swell the number of cases of diphtheria without adding to the deaths which occur, thus reducing the rate of mortality. All these facts must be taken into account when we endeavour to form a scientific judgment as to the inter- pretation to be placed upon the available statistics with regard to the influence of antitoxine on the course of diph- theria. With this preliminary caution we may proceed to consider the figures actually given by different authorities, A very instructive table is to be found in the Reports of the Metropolitan Asylums Board, giving the total number of admissions of cases suffering from each of the notifiable diseases, and the mortality which occurred in the Board's hospitals and throughout the country in each class. From it we extract the following data with regard to diphtheria. From the table (page 79) we see that a very marked diminution has occurred in the case-mortality in the hospitals under the Metropolitan Asylums Board since the use of antitoxine came into fashion. Reasons have already been given for thinking that not all of this apparent diminution can be rightly attributed to the new remedy, and if these statistics stood by themselves some doubt might still exist as to the value of antitoxine. But these figures are confirmed by reference to those obtainable from other parts of the country and of the world. Almost everywliere ANTITOXINE IN DIPHTHERIA. 79 the inortality from diphtheria seems to have fallen at about the same time, and this simultaneous effect can hardly be entirely a coincidence. Taijle 1 Sho-\vixg Admissions ikom Diphthekia to Mktuo- poLiTAX Asylums Board Hospitals roii the Yeaks 1888-1901, AViTH Mortality-rate ix these Hospitals axd Throughout the COUXTRY. Year. Admissions. Deaths. Percentage Mortality ill Hospital. Aininal Mortality per 1,000 Estimated Population. 1888 99- 46- 59-35 -^ 0-32^1 .^l 1889 722 275 4074V^SC? 0-39 -^- 1890 9i2 316 33-55 =^3 0-33 o?^ 1891 1312 397 30-63 I ai,t 0-32 f- r i 1892 2009 583 29-35 g^^ 0-46 1 ^ € 1893 2848 865 30-42j^^.i 0-76 1 '^l 18913 3666 1035 29-29 0-62 1895 3638 820 22-85"^ 0-54^ u 1896 4508 948 21-20 j^+j . 0-60 1897 5073 987 17-69 °3 s 0-51 ■~ -t 1898 6566 991 15-37 ^ SP.."^ 0-39 'So| 1899 8676 1182 13-95 II « 0-43 i^^ 1900 7873 988 12-27 ^^.S 0-34 > s < ^ 1901 7622 849 11-15; 0-29J Totals 56,151 10,282 18-50 It is noteworthy that, while the case-mortality in these hospitals has so distinctly fallen, as shown by the table, yet the general mortality throughout the country has not decreased. This is due, without doubt, to the wider distribution of the disease. The total number of cases has ^ Extracted from the Eeport of the Statistical Committee of the Metropolitan Asylums Board. Annual Report, 1901, Vol. ii., p. 33. - Number of cases too small to be of value. These figures are therefore neglected in computing averages. ■^ Treatment Avith antitoxine introduced during this year. 80 SERUMS, VACCINES, AND TOXINES. risen ; and although the fatality (case-mortality) has de- creased, the whole number of deaths is as large as ever, or even larger. This may })erhaps be due to the increased herding-together of children in scliools, owing to more efficient working of the Education Acts wliicli ensure their attendance. In any case it does not affect the question of the curative value of antitoxine. Whether a larger prophy- lactic use of the remedy might not reduce the incidence of the disease is another question. To show that the fall in diphtheria-mortality has been general throughout the world, and not confined to any one place, it may be worth while to quote statistics derived from a variety of sources. Speaking of New York, Billings ^ states that since the introduction of antitoxine a steady fall in both the number of cases and the number of deaths took place. He gives the following table, which may be com- pared with that on p. 79. MORTALI) New York before axd after the iXTRODrcTiox OF Antitoxine (Billings). Year. Cases. Deaths. Mortality per cent. 1891 0,364 1,970 36-7 1892 0,184 2,196 40-6 1893 7,021 2,558 36-4 1894 9,641 2,870 29-7 1895 '^ 10,353 1,976 19-1 1896 3 11,399 1,763 15-4 1897 10,896 1,590 14-6 1898 7,593 923 12-2 1899 8,240 1,087 13-1 The death-rate per 10,000 inhabitants previous to the advent of antitoxine was, according to Park, from 15 to 18*8. After its introduction it fell to 7, the average number of deaths in New York falling from 2,733 to 1,341 (taking the averages of fifteen years before and four years after anti- 1 New York Med. Jour». , 2 Antitoxine introduced. 1900, Feb. 17. ^ Use of antitoxine became general. RESULTS OF A-N^TITO*OTE- TREATMENT. 81 toxine). In Berlin^ the average deaths per 100,000 inhabitants in pre-antitoxine days were 90 6; they fell to 3(S*5 in the five succeeding years. In Paris the fall was from G2-2 to 13-3. It cannot be maintained, indeed, that all this reduction in mortality was due to antitoxine ; sanitary measures probably helped to reduce the death-rate, and the virulence of the disease may have diminished ; but the coincidence of a fall all over the world about the time of the introduction of antitoxine is too remarkable to be altogether accidental. \Yith regard to case-mortality, Rosenthal ^ collected from various sources figures showing that of 183,256 cases treated before antitoxine was introduced, the mortality amounted to 38*4 per cent. Among 132,548 cases after its use became general the mortality was only 14*6. Felix ^ states that in Roumania before the remedy was known the mortality of diphtherial cases was from 41 to 63 percent. ; the introduction of antitoxine has reduced the fatality among cases treated with serum to 12 per cent. Jaeger'* states that in Mulhausen the death-rate was 52 to 55 per cent, in ordinary cases_, and 65 to 68 per cent, in laryngeal cases in pre-antitoxine days ; whereas it fell to 1 6 to 20 per cent., and 20 to 25 per cent, respectively after its use became general. Similarly for Vienna Siegert' states that : From 1892-4, of 4,894 cases of diphtheria, over 2,000 died ; mortality nearly 50 per cent. From 1895-7, of 4^143 cases of diphtheria, only 817 died ; mortality about 25 per cent. Enough has now been said to show that the diminution in mortality is not confined to any one part of the world. Other evidence in favour of the remedy may be quoted of 1 Cobbett, Edinburgh Med. Journ., 1900, I., p. 521. 2 Med. Press and Circ, 1900, II., p. 293. 3 Spitahd., 1902, No. 5 (Abstr. Centralbl. f. inn. Med., 1902, 799). 4 Bentsch. Arch.f. klin. Med., LXXIII. 5 Jahrbuchf. Kinderheilk., Jan., 1902. G 82 SERUMS, VACCINES, AND TOXINES. When diphtheria affects the larynx the cases are generally more severe than those which are confined to the fauces. Goodall ^ gives some figures as to the efficacy of antitoxine in these cases. Before the days of antitoxine, of 3,275 cases of laryngeal diphtheria, 1,008 recovered (33-8 per cent.), giving a mortality of 66-2 per cent. After the introduction of the remedy, of 3,486 cases of the same nature, 2,522 recovered (72-3 per cent.), a mortality of 27*7 per cent. Taking cases of tracheotomy, the pre-antitoxine rate of recovery was under 30 per cent. ; after serum- treat- ment was inaugurated, the percentage of recoveries rose to 63*4 per cent. The improvement is very remarkable. Goodall concludes : — " Whereas in the pre-antitoxine days, of 100 tracheotomies you could not expect to save more than 29, you can now expect to save no fewer than 53. . . I think I am fully justified in claiming for antitoxine the great reduction in mortality among cases of laryngeal diphtheria that these figures reveal." Park ~ records 802 laryngeal cases with a mortality of 23 per cent., and Piekema^ 3G9 cases of tracheotomy or intubation with 28-2 per cent, of deaths. Both these authors ascribe the success met with to the use of antitoxine. Still more conclusive evidence is afforded by the statistics of the comparative rates of mortality in cases in which the remedy is administered early in the disease, and in those in which a delay of some days has occurred. The reason for this has been already pointed out (p. 75). The table on page 83 gives the results obtained by the Collective Investigation Committee of the American Pedia- tric Society i (1896). Larkins -^ records 132 cases treated with antitoxine; 1 Brit. Med. Journ., 1899, I., p. 197. 2 Jour)}, of the American Med. Assoc, 1900, April 14, p. 902. 3 Inaugural Dissertation, Utrecht, 1900 (Abstr. Centralhl. f. inn. Med., 1902, p. 799). ^ Journ. of the American Med. Assoc, 1896, II., 27; cf. Biggs' Med. Kews, 1899, July 22 and 29, pp. 97 and 137. ■5 Ihid., p. 7. RESULTS OF ANTITOXINE TREATMENT. 83 among G3 of these, who received injections on the first or second day of the disease, none died. Park ^ states that among 319 cases injected on the first day of the illness the mortality was 4 per cent. ; of 850 cases treated on the second day 57 died (mortality : 6-7 per cent.) ; among 573 injected on the third day the mortality was 12 per cent. The report of the Brook Hospital (1902) under the Metropolitan Asylums Board shows very similar results. The table on page 84 sets forth the results obtained at this liospital during the six years 1897-1902. Thus for six years no death has occurred at this hospital among the cases treated on the first day of the disease. The agreement shown among these sets of figures is very striking. There is invariably a progressive in- crease in the mortality as the remedy is given later and later after the onset of the attack. No stronger evidence could be found in favour of the use of antitoxine in this malady, for there 1 Jour. Amer. Med. Assoc, 1900, April 14, p. 902. ■~'i ^ a, o o ^ lli. OO OS ■* .o >"* 1 CO C-1 M CO < o ! 1 C-l O CO '^ 1 JO O CO ?i cc O CO 1^ o cb o o C-. ffl CO CN ■s u ■ . 1 |;g's O O rH ^ > -?;^ 0^ o -^ o < C-1 (>» I-H •M o ^ i i \Z ^ ^ t- 1 ■-■ OD O CO o QO <^ O 8 CO ^ c5 1- 1 1 .i^c I- O 1- X > -^M ^^ i ^1 t-i t-i '^ < 1-1 i C5 t- CO 'i' Q 1- CO rl CO I rr. O CO o CO H s-i ir^ c-i o O !M O si ■o ■^ o = o rt O ip ^ •^ ^t<. 'j:) ^\ ^ t- < f-i i-i ■^ Oi -o o o CO 'M "^ i a o i o o -s » -O r-l CO ri 0_ C-1 CO — __ 5 " ■"^ , . i C 5
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84
SERUMS, VACCINES, AND TOXINES.
is absolute!}^ no explanation which we can adduce for this
steadily-increasing death-rate according to the delay in the
use of the remedy, other than the hypothesis of its curative
power when administered sufficiently early. This accords
exactly with what is theoretically to be expected of the
serum and with experimental results obtained on the lower
animals.
Table showing Percentage MoHTALixr at the Brook Hospital
(1897-1902) ACCORDING TO the Day on which Treatment
WAS BEGrx.^
Day of the
Mortality
Per Cent
Disease on which
Treatment
COMMENCED. .
1897.
1S98.
1899.
1900.
1901.
1902.
Fnst
0-0
0-0
1
1 0-0
0-0
0-0
0-0
Second
5-4
5-0
! 3-8
3-6
4-1
4-6
Third
11-5
14-3
1 12-2
6 7
11-9
lO-o
Fourth
19-0
18-1
20-0
11-9
12-4
19-8
Fifth and after
21-0
22-5
20-4
21-2
16-6
19-4
While pronouncing thus unhesitatingly in favour of the
use of antitoxine, it is necessary to bear in mind that some
authorities who have had good opportunities of judging of
its value are still sceptical as to its usefulness. Among
American writers we may mention Hermann - and ilupp,-^
both of whom decline to subscribe to the general verdict
in favour of the remedy. On the Continent of Europe
Kassow4tz ^ is equally opposed to the prevailing view. He
points out that although a fall in the mortality from di})h-
theria was experienced in many parts of the world
synchronously with the introduction of antitoxine, yet
latterly the death-rate has risen again in many places in
spite of its continued use. Hence the f ill in the death-rate
cannot be ascribed to the antitoxine. There is much truth
n this argument, as has already been admitted ; and if we
1 Lancet, Sept. 12, 1903, p. 777.
- lied. Record, Jaa. 20, 1900.
3 Neiv York Med. Journ., Jan. 27, 1900.
4 Therapeutisc/ie Monatshefte, 1902, pp. 223, 499.
ADMINISTRATION OF ANTITOXINE. 85
had only this means of judging of the value of antitoxine,
it would be necessary to return a verdict of "not proven."
But the evidence available as to the progressively-greater
mortality in cases of diphtheria, according as tliey are left
for increasing periods of time without antitoxine, appears
to constitute irrefragable evidence of the value of the
remedy.
Mode of Administration of Antitoxine.^— As a
rule diphtherial antitoxine is administered suhcutaneously,
the injections being given in the flank or groin. The site
of injection is immaterial. If the child is restless it may
be advisable to interpose a piece of rubber tubing between
the needle and the syringe in order to lessen the risk of
breaking the former in the wound. Antiseptic precautions
must be observed, the skin being cleaned up w^th soap and
water, and then with lysol or some similar antiseptic.
In severe cases it has been recommended to give the
remedy intravenously ^ in order that it may be more quickly
absorbed and so manifest its effects more speedily. For the
purpose of intravenous injection a general anaesthetic must
be administered in the case of a child. For an adult
cocaine-anpesthesia would probably be sufficient. Any vein
may be selected, the median basilic at the bend of the
elbow being the most usually chosen. An incision must be
made over the vein, and the vessel isolated. An opening is
made into the vein, and the needle (which should not be a
1 Diphtherial antitoxine can he obtained from the Lister Institute
of Preventive Medicine, or from its agents, Messrs. Allen & Hanbury,
in bottles containing 1,000 units (liquid), price 2s. 6d, ; or from
Messrs. Burroughs, Wellcome & Co. in phials containing 2,000
units (price 2s. 6d.) or 1,000 units (price Is. 3d.), or a serum of a
higher potency containing 1,000 units in 1 cc. (pi'ice 2s. 6d. per 1,000
units); or from ^fessrs. Parke, Davis & Co., either as "standard"
serum (Xj containing under 400 units per cc. (price Is. for 500 units,
2s. 6d. for 1,500 units, &;c.), or a "special" scrum (XX) of higher
potency (1,500 units, price 4s. 9d., up to 6,000 units for 15s.); or
from Messrs. Meister, Lucius & Briining (Behring's serum) in
strengths of either 500 or 600 units per cc. (200 units, prophylactic,
price Is.; 600 units, 2s. 9d. ; 1,500 units, 6s. lOd. in the weaker
serum) .
86 SERUMS, VACCINES, AND TOXINES.
sharp-pointed one) is thrust into the vessel through this.
A ligature is passed round both vein and needle, and tied ;
and the antitoxine is slowly injected. The vein may
subsequently be ligatured on either side of the incision, if
there appears any tendency to bleeding ; but if the opening
has been a small one, this will not be necessary in most
cases, pressure alone sufficing to stpj^ haemorrhage. In the
case of adults in whom the veins are larger, it may be
possible to thrust a sharp needle directly into the vein
through the skin, when the vessel has been rendered
prominent by a ligature.
In order to avoid any j^isk of injecting air into the
vein, it is necessary to see that the needle is full to the end
with the serum before it is passed into the vein. There is
in reality no danger to be apprehended, if only a bubble or
two of air enter a vein ; and in veins at a distance from the
heart there is not a sufficient negative pressure to suck air
in, apart from any injected. Still, care should be taken in
this respect, a finger being kept on the vein on the central
(cardiac) side of the point incised to prevent any possibility
of mishap.
The serum should be warmed to body-temperature
before it is used intravenously ; and if it exhibit any undue
opacity or deposit, it should be strained through sterilised
muslin before it is drawn into the syringe for injection.
It would seem advisable to make use of the intravenous
method of injection in cases in which the symptoms are
severe, and especially in those in which the use of the
serum has been unduly postponed. Good results are re-
ported by Cairns^ from this mode of procedure, ver}^ large
doses of antitoxine being used in some cases. (See below.)
The antitoxine has also been given by some physicians
by the rectum and by the mouth. Parkinson ~ states that
for the last two j^ears rectal administration has been
carried out at the London Temperance Hospital, and that
the results obtained have been \eYy satisfactory. He
1 Lancet, 1902, II., Dec. 20. "^ Brit. Med. Jonrn., 1903, June 20.
ANTITOXINS AS A PROPHYLACTIC. 87
recommends tbis method as being free from some of the
discad vantages of subcutaneous injection, such as local
abscess-formation. Paton,^ who advises the use of dii)h-
therial antitoxine in septic conditions not due to the B.
diphtheriae, administers the remedy by the mouth, stating
that it is not affected by digestion ; and Zahorsky - advises
the administration of antitoxine in milk to chikh'en when
a prophylactic dose is needed. On the other hand, Hewlett •'
states, as the result of experiments on rabbits and guinea-
l^igs, that antitoxine is not absorbed from the stomach or
rectum. More experience seems to be needed on this point.
Tiiere is, at any rate, no doubt that antitoxine acts well
when administered by hypodermic injection, and as the
drawbacks to this method are very insignificant, it seems
wiser to use this method (or the intravenous) at present for
the treatment of the disease.
Prophylactic Use of Antitoxine.— It has been
ascertained beyond reasonable douljt that the administra-
tion of a comparatively small dose of antitoxine will pro-
duce immunity to the disease for a certain joeriod of time.
According to Netter,^ the protection begins about the end
of the first day (24 hours) after the injection, and lasts for
about three weeks. The protective substance is probabl}^
excreted in the urine, &c., or possibly anti-antitoxines may
be formed. There seems no reason to hold that the length
of the immunity is at all proportional to the dose adminis-
tered. As a result of Netter's communication and the
discussion which followed it, the Academy of Medicine
(Paris) passed resolutions ■' to the following effect : —
1. Preventive injections of 1,000, or at most 2,000,
units of diphtherial antitoxine produce immunity
to the disease. This protection is transitory in
character.
1 Atcsf rains. Med. Gazette, 1902, Feb. 20. '
- Archives of Ftdiatrics, March, 1899.
3 Lancet, 1902, I., p. 375 {Proc. Path. Soc. Loud.).
■* Bull, de VAcad. de Med., Paris, March 18, 1902.
5 Brit. Med. Journ., 1902, I., 997.
88 SERUMS, VACCINES, AND TOXINES.
2. Such preventive injections are specially to be
recommended in the members of families in
which cases have occurred, in order to immunise
the other children.
3. They are also called for in schools, creches, hos-
pitals, ttc, where children are collected together,
so that infection is easily spread from one to
another.
i. The injections of antitoxine are useful in patients
suffering from scarlet fever and measles, in which
affections di^Dhtheria is a frequent complication.
5. The prophylactic use of antitoxine does not pre-
clude the carrying out of ordinary measures of
disinfection and isolation.
In the presence of an epidemic of diphtheria, among 491
children exposed to infection, who did not receive protective
injections, 87 contracted the disease ; while of 502 children,
who had been similarly exposed and were given protective
injections, only 13 became infected. Of these, 7 developed
the disease within 24 hours, and 6 more than a month after
the injections. Since the immunity does not begin for 24
hours and passes off in approximately three weeks, these
figures strongly support the prophylactic use of the remedy.^
It might be w^ell, owing to the transient nature of the
immunity obtained, to repeat the dose if the epidemic con-
tinued, so as to renew the protection. Even apart from
re-infection, virulent bacilli have been known to remain
present in the throats of children for long periods of time.
American writers are strongly in favour of the preven-
tive use of antitoxine. Biggs- states that out of 3,109
cases in which these were given, only 9 children acquired
diphtheria; and Park ^ records 6,506 cases of immunisation,
among which 28 developed the disease within 24 hours,
before the protection was effective, while only 27 were
^ Netter, loc. cit. - Quoted by Billings.
3 JoiiT)}. of the A))ier. Med. Aisoe., 1900, i., p. 902.
DOSE OF ANTITOXINE. 89
attacked after this limit of time, of whom one died of scarlet
fever. Billings^ attributes the rise which has occurred in
the diphtheria-rate to the neglect of the prophylactic use of
the remedy.
On the other hand, Violi - considers that antitoxine is
not a sure preventive of diphtheria, and advises that it
should only be given when there is a certainty that a child
has been exposed to infection. Netter '^ admits that such
injections need not be given if the children can be kept under
observation and apart from others whom they might infect.
It cannot be denied that there is a real, though exceed-
ingly small, risk in the administration of antitoxine. Hence,
in private practice among well-to-do patients it does not
seem advisable to give preventive injections as a routine
procedure. In the case of institutions, such as schools and
hospitals, the question stands on a different footing ; here
there is a very great liability to spread the infection from
one patient to another, and some measures of precaution are
certainly advisable. A via media may perhaps be found
in such cases in taking " swabs " from the throats of
children exposed to infection, and only giving prophylactic
injections to those who are found to have diphtheria-bacilli
in their fauces. It is well to remember the frequency with
which epidemics of diphtheria are kept alive by means of
children who are the subjects of chronic nasal discharges.
This condition seems to have little effect on the health of
the child itself, but the virulent bacilli contained in the
nasal secretion are capable of infecting others. Hence,
children who have been brought into contact with such
nasal cases must be looked upon as having been exposed to
infection and treated accordingly, whether it be thought
better to administer antitoxine at once or to w^ait for the
result of swabbing the throat.
Dose of Antitoxine. — The tendency at the i3resent
time seems to be in the direction of giving large doses of
^ JVew York Med. Journ. 1900, Ixxi., p. 'IZi.
^ rediatria^ June, 1900. ^ Loc. clt.
90 SERUMS, VACCINES, AND TOXIXES.
antitoxine. For jjrophylactic use 150 units seem to have
been generally employed in America, but this dose was
found to be too small, and the Health Authorities ^ there
now recommend the use of not less than 300 units. Jump ^
recommends 250 units for children under two years, 500
for older children and adults. The Paris Academy, whose
resolutions were alluded to on pages 87-88, speak of 1,000 to
2,000 units as a protective dose. There can be little doubt
that these last are unnecessarily large. Perhaps, on the
ground of American experience, we may consider that 300
units is the proper dose for a child, adults receiving more —
sa}^, 500 units. It seems advisable to repeat the dose, if
liability to infection continues, or if virulent bacilli should
still be found in the child's throat.
For purposes of treatment much larger amounts are
required. Villy ^ advises that in cases of moderate severity
2,000 units should be given at once, and repeated in 12
hours if necessary. In severe cases 8,000 to 12,000 units
maybe the first dose, followed by 2,000 to 8,000 units every
12 hours. McCullom^ gave an initial dose of 8,000 units
in a severe and apparently hopeless case, following this up
with 4,000 units some hours later, and repeating the dose
every four to six hours till 92,000 units in all had been
administered. The patient recovered completely, Satter-
thwaite -^ states that the initial dose for an infant under
one year is now established at 2.000 units; for one over
one year, 3,000 ; and for an adult, 4.000 to 6,000 units.
Cairns ^ puts the doses for subcutaneous injection at from
4,000 to 20,000 units ; while intravenously he administers
from 20,000 to 35,000 units. He holds that in severe cases
an initial dose of 20,000 units is not excessive. Welden~ finds
that the best results are obtained by administering 2,000
1 Billings, Keu- York 3Ied. Jonm., 1900, Ixxi., p. 2:34.
- Fhilad. Med. Journal, Jan. 11. 1902.
3 Med. Chronicle, 1900, ii., p. 241. ^ Quoted bj' Satterthwaite.
5 2Ied. News, May 16, 1903, p. 936. « Lancet, Dec. 20, 1902.
" Xciv YorJi Med. Journ. and Philadelphia Med. Joarii., Nov. 14,
1903, p. 927.
ANTITOXINE AND PARALYSIS. 91
units every three hours until the severity of the symptoms
diminishes.
On the other hand, some authorities recommend the use
of small doses for treatment. Thus Musser ^ advises that
children from G to 8 years old should receive only 500 units,
while those over 8 should have 1,000, repeated if necessary
in 8 to 12 hours. Geffrier & Rozet ^ also recommend that
small doses should be used ; and these writers do not
advise the use of prophylactic injections, on account of their
occasional l)ad effects.
Antitoxine and Post-Diphtheritic Paralysis.—
There is reason to believe that since the introduction of
antitoxine the percentage of cases which suffer from paraly-
sis after diphtheria has definitely increased. This effect
was at first attributed to the remedy, and some prejudice
against the use of it w'as thereby excited. As a matter of
fact, it seems that the antitoxine has little, if any, power of
counteracting the poison which produces the paralysis.
It does, ho\vever, as we have just endeavoured to prove,
save the lives of many patients who would otherwise have
died. These severe cases are those which are most likely to
exhibit paralysis later on. Hence arises the increase in the
percentage of cases of paralysis ; it is in reality a testimony to
the value of the antitoxine/ and not a drawback to its use.
With regard to the incidence of paralysis after diph-
therial intoxication, Ransom •" comes to the following con-
clusions as the result of experimental researches : —
" 1. Paralysis may certainly be expected after intoxica-
tion with not less than one-quarter of a minimal fatal dose.
With doses between one-quarter and one-eighth of this
amount paralyses occur, but are not constant, and below
one-eighth no paralysis was noticed.
" 2. The larger the dose of toxine the severer will be the
paralysis, if the animal survives long enough.
1 University Med. Magazine (Philadelphia), March, 1900.
2 Arch, de Med. des infants, Feb., 1900.
3 Journ. of Fathology and Bacteriology, 1900, p. 397.
92 SERUMS, VACCINES, AND TOXINES.
" 3. Neutralised mixtures of toxine and antitoxine con-
taining only about one lethal dose or less do not appear to
cause p3,ralysis.
"4. Antitoxine, given 15 to 22 hours after intoxication
with doses of toxine not greater than the lethal dose,
exercises in large doses a mollifying influence on the sub-
sequent paralysis. . . . Small doses of antitoxine have no
evident effect in diminishing the paralysis.
" 5. Transferring the results to practice among human
beings, we may expect liberal doses of antitoxine given
early in the illness to influence favourably the subsequent
paralysis j and this favourable influence is likely to mani-
fest itself, not so much in the local paralyses (soft palate,
&c.), as in such fatal symptoms as failure of the heart.
Severe cases are, however, likely to be followed by some
paralysis in spite of even large doses of antitoxine."
It would appear from these results that so far from
having any part in the production of paralysis, antitoxine
has some power of restraining it. It does not seem to
neutralise directly the toxic material causing the paralysis.
Possibly the principal toxine, which causes death, also
modifies the cells of the nervous system in some way which
renders them more susceptible to the paralysing toxine ; so
that, if the former is neutralised by antitoxine, the latter
does not so readily produce its effects, but is still capable of
doing so if it exists in sufficient amount in an individual
case.
Ill Effects of Antitoxine. — The modes in which the
injection of the serum of any species of animal into an
individual of another species is liable to be followed by
toxic symptoms, has already been considered (p. 55).
It cannot be denied that in a certain number of in-
stances the injection of diphtherial antitoxine has been
followed by death, directly attributable to the action of the
serum. A melancholy instance was afforded by the sudden
death of Professor Langerhans' infant son after a prophy-
lactic dose of the serum. Most of the fatal cases recorded
ILL EFFECTS OF ANTITOXINE. 93
have been of the same sudden character. A case in which
death took place at a later period, and was due to the same
vascular disturbance which gives rise to the rashes often
seen after injections of antitoxine, is recorded by Gerlach.i
In this instance an erythematous eruption appeared on the
eleventh day after the injection; on the twelfth day there
were clonic spasms, without the presence of albumen in the
urine or any sym])toms of nra:?mia. At the necropsy there
was found an extradural meningeal luemorrhage, which
was attributed to a leakage from the vessels, analogous to
the escape of serum, and sometimes of blood, seen in
erythema niultiforme. Gerlach alludes to another case in
which cerebral symptoms came on after an injection, but
which did not end fatally.
Holladay records a case ~ in which a man of twenty- six
received an injection of only 500 units. This was followed
by tingling in the arm, where the dose had been adminis-
tered, cyanosis, constriction at the chest, and collapse ;
recovery subsequently ensued.
Saward -^ records two cases in which sulden syncope
occurred after injections of antitoxine. It must be re-
membered, however, that the toxine of diphtheria acts on
the cardiac muscle, producing a tendency to syncopal
attacks ; it is therefore very doubtful whether these cases
were really to be attributed to the antitoxine, since they
might equally well have been caused by the disease itself.
The most frequent ill effects brought about by injection
of serum are cutaneous eruptions ^ of various kinds. Park ^
observed them in 3 per cent, of his cases, Stanley ^ in over
25 per cent., and Villy'in 35 '2 per cent. The rashes are
1 Therapeut. Monats., April, 1903, p. 198.
- Virginia Med. Stmi-MonthJy (quoted Indian Lancet, March 23,
1903, p. 481).
3 Brit. Med. Journ., 1902, i., 1025.
^ Galitsis, These de Paris, quoted in Jonrn. de Med. et Chir.
Fratiques, Sept. 25, 1903, p. 692, states that some eruptions occur-
ring in diphtheria are due to infection with a special organism,
Diplococcus hemipholus, and are wrongly attributed to the serum.
'" Brit. Med. Journ., 1902, i., 386. . ^ Qp. cit. ' Op. cit.
94 SEKUMS, VACCINES, AND TOXINES.
generally classified as (1) Erythematous, (2) Scarlatiniform,
(3) Morbilliform, and (4) Urticarial. They are all mani-
festations of a condition which may be termed erythema
multiforme, consisting in a tendency to vascular dilatation
of difterent degrees and distribution, and escape of serum
into the tissues (urticaria).
(1) Erythema, or simple localised hypertemia, is the
commonest form of rash. It may take the shape of a slight
blush, either at the point of injection or elsewhere, or may
consist of slightly-raised circinate patches, which may
coalesce to form gyrate patterns. Favourite seats of this
variety are the extensor surface of the limbs. Out of 112
cases of rash noted by Stanley, lifty-eight were simple ery-
thema. Its average date of appearance after the injection
was the twelfth day, varying from the fourth to the twenty-
ninth. It may be combined with urticaria (fiifteen cases).
(2) The scarlatiniform rash is a more pronounced
form of the erythematous ; it is more intense and becomes
widely generalised. Stanley noted this variety in six cases
out of 112. It is of ten followed by desquamation. Leiner^
states that this form of eruption tends to come out within
the first five days after injection — an earlier period than
that of most of the other varieties— that it starts from the
point of injection ; that it is followed by peeling ; that it is
contagious, and that it seems to protect against infection
witli scarlatina. The infection is difficult to eradicate
from a ward in which cases have occurred. He concludes
that it is true scarlatina, and compares it with the surgical
variety of this disease. It is very possible that the cases
observed by Leiner were of this nature, and there is no
doubt that the diagnosis between a serum-rash and an attack
of true scarlet fever must be difficult; but there is no
reason to suppose that all cases of scarlatiniform rash after
the administration of antitoxine are of this character.
(3) Morbilliform eruj^tions formed less than 3 per
cent, of Stanley's cases. They may be accompanied by
1 Wien. JcUn. Woch., 1902, No. 43.
ANTTTOXINE-RASHES. 95
swelling of the face, conjunctivitis, and lachrymation, so as
exactly to resemble measles. Even pyrexia may occur.
Distinguishing points from true measles are that the rash
comes out first on the limbs, instead of on the face and
behind the ears ; that gyrate patterns are met with ; and
that there is no accompanying bronchitis (A'illy).
(4) Urticarial erujHions are common. They occurred
in 30 of Stanley's 112 cases. They are met with at an
earlier period after injection than the erythemata — from
the fourth to the nineteenth day (average, 9th, Stanley).
The urticaria may be quite transient, or may last several
days. The itching is sometimes very severe. A local
urticaria round the site of injection may be seen.
Pains in the joints are another inconvenience which
may arise after injections of serum. They are not usually
severe, but Taillens ^ records two cases in which they were
accomjDanied by high fever and a rash ; in one of his cases
the pains were so intense that the child could not move at
all, or bear the weight of the bed-clothes to rest on her.
Villy noted joint-pains in 6-5 per cent, of his cases.
Albuminuria may follow the use of antitoxine, but is
not of any serious import. Actual nej)hritis is said to be
rather diminished than increased by the use of this remedy.
Suppression of urine is also more rarely seen in these days
(Villy). A rise of teinperature as a result of the antitoxine
was observed by this writer in 19*8 per cent, of his cases.
An interesting case illustrating the effects of idiosyn-
crasy in relation to antitoxic serum is reported by Reckles.^
He administered an injection of 4,000 units of antitoxine to
a woman who was suffering from faucial diphtheria. A few
days later, when she aj^peared to be convalescent, a severe
urticaria developed, and shortly afterwards she was seized
with a sudden attack of pr^ecordial pain and dyspnoea, with
marked lividity of the face. In the next few days nine
successive attacks of this nature were experienced, but in
^ Revue Med. de la Suisse Romande, July 20, 1903, p. 463.
- Quarterly Med. Journ., Feb., 1903.
96 SERUMS, VACCINES, AND TOXINES.
the end the patient recovered satisfactorily. It was subse-
quently learnt that two years previously she had received
an injection of antitoxine for diphtheria, and that she had
on that occasion also suffered from attacks of dyspnoea and
cyanosis.
Borchman i states that antitoxic serum produces less ill
effects if it is warmed to 58*^ C. before it is injected. This
has onl}' a slight effect in diminishing its potency. He
quotes his experience in 578 cases: in 193 of these the
remedy was given in the ordinary way, cold, and among
these there were 22-7 per cent, of rashes. On the other
hand, among 385 patients who received the warmed serum,
only 16*3 per cent, developed these troubles.
Diphtherial Antitoxine in other Diseases.— Various
writers have detailed their experience with antitoxine in
diseases other than diphtheria. Thus Talamon has used it
in pneumonia (see p. 232) ; Paton recommends it for all
septic conditions, and thinks it has a definite influence on
inflammatory tissue (p. 219); Schapi^ and Tsvietaieff
have tried it in erysipelas ; and Konarzsherski believes
that it will cure whooping-cough. It is diflicult to
take these results seriously, since the most definitely-
established fact at present with regard to serums is their
specific nature, i.e. the power possessed by each of counter-
acting the poisons or killing the bacteria of that disease
alone for which it is manufactured. It has, indeed, been
suggested that diphtherial antitoxine may act as a stimulant
to the production of leucocytes general^, and so may be
useful in other diseases in which leucocytosis is beneficial,
much in the same way as cinnamic acid is said to act in
tuberculosis. This may possibly be the case, but at present
the probabilities point rather to the supposed benefits of
antitoxine in other diseases being due to fallacies of observa-
tion, which are difficult to avoid in studying the action of
any remedy.
^ Bietskaya Meditsina, 1900, Y., No. 3. [(Abstr. in Pediatrics,
1900, Yol. X., p. 316.)
COMPLICATIONS OF DIPHTHERIA. 97
Antitoxine in Conjunctival Diphtheria. Anti-
toxine has proved of the greatest sen-vice in diphtherial
hif action of the conjunctiva. Emmett Holt i states that
without it total destruction of the eye generally results,
whereas with its aid good results may be obtained. He
administers 2,400 units as a dose. Stevenson ~ also bears
testimony to the value of the remedy; as the result of a
study of 43 cases ; he advises the use, along with the
antitoxine, of a lotion of perchloride of mercury (1 : 5,000).
The diphtherial nature of the affection must be established
by bacteriological examination.
Post-scarlatinal Ear-disease.— On the other hand,
curiously enough, in cases of middle-ear disease following
scarlatina, in which diphtheria-bacilli appear tc be the
pathogenic organisms, antitoxine is said to act neither as a
prophylactic nor as a curative agent, although the patients
seem to obtain immunity to faucial infection by the bacilli.^
Diphtheria as a complication of other Diseases. —
Diphtheria is liable to occur as a complication of scarlet
fever and of measles. In the former malady the superven-
tion of diphtheria constitutes a very grave condition, the
mortality being very high. In measles, also, infection with
diphtheria is very likely to occur. It has, therefore, been
suggested that prophylactic injections of antitoxine should
be given as routine treatment in both these diseases. If
many cases of such superinfection have occurred in a
scarlet-fever ward, then such treatment would certainly be
advisable. Heubner^ states that a larger dose of the
serum is needed to produce immunity in cases of measles
than under other conditions (twice the amount), and that
the duration of the protection is also shorter. Richardiere ^
records that in the first four months of the year 1901 two
to four cases of diphtheria used to occur each month in the
1 Pediatrics, May, 1902.
■2 Brit. Med. /o«r>?., March 22, 1902.
^ Duiican Forbes, Journal of .Pathology and Bacteriology, 1903.
^ Quoted bv Netter, Bull, de I Acad, de Mid., Paris, March 18, 1902.
5 Ibid.
98 SERUMS, VACCINES, AND TOXINES.
measles-ward of a hospital. After this time all the children
as they came into the ward were injected with antitoxine,
and no more cases of diphtheria occurred.
Nasal Diphtheria.- — The ordinary acute faucial diph-
tlieria may spread to the nose, and such cases show a very
hicjli rate of mortality. Larf(e doses of antitoxine are
called for by way of treatment. There is also a chronic
nasal inflammation associated with the formation of mem-
brane (membranous rhinitis), which affects the nose, and in
which virulent diphtheria-bacilli are found. The patients
do not sufier from the toxic symptoms characteristic of
diphtheria, nor do they exhibit as a rule any distinct signs
of ill health ; but the discharge from the nose is capable
of conveying the infection to others. Indeed, it is pro])al)le
that many epidemics of diphtheria, of which the origin has
been obscure, might be traced to such chronic nasal disease,
which may pass as an ordinary " cold in the head." It is
doubtful whether antitoxine has any beneficial effect on
these cases. One authority- speaks of using this treat-
ment apparently with benefit ; but Lambert .Lack "^ did
not see any good results in a case in which he used
the antitoxine. The serum cannot, however, do any
harm in such a case, so that it would be well to make a
trial of it in any condition in which the presence of the B.
diphtherias was established ; but as it is antitoxic and not
antibacterial, the prosjDect of benefit from its use is remote,
1 In this connection we may note that Mj-guid [Journal of Laryng-
ology, Aug., 1898) made use of injections of diphtheriHl antitoxine in
ten cases of ozcena. He found that the injections produced congestion
of the mucosa of the nose and subsequent formation of crusts, while'
the foetid odour disappeared. It does not seem to be suggested that
the B. diphtheriiB is the cause of oza^na, the nature of which is not
well understood. Myguid's experience with antitoxine is analogous
to tliat of other observers in the different maladies ahove alluded to
(p. 96), in which it is difficult to believe that the good results seen
were in reality due to the antitoxine. Our present knowledge of the
action of serums is, however, too smull to enable us to pronounce with
any degree of confidence as to what they can or cannot do.
- M. Keusie (r McKenzie) quoted in Ann. de Med. et de Cliir.
Infantiles, June 1, 1903, p. 393. (Reference not given.) ,
3 Personal communication. I
ANTIBACTERIAL SERUM. 99
;^,ince it is destruction of the l)acilli which is desired in
this case, not the neutralisation of an}'^ absorbed toxine,
and in these cases there are no symptoms of intoxication.
ANTIBACTEKIAL SERUM.
Antitoxic serum, as ah'eady exphiined, does not tend
to kill the bacilli which cause diphtheria. They will
grow readily in the fluid itself, and continue to exist in a
virulent form in the throats of persons who have been
injected with antitoxine. Wassermann ^ has recently an-
nounced the production of a serum which is bactericidal.
He prepares a fluid somew^hat analogous to Koch's " new
tuberculin," by pounding up the bacilli and extracting
them with ethylene-diamine, 20 cc. of this solvent being
added to 1 gramme of pulverised bacilli. The mixture
is well shaken in a special apparatus, and is then sub-
mitted to the centrifuge. The supernatant fluid is of a
yellow colour, and contains the intracellular toxines (I) of
the bacilli. It is capable of killing rabbits wdien injected
into them. If, how^ever, the toxine is mixed w'ith a pro-
portion of antitoxine and repeatedly injected into these
animals the serum obtained from them is strongly ag-
glutinative of diphtheria-bacilli. Wassermann thinks that
this serum will aflTord a means of distinguishing B. diph-
therise from pseudo-diphtheria bacilli, and that it may
possibly have curative properties.
AGGLUTINATION-EEACTION-
The agglutination of diphtheria-bacilli just alluded to
is not very easily obtained, as the bacteria naturally occur
in masses, closely adherent together. Lubowski ^ obtained
the bacilli in a state of division by shaking up an emulsion
of them with small glass balls, and diluting the resulting
fluid with a 10-per-cent. solution of glycerine. The reaction
is of no practical value in the diagnosis of diphtheria.
1 Leutsch, med. JFoch., Oct. 30, lb02.
••^ ZeiUch)\f. Hygiene, 1900. Bd. 35.
100 SERUMS, VACCINES, AND TOXINES.
COXCLUSIOXS. H
1. FropJiylaxis. — Diphtherial antitoxine has a definite'
power of preventing the onset of diphtheria. The pro-
phylactic dose for adults and children over one year
should be 500 units; for infants under one year, 300 units.
Antitoxine should be used 'with this object in institutionsi
where children are congregated together, if there is any
tendency to an endemic prevalence of the disease in thes
institution. In other cases, if the children can be kept
under close observation, these protective doses are un-
advisable, as the administration is not absolutely free from-;
risk, and the prompt administration of the serum on thej
first appearance of the disease is an almost absolute safe-
guard against a fatal issue. If " swabs " can be taken from,
the throats of children exposed to infection, and examined;
for bacilli, the prophylactic doses of serum need only be
given to such as exhibit the organisms in their fauces.
2. Treatment. — The curative efiects of the serum are,
well established. The remedy should be given as early as
possible in the course of the disease, as the mortality isj
progressively greater according as the serum is adminis-
tered on later and later days. The dose for an ordinary
case in an infant under 1 year may be 1,000 units ; for
child over 1 year, 2,000 units ; for an adult, 4,000 units.
In severer cases these quantities should be largely exceeded
The dose may be repeated as often as necessary at intervali
of 4, Q, 12, or 24 hours.
3. Method of Adininisiration. — In ordinary cases sul
cutaneous injection of the serum is advisable. In ver]
severe cases it may be given intravenously (warmed), ii
order to get the patient under its influence as soon
possible. The efficacy of the use of antitoxine by the
mouth or rectum needs further elucidation ; at present it i
not advisable to make use of this method of administration
4. Ordinary measures of prophylaxis (isolation, etc.)j
and of treatment, should not be omitted because antitoxin^
is used.
101
CHAPTER VI.
TETANUS.
Causal Agent. — The micro-organism which is the cause
of tetanus was discovered by ]Sricolaier in 1881:, and was
first cultivated by Kitasato in 1890. It is a rod-shaped
bacillus, about half as large again as the tubercle-bacillus,
motile, and provided with numerous flagella. It forms
spores, each of whicli is usually situated at one extremity of
a bacillus, so that the characteristic " drum-stick " shape is
produced. The organism stains readily by ordinary
methods, and retains the dye when treated with Gram's
reagent. When first isolated from the human or animal
body, the B. tetani will only grow in the absence of free
oxygen (anaerobically). When, however, it has lived for
some time on artificial media, it can flourish in the 'j^iresence
of air. It is found in the soil, especially cultivated soil,
such as that of gardens, and occurs almost invariably in the
excreta of horses and other herbivorous animals. It is
much more prevalent in some parts of the world than in
others, if we may judge by the frequency of the disease.
Thus in tropical and sub-tropical climates tetanus is a
iiiueli more common complication of wounds than in
Ki Inland.
Infection by the bacillus. — It has been shown that
ti tanus-bacilli inoculated directly into healthy tissues do
Hot live and produce disease. In order that this may occur,
it is necessary either that some of the toxines of the bacteria
should be inoculated along w^ith them, or that some
: foreign substance, such as a splinter of wood or some of the
jSoil, should be introduced with them, by which a certain
I degree of irritation and damage to the tissues is produced.
JO:^ SERUMS, "VACCINES, AND TOXINES.
The most probable form of wound to give rise to tetanus is,
therefore, a crushed wound, in which dirt and foreign
substances are deeply embedded. Wounds from toy pistols
have in America frequently been complicated by tetanus.
Suppuration seems to favour its occurrence, and the bacilli
have a better chance of thriving if the wound is deep and
narrow, so that air is excluded. So-called "idiopathic"
tetanus is caused by the entrance of the bacilli through
slight wounds which heal up and are forgotten. Lambert i
finds that infection with tetanus may take place through
the mucous membrane of the nose or mouth, if they are.
wounded, or if the former is the seat of catarrh. He
suggests that so-called "rheumatic tetanus " may be due to
an infection through the bronchi by inhaled germs.
Infection does not, according to this writer, occur by the
stomach or bladder, even if their living membranes are
injured. Cano-Brusco and Frassetto^ state that the toxine
is destroyed, not by the digestive ferments, but by the
epithelial cells of the intestine. Commercial gelatine is
very liable to contain the spores of tetanus, and a number
of cases have been -recorded in which the disease occurred
after gelatine-injections, made for the treatment of
aneurysm. An outbreak also occurred in the United
States, due to contamination of diphtherial antitoxine with
the toxines of tetanus. Cases have occurred after vaccina-
tion, either due to the presence of the organisms or their
spores in vaccine-lymph, or to subsequent inoculation of the
vaccination-lesions from accidental sources. Vaccination-
wounds may perhaps afi*ord a specially favourable nidus for
the organisms, as they form an ulcerated surface beneath a
scab.
Production of Symptoms. — In the body the bacilli
do not become generalised ])y the blood-stream,^ but remain
localised at the point of infection, where they form their
1 Med. Xeu-s, 1900, July, p. 12.
- Gazz. degli OspedaU, 1900, xxi., p. 879.
'^ Lambert [op. cit.) states thiit the bacilli may wander into the
general circulation.
ABSORPTION OF TOXINES. 103
toxines. These are taken up by tlio peripheral nerves, and
;ire carried along the axis-cylinders to the central nervous
system. Exactly the same mode of conveyance seems to
hold good in the case of the poison of hydrophobia, and it
seems that there must be a continual streamino- of the
])rotoi3lasm of the nerve-fibrils towards the cells from
which they are prolongations, llesection of a portion of a
nerve may prevent the onset of tetanus in animals, if only a
very small dose of the toxines has been administered. If
large doses are given, part of the poison is absorbed into the
blood-stream, and reaches the nervous system by a more
circuitous route. Meyer and Ransom,^ however, as the
result of many ingenious experiments, consider that it is only
by the peripheral nerves that the poison can reach the spinal
cord or brain. Poison circulating in the blood is taken up
by the nerve-endings, and so passed on to the central
l)ortions of the nervous system, but it does not penetrate
directly into the latter from the lymph. They thus explain
the greater part of the incubation -period met with in
poisoning by tetanus- toxines. They point out that the
incubation is longer in direct proportion to the size of the
animal and the consequent length of the nerves. Thus a
mouse exhibits symptoms in 8 to 12 hours, a guinea-pig in
13 to 18, a rabbit in 18 to 36, while in man four days is
about the shortest period, and in the horse five days. If an
animal is injected with the poison in the nerve of a hinder
limb, the spread of the poison to the important centres in the
medulla can be prevented by section of the spinal cord. In
this, as in the peripheral nerves, there appears to be a
centripetal movement of protoplasm.
In cases of tetanus in man, the incubation-period is very
variable. Symptoms may appear in four or five days, or
they may be delayed for months. The more rapid the onset,
the more acute are the symptoms, and the more grave the
prognosis.
1 Arch. f. Exper. PaUtoL u. Vhurmahol., 1903. Bd. 49, Heft. 6,
p. 369. Cf. Marie and ^Eorax, Ann. de Vlnat. Fasten/; 1902, p. 818 ;
1903, p. 335.
104 SERUMS, VACCINES, AND TOXINES.
Experimentally, in animals, some peculiar phenomena
have been produced. Thus if the jjoison be injected directly
into the cerebral substance, there results a condition of mental
excitement, aj)parently accompanied by hallucinations, as-
the animal may snap at imaginarj^ objects. Injections into
the spinal cord are followed by agonising pains, referred
to a point of the body corresponding with the site of
inoculation (tetarius dolorosus). Injections of small doses
of poison may cause only localised spasms without general
stiffness or convulsions.
Toxines of the Tetanus-bacillus.— The toxines of
the bacillus may be obtained by growing the organisms in
an atmosphere of hydrogen in glucose-broth, from which all
oxygen has been expelled by causing hydrogen gas to
bubble through it. Instead of this method, a broth
containing sulphindigotate of soda may be used. Morax
and Marie ^ state that a potent poison can be prepared by
cultivating the bacilli in the presence of air along with
Bacillus subtilis. The fluid obtained after about a month's
growth is filtered through a porcelain filter, and the poison
is ready for use. The fluid solution does not keep at all
well, the poison gradually losing strength, probably owing
to the formation of toxones similar to those described in
connection with diphtheria (p. 72). The toxines are not
destroyed by heating to 135° C. for ten minutes; their
action is, however, slightly retarded. Their potency is
rapidly destroyed by sunlight. If the albuminous con-
stituents of the culture-fluid are precipitated by alcohol or
other reagents, the toxine is found in the precipitate; but
it is probably not of an albuminous nature itself. Dean ~
concludes as follows with regard to it : — " Tetanus-toxine
has thus many points of resemblance to the soluble
ferments ; it is difiicult to dialyse, is soluble in water, is
precipitated by alcohol and tends to adhere to precipitates,
1 Amu de VInst. Past cur, 1902. p. 418.
- Art. ''Tetanus " in Quain's " Dictionary of Medicine," edited by
Montague Murray, 1902, p. 1688,
TOXINES OF TETANUS. 105
is Diodified or destroyed by the action of air, suuliglit, and
comparatively low temperatures, and requires an in-
cubation-period for its action."
The mixed poison, as obtained from cultures, is almost
certainly very complex in character. Tizzoni and Collina ^
state that it contains two toxines, one of which acts
specially if it is administered subcutaneously and produces
convulsions, while the second becomes prominent in case of
intravenous injection, giving rise to local tonic spasm.
Erhlich ~ has shown that there is also present a substance
which jDroduces hsemolysis (tetanolysin), and that an
antilysin which neutralises this is found in tetanus-anti-
toxine. Tetanus-toxine, or tetanine, as it is called by
French writers — using the word to apply to the poisons
collectively — has a great affinity for the nervous system.
If an animal has died of tetanus, the nerves leading from
the seat of infection, and also the brain and spinal cord,
contain the poison. The affinity of the brain-tissue for the
toxine has also been shown in another way by Wassermann
and Takaki,-^ who found that if an emulsion were made
of brain-substance, and this were mixed with the toxines
before injection into animals, no ill effects were produced.
A protective influence was exercised even if the emulsion
were injected at a different point from the toxine, without
previous mixture. Cerebral substance seems thus to
contain an antitoxine to the poison : in other words, the
toxine has the power of combining with the side-chains of
the cerebral cells ; when, therefore, these cells are injected
into another animal, they are capable of uniting with the
free poison and so preventing it from attacking the living
tissues of the animal.
The pathological effects of tetanus are seen in the cells
of the nervous system, and consist in swelling of the
chromatic bodies and cell-body, followed by progres-
sive chromatolysis. These lesions are of rather irregular
^ Ga::. (legli OsjxdaH, 1901, No. 138. - Quoted by Dean, loc. cH.
^Berlin, klin. TFoch., 1898. No. 1.
106 SERUMS, VACCINES, AND TOXINES.
distribution, being more uniform and intense in the brain
than in the cord ; and with the possible exception of the
early stages of the process, the changes are not specific of
the disease.
The toxine exists in other organs besides the nervous
system, and may be extracted from them by glycerine.^
The peripheral nerves contain it, but have not the same
neutralising action as is possessed by the brain and spinal
cord.
TETANUS- ANTITOXINE.
Preparation of AntitOXine.- — For the practical pre-
l^aration of antitoxine for tetanus, horses are used. These
animals are very sensitive to the toxines of this disease,
and great care is necessary in the process of immunisation.
At the beginning of the treatment use is made of a toxine
which has been attenuated by means either of heat or of
some chemical agency. Iodine trichloride is used by
Behring. After the blood of the animal has been for.nd
to contain a considerable amount of antitoxine, as a result
of these injections, the undiluted toxine may be adminis-
tered. The method of procedure is the same as that
already described in the case of diphtheria.
Standardisation of Antitoxine.— A method of stan-
dardising tetanus-antitoxine similar to that in use for diph-
therial serum has been introduced by Behring. A test-toxine
is prepared of such a strength that O'Olcc. will kill a guinea-
pig of 500 grammes in about four days. This amount of
toxine is neutralised by Yzho ^^ ^ ^^^^^ ^^ antitoxine. In
other words, one unit of antitoxine will protect 1,000
guinea-pigs against the minimal lethal dose of toxine.
1 Waring, Froc. rath. See. Lond., Brit. Med. Jonrn., 1902, i., 965.
- Tetanus-antitoxine may bo obtained from the Lister Institute of
Preventive Medicines, or from their agents, Messrs. Allen and Hanbury,
in liquid form (three bottles of lOcc. each, 12s.) or in solid form
(1 gim. =: lOcc, 48.); or from Messrs. Burroughs, AVellcome & Co.
(Pasteur Institute Serum) liquid or dry (1 bottle, 3s. 4d. ; 1 grm.
solid, 4s.) ; or from Mes.^rs. Parke, Davis & Co., for human use, in
bottlts of lOce. (3 for 12s.), or for veterinary use (1 fluid ounce, 4.';.;.
TETANUS-ANTITOXINE. 107
According to Roux's metLod of standardisation, the
value of the antitoxine is expressed according to the
amount of guinea-pigs (calculated in grammes) which Ice.
of the serum will protect from a minimal fatal dose (for
that number of guinea-pigs). Thus if Ice. of a certain
serum will protect 100 guinea-pigs, each weighing 500
grammes, against the minimal amount of toxine which
would otherwise kill them in four days, the value of the
serum is said to be 50,000 (100 x 500). This would be
a very weak serum for use, 1,000>000 units per cc. being
an average strength.
Unfortunately no standard strength is adopted in the
serums on the market. The dose is generally calculated in
cubic eentimetres, without any statement of the number of
units contained, so that not only is accurate dosage im-
possible, but cases recorded as treated with antitoxine have
very little value owing to the impossibility of knowing how
many units of antitoxine were really used.
Experimental Value of Antitoxine.— From experi-
ments made in the laboratory, in which all the factors are
under control, there can be no doubt that tetanus-ant itoxine,
if given along with, or shortly after, a dose of the toxine, has
the power of preventing the occurrence of the characteristic
symptoms and of death. Donitz ^ finds that, whereas a
certain dose of antitoxine suffices to neutralise a definite
quantity of the toxine when it is injected at the same time,
if a space of four minutes is allowed to elapse between the
administration of the toxine and that of the antidote, then
a slightly larger amount of the latter is needed. If eight
minutes intervene, then six times the original neutralising
dose of antitoxine is required ; if sixteen minutes, then
tw^elve times the dose ; and at the end of one hour, twenty-
four times the amount of antitoxine is requisite. Here, as
in the case of diphtheria, a "mass" action of the antitoxine
may be exerted for a certain time after the poison is
injected ; but there coiiies a time in both cases in which
1 Dcutsch. med. Woch., 1897, p. 430.
108 SERUMS, VACCINES, AND TOXINES.
this is no longer possible — when the toxine has entered so
closely into combination with the cells that no amount of
antitoxine is capable of Avithdrawing it. After that time
even the minimal lethal dose is of necessity fatal. If the
blood of an animal is rendered antitoxic to tetanine,
injection of this j^oison into the substance of its brain will
produce a fatal eiFect as surely as if no protective power
had been gained.
General Considerations on the Use of Antitoxine.
— Tetanus is a rare disease in this country, and is
comparatively uncommon in most civilised parts of the
world. Hence statistics are difficult to collect, and are
generally founded on insufficient numbers of cases for
accuracy. Further, the disease may occur in an acute or in
a more or less chronic form, these varieties merging one into
the other with no distinct dividing line. The mortality in
chronic cases is much less than in the acute. Owing to
these peculiarities, it is difficult to calculate what the
mortality from the disease was before the introduction of
antitoxine, and the statistical method used for demonstrat-
ing the value of diphtherial antitoxine is here even more
liable to be vitiated by errors of observation.
As previously mentioned, the acuteness of a case of
tetanus is to some extent proportional to the rapidity of the
onset of symptoms after infection. Cases which develop
within the first week are usually very severe ; those which
are delayed till after the tenth day show a much greater
percentage of recoveries. There is some reason to believe
that the disease is more virulent in some parts of the world
than in others — a fact which further vitiates the statistics.
Any calculation of the total average mortality from tetanus
is, therefore, of little or no value. It may, perhaj)s, be put
at about 70 per cent. Lambert^ calculates the mortality
in acute cases as varying from 78 to 96 per cent., with an
average of at least 88 per cent. In chronic cases he puts
the mortality between 18 and 55 jDer cent., the average
1 Jled. JVeics, 1900, July, p. 12.
MORTALITY FROM TETANUS. 109
being about -iO per cent. lie quotes Beliring's collection of
71 G cases, among which 88 per cent. died. Lambert col-
lected 1,226 cases occurring in war, with a mortality of
88*6 per cent. ; white among 280 })atients in peace-time the
death-rate was 76 per cent. The death-rate is considerabl}^
higher among the " traumatic " cases (i.e. those in which
the disease follows a wound of sufficient extent to be
remembered, or which is not healed up at the time of the
onset of the tetanus) than in the "idiopathic" or "rheu-
matic " cases. Gowers puts the mortality in the former at
about 90 per cent., and in the latter at about 50 per cent.
In recorded cases of tetanus it is generally impossible to
gather a definite opinion as to the severity of the case, and
hence it is difficult to realise what would have been the
chance of recovery without the use of antitoxine ; while a
comparison of a small series of six or eight cases with a
theoretical general-mortality figure, of the vague description
which we have just indicated, gives very little information.
On the other hand, the observer's opinion in any individual
instance, as to the effects produced by the remedy in abating
symptoms or conducing to recovery, is unreliable.
The data obtained by means of experiments on animals,
as to the behaviour of the poison in the body and as to the
neutralising eflfects of the antitoxine, afford very clear
indications as to what we may expect from the latter in
the treatment of the disease. Nothing is more definitely
established than that there comes a period of time at which
the toxine is so closely attached to the cells of the nerv^ous
system that no amount of antitoxine will suffice to with-
draw it or to counteract its effects. This, unfortunately,
occurs very early in the disease ; indeed, the vital question
is, whether it is not already too late for the antitoxine to
have any good effect when the disease has declared itself,
i.e. when the symptoms, by which alone it can be recog-
nised, have appeared.
This will perhaps be clearer if we recapitulate briefly
what is taking place in the body of the patient, in the light
no SERUMS, VACCINES, AND TOXINES.
of the researches to which allusion has already been made
(page 103). According to the views of Meyer and Ransom,
which agree with those of Marie and Morax, the toxine is
only absorbed by the terminations of the peripheral nerves,
and is by the latter conveyed directly to the central
portions of the cerebro-spinal axis. Death occurs from
intoxication of the important centres in the medulla. A
stream of poison is passing along the nerves and up the
spinal cord all the time. The appearance of symptoms
indicates that this stream has already reached the spinal
cells ; and at this time there is still a further portion of
toxine which has entered the peripheral nerves and is on
its way to the cord. An injection of antitoxine given at
this time will^ indeed, be capable of neutralising any toxine
which is circulating in the blood, and thus of preventing
any fresh dose from entering the nerve-terminations ; but it
cannot prevent the action of the j^oison which has already
been absorbed by them. It is true that the presence of a
large relative projDortion of antitoxine might withdraw
some part of the j^oison from the cells, but it does not
appear to be known how the antitoxine reaches the nerve-
cells. It is possible that it, too, has to be taken up by the
nerve-terminations and passed along to the centres, in
which case its action must be very slow. Even if it can
l^enetrate directly into the spinal cord and brain from the
circulating lymph, it can only do so slowly ; whereas the
toxine is at work all the time. From these considerations
it is apparent that we cannot hope for the same good results
in the treatment of tetanus by antitoxine as are obtained
in diphtheria, in which it is possible to recognise the
existence of the disease by means of the false membrane in
the throat before any considerable degree of intoxication
has occurred.
It does not, however, follow that the remedy is of no
effect at all. It is impossible to know in any given case
whether the amount of toxine which has been taken up by
the nerves is sufficient to cause a fatal termination. By
VALUE OF TETANUS-ANTITOXINE. Ill
adininisterinf;- aiititoxine we cut ott" the supply of the poison
and prevent further absorption. It might be that a
quantity of toxine just short of the minimal fatal dose had
been absorbed at the time when the case came under
treatment. If tlie remedy were given at once, the further
absorption of the poison would be stopped, and the balance
would be turned in the patient's favour ; whereas if aiiv
delay in giving the remedy were allowed to arise, time
might be afforded for the absorption of the additional
amount of toxine necessary to cause death. Hence theo-
retical reasons lead us to the conclusion that the antitoxine
should be used in all cases directly they come under treat-
ment, but that no great fall in the mortality is to be
expected to result. A few cases only will be saved ; no
brilliant statistics of cures are to be looked for ; but we can
never know till death has actually ensued that the case
before us is not one of the exceptions in which the use of
antitoxine will turn the scale towards recovery.
Statistics of the Use of Antitoxine in Tetanus.—
We have already pointed out that tetanus is too rare a
diseaoe for the statistics of it to be valuable ; further, the
use of antitoxine is of comparatively recent introduction, so
that no large number of cases are available for the study of
its effects. Lambert i collected 262 cases, of w^iich 151
recovered, giving a total mortality of 42-36 per cent. These
were divided into 124 acute cases, of which 35 recovered,
giving a percentage mortality of 7 1*7 7; and 138 chronic
cases, of whom 116 recovered, leaving a mortality of only
15-94 per cent. On these figures he pronounces strongly
in favour of the use of the remedy, especially in the chronic
cases, in which the previous mortality was about 40 per
cent. ; in these his figures show a reduction in mortality
(due to antitoxine) of approximately 24 per cent. Abbe -
saw six cases of tetanus in pre-anti toxine days, of which
only one recovered ; whereas of nine others treated with
this remedy seven survived. Five of these latter cases
^ Op. cit. - Ami. of Surg., 1900, xxxi., p. 273.
112 SERUMS, VACCINES, AND TOXINES.
were severe in type, and of them three recovered. For
reasons already given it does not seem worth while to set
forth in detail the figures given by different writers.
Mode of Admimstra,tion.— Subcutaneous injection of
antitoxine was that first employed in tetanus, as in diph-
theria, and the majority of the cases on record have heen
treated by this method. It was, however, pointed out that
the antitoxine was only slowly absorbed by this route,
whereas it was important to neutralise the toxine, which
had already got the start of the antidote, as quickly as
possible. Hence intravenous injection has been advised as
a speedier method. It certainly seems to be preferable to
the subcutaneous route.
Both of these methods affect a neutralisation of the
poison circulating in the blood, but they do not avail to
counteract that which has already reached the nervous
system. Two methods adapted to achieve this end, if it be
possible in any way, have been recommended, namely, the
injection of the antitoxine into the space between the dura
mater and the brain or spinal cord, and injection directly
into the brain-substance. The former is known as sub-
dural, the latter as intracerebral injection. Both of these
have their advocates.
In favour of the subdural method, Leyden i points to
the results of eleven cases which he has collected, among
which there were six deaths and five recoveries. Two of
the latter were cases of his own. It is certain that the
cerebro-spiiial fluid contains toxine in fatal cases of tetanus,
and the subdural injection will neutralise this. It is,
however, uncertain whether the toxine is taken up by the
central nervous syste n directly from the fluid in which it is
bathed. Meyer and Ransom, as already quoted, do not
think that it is ; but there is no doubt that other substances,
such as cocaine, are thus absorbed. Further experiment
seems necessary to decide this question positively.
1 Deutsch. med. Woch., 1901, No. 29, p. 177.
METHOD OF ADMINISTRATION. 11:5
Jacob ^ states that he was successful in saving two-thirds
of his cases at the Charite Hospital in St. Petersburg,
which were treated by the subdural method. He withdraws
10 cc. of cerebro-spinal fluid by lumbar puncture, and then
slowly injects 10 to 20 cc. of antitoxine. Penna- gave
the remedy beneath the cerebral membranes, using large
quantities of serum — in one case 60 cc. at a dose. Daily
injections were given, amounting in all to 100-240 cc, per
case. Of five patients, three recovered and two died —
the latter of intercurrent pneumonia, not of tetanus.
AVallace and Sargent '" report four cases treated by injection
into the spinal theca ; of these three recovered, one being
a severe case and two subacute.
Theory is very strongly in favour of the method of intra-
cerebral injection. It has been pointed out that if the
toxine is injected directly into the brain, no amount of anti-
toxic power in the blood will avert a fatal issue. The
toxine appears to be passed on from one cell to another in
the central nervous system, and it is to be supposed that
the antitoxine will also be thus diffused. Roux ^ saved 35
out of 45 guinea-pigs inoculated with tetanine by this
method, whereas by the subcutaneous injection he had only
two recoveries out of 17 animals. Letoux ^ records four
cases of rtcovery in human patients after intracerebral
injection. He administered 10 cc. of antitoxine into each
hemisphere.
Abbe ^ speaks in favour of the intracerebral route. The
procedure adopted is as follows : — A line is drawn from one
auditory meatus to the other across the vertex of the skull.
From the point at which this meets the mid-line a second
line is drawn to the outer angle of the orbit. The middle
point of this last line gives the site for the injection [Rouxt^
j)oint). An incision is made in the scalp, and a small
1 RottsskyVratch, Jan. and Feb, , 1902 (abstr. in Journ. of the Amer.
Xed. Assoc.^ 1002, i., 977) '
- Semana Medica, Oct, 31, 1901 (abstr. ihid., p. 602).
3 Lancet, 1901, i., 642. ■» Quoted by Abbe, op. cit.
'" Semaine Med., 1901, p. 349. « Op. cit.
114 SERUMS, VACCINES, AND TOXINES.
tre[)hine is employed to remove a piece of the skull. Abl>e
says that the operation can be done satisfactorily under
cocaine-anaesthesia. There does not seem, however, to be
any advantage in this over chloroform, as the latter will
control any spasm which may occur during the operation,
while cocaine will not. An opening is made in the dura
mater and a blunt needle is thrust into the cerebral sub-
stance. The antitoxine must be very slowly forced into the
brain, live cubic centimetres being enough to use on each
side. It would seem better to endeavour to inject the anti-
toxine into the actual brain-substance thanintothe lateral ven-
tricle,! as this latter method is practically equivalent to sub-
dural injection, which can be more easily carried out, if it is
desired, externally to the brain. A procedure similar to the
above may be adopted for subdural injection, if it be decided
to give this intracranially, but the method by spinal puncture
is in all probability as effective. An actual trephine-opening
in the skull is not necessary for the intracerebral injection,
which can be given through a simple hole bored with a drill.
The large cerebral sinuses must, of course, be avoided in
these procedures, and a blunt needle is preferable, in order
to avoid wounding the smaller vessels in the substance of
the cerebrum.
For the purpose of intracerebral injection it is advisable
to prepare a specially strong serum- by dissolving the solid
(dried) substance in half the usual quantity of distilled
water, or by evaporating the liquid serum to half its bulk
at a low temperature in vacuo.
Behring ^ considers that no good results are to l)e
hoped for from the use of antitoxine, if it is administered
more than 30 hours after the onset of symptoms, or if
less than 100 units (on his system) are given. Accord-
ing to the experience of Moeller,* even if these
1 See Kocher, Centralhl. f. Chirurff., 1899, No. 22, as to technique.
- Church, ]Sew York Med. Journ., Dec. 17, 1898.
3 Dentsch. med. JFock., 1900, No. 2.
■* Ibid., 1901, No. 47, p. 814.
PROPHYLAXIS OF TETANUS. 115
postulates are fultilled, no great fall in tlio inoitality is
to be looked for.
A considerable number of isolated cases are re}>oi't(!d,
but as they are by different observers, using diflerent brands
of serum and employing a variety of methods, little would
be gained by tabulating them. Tizzoni i considers that the
serum which he prepares is superior to that of Behring,
while German writers " apparently hold that the disease in
Italy is less virulent than in their own country. No exact
data seem to be available for estimating the truth of these
international amenities.
On the whole, it must be confessed that, as was to be
anticipated on theoretical grounds, the curative treatment
of tetanus by antitoxine is disappointing, as compared, at
least, with the results obtained in diphtheria. Nevertheless,
it is our duty to give the remedy a trial in all cases, since it
is impossible to be sure that it will not do good, and it can
in any case do practically no harm.
Prophylactic Use of Antitoxine.— There is hope
that antitoxine will prove of greater service by way of pre-
vention than as a cure for the developed disease. Experi-
ments on animals point strongly in this direction. Actual
results are also encouraging. In veterinary practice some
striking statistics are quoted by MacFarland.^ In one of
the large factories for the production of antitoxine (diph-
therial) much trouble was at one time caused by the inci-
dence of tetanus among the horses. At last systematic use
of prophylactic injections of antitoxine were instituted, and
the result was that, whereas in one year (1898) the death-
rate among the animals was 10 per cent , in the year
following it fell to 1 per cent., and in the year after that to
a mere fraction. Nocard "^ reports that he injected 2,727
horses with antitoxine in a certain district ; none of them
1 Rifonna Medica, 1901, i., p. 3G6.
- See Pfeifl'er, Zeitschr. f. Heilkunde, Bd. xxxiii , TTeft 2.
^ Jour It. of the Amer. Med. A.s.soe.. July 4, 1903, p. 34.
•* Quoted by Lambert, op. cit.
116 SERUMS, VACCINES, AND TOXINES.
developed tetanus, although among other animals in the
same part of the country there were 259 cases of the
disease. Herhold,i who was surgeon to the German
contingent on the expedition to Pekin, at first lost several
jDatients by tetanus. Afterwards he used prophylactic injec-
tions in all cases of cruslied wounds contaminated with
dirt, and no more cases of the disease occurred. Fisch ~
considers that the preventive use of the antitoxin e was of
considerable service in the epidemic v/hicli occurred at
St. Louis from contaminated diphtherial antitoxine.
At the same time, it seems necessary to admit that an
injection of antitoxine is not a certain preventive of tetanus,
as Reynier 3 records that in a 'hospital in Paris, in conse-
quence of an outbreak of the disease, a patient was given a
prophylactic injection. In spite of this she developed
tetanus, though she finally recovered. Two other patients
died of the disease ; so it is possible that even here some
good was done, and that what would otherwise have been a
fatal attack was rendered milder.
We may conclude that in countries where tetanus is a
comparatively common complication, all cases of crushed or
dirty wounds should receive a prophylactic injection of
antitoxine. How long the protection will last is not known.
As the serum, being derived from the horse, is exactly
analogous to diphtherial antitoxine, it seems legitimate to
assume that it will be excreted or neutralised at the same
rate as the latter ; hence we may conclude that the prctec-
tion will remain effective for about three weeks.
Dose of Antitoxine. — As was mentioned above, re-
cords of cases generally state the number of cubic centi-
metres of serum injected. A dose is usually from 10 to
20 cc. The amount of antitoxic units contained in the
serum is not often noted on the bottle, and hence is not
known to the administi-ator. This is unfortunate, as no
1 Beutsch. med. TFoch., 1901, Xo. 30, p. 479.
- Interstate Med. Jonni., Dec, 1901.
3 Quoted by Moeller, Beutsch. med. Woch., 1901, Xo. 47, p. 814.
1
CEREBRAL EMULSION IN TETANUS. 117
accurate dosage or comparison of results is possible. Behriiig
advises that not less than 100 of his units should be given
at once. Amounts of 10 to 20 cc. are easily administered
subdurally, around either the spinal cord or the brain.
Three to live cubic centimetres can be injected into the
substance of the brain by slow instillation. Prophylacti-
cally, 10 cc. may be given.
Ill Effects of AntitOXine.— The same by-effects may
be expected to occur after the use of tetanus antitoxine as
after diphtherial serum, since it is horse's serum which is
us 3d in both cases. Di Gaspero ^ describes a case in which a
fatal issue occurred, due, as he thinks, to the serum. This
inference is very doubtful. Rashes of various kinds,
scarlatiniform, bullous, urticarial, Slc, are also recorded.
TREATMENT WITH CEREBRAL EMULSIOX.
In view of the great affinity of the substance of the
central nervous system (brain and spinal cord) for tetanus-
toxine, use has been made of emulsion of fresh brain-
substance as treatment for the disease. Krokiewicz ~ records
16 cases in which use was made of this preparation, an
unfiltered emulsion being injected hypodermically. Of the
16 cases, 13 recovered, three of them being severe attacks.
When we consider the difficulty with which an emulsion
of cerebral substance must be absorbed and reach the
circulation, it is easy to realise that the curative action of
such a remedy must necessarily be slower than that of
antitoxic serum. The latter should therefore probably be
given the preference, if it be available. If, however, no
antitoxine be at hand, it would certainly be, not only
legitimate, but also advisable, to make trial of cerebral
emulsion. The mode of action of the two remedies is
theoretically the same, the receptors of the cerebral cells
being available in the emulsion to anchor the toxine and
neutralise it, just as the free receptors (side-chains) do in
1 LieTherap. der Gegenwart, 1902, p. 139.
•2 Klin, therap. JFoeh., Feb. 8, 1903.
118 SERUMS, A^ACCINES, AND TOXINES.
the antitoxine. The diffusion of the emulsified cells must
be less rapid than that of the cast off receptors — which
are separate molecules, not whole cells.
CONCLUSIONS.
1. Tetanus-antitoxine cannot be expected to cure any
large proportion of cases in whom the disease has developed.
It should, however, be given in all cases of tetanus, since it
may just turn the scale in the patient's favour by neutralis-
ing the poison circulating in the blood, although it may not
reach that already absorbed by the nervous system.
2. The dose should be from 10 to 20 cc. if the strength
of the serum is not stated. If this be known, not less than
1 00 units (Behring) should be administered at once. The
injection may be repeated in 6 to 12 hours. Intracerebral
injection probably offers the best chance of neutralising the
poison already absorbed ; this method should be used in
severe cases, subcutaneous or intravenous injections being
employed at the same time.
3. Frophylactic injections of antitoxine should be used in
countries where tetanus is common, in all cases of crushed
wounds in which dirt or other foreign matter has been
ground into the wound. The antitoxine may be given
subcutaneously in such instances.
IIJ)
CHAPTER Vil.
SNAKE-BITE.
Classification of Snakes. — The poisonous varieties of
snakes belong chiefly, if not entirely, to the families
Coliihridfc and Viperidce. The best known kinds belonging
to tlie Colubrine group are the Cobras [Naja), the Coralline
snakes (Flaps), the Kraits {Bungarus), and the Death-
adder (Pseudechis). To the Viperine division belong the
Common Viper (Vipera verus), Russell's Viper (Daboia),
the Puff- Adder (V. arietans), the Rattlesnakes {Crotalus),
the Bush-master (Lachesis), the Tiger Snake (Hoplocephaliis),
and the Copperhead (Ancistrodon). The classification
depends mainly on small difierences in the teeth and the
bones of the head. The poison-apparatus is much the
same in all, consisting of a secretory gland on each side,
with a duct leading to the root of a long fang situated in
the upper jaw. Each fang is perforated by a canal which
reaches nearly to the tip, where it opens on the surface. In
the act of striking, the poison is forced along the canals in
the teeth, and is thus injected into the wound. The amount
of poison injected in an individual case depends on the
efficiency with which the stroke was delivered, while the
A'irulence of the poison varies with the health and vigour of
the snake at the moment, and to some extent with the
season of the year.
Different animals vary somewhat in the degree of suscep-
tibility which they present to the action of snake-poison.
Rabbits, guinea-pigs, and herbivorous animals in general,
seem more susceptible than carnivora, such as the cat or
dog. Man is probably about equal in this respect to the
latter class. The minute dose of poison which is capable of
causing symptoms is illustrated l>y the experience of the
120 SERUMS, VACCINES, AND TOXINES.
late Mr. Frank Buckland, who accidentally pricked his
finger in the process of dissecting the body of a rat killed by
the bite of a poisonous snake. Only an infinitesimal amount
of fluid could have been inoculated, and the original dose of
poison had been diluted by diflfusion throughout the body
of the animal ; nevertheless alarming symptoms of faintness
and collapse ensued.
Nature of the Poison. — The poison may be collected
V)y killing the snake and dissecting out the gland and recep-
tacle ; or the snake may be " milked " by pressure on the
sac ; or it may be made to bite on a watch-glass covered with
an indiarubber membrane, and the poison thus collected in
the glass. The last method is probably the best. The
poison is then dried in vacuo, and may be preserved
indefinitely in this condition.
It has long been known that the poisons of different
snakes are not identical in their effects, the poisons of the
viperine group having, as a rule, a more intense local action,
those of the colubrine family rapidly producing a general
intoxication. Calmette, however, to whom we are
indebted for the original preparation of an anti-venomous
serum {antivenene), considered that the main toxine, at all
events, was the same in all venoms, and could be counter-
acted by one and the same antitoxine. This view was
combated by Martin,^ who, as the result of his experiments,
came to the conclusion that there were two substances
present in the venom of all snakes, varying, however, in
proportions in different species. One was a globulin, coagu-
lable by heat; the other a peptone, which was resistant to
it. The former is responsible for the general nervine in-
toxication, the latter for the local effects and blood changes.
More recently Lamb" has given reasons for rejecting
these views. He is inclined to think that the poison of
each genus of snakes is specific, containing a kind of
1 Allbutt's " System of Medicine," Vol. ii., art. " Snake Poison and
Snake Bite."
- Scientific Memoirs by Officers of the Med. and Sanit. Depts. of the
Gov. of India. No. 5, "Specificity of Anti-venomous Sera," 1903.
ACTION OF SNAKE-VENOM. 121
poison not met with in other varieties. Tlius he holds tliat,
though the poisons of the cobra and the daljoia are eacli
complex, no single constituent is common to both. The
reasons for this will be clearer when we come to consider
the action of antitoxic serum as an antidote to the poisons
of different snakes„ Lamb considers that in all proi)ability
the poisonous element is not proteid in nature. He shows
that the proteids in the different venoms are, as far as can
be determined, the same in all, while tiie effects produced
are diverse. This is opposed to the current view that the
poison is a globulin, but it would bring snake-poison into
the same category as the toxines of diphtheria and tetanus,
of which the former was shown to be developed in a non-
albuminous medium, while the latter, according to Dean, is
also of the nature of a ferment rather than an albumen.
Action of Venom.— Locally^ the effects produced by
snake-bite are swelling, redness, and ecchymosis ; if the
patient survive the first effects, cellulitis and sloughing of
the parts may occur, and the cedema may spread up the
affected limb. The general effect of the poison is shown in
its action on the nervous system, which takes the form of
depression, collapse, nausea or vomiting, incoordination,
paralysis, and convulsions, ending in coma and death by
asphyxia or heart-failure. Ha^morrhagic discharges from
mucous membranes are sometimes seen.
Experimentally, snake-poison is found to ha^■e a solvent
action on the red corpuscles of the blood (haemolysis).
Lamb finds that both cobra- and daboia-venom have this
property. Cobra-poison, however, acts more powerfully
outside the body than within it, while daboia-venom is more
h?emolytic in vivo than in vitro. The h?emolytic substance
in cobra-venom is not coagulable by heat, Flexner and
Noguchi 1 assert that the haemolytic agent in venom is of
the nature of a coj^ula or intermediary body (see page 24).
The alexine or complement which is necessary to complete
^ University of rennsylvania Medical Bulletin, 190'J, xiv., 438;
and XV., 345.
122 SERUMS, VACCINES, AND TOXINES.
its action on the blood-corpuscles is contained within the
corpuscles themselves (endo-complement). Snake-poison
also possesses the power of agglutinating red corpuscles,
the clumps thus formed being disintegrated again ])y the
action of permanganate of potassium. The agglutinative
power is removed by heating the venom to 75° or 80° C,
whereas the hsemolytic power still remains after this treat-
ment, showing that the two properties are dependent on
distinct toxines. Leucocytes are destroyed by the poison as
well as red corpuscles (leucolysis). The different kinds of
leucocytes contained in rabbit's blood are unequally affected
by the leucolytic substance, the lymphocytes being the
least susceptible.
The ecchymoses seen in cases of snake-bite are due to
the action of the poison on the endothelial cells forming the
lining membrane of the capillaries. This action is
analogous to haemolysis, and is due to a destructive "lysis "
of these cells by a special substance, which is called by
Flexner and Xo2;uchi " haemorrhagin," from its effect
in producing haemorrhages. It is suggested b}^- these
observers that perhaps similar substances may exist in the
blood of patients suffering from the various forms of i)ur-
pura. In a later communication, these authors %state that
venom has the power of dissolving other cells beside blood-
corpuscles {e.g. liver-cells, spermatozoa, &c.). Daboia- venom
also liquefies gelatine by means of a special ferment.
According to Lamb ^ the venom, of the daboia produces
intravascular clotting of the blood, whereas cobra-poison,
on the other hand, has an exactly opposite effect, causing a
diminution in the coagulability of the blood. Death in cases
of bite by the daboia results from the extensive character
of this clotting. Curiously enough, it appears that a small
dose of the poison, insufficient to cause this phenomenon, is
followed by a diminution of coagulative power ; and if this
1 Scientific Memoirs of the Government of India, No. 4. 1903,
" On the Action of the Yenoras of the Cobra (Xaja tripudians) and of
the Daboia (D. Russelliil," ^tc.
CONSTITUENTS OF VENOM. 123
has once been produced, no subsequent injection of further
doses of the poison will any longer produce clotting, Tliis
peculiar phenomenon is not reproduced in vitro, and is
dependent on some obscure vital action. The substance in
cobra-venom which reduces the coagulative power of the
blood is not a proteid coagulable by heat. The addition
of snake-venom to blood fias the effect of reducing the
bactericidal power of the latter by depriving it of the
necessary alexines, which become tixed to the copulas pre-
sent in the venom. The latter does not contain alexine.
Kyes has found that lecithin is capable of acting as alexine
in presence of the copula contained in venom.
The element in venom which acts on the nervous systc^m
is called by Flexner and Noguchi " neurotoxin." It, too,
is of the nature of a copula, and acts by fixing a suitable
alexine to the nerve-cells. These observers also found
that the brain was the organ in the body which con-
tained the most neutralising substance for venom, i.e. that
its cells have the greatest affinity for the toxine. An animal
injected with a minimal lethal dose of venom mixed with
emulsion of brain lived many hours longer than one which
received the same quantity of poison in blood-serum or
similar fluid. Rogers ^ states that the poison of the sea-
snake, Enhydrina, has an action resembling that of curare.
The principal poisonous substances in snake-poison are
therefore (1) Hsemolytic, (2) Leucolytic, (3) HcTmorrhagic,
and (4) Neurotoxic. The bodies having these separate
actions are probably different in the various kinds of
poisonous snakes. Hence it is evident that the term
" snake-poison " or venom includes a very complex group of
chemical substances.
ANTIVENENE.
Preparation of Antivenene.-— The possibility of pre-
paring an antitoxic serum (antivenene) for the treatment
1 ZaNcet, Feb. 6, 1904, p. 319.
- Calmette's anti venomous serum may be obtained from Messrs.
Burroughs, Wellcome & Co., in bottles, at 4s. each.
124 SERUMS, VACCINES, AND TOXINES.
of snake bite was first practically demonstrated by Calmette
of Lille. His serum is manufactured by injecting a horse
with gradually increasing quantities of a mixed venom,
containing 80 per cent, of cobra- poison and 20 per
cent, of viperine venom. The mixture is heated before the
injections are given, as the crude poison is so intensely
toxic that the horses are often killed by the minute
quantities used for immunisation. Thus MacFarland ^
states that he lost two out of three horses in which he
practised the inoculations. It is therefore necessary to
proceed with the greatest caution in these injections. A
further difficulty is met with in the process, owing to the need
of procuring large quantities of venom for the later injections
— a need which is not easily satisfied for obvious reasons.
It is advisable to administer to the horse before inoculation
one or more protective doses of antitoxine, in order to
enable it to withstand the first injections of the poison.
Otherwise the method adopted is practically the same as
that already described for the antitoxines of tetanus and
diphtheria. Tidswell ^ took more than three years in im-
munising a horse against the venom of the Australian
tiger-snake, owing to a combination of the above-mentioned
difficulties.
Action of Antivenene. — Calmette claims that the
antivenene which he prepares is capable of neutralising the
effects of the venom of any snake, whatever the species to
which it belongs. The mixture of venoms which he uses
for the inoculation is calculated to render the horse resistant
to the poisons of viperine as well as colubrine snakes, even
if it be not the case, as he apparently holds, that the venoms
of all kinds of snakes are identical. It has, liowever, been
pointed out by Hanna and Lamb ^ that the heating process
to which the mixed poison is subjected, in order to render it
less virulent, before injection, is capable of destroying the
potency of viperine poison altogether, or almost entirely.
^ Journ. of the American Med. Asioc., 1901, xxxvii., p. 1597.
- Australasian Med. Gaz., April, 21, 1902.
•^ Journ. of Fathol. and JBacter,, 1902, viii., 1.
ACTION OF ANTIVENENE. 125
Hence only the cobra-venom is actually left to immunise
the horse.
Experience seems to confirm this view to a great
extent. Thus Tidswell finds that Calmette's serum has
no effect in neutralising the poison of the Australian
tiger-snake {^Iloplocepliahift curtus); nor had an antivenene
prepared from the venom of this latter any antidotal power
against the poisons of other snakes met with in the same
continent (Echis, P sendee] ds). Lamb similarly found that
cobra- antivenene has no antitoxic power against the bite of
the daboia ; and in a later memoir he proved that it was
unavailing against the poisons of the snakes known as
Bnngarus can'ideus and Ecliis carinata. Serum prepared with
the venom of the hoplocephalus by Tidswell had no neutral-
ising effect on the venom of bungarus, cobra, or daboia.
These results are of considerable practical importance.
If the venoms of the different snakes are thus specific in
nature, so that a serum prepared from one of them has no
neutralising effect on poison derived from another species,
the question of the practical therapeutics of snake-bite
becomes much less simple than was originally hoj^ed by
Calmette. It would seem necessary to have at hand in all
cases a supply of serums for all the different varieties of
snakes found in any district, or else to prepare a polyvalent
serum by injections of the poisons of all of these reptiles.
It does not seem to be known at present how far the latter
suggestion is feasible. On the other hand, it is evident that
if separate serums were prepared for each kind of snake it
would often be necessary to inject all of them in a case of
snake-bite, since the patient could not be expected to know
what was the kind of snake which bit him. Bites from
snakes often occur at night, when it would be impossible
for anyone to identify the assailant ; while those who are
not skilled biologists would in any case not be likely to
know one poisonous variety from another.
With regard to the neutralising action of antivenene on
the different constituents of snake-poison, there seems some
126 SERUMS, VACCINES, AND TOXINES.
divergence of opinion. Flexner and Noguchi ^ state that
antivenene is capable of inhibiting the effects, not only of
the neurotoxine or main poisonous element, but also of the
hsemolytic and other materials. On the other hand, Mac-
Farland - finds that it is very difficult, if not impossible, to
l^roduce immunity to the local irritation of the poison. He
considers, however, that the remedy should be used in all
cases, as the counteraction of the most deadly toxine allows
the body to concentrate all its resistant powers on repelling
the local irritant. According to Auche and Yaillant-Hovius "
the presence of antivenene does not prevent haemolysis
altogether, but renders it more transitory and less intense.
If the neural toxine of snake-poison be removed, the body
which gives rise to coagulation of the blood may still cause
death, if it is present in a sufficient amount.
Standardisation of Antivenene. — For the purpose
of experimental study of the action of venom and of anti-
venene on animals it is necessary to arrive at some standard
of virulence and protective power respectively. This is done
by determining the minimal lethal dose for a certain kind of
animal (rabbit or rat), calculating the weight of the latter
in grammes. Thus Lamb found that 0*05 milligramme
(0-00005 gramme) of the venom of Uchis carinata per
kilogramme of body-weight was fatal to rabbits ; in other
words, a rabbit weighing 1,000 grammes would be killed by
the above quantity of poison, while one weighing 1,500
grammes would require half as much again. Of the venom
of Bungarus fasciatus 0*7 mg. was required to produce the
same effect. The venoms of different snakes thus differ
markedly in their actual toxicity. Further, as was pre-
viously stated, different species of animals vary somewhat
in their susceptibility to snake-poison, while the actual
toxines are probably very ditierent in the various kinds of
snakes. Hence no accurate measurement of toxines and
antitoxines applicable to all can jDossil^ly })e arranged.
^ Journ. of Expe)imental Medicine, March 17, 1902. - Op. cit.
3 Arch, de Med. Experimentale et d'Anat. Fathol., 1902, xiv., 221.
DOSE OF ANTIVENENE. 127
The only antitoxic serum on the market is Calmette's
antivenene, which is effective for cobra-poison. This is
standardised by experiments on rabbits. The amount of
serum which will protect a rabbit weigliin,i>- 2,000 i>Tammes
against the smallest amount of the toxine which would
otherwise kill it, is said to contain 2,000 units of antitoxine.
Tlie whole matter is as yet in so experimental a sta<,'e that
the standardisation of the antivenene is scarcely of practical
therapeutic importance.
Dose of Antivenene- — Doses of 10 to 20 cc. are gener-
ally administered. Lamb, however, considers that this
amount of a serum of the ordinaiy strength is not sufficient
to protect against the whole amount of poison which a full-
grown, healthy snake can inject at a bite, and he advises
the use of not less than 40 cc. if the quantity be available.
Kogers calculates that as much as 400-800 cc. may be re-
quired. If any time has elapsed since the bite, the remedy
should be given intravenously. If the case be seen at once,
injection into the neighbourhood of the bite may be
employed. Ordinary measures, such as the constriction of
the bitten limb by a tight ligature above the seat of injury,
})ressure to squeeze out any poison lying free in the punc-
tures, and stimulating remedies, must not be omitted.
CONCLUSIONS.
1. Calmette's Antivenene should be used promptly in
all cases of snake-bite. It protects effectually against cobra-
bites. It probably has no eflfect against the venom of snakes
belonging to other genera, but this matter is still under
investigation, and in any case it is difficult, if not impossible,
to })e certain of the kind of snake which has inflicted the
bite in countries where several kinds are met with. The
dose should be 40 cc. or more, if possible.
2. Fuller investigation is necessary with i-egard to the
manufacture of a polyvalent serum applicable to the bites
of more than one kind of poisonous snake.
128
CHAPTER YIII.
■S:\IALL-POX AND VACCINIA.
Causal Agent. — That small-pox is due to some living
agent similar in nature to the organisms of other infective
diseases there can be no doubt, but the actual germ has not
been certainly isolated. Peculiar refractive bodies have
been described in the blood of patients sujffering from the
disease, and in the lymph used for vaccination. Thus
Buttersack^ described in vaccine-lymph certain threadlike
structures and round bodies resembling spores. It is,
however, doubtful whether these are not artificial. Van
de Loefi' and Guarnieri ^ found appearances which they took
to be protozoa, and their results are confirmed by Pfeifter.^
This organism (?), to which the name Cytoryctes variolce
was given, is said to be found in the blood of small-pox
patients and of vaccinated children. It is about one-quarter
the size of a blood-corpuscle, and exhibits active movements,
throwing out pseudopodia or flagella. It does not penetrate
the blood-corpuscle as does the malarial parasite, but sticks
to the side of it. The parasite is deposited in the skin by
some process allied to embolism, and thus causes the rash.
In the epidermis it penetrates the cells of the rete Malpighii,
and gradually destroys the cell -protoplasm, pushing the
nucleus to one side. Funk -^ described the existence in
vaccine of adult organisms (2.5 fx in diameter), together
with cysts full of spores, and also free spores (1.3 yu in
diam.). He considered that he had been able to produce
1 Quoted h}^ Immermann. Nothnagel's " Encyclopaedia of Pract.
Medicine," English Edition, 1902.
2 Centralhl. f. Bal-t,, xvi„ 299.
3 Monatsh,/. pyaTit. Derm., iv., 43o.
^ La Semaine Medicale, 1901, p. 57.
PARASITES IN SMALL-POX. 129
the disease by inoculation of these bodies, and he gave the
organism the name of Sporidium vaccinale.
Recently Councilman ^ has described organisms, also
belonging to the order Protozoa, which he holds to be the
infective agents in small-pox. These parasites are said to
pursue a double life-cycle within the cells, one phase being
extranuclear, the other intranuclear. The latter is supposed
to correspond with the sexual cycle of the malarial parasite.
These organisms, which are acknowledged as protozoa by
Calkins,- a well-known authority on these minute animals, and
which Councilman considers to be identical with Guarnieri's
Cytonjctes, appear first as small homogeneous bodies about 1 ^
in diameter, in the protoplasm of the cells. They quickly
increase in size, and coincidently the cell seems to undergo
a process of degeneration. As they enlarge, the parasites
become granular in appearance and irregular in outline ;
and finally they break up into a number of still smaller
fragments, which are regarded as spores. These proceed to
invade the nuclei of the cells, which have so far been left
alone. As the invaders once more enlarge, the nuclei are
in their turn destroyed, and the parasites are set free. A
second division into minute fragments may take place.
These free bodies are supposed to be the infective agents by
which the disease is communicated to other individuals. It
is suggested tlat vaccinia represents the extranuclear phase
of the organism, whereas small-pox consists essentially in
the invasion and destruction of the nuclei. If these obser-
vations are confirmed, the discovery will be a matter of great
interest. We shall have an instance of the attenuation of
a protozoan jDarasite taking place by passage through
another animal, just as occurs in the case of vegetable
parasites (bacteria). Further, a vaccine will have been
prepared against a protozoon '^ as well as against bacteria,
^ Boston Med. and Surg. Journal, April 30, 1903.
•- Journ. of Med. Research, Feb., 1904, p. 136.
'^ Ledoux-Lebard (Comptes Rendues de rAcadenile des Sciences, 1902,
cxxxv., p. 298) sta es that he has prepared a specific antiserum to the
J
130 SERUMS, VACCINES, AND TOXINES.
showing that the human body has the power of forming
l^rotective substances against this order of pathogenic
agents, as well as against vegetable organisms. Again, the
suggestion of an organism undergoing two different cycles
within the same animal host, but in different positions (cell
and nucleus), is of considerable interest.
Sugg and de Waele ^ found strej^tococci in the blood
in 27 fatal cases, and believe that these organisms (which are
agglutinated by the serum of vaccinated persons) are the
causal agents in small-pox.
Identity of Small-pox and Cow-pox. — The question
of the identity or difference of small-pox and cow-pox was
long disj)uted, but there can now be little doubt that
Jenner was right in holding that the latter is only small-
pox moditied by transmission to a different animal, which
is less susceptible to the disease.- Many attempts have
been made to transmit small-pox directly to cattle, and a
certain number of successful results have now been
recorded. Adult cows take the disease with difficulty ;
calves are more easily infected. A condition in horses
analogous to vaccinia (equine variola) appears to be really
the same disease, and caf)able of effecting vaccination.-^
Complications of Small-pox. — ^lost of the com-
plications arising in small-pox can be distinctly traced to
intercurrent infection with pyogenic micrococci (strej)-
tococci or staphylococci). It has even been said that the
pustular stage of the lesions, which has been regarded as so
characteristic of small -pox, as opposed to chicken-pox, can
be almost entirely prevented by careful and thorough anti-
septic treatment of the skin. If this be true, it would seem
protozoan crganism. Fnrca/ia'cium candatum, which is pathogenic to
some of the lower animals (rabbits and guinea pigs). Cf. also p. 329.
^ Arch, interna t. de Pharmacod. el de Therap., xii., Nos. 3 and 4,
- See Blaxall: Z\st Ann. Report of the Local Gov. Board. 1901-2.
Supplement containing the Report of the Medical Officer. Appendix
C, i., p. 568.
3 It has been suggested that vaccine contains the poison of \ariola
in the " toxone'''' condition, a modified form of toxine. See Grriinbaum,
The Practitioner, Dec, 1903, p. 809.
THEORY OF VACCINATION. 131
that the cutaneous manifestation of tlie disease is essentially
a vesicular eruption, and only accidentally becomes pus-
tular. Abscesses are the commonest complication met
with ; erysipelas and cellulitis are by no means rare. The
ocular affections (keratitis and conjunctivitis) may possibly
be due to the virus of the original disease, but here again
the action of secondary infections can hardly be excluded.
Pneumonia, pleurisy, and empyema are also probal)ly in-
stances of intercurrent infection.
Infection in Small-pox. — The exact method by
which the infective agent in small-pox enters the body is
not known. There seems little doubt that it is carried l^y
the air, as statistics prove that a small-pox hospital is a
source of infection to the district surrounding it, the
liability to infection gradually diminishing as distance from
the hospital increases. The contagium is apparently capable
of being transported for a considerable distance. It seems
probable, therefore, that it enters by the respiratory tract.
The incubation-period in cases contracted by the ordinary
method of infection is twelve days.
VACCINATION.
Theory of Vaccination. — As already explained,
vaccination consists in inoculation of an attenuated form of
small-pox germs, the diminution in virulence being brought
about by passage through the body of a calf, a less suscep-
tible animal than man. The attenuated germs are present
in the lymph of the vesicles formed on the vaccinated
person, and this lymph may be used for inoculation of
other individuals, as the germs do not regain their virulence
by re-passage through man. Vaccinia remains a localised
disease, the attenuated germs remaining in the place of
inoculation, and not becoming generalised by the blood-
stream. At the point of inoculation they form their
toxines, which are conveyed all over the body, and
stimulate the tissues to form germicidal substances. ■ The
cells thus educated retain the property of secreting these
132 SERUMS, VACCINES, AND TOXINES.
substances for a considerable length of time ; in other words,
the person vaccinated has gained an active immunity to
sinall-pox and vaccinia.
Preparation of Lymph. — It is immaterial, theoretic-
ally, from what source, human or bovine, the lymph is
derived, but for reasons set forth below, the use of material
got from an " animal " source is to be preferred in practice.
What is known as " glycerinated calf-lymph" is now in-
variably used in this country. i This is prepared in the
following manner : — A supply of stock lymph being already
available, a calf is taken, and its abdomen is shaved. A
series of incisions are made in it of considerable length, and
the stock lymph is rubbed into them. By the fifth day large
vesicles have developed along the course of the incisions,
and are full of clear fluid, which does not yet exhibit any
tendency to become pustular. At this stage the vesicles
and their contents are scraped off with a sterile sharp spoon,
with all aseptic precautions, and the resulting material is
collected in suitable bottles. It is next finely broken up,
and triturated w4th four times its weight of glycerine ^ and
water (50-per-cent. solution). The thick, creamy fluid pro-
duced is run into tubes ; and these are kept in a cold, dark
^ In India, in addition to glyceiinated lymph, mixtures with vase-
line and with lanoline are also employed, apparently with good results,
'^ A preliminary communication has recently appeared fro7n
Dr. Alan B. Green, in which he recommends the use of chlorofoi-m
instead of glycerine for the purpose of killing extraneous organisms
in vaccine-lymph. Air charged with the vapour of chloroform is made
to pass through a series of tubes of vaccine, an 1 the chloroform is sub-
sequently expelled from the tubes by means of a current of air. The
lymph is first mixed with water for the purpose of this procedure, and
only the pi'oportion of chlorofor in which the water can hold in solution
(1 : 400) can come in contact with the vaccine. This quantit)'' suffices
to kill the bacteria (chiefly staphylococci) generally present, but has no
ill effect on the vaccine. The addition to this of any traces of liquid
chloroform appears, however, to diminish its activitv. The ad-
vantages claimed for this methoi ai'e the speed with which sterilisa-
tion is effected, so that in cases of emergency large quantities of "
vaccine can ba rapidly rendered fit for use, and the consequent avoid-
ance of any possible deterioration of strength, such as may perhaps
occur during the month or more for which ordinary glycerinated
lymph has to stand. — Lancet^ June 20, 1903, p. 1,738.
TECHNIQUE OF VACCINATION. 133
place for some weeks. The result of this treatment is to
kill oti' most of the common pyogenic and similar organisms
which might do harm if inoculated ; but few if any speci-
mens of lymph are actually germ-free. The contagium
of vaccinia is left apparently uninjured. It is possible that
it exists at this stage in the form of spores, which are
resistant to the action of the glycerine. After about a
month the lymph is tested bacteriologically, to prove it
free from the organisms alluded to ; and if it is found to
be sterile, it is drawn into capillary tubes, and is ready
for use. The lymph thus prepared is a thick, syrupy fluid,
which tends to separate to some extent into a clear and an
opaque portion. It is probable that the latter is the active
part, and care should therefore be taken not to use only
the clear portion in vaccinating.
Technique of Vaccination. — The essential part of
the process of vaccination is that the infective material —
the lymph — should be introduced through the epidermis, so
as to be absorljed by the lymphatics and blood-vessels of
the corium. The skin should first be cleansed with soaj)
and water, and, if there is reason to fear the occurrence of
sepsis owing to the surroundings or person of the patient,
with some antiseptic such as carbolic acid (1 : 20). It is
necessary, however, to remove the antiseptic from the skin
before making the inoculation, lest it inhibit the growth of
the living matter of the vaccine. The skin is then
scratched through with a sharp instrument (lancet or
needle), previously sterilised by boiling or by some suitable
germicide. The skin should be put upon the stretch by the
left hand of the operator, and the strokes of the instrument
made quickly and lightly. The appearance of a trace of
V)lood in the scratches shows when the corium is reached.
The ends of the tube of lymph are then broken off and the
material blown on to the scarified areas, or on to one of
them, the material being then distributed as equally as
possible to all of them. It is next lightly rubbed in with
the lancet or wdth any smooth instrument preferred,
134 SERUMS, VACCINES, AND TOXINES.
provitled that this is scrupulously clean ; and the lymph is
left for a few minutes to soak in.
Another mode in which vaccination may be effected is
by blowing the lymph directly on to the skin after pre-
liminary cleansing, and then making the necessary scari-
fications through the droj)s of lymph into the corium. The
several sites of inoculation should not be too close together
— preferably an inch or an inch and a half should be left
between them — in order that the resulting vesicles may not
coalesce. Should they do so, an unduly sore arm may
ensue, and a consideral^le amount of scarring he finally left.
Table showing the Age-Ixcidence of Yaccixated Cases
classified according to the character of the scahs ; each
Class is represented as Comprising a Total of 1,000 Cases.
(Sandilands.I)
Character of Scar.
Under ! ,q_
10 years.
20-
30-
40-
50 and
upwards.
Large (A 1)
Medium (A 2) ...
Small (A3)
15 187
22 109
45 112
411
248
199
253
268
241
98 ! 36
222 ! 131
211 192
Four or more
Three
Two
One
23 217
11 .48
]5 114
31 129
441
350
273
270
207
309
292
236
77
127
190
203
35
55
116
131
Half or more than ]
half foveated ... i
Less than half
foveated
Pi 1 in scars
21 : 188
15 183
27 127
418 246
387 263
293 238
99
107
182
28
45
133
Scars absent
83 204
210
157
127
219
Dr. Sandilands points out that "the figures in this table demon-
strate a point of some importance — that the incidence in later life
is very much greater in the classes with inferior vaccination scars."
Glycerinated lymph does not dry up, and it is of no use
waiting for this to occur. No special protection is
^ " An Analvsis of the Vaccination Statistics of the Metropolitan
Asylums Board for 1901 and 1902." {Lco/cef, 1903, ii., 378.)
MODE OF VACCINATING. 135
necessary for the areas of skin inoculated, a shield of any
kind not being advisable; but in patients whose sur-
roundings are insanitary a dressing of sterile (not anti-
septic) wool may be applied and secured with a bandage.
Statistics seem definitely to prove that tlie protection
afforded by vaccination is to some extent proportional to
the number of spots at which the lymph is inserted, two
" scars " protecting better than one, three than two, and
four than three. The practice of making only one insertion
is to be condemned as inefficient and conveying a felse
security. The scars resulting from the vaccination should
together make up an area of not less than half a square
inch. The table on page 134 shows the nature of the statis-
tical evidence upon which these statements are founded.
It seems at first sight rather difficult to understand the
reason for the relation of the amount of protection afforded
to the area of vesicles resulting, since it might be sujjposed
that, vaccinia being an infective disease, the virus would
multiply in the body in any case to an extent only limited
l)y the resistance of the individual, and that therefore one
insertion would be as effective as many.^ The facts being
as stated, it appears necessary to believe that the infective
organism, whatever its nature, remains localised, in the
majority of instances at any rate, within the tissues near
the site of inoculation, multiplying to some extent therein,
and producing poisons which are carried throughout the
system. It would thus bear a close resemblance to the bacilli
of diphtheria and tetanus in its mode of behaviour. It is
requisite that a certain amount of the poison should be
manufactured, in order to cause a sufficient action on the
cells of the body to stimulate the formation of the pro-
tective substances. Hence the need for a consideral)le
quantity of the virus to be inoculated, since possibly the
^ It seems not impossible that a fallacy of observation may lurk in
the inference drawn from the statistics. It is at least conceivable that
it is not so much the number of insertions of the l\ mph that protects,
as the careful performance of the act of vaccination, of which the
number of scars is some criterion.
136 SERUMS, VACCINES, AND TOXINES.
organisms tend to die out somewhat rapidly, l^eing ex
liypotliesi of an attenuated kind.
It is well to remember that the lymph remaining on tlie
arm or other part vaccinated may be conveyed accidentally to
other regions of the body, and that if there is any excoriation
at the point of contact, a vaccination-lesion will result.
Should such an occurrence take place on the face, a some-
what alarming condition is often produced, the affiected part
swelling markedly and the neighbouring glands enlarging
to a considerable size. The condition is in no way danger-
ous, but an unsightly scar may be left. A generalised
erujDtion is sometimes produced by such accidental inocula-
tion if it occur in several jolaces.
Site of Vaccination. — The point usually chosen for
vaccination is the skin over the insertion of the tendon of
the deltoid muscle on the outer side of the upper arm.
This is probably the most convenient spot for general use.
It has been suggested that in ladies who may wish in after
years to wear dresses without sleeves, it would be better to
select some other site, such as the outer side of the thigh.
It is immaterial from the point of view of protection what
spot is selected. It is well to avoid any part very richly
supplied with blood, or in very close relation with lymphatic
glands.
Phenomena of Vaccination. — In a subject who has
not been previously vaccinated and who has not suffered from
small-pox, the series of events after vaccination is generally
as follows : — For the first two days nothing is usually noticed
at the site of inoculation. About the end of this time a
small paj^ule begins to form on each of the vaccinated
areas. This next becomes transformed into a vesicle Avith
clear contents, possessing a raised border and a slightly
infiltrated base, surrounded by only a very faint and narrow
halo of hypersemia. The vesicle thus formed tends to sink
in at the centre, forming a cup-shaped depression, while it
continues to spread at its periphery. Meanwhile the sur-
rounding pinkish ring becomes deep-red in colour, and
PHENOMENA OF VACCINATION. 137
enlarges till it may extend for an inch or more all round
the central lesion.^ The contents of the latter become
gradually more opaque and even pustular, and finally dry
up into a scab, while the areola fades after the tenth day.
A more or less circular cicatrix is left when the scab
separates, which is often pitted — "foveated," If the scari-
fied area be large in extent, more than one vesicle may form
on it, a ring of small blebs rising round its periphery ;
while if the points of insertion are near together, they may
tend to coalesce, forming irregular rings of vesicles, more or
less fused one with another, with central depressions. Ac-
companying these local changes there is fever, which begins
al)out the third day and generally increases till the eighth
or tenth, after which it subsides. There may be some
feeling of illness, and occasionally sickness or diarrhoea.
The neighbouring (axillary) lymphatic glands may become
palpably enlarged. In some cases an erythematous rash
appears (roseola), or a generalised papular or vesicular
eruption may be met with. These cutaneous conditions
are of no practical importance.
In those who have already been once or more vaccin-
ated the phenomena are similar, but less marked. Only a
papule may appear, Or a poorly-developed vesicle with
subsequent scabbing. Itching may be the most marked
feature. Not very unfrequently in such persons revaccina-
tion fails entirely.
Risks of Vaccination. — In the days when it was the
practice to vaccinate one child from another by the " arm
to arm " method, there was a certain element of risk lest
some disease vshould be transferred from one to the other
at the same time. Thus it can hardly be denied that
syphilis has been conveyed in this manner ; it seems
definitely established that the clear lymph ^ of a vesicle
1 This is ]U'obably due to the action of i)yogenic organisms
accidentally present in the lymph. Absolutely sterile vaccine is said
not to give rise to any marked areola, but scarcely any specimens of
lymph are of this de;»ree of purity.
- Cory, quoted by Immermann, op. cit.
138 SERUMS, VACCINES, AND TOXINES.
may convey the infection, even apart from contamination
with blood. This risk no longer exists when calf-lymph
is used.
Secondary infection may take place at the site of a
A^accination-puncture, as it may by any other abrasion of
the skin. Thus, in a certain number of cases, erysipelas has
supervened, owing to the subsequent entry of streptococci
derived from the insanitary surroundings of the child. It
is said that the vaccination in such cases is generally un-
successful. Milder seiHic infection (probably with staphy-
lococci) may result in a sore arm of unusual severity, and
even give rise to glandular abcesses.
In countries where tetanus is common, this complication
has followed vaccination. McFarland,^ from a study of
ninety-five such cases, concludes that, although it is after
vaccination that the infection with tetanus most often takes
place, in some cases the actual lymph may have been con-
taminated from hay, manure, etc.
The constitutional disturbance produced by vaccination
may in some cases be prolonged, taking the form of some-
what severe aiicemia. Bellotti,- who calls attention to
this possible sequel, states that children who have been
previously rosy and healthy in appearance most often exhibit
this condition. He suggests that the organisms of vaccinia
may in these rare cases exert a special heemolytic action.
The names vaccinia hcemorrhagica and vaccinia gan-
grwnosa have been applied to conditions in which symptoms
of unusual severity attend vaccination. In the former a
generalised hsemorrhagic erui)tion develops, which may be
accompanied by bleeding from mucous surfaces ; in the
latter the local lesions, instead of healing, extend deeply
and widely, causing necrosis of the tissues and large areas
of ulceration, along with severe constitutional disturbance.
It is probable that these conditions are both dependent in
the first place upon a debilitated condition of the child,
1 Journ. of Med. Research, May, 1902. ';
2 Gaz. degli Ospedali, May 10, 1903. j
RISKS OF VACCINATION. 139
j^roduced by ill feeding, rickets, or tuberculosis ; and in
the second place upon an invasion by other organisms, such
as pyogenic cocci, which either produce local gangrene in
the weakened tissues, or give rise to a general septicsemic
condition, with haeniorrhagic symptoms. Of the close con-
nection between hajmorrhagic eruiDtions and general se})ti-
ciemic states there can l)e no doul)t whatever.
A keloid condition sometimes results from the scarring
produced in vaccination. This probably has nothing to do
with the virus employed, but depends upon a constitutional
peculiarity of the individual, in whom any slight trauma-
tism may give rise to a chroaic inflammatory over-production
of scar-tissue.
In the absence of an epidemic of small-pox a child should
not be vaccinated when it is obviously in l^ad health. Not
only will the parents attribute to the operation any increase
in the symptoms of the existing condition which may
ensue, however accidentally, so that the procedure will
incur some degree of disrepute with them and with their
ignorant neighbours, which it is better to avoid ; but it is
probable that in some instances the constitutional disturb-
ance produced by the inoculation may unduly depress a
child already weakened by existing disease. Children
suffering from eczema, herpes, or other skin-diseases should
not be vaccinated, if the matter is not urgent. Generalisa-
tion of the vaccinial eruption is said to occur in such
patients, but the evidence is not very clear. H?emophilic
subjects should not be vaccinated, the risk to them being
greater from any source of bleeding than from the increased
liability to small-pox.
Insusceptibility to Vaccination.— It is said that
some persons are by nature insusceptible to vaccination.
This may possibly be the case occasionally, but instances of
such a condition which will stand investigation must be
very rare indeed. Thus Thorne ^ states that 107,180
^ 'llfh AiiNual Report of the Med. Off. of the Local Government Board,
p. viii.
140 SERUMS, VACCINES, AND TOXINES.
vaccinations have been done by public vaccinators under
the Local Government Board without one instance of
failure. Cory ^ reports one case among 38,000, in which
he was twice unsuccessful in attempting to vaccinate an
infant. Bryce " records ninety-eight unsuccessful attempts
to vaccinate with calf-lymph out of 126,000 vaccinations.
It is not unfrequent, in attempting to re-vaccinate an adult,
to find it impossible to produce any effect recognisable as
vaccinia. The same is of course true, and to a still greater
degree, of those who have suffered from small-pox.
It is necessary to make three attempts at (primary)
vaccination before pronouncing any individual insusceptible.
Supply of Lymph. — In the present state of the law
in this country, public vaccinators are supplied by the
Local Government Board with lymph which is prepared
under careful State supervision. This lymph is not to be
obtained by other practitioners, who are dependent for their
material upon the lymph offered in the market by private
trading establishments. No supervision of any kind is
exercised over these manufactories, so that only the pressure
of competition with other firms, and the risk of losing
custom if their product is found inert, are to be relied upon
to ensure the purity and efficacy of these lymphs. Such a
state of things appears entirely indefensible. It is much to
be hoped that in future Acts of Parliament dealing with
vaccination, provision will be made for the inspection of
private vaccine-establishments, and for the testing by State
officials of all lymph put upon the market. •
Protection afforded by Vaccination.— Of the value
of the protection afforded by vaccination against small-pox
there can be no doubt in the mind of anyone who is willing
to look facts in the face and draw conclusions without
pre-existing bias. Befoi-e Jenner introduced his great dis-
covery to the world the disease was universally prevalent.
It was regarded as a children's disease, owing to the fact that
^ "Lectures on Yaccination," p. 73.
- Boston Med. and Surg. Journ., Feb. 26, 1903.
PROTECTION GAINED BY VACCINATION. 141
all were susceptible and contracted small -pox at the earliest
opportunity. It thus caused an immense infantile mortality ;
but it also attacked adults of all ages and all positions in
life. Princes were no more sacred from its attack than the
poor; scarred faces were the rule rather than the exception.
Hence the new protective was welcomed with delight
throughout the world, and special measures were taken to
introduce it and to carry a suppl}^ of lymph into the jnost
distant countries.
At the present day, owing to the general practice of
vaccination, small-pox is a rare disease, and its very rarity
has caused a certain degree of carelessness in carrying out
the prophylactic procedure. Hence there are signs that in
this country the disease is making attempts to re- assert
itself ; and places, such as Gloucester and Leicester, where
the fanatical opponents of vaccination have gained the
ascendancy and succeeded in causing general neglect of the
precaution, have paid the penalty for their folly by suffering
from severe epidemics.
The general recognition of the value of vaccination is
shown in the regulations adopted by most life-insurance
offices, which charge an additional premium to all those who
have not been vaccinated. In view of the general protection
of the community, the risk is small and the addition slight ;
but there can be no doubt that, if small-pox once more
became prevalent, this additional percentage would be con-
siderably increased. Vaccination, or revaccination, is also
compulsory upon all recruits for the army and navy, and upon
all those employed in the postal service. The statistics of
the German Army and of the civil j^opulationi in that
country afford convincing evidence of the benefits derived
from vaccination, if any be still needed. Directly vaccination
was introduced into the army the average deaths per 100,000
(taking an average of the 10 years before and the 10 years
after its initiation) fell from 36 to 3, whereas in the civil
^ See Statistical Chart quoted in 'Marx, Die Experimentelle Diagxostik,
Serum-therapie u. Frophylaxe der InfectionsTcranJc, 1902.
142 SERUMS, VACCINES, AND TOXINES.
population the relative numbers were 26 "9 and 19 '4 respec-
tively, showing no such tendency to fall. At the same
time the existence of small pox among the civil population
was a source of infection even to the protected members of the
army, a small number of cases continuing to occur. It was
only after vaccination was enforced universally that the
disease practically disappeared from the army, while among
the civil population it at once fell almost to vanishing
point.
We may see the value of the protection thus afforded
by vaccination, by means of a comparison of the German
Army with others not so protected. In the German army
from 1875-1887 only 148 cases of small-pox occurred, whereas
in the Austrian army, not protected by systematic vaccina-
tion, there were 10,238 cases between 1873 and 1886, and
in the French army, from 187-") to 1881, 5,605 attacks. i In
Sweden, in pre- vaccination days, 2,050 deaths occurred
annually from variola ; after its introduction the average
mortality fell to 169 per annum. In Bohemia, with a
population of 3,039,722, the avierage annual deaths for
small-pox w^ere 7,663 ; after vaccination was introduced
they fell to 28 ?, though the population had risen meanwhile
to 4,248,155. Thus the small-pox mortality fell, owing to
vaccination, from 1 in 397 of the population to 1 in 14,741
— a sufficiently striking decrease.
There is no doubt that Jenner was w^rong in considering
that vaccination once performed conferred an absolute
immunity against small-pox ; and failure to recognise certain
limitations in this respect has done harm by enabling dis-
believers in the practice to create a distrust in the minds of
the ignorant by pointing to individual instances of failure,
in which complete protection had l)een promised. That a
person who has been once vaccinated may afterwards sutler
from small-pox is undoubted, although it is almost always
the case that the subsequent attack of the disease is relatively
^ Immermann, art. "Vaccination." Xothnagel's '-Eiievcl. of
Pract. Med.," English Ed., 1902.
PROTECTION GAINED BY VACCINATION. 143
mild (modified small-pox). A certain number of deaths do
however occur even among those who have been vaccinated.
Even revaccination does not necessarily confer al)S()lute
immunity.
In the first place, there is now no doubt that in many
persons the period of immunity after vaccination is
not indefinitely prolonged. Perhaps seven years may
be taken as the average period of fairly complete
protection, but probably even during this time the degree
of resistance is constantly diminishing. In the second
place, it is most probable that modifications of general
health may afiect the individual's resistance to this, as to
other diseases, even when immunity has been produced.
Fatigue or ill-health may perhaps temporarily reduce the
powers of defence. The longer, therefore, the period which
has elapsed after vaccination, the less the degree of
])rotection that is likely to persist, and the more easily will
depressing circumstances sufiice to reduce it below the point
necessary to confer immunity. The following table sho^vs
the gradually -diminishing protection afi'orded by vaccination,
and the consequent increase of mortality as age advances : —
Taule showing the Percentage Mortality at Several Age-
Periods AMONG the same Scar-bearing Vaccinated Cases
as are shown in the former Table (Sandiland).^
Age-periods.
Under
10
years.
10-
20-
30-
40-
50-
60-
TO and
upwards.
Mortality
3
2
7
22
21
24
20
Hence it cannot be too strongly insisted upon that not
only vaccination, but revaccination, is needful to j^rotect the
individual and society against small-pox. Children should
not only be vaccinated soon after birth — within the first three
months of life — but revaccinated perhaps on going to school,
^ 0}). supra cit.
144 SERUMS, VACCINES, AND TOXINES.
and' certainly on leaving it. Should small-pox be at all
prevalent, adults will be wise to have the operation repeated,
if more than seven years have elapsed since the}'- last under-
went it. If as a matter of fact they are still immune, the
vaccination will not '' take," and they will suffer no incon-
venience ; while if it succeed they will have the satisfaction
of knowing that they have gained a new lease of immunity.
In this connection we may quote the following remarks
by Dr. Sandiland i with regard to the protection of the com-
munity at large by vaccination : —
"It cannot be too much emphasised that the extraordinary
diminution in the mortality from small-pox in the last century has
been due, not so much to the protection of a majority of the population,
as to the absolute immunity of a minority, probably made up from
persons at all periods of life, who are continually standing in the way
of small-pox infection, and compelling it to ti'avel by long- and cir-
cuitous routes before alighting, scattered and diluted, on patches of
soil in which it can take root and flourish.
Again, a person saved from small-pox by vaccination should not,
so to speak, be counted as one, but rather should be represented by a
figure standing for himself and all those whom he would have infected j
had he been overtaken by the disease. It is this process of the multi-
l^lication of the benefits of vaccination which has reduced the small-:
pox mortality in England out of all proportion to the protective power]
of infantile vaccination, and which makes it i-easonable to anticipati
with confidence that if revaccination in adolescence were added to
infantile vaccination, small-pox would disappear, as indeed it has
disappeared in Germany."
Modified Small-pox. — Small-pox occurring in persons
who have been vaccinated is generally of the kind known as j
"modified" small-pox. The eruption is generally scanty J
and comes out rapidly, becoming vesicular within 12-24
hours. Some of the papules may never develop into;
vesicles. The vesicles which do form are often smaller than!
those seen in the unmodified disease, and many of them dry
up without becoming pustular. The crusts fall off more
rapidly than in ordinary small-jDox, and less pitting is
1 St. Bartholomeiv's Hosp. Journ., July, 1903, p. loo.
SERUM-TREATMEXT OF SMALL-POX. 1^5
generally left behind. The constitutional syn:!ptouis arc
much less pronounced, and often subside entirely within two
or three days, the patient being practically well within a
fortnight of the onset. Complications are unfr(H|uent and
scarcely ever severe.
Rapidity of Protection Gained.— With regard to
the exact period at which immunity to small-pox is pro-
duced by vaccination— ^.e. on which day after the perform-
ance of the inoculation — it is difficult to be certain. ' No
doubt the immunity is a gradually-increasing one, but it is
probably slight before the vesicles are well developed, and
is mainly brought about from this period to the time when
they become purulent. According to Bryce,^ protection is
complete by the fourth day after vaccination, and only a
modified small-pox is likely to ensue in cases in which
exposure to infection is contemporaneous with vaccination,
a fatal issue being improbable in such a case. It will be
remembered that the inculiation-period of small-pox is
usually about twelve days, so that vaccinia will have time
to develop to its full extent in the interval between infec-
tion and the onset of symptoms. According to E. Hart,^
immunity reaches its maximum about the fourth week
after vaccination. There is little doubt that individuals
vary as to the rapidity with which protection is gained, as
well as with regard to the length for which it remains.
The "memory" of tissue-cells with regard to the production
of immunising substances is as liable to vary as the mental
memory for events.
SEPtUM-TPtEATMEXT.
Serum of Immune Cattle.— Thomson and Brownlee 3
made experiments with regard to a possible antitoxic
influence, upon patients suffering from small-pox, of the
serum derived from heifers which were immune to vaccinia.
1 Boston Med. and Surg. Journal, Feb. 26, 1903.
- AUbutt's " System of MeJicino,'' Vol. ii., p. 578.
^Lancet, Aprir4, 11103.
K
146 SERUMS, VACCINES, AND TOXINES.
Large quantities of the serum were injected, but the results
were apparently quite negative. In certain cases a modified
form of the disease occurred ; but, as the patients had been
vaccinated, it was probably to be attributed to this latter
cause. The serum did not appear to influence the course
of vaccination (re-vaccination) in one case.
Antistreptococcic Serum. — With a view to diminish-
ing complications the use of antistreptococcic serum has
been suggested (Lindsey). Schoull i has made a practice of
injecting 60 cc. of this serum in doses of 20 cc, and gives
even more than this in severe cases. No pain or re-action
is induced by the injection, which is given in the flank, all
antiseptic precautions being taken. He claims that rapid
improvement results in all the symptoms which are con-
nected with the eruption. The painful condition of the
face subsides ; photophobia, dysphagia, and hoarseness
diminish ; pruritus is checked. In some instances a single
injection of the antistreptococcic serum produced an
immediate fall of temperature. Even hsemorrhagic cases
may recover under this treatment. In all Schoull treated
five h?emorrhagic, eight confluent, and nine discrete cases.
Out of these, two patients died (9 per cent.), whereas the
general mortality in cases not so treated was 20 "5 per cent.
Alfred Smith - speaks enthusiastically of this method of
treatment, as shortening the duration of the disease and
preventing pitting and complications. The serum should
be used early, and in sufficient quantities (20 cc, repeated).
COXCLU SIGNS.
1. Vaccination confers an active immunity against
small-pox, and protects almost absolutely for a certain
period of time. This immunity gradually diminishes, and
in man}" cases disappears after a longer or shorter interval,
which varies in diflferent individuals. The immunity may
be renewed b}" revaccination.
1 La Semaine MkL, March 11, 1903; Med. Xetvs, April 25, 1903,
p. 794.
' Med. Record, April 2, 1904, p. 533.
CONCLUSIONS. 147
2. In a person who has been even once vaccinated
small-pox generally occurs, if at all, in a modified form,
which is comparatively seldom fatal.
3. If calf-lymph, duly sterilised, be used, the danger of
;iny ill effects resulting is very small indeed. Complica-
tions are generally due to want of cleanliness and lack of
care in the after-treatment of the lesions resulting from
the inoculation.
4. Complications in the course of small-pox are gener-
ally due to intercurrent infection with pyogenic organisms,
and there is reason to believe that the use of antistrepto-
coccic serum may prove beneficial in averting or modifying
them.
5. Attempts to treat the disease with a serum derived
from immune cattle (anti-bacterial ?) have been unsuc-
cessful.
6. No antitoxic serum is known.
148
CHAPTER IX.
HYDROPHOBIA (RABIES).
Causation. — Up to the present time the actual cause
of hydrophobia, or rabies as it is called when it affects the
lower animals, is absolutely unknown. A large number of
organisms have at one time and another been announced as
the excitants of the disease (bacteria, protozoa, &c.), but no
one of them has so far withstood the test when its claims
were more fully investigated.
Most recently of all Negri ^ has described parasites in
the large nerve-cells of the cerebral cortex, cerebellum, &g.
Some of the organisms contain a number of small refringent
bodies like spores. They appeared as a rule just before the
onset of symptoms in the rabbit, and were found in one
case of human hydrophobia. Negri regards the bodies as
protozoa, and states that they occur only in hydrophobia
and not in other conditions. They are easily demonstrable
by ordinary staining methods. Remlinger and Riffat Bey "
state that they have succeeded in passing the virus of
rabies through a Berkefeld filter ; if this be confirmed, it is
necessary to conclude that the infective agent is capable of
existing in a very minute form, at one period at all events
of its life-cycle.
The virus or infective material, whatever its nature,
resides in the saliva of infected animals, as is evident from
the fact that the disease is most often conveyed by bites of
rabid animals ; but it exists in still greater concentration in
the central nervous system (brain and spinal cord). It is
probably not present in the blood or in most of the organs
of an animal which has died from the disease, but is found
1 Zeitschr. f. Hygiene u. InfectionsTcranh. , 1903, xliv., 507.
2 Comptes vendues de la Soc. de Biol., 1903, Iv., 730.
CAUSATION OF HYDROPHOBIA. 149
on the secretions of certain glands (lachrymal, mammary,
pancreatic) ; possibly the poison is excreted by these
channels. The toxine of rabies bears very close resem-
blance to that of tetanus in many of its properties. Thus
it has a marked affinity for the nervous system, passing to
the central portions of this by way of the peripheral nerves
{('/. p. 103) ; it produces first a stimulation of the reflex
activity of the nervous centres, though this is followed
later on by paralysis ; its effects on the cerebrum are
manifested by excitement and delirium analogous to the
j)henomena noted as the result of injections of tetanine
into the brain substance. Further, the occurrences in the
wound itself bear some resemblance to those met with in
tetanus, as in each case the injured point may cicatrise, but
with the onset of the disease pain may occur in the scar ;
while Pace ^ has shown that the virus of rabies remains
locally at the seat of inoculation, as do the bacilli of
tetanus. Deep wounds of a lacerated nature are those
most liable to give rise to hydrophobia, just as injuries of
this sort are those most commonly followed by tetanus.
The fact that attempts to cultivate any causal organism
from tissues known to contain the virus of rabies have
invariably failed, might suggest the probability that the causal
agent is of the nature of a protozoan rather than a vege-
table parasite. The virus can be preserved in an active
condition for long periods in glycerine, in which property it
resembles that of vaccinia. If it be proved that, as is held
by Councilman, the causal agent of vaccinia is a protozoon,
an additional argument might be drawn from this fact in
favour of the organism of rabies also belonging to this
class. That a living germ is present seems practically
demonstrated, apart from probabilities based on the analogy
of other infective diseases, by the fact that sterilised virus
is incapable of acting as a vaccine.
Infection. — In man the disease is practically always
conveyed by the bite of a rabid animal, though instances
^ Ann. de VInst. Pasteur^ 1903, Vol. xvii., p. 293.
150 SERUMS, VACCINES, AND TOXINES.
have been recorded in which infection was caused by the
mere licking of the hand by a rabid dog, and a case is men-
tioned by Gowers in which a man contracted the disease
by untying with his teeth a cord used to fasten up a dog
suffering from rabies. Other animals besides dogs may
become rabid and inflict bites ; thus Ferre ^ states that
among 100 cases treated at Bordeaux 88 were due to
bites from dogs, 10 to cats, 1 to a bite from a pig, and
1 to a rabbit. Pampoukis' ^ statistics of 1,300 cases show
92-8 per cent, due to dogs, 4-4 per cent, to cats, 1*2 per
cent, to bites of other animals, and 1 '3 per cent, to mere
" contamination with saliva " (licking ?). Bites from rabid
wolves are specially dangerous, owing to their severe,
lacerated character. In India, jackals are a source of
special danger ; thus most of the fatal cases during the
year 1901 occurred among a batch of 35 cases treated
for jackal-bites during August of that year.'^
Statistics as to the percentage number of all cases bitten
oy rabid animals wdiich subsequently develop hydrophobia
are somewhat difficult to obtain. J. R. Bradford * puts it
at 16 to 25 per cent. ; some authorities give rather higher,
others lower, figures. It is, at any rate, certain that all
who are bitten do not develop the disease, even apart from
treatment. This fact is of importance in estimating the
benefits derived from preventive inoculations. When once
it has appeared, the disease is invariably fatal. Persons
bitten through their clothes are not very likely to be
attacked by hydrophobia, as the virus is wiped off the
teeth of the animal in passing through the dress-material.
Natives of India and other hot countries are thus more liable
to suffer from hydrophobia than are Europeans resident in
the same districts, owing to the scantiness of their clothing.
1 A>m. de Vlnst. Pasteur, 1902, p. 391.
- Op. infra cit.
^ Ann. Bep. of the Sanitary Commissioner ivith the Government
of India, 1901, p.' 128.
■* Art. " Hydrophobia " in Quain's " Dictionary of Medicine,"
Third Edition, by Montague Murray, 1902.
INCUBATION-PERIOD. 151
Incubation-period. — The incubation-period of hydro-
phobia is very long, varying from about tliree weeks to
(possibly) some years. As to the extremely long periods
assigned to the incubation of this disease, there is consider-
able doubt. Kaspareck and Teuner i relate a case in which
the disease occurred seven months after infection, in spite
of prophylactic inoculation. Pampoukis,- out of a number
of cases not treated in any way, found tliat 9-3 per cent,
occurred within the first month after the bite, 53 --i per
cent, in the second month, and 37-2 in the third month.
Probably six weeks may be looked on as the average period
of time between the injury and the onset of symptoms.
It is hardly necessary to point out that the popular
belief that hydrophobia may result from the bite of a dog
which suVxsequently becomes rabid is erroneous. It is
founded probably (if it be not a mere superstition) on the
fact that the virus of rabies is present in the saliva of
animals for some days before actual symptoms of disease
are manifested. The period is generally put at two or
three days, but Pampoukis states that it may be present as
long as eight days previously. The belief just alluded to
is unfortunate, as leading in many cases to the immediate
destruction of a dog which has inflicted a bite, a course
which renders it difficult to say whether it was really rabid
or not at the time. If there is any doubt as to the condition
of a dog which has bitten an}^ one, the animal should be pre-
served alive and carefully watched. If it be rabid, symp-
toms will develop within a week, a period of time the lapse
of which will not necessarily prevent subsequent prophy-
lactic treatment from being effectual. On the other hand,
if the dog be killed, it will only be possible to ascertain that
it was suffering from rabies by inoculation-experiments made
with its brain or spinal cord. Three weeks will be neces-
sary for this purpose, this being the incubation-period in
rabbits for inoculated rabies. ]Much valuable time will
1 Berlin, kl'm. Woch., 1902, September 8, p. 844.
'-' Ann. de VInst. Fasteur, I'JOO, p. 111.
152 SERUMS, VACCINES, AND TOXINES.
thus be lost, and it may then be too late to undertake
prophylactic vaccination with success.
Anatomical Changes Produced by Rabies. — The
chief pathological appearances are found in the nervous
system. The changes are most intense in the lower part of
the medulla oblongata, but they are also found in the cerebral
hemispheres and elsewhere. The meninges are inflamed
and oedematous ; and on section of the cord or brain,
punctiform hseraorrhages may be visible. These are pro-
duced by a degeneration of the walls of the veins and
capillaries. Small collections of leucocytes may be found
around the blood-vessels or nerve-cells, forming the rabic
"tubercles" of Babes. The ganglion- cells show changes
somewhat similar to those met with in tetanus ; the
chromatin breaks up, and the processes of the cell disappear.
Ultimately the cell itself may be disintegrated, and its
place taken by a collection of leucocytes. These changes
may be specially marked at points corresponding with the
entrance of nerves coming from the seat of the bite.
The larynx and trachea are hyper?emic ; as are also the
saliv^ary glands and the mucous membrane of the stomach.
Petechial haemorrhages may be seen- in these parts. The
stomach is frequently found in rabid animals to contain
sticks, straw, stones, hair, and other foreign bodies, which
have been swallowed by the animal owing to the disordered
appetite which is characteristic of the disease. The presence
of such things is a strong evidence of rabies.
Modification of the Virus of Rabies. — Although
nothing is known of the poisonous material which gives
rise to this malady, yet experiments show that it resides
chiefly in the nervous system of infected animals, and that
it can be modified in various ways. Thus, light, air, and
desiccation rapidly destroy the virulence of rabic matter.
Heat, also, has the same effect, and so has the addition of
antiseptic drugs, though the resistance offered to these last
is considerable. Carbolic acid (1 : 20) cannot be relied upon
to destroy the virulence of emulsions of brain-substance in
ANTIRABIC VACCINATION. 153
less than an hour, and perchloride of mercury (1 : 1,000)
takes three hours to sterilise this fluid. Digestion with
£,'astric juice diminishes the virulence of infected spinal
cords ; and this method of producing a vaccine has been
employed in Italy, and is known as the "Italian method."
Post-mortem decomposition has little effect in destroying
the virus of rabies, which may remain potent for at least
a month after burial of a carcase. As already mentioned,
glycerine is a good preservative of the virus.
Exaltation of virulence may be effected by passing the
^•irus through a succession of rabbits, which are very sensi-
tive to the disease. After passage through a large number
of these animals the incubation-period is gradually shortened
from about three weeks or a little less to a constant period
of six or seven days. Virus of this degree of virulence is
called by Pasteur " virus fixe," i and is used in the prepara-
tion of his vaccine. Some authorities maintain that the
virus which has thus been exalted in virulence for rabbits
is really attenuated for mankind.
AXTIRABIC VACCIXATIOX.
Pasteur's Vaccine.— Pasteur discovered that by drying
the spinal cords derived from rabid animals for varying
periods of time he could prepare a series of viruses of
graduated strengths. Thus, if such a cord is dried for 14
days, it loses all its toxic potency ; if it is submitted to this
process for only three or four days, the virulence is but
little reduced. Immunity to rabies, as to other infective
diseases, can be induced by injecting at first minute doses
of the organism or toxine, and gradually increasing the
doses until quite strong \4rus can be employed. Graduation
of the dose is effected l)y taking equal amounts of
nervous matter from spinal cords which have been dried
for varying lengths of time. The actual vaccine consists of
a small quantity (1 cm. length) of the substance of the
^ As opposed to the virus of uncei'tain strength {vims de la rue ;
StrasscnivHih) derived from uccidcntally-inl'ectei animals.
154 SERUMS, VACCINES, AND TOXINES.
sjjinal cord of a rabbit which has been killed by inoculation
with the " fixed virus," rubbed up into an emulsion with
5 cc. of sterile l^roth or salt-solution. About 3 cc. of the
resulting fluid are injected. A cord dried for fourteen days
is used for the first injection : on succeeding occasions emul-
sions of less attenuated virus are used, till finally a portion
of a sj^inal cord dried for only three or four days is
employed. A scheme of the actual doses may be thus
drawn up : —
Okuixary Tkeatmfxt.
IXTEXSIVE TkEATMEXT.
-Morning
Cord Dried
2 injections
Cord Dried.
First day-
14 days
14 and 13 davs
Evening
13 „
j>
12 and 11 „'
Second „
Morning
12 „
10 and 9 ,,
Evening-
11 „
,;
8 and 7 „
Third „
Morning
Evening-
10 „
9 „
1 injection
6 days
Fourth „
Morning
8 „
) >
days
Evening
7 „
Fifth „
Morning
6 „
J5
»
Evening
6 „
Sixth
Morning
5 „
)>
4 days '
Seventh .,
.^
5 »
''
3 days
Eighth „
M
4 ,,
4 days
Ninth ,,
)5
3 „
>>
3 days
Tenth „
'>
•5 „
J>
5 days
Eleventh day ,,
5 „
, )
?>
Twelfth
„
4 „
4 days
Thirteenth „
4 „
)?
Fourteenth ,,
,,
3 „
i)
3 days
Fifteenth „
5J
3 „
On the followint
injections of 5-
cords respectiv
5)
; 6 days six more
4-, 3-, 5-, 4-, 3-day
ely.
A more rapid form of vaccination is used in cases in
which the bites are about the face and head, as in these
cases the incubation-period is usually shorter, and therefore
it is important to produce a full degree of immunity as
quickly as possible. This is known as " intensive "' treat-
ment. It will be seen in the scheme given that the virulent
toxine contained in a cord only dried for three days is here
ANTIRABIC VACCINATION. 155
admiiiifctered on the sixth day, instead of on the ninth, as
in the ordinary method.
The exact arrangement of the doses varies a little at
different institutions. Marx states that in Berlin it is con-
sidered that the virulence of the dried cord is lost about the
eighth day, instead of the fourteenth. Hence the Berlin
authorities consider that the first few days of the Paris
treatment are wasted, only material which is quite inert
being inoculated ; they, therefore, adopt a scheme according
to which on the first day cords of the seventh and eighth
days are administered ; on the second day, cord of six days'
drying, and so on, reaching a cord dried for three days on
tlie sixth day of treatment. Then cords of five, four, and
three days' drying respectively are each administered for
two days, and on the fourteenth and fifteenth days cords
only dried for two days. Then for the last four days of the
treatment slightly less virulent material is again employed
In the intensive treatment at Berlin a cord of three days'
drying is reached on the evening of the third day of treat-
ment, and one of two days' on the eighth day. The whole
i]itensive course, here also, lasts 21 days.
Institutes for antirabic inoculation are now numerous.
Besides the Paris " Pasteur Institute," there exist others
at Lille, Marseilles, Montpellier, Lyons, and Bordeaux, in
France; Berlin, Vienna, Buda-Pesth, Berne, Odessa, Algiers,
Kasauli (India), ttc. Different modes of preparing a virus
of diminished virulence for purposes of inoculation are
adopted in different countries. Thus, the Italian method
of Tizzoni and Centanni is to treat the spinal cords with
gastric juice, which has an attenuating effect on the virus.
Hogyes, in Buda-Pesth, merely dilutes an emulsion of viru-
lent material to different degrees, using a high dilution for
the first injections, and o-radually raising the strength on
succeeding days. The theory underlying this procedure is
that the usual method of attenuation by drying alters the
quantity of. the virus, but not its quality ; in other words,
it kills a certain proportion of the germs present, so that a
156 SERUMS, VACCINES, AND TOXINES.
smaller number of them are injected at a dose, but it does
not alter tlieir virulence. Hence, the same result may be
obtained by simple dilution. The practical results of tlu8
method seem to bear out the theory on which it is founded,
as very favourable statistics are shown of the work of
Hogyes' institute.
Effects of Antirabic Vaccination.— The effect of Pas-
teur's method of vaccination in cases of bites by rabid animals
is to produce an active immunit}^ Since the infective agent
in rabies is not known, it is impossible to say with certainty
whether the immunity depends on an antitoxine or on a
germicidal state of the serum and tissues. The latter is the
more probable, as it has already been shown that the virus
must contain a living organism, not merely a toxine.
Owing to the long incubation-period of hydrophobia it is
possible to induce immunity to the disease between the time
at which the bite was inflicted and that at which the
symptoms commence. Thus the treatment is in reality
prophylactic, and not in any way curative. If the symp-
toms have already set in, Pasteur's treatment is of no avail.
The analogy to ordinary vaccination (against small-pox) is
exact. In the latter, vaccination carried out at the time
of exposure to infection may protect against the disease,
since the incubation of vaccinia is shorter than that of
small-pox. The difference here, however, is not very great,
and more often such vaccination will only lessen the
severity of the ensuing attack of small-pox. In the case
of rabies, which has an incubation-period of about six weeks
as a rule, there is full time for immunity to be produced
before the disease appears, and protection is thus usually
complete.
Results of the Treatment.— A good deal of scepticism
was expressed as to the value of Pasteur's treatment when
it was first introduced ; it was even suggested that it might
result in conveying the disease instead of preventing it, and
it is possible that accidents of this kind have actually,
occurred. At tlie present time there can no longer be any
ANTIRABIC VACCINATION.
157
doubt as to its efficacy, or as to the boon confernxl on tlio
liunian race by its discovery. The exact mortality from
liydrophobia in all cases of bites by rabid animals, in times
before the inoculation-treatment was introduced, cannot be*
exactly calculated, but it may safely be put at not less than
10 j)er cent., whereas now among the cases treated at the
various Pasteur institutes the death-rate has been reduced
to a fraction of 1 per cent. The following table shows
the annual mortality at two separate institutions — the
original Pasteur Institute in Paris,i and the similar founda-
tion in New York- : —
Tai'.i.e showing Deaths pkom Hydrophobia amoxg Cases tkeated
IN P.uiis AXD New Yokk.
1
Paris.
New York.
Year.
Xo. of Cases.
Dcatlis. !
Per-
centage.
Xo.of Cases.
Deatlis.
Per-
centage.
1886
2,671
25
0-94
_
1887
1,771
14 1
0-79
—
—
—
1888
1,622
9
0-55
—
—
—
1889
1,830
7
0-38
—
—
—
1890
l,a40
5
0-38
160
1891
1,559
4
0-32
100
2
2
1892
1,790
4
0-25
104
1893
1,648
6
0-22
85
1894
1,387
7
0-36
89
1
M2
1895
1,520
5
0-33
167
2
1-19
1896
1,308
4
0-30
236
1897
1,521
6
0-39
133
1
0-74
1898
1,465
3
0-20
125
1
0-80
1899
1,614
4
0-25
159
• 2
0-25
1900
1,420
4
0-28
U4I
1
0-43
1901
1,321
5
0-38
)
It will be seen from these figures that the death-rate
has never reached 1 per cent, in Paris since the Institute
was started, while in New York the percentage has only
twice been over that amount.
1 Viala, A)in. de VTnst. Pasteur, 1902, p. 452.
'-' Rambaud, Med. Xews, 1902, i., 635.
158 SERUMS, VACCINES, AND TOXINES.
Yery full statistics are published Ly all the Pasteur
institutions as to the exact nature of the cases treated, in
which these are tabulated accordino- to the reojion of the
bite and the evidence available as to the reality of the
disease from which the dog or other animal which inflicted
the injury was suffering. In the tables below, Class A
contains cases in which the dog was proved by conclusive
evidence to be rabid ; Class B, those in which rabies w^as
certified by a veterinary surgeon, as a result of examination ;
and Class C, those in which the nature of the disease in the
animal w^as doubtful. The injuries are classified, as a rule,
according as they were on the face, hands, or lower limbs,
the last being usually covered with clothes.
Table of Cases Tkeated ix the PASXErii Ixstitutes of Paris
(1901) AND New Yokk (1900-1).
Bitten
on
Bitten on
Bitten on Lower
Head.
Hands.
Limbs
.
>.
>,
^
>>
'6
•;..
1
1
1
1
1
4^ ■
2
3
1
1
H
^
H
^
H
^
^
S
Class A
20
93
58
171
13
1
7 69
62
13
88
1
1 13
Class B
80
521
4
0-77
184
785
4
51
7
47
6
60
Class C
23
1
4-34
186
153
862
1
0-23
13
53
27
93
Total
123
1
0-79
800
4
0-50
395
1318
6
0-38
33
1
3 03
162
46
»
241
1
0-4
In the above table the figures derived from the New York Institute
are in dark type.
In the table on page 159 are given the figures supplied
by the Indian Pasteur Institute at Kasauli, ^ under Major D.
1 Annual Report of the Sa)iitary Comniissionei' with the Government
of India, 1901, p. 128.
159
1
1
<
H
•X^iiv^.ion
o -o
CO
o o
«p
•so.ini!r^,j
OCO
i
© o
O Tl
©to
•paaBa.ii
sgg
lO 00
« CO
SUB-CLASS IlL
Bitten TiiROunii the
Ci.othino.
-J3J
o o
O O
o o
©o
•S3xnia'j[
o o
o o
O c;
o o
•P8]B9JX
t- so
t^
SUB-CLASS II.
Bitten Through the Ex-
posed Skin on any Part
OF THE Body other than
the Head or Face.
9o'BJlI90
oo
O Ci
© CO
CO
•S9.railT3J
o CO
o o
O -M
©^
!
•pa;ijp.iX
P3 .-.
« 00
eo 00
*2
2^
rH (M
© o
© o
SUB-CLASS I.
. Bitten on the Heap
or Face.
-.18^
o o
o o
o o
•SMiixrej
o o
o o
o o
•P8|B8JX
o> o
« r-l
t- VO
00 (M
Class A—
Bitten by animals \
proved rabid )
Class B—
Bitten by animals )
certified rabid j
Class C—
Bitten by animals )
suspected rabid (
1
H
160 SERUMS, VACCINES, AND TOXINES.
Semple, M.D,, R.A.M.C, which are on a slightly different
system, the classes being, however, the same. The statistics
for Europeans and natives are given separately, the latter
being liable to more extensive and dangerous bites owing to
their liijhter clothins;,
- Ferre i records that at Bordeaux there were treated, in
1901, 100 cases of bites by rabid animals, with no deaths.
Trolard,2 in Algiers, treated 1,836 patients, among whom
there occurred nine deaths (0-49 per cent.).
The immunising injections are generally administered
subcutaneously over the abdomen, as here it is easy to
avoid injury to any nerves. Krasmitski ■" recommends
intravenous injection of a filtered emulsion, as producing a
more rapid protection. He states that he has successfully
treated seventy cases in this manner without any ill effects.
The importance of early treatment after the injury has
been inflicted is proved by the statistics of the Odessa
Institute,^ which show that of 4,602 cases treated within
the first week, 26 deaths occurred, giving a mortality of
0-56 per cent. ; among 961 treated in the second week,
16 died, or 1-66 per cent. ; while among 313 treated in the
third week, 10 deaths ensued, a mortality of 3-19 per cent.
SERUM-TREATMEXT.
Antirabic Serum. — The serum of animals immunised
by the Pasteurian method is capable of neutralising the
virus of the disease. If a sufficient amount of the serum be
mixed with an emulsion of virulent spinal cord and injected
into a rabbit, no symptoms of disease will develop. As
previously mentioned, in the absence of all knowledge of
the causal agent of hydrophobia it is impossible to ascertain
whether the serum is antitoxic or germicidal ; but proba-
1 Ann. cle VInst. Pasteur, 1902, p. 391.
2 Ibii., 1900, xir., p. 190.
3 Ibid., p. 393.
4 Quoted by Deutsch and Feistmantel, " Impstoffe und Sera,"
Leipsig, 1903.
SERUM-TREATMENT OF RABIES. 161
■ bilities are in favour of the latter. Tizzoni and C'entanni,^
as the result of prolonged experiments, suggested the use of
this serum as a protective against the disease in persons
wlio had been bitten, instead of the Pasteurian treatment.
They consider that their method is quicker and equally
certain. In some cases, also, this serum may act as a cur.e
(in rabbits) when the symptoms of the disease are just
beginning, a period at which ordinary immunising treat-
ment would be absolutely useless.
Preparation. — The method of preparing the seium is
by inoculating sheep with rabic material attenuated by the
" Lilian method " (p. 155). For the first series of inocula-
tions, 17 injections in all are given over a period of 20 days,
each dose consisting of 0*25 grm. of virus for every
kilogramme of body- weight. The injections are given sub-
cutaneously. Later on, immunity is kept up by further
inoculations at intervals of 2 or 2^ months. The serum is
withdrawn on the 25th day after the last injection. The
fresh serum may be dried at a gentle heat over sulphuric
acid, and preserved in this form indeJSnitely.
The serum thus prepared will protect animals against
rabies when administered in doses equivalent to 1/25,000 of
the body-weight. One-and-a-half drops may protect an
animal weighing 2 kilogrammes. A serum of this strength
is called " typical serum " (S. T.).
Tizzoni and Centanni state that their serum is applicable
to man, and recommend that doses of 20 cc. should be used^
given in three injections — one-half first, then the remain-
ing half in two other doses at intervals of three days.
The above amount is advised for cases which come under
treatment within the first four days after the bite. For
cases seen between the fourth and fifteenth days the amount
of serum should be doubled, and very large quantities
should be given in cases of bites about the face and head.
Serum-treatment does not appear to have been actually
tried on man. It would, however, seem advisable to have
1 Lancet, 1895, i!., 659, 727, nnd 780.
L
162 SERUMS, VACCINES, AND TOXINES.
recourse to it in cases which are only seen some time after
the injury has taken place, and in which there is therefore
reason to fear that there will not be time enough to pro-
duce immunity by the Pasteurian method. It would be
well to inject some of the serum in any case in which pain
or discomfort began to be felt in a wound inflicted by a
bite, after tliis has healed up, even before any symptoms
of hydrophobia were manifested. The serum is quite
harmless in any case.
CONCLUSIONS.
1. In all cases of bites by rabid animals, recourse
should be had as soon as possible to antirabic inoculation.
It is important that this should not be delayed. There is
practically no danger in the procedure.
2. If possible, in cases where there is doubt as to
whether a dog which has bitten anyone is rabid or not, the
animal should not be killed at once, but should be kept
under close observation. In this way a positive diagnosis
can be made in a few days ; otherwise it may be necessary
to have recourse to experimental inoculations to decide the
question, and such experiments take some weeks. It would
not be safe to await the results of these before underfjoing
treatment.
3. If for any reason the preventive treatment has been
put oft' till unduly late, it would seem advisable to inject
antirabic serum as a prophylactic measure, if it should be
available.
163
CHAPTER X.
PLAGUE.
Causal Organism. — The Bacillus pestis was dis-
covered by Yersin in 1894. It is a short, thick bacillus,
with rounded ends. It is non-motile, but possesses several
llagella. It grows well on ordinary laboratory media,
either aerobically or in the absence of oxygen. It is not
known to form spores. The B. pestis stains readily
with aniline dyes, but is decolorised by Gram's method.
The ends of the bacilli take the stain much more strongly
than the central portions (polar staining), so that the
organisms may at first sight somewhat resemble diplococci.
^Yithin the body they occur, as a rule, singly or in pairs
(diplo-bacilli), but in artificial media they may form chains.
They sometimes exhibit a definite capsule.
A very characteristic growth is formed by the bacillus
in broth to which a few drops of oil or fat have been
added. Each drop as it floats on the surface of the liquid
acts as a focus for the development of the organisms,
which form colonies hanging down into the medium in the
shape of stalactites. This mode of growth seems to be
confined to virulent bacilli, and the method is used for the
preparation of vaccine. Grown artificially, the organisms
soon lose their toxicity, but this is restored by passage
through a susceptible animal.
The ordinary laboratory animals (rabbits, guinea-pigs,
mice, and rats) are all suscejDtible to infection with plague-
bacilli. Horses, cattle, sheep, and goats are immune, but
may be affected by injection of the toxines of the bacilli in
the form of a solution of their dead bodies. Rats are
spontaneously aff'ected hy the disease, a great mortality
164 SERUMS, VACCINES, AND TOXINES.
among these animals being often a forerunner of an outljreak
among mankind.
Toxines and Pathogenic Effects.— The Bacillus
pestis does not appear to form virulent poisons in culture-
media,^ but the bodies of the bacteria themselves are
highly toxic. By its action as a parasite, the organism
produces a " hsemorrhagic septicaemia," that is to say, a
general infection (the organisms multiplying in the blood-
stream), with interstitial haemorrhages in the various organs.
The common form of plague is characterised by the appear-
ance of a bubo, or mass of swollen and inflamed lymphatic
glands, in some part of the body. This occurs most fre-
quently in the groin, but may be produced anywhere,
according to the point of entry of the germs. Infection
probably occurs through the skin by means of some breach
of surface, such as the bite of an insect. The view that
it is directly transmitted by bites of fleas, derived from
rats suffering from the disease, is not well authenticated ;
it is, however, not impossible.
In the pneumonic variety of plague direct infection
may occur, owing to the number of bacilli which are pre-
sent in the expectoration. In the other varieties this direct
infection is not common, at any rate, in hospitals, where
precautions against spread of the disease are taken.
Curative serums have been prepared for the treatment of
the disease, and protective vaccination has been carried
out.
HAFFKIXE'S PROPHYLACTIC.
Preparation of Vaccine. — Haffkine prepares his
vaccine by growing tJie B. pestis in flasks of broth in the
manner already described (stalactite-formation). The vessels
are shaken from time to time, by which means the hanging
colonies are thrown down into the fluid, and others form
in their places. After growth has gone on for a month
^ Klein's experiments, however, seem to show th.;it some toxic
material is contained in the fluid of broth-cultures of the bacilli
(see p. 166).
HAFFKINE'S PROPHYLACTIC. 165
or six weeks, the bacilli are killed by heating to 70° C.
for one to three hours, and the Huid is tested by culture to
make certain that it is sterile ; after which it is ready for
use as vaccine. The usual dose for an adult man is 3cc.,
for a woman rather less (2 to 2h cc.) ; children receive still
smaller amounts. The vaccine is given by subcutaneous
injection in the arm. The administration is followed by
redness and swelling at the seat of inoculation, and con-
stitutional symptoms in the form of rise of temperature
and feeling of illness. The latter pass off in about twenty-
four hours, but the patient should spend the first day after
the treatment at rest, not resuming his ordinary avoca-
tions till the second day.
Results of Inoculation. — HafFkine considers that pro-
tection against plague is produced rapidly — at the end of
2-1 hours. In view^ of the facts ascertained by Wright with
regard to antityphoid inoculation, it seems likely that there
may be at first a period of increased susceptibility to
infection, and this has been asserted by Calmette. Banner.
mann, however, denies that this is the case, and considers
that the injection does not aggravate an attack if made
during the incubation-period. Of the figures given by
Haffkine as to the results oljtained with his inoculations,
we may quote those relating to the village of Undhera.^
Among 64: uninoculated persons, there were 27 cases of
plague, and 26 of these jDroved fatal ; while among 71 inocu-
lated persons — members of the same families as the former
and living under exactly the same conditions — there
were 8 cases, 3 of which were fatal. The deaths among
the uninoculated thus exceeded those among the inoculated
by 89-65 per cent.
Leuman ~ records that of 1,173 mill-hands, 1,040 were
inoculated twice : among these there were 22 deaths (2*11
per cent.); of 58 inoculated once, 8 died (13*79 per cent);
1 latwei, 1899, i., p. 1,697,
2 Quoted by Miss [Slaughter. See next note.
166 SERUMS, VACCINES, AND TOXINES.
of 75 not inoculated, 20 died (26-6 per cent.). Bannermann ^
states that in a total of 6,000 cases the mortality among the
inoculated was 43-5 per cent., while among the uninoculated
it was 73-7 per cent.
The Indian Plague Commission reported as follows with
regard to this method of prophylaxis : —
(1) Inoculation sensibly diminishes the incidence of
attacks of plague. It is, however, not an absolute protection
against the disease.
• (2) The death-rate is markedly diminished by its means,
not only the incidence of the disease, but also the fatality
(case-mortality) being reduced.
(3) The protection is not conferred on those inoculated
for the first few days after the injection.
(4) The duration of the immunity is uncertain, but it
seems to last for a number of weeks, if not for months.
The mode of action of HaflPkine's prophylactic is presum-
ably the same as that of other vaccines, viz. it depends for
its efficacy on the presence of the actual bacteria contained
in it. It has, therefore, generally been supposed that the
precipitate that forms in tubes of the vaccine which are
allowed to stand, consisting of the bodies of tlie dead
bacteria, is the effective part of the preparation, Klein -
has recently thrown some doubt on the inert nature of the
supernatant fluid. He finds that it has a certain, though
small, protective influence on rats. Further, he finds that
the blood of immunised animals is agglutinative towards the
B. pestis, but not bactericidal.
Wurtz and Bourges,from experiments on white mice," find
that the protective power of the prophylactic is consider-
able, and lasts for a moderate jDcriod of time (two or three
months). Haffkine considers that the protection afforded by
1 Quoted by Miss Slaughter, Johns Hopkins Rosp. Bid.. Xov.,
1903, p. 307.
2 Thirly-Jirst Annuil Report of the Local Government Board, 1901-2 ;
Supplement containino- thi report of the Medical Officer, 1903, pp.
357-394.
2 Arch, de Med. £xperme?itale, kc, 1902, p. 145.
TERNI AND BANDI'S VACCINE. 167
his prophylactic lasts as long as six months. Tiie geneinl
opinion in India is that it is " absolutely safe for three
months. " liCuman found that the protection gained by those
twice inoculated was ten per cent, greater than that of
the once-inoculated.
Pfeiffer i considers that the bacilli lose some of their
\irulence by being cultivated in broth, and that their
efficacy as a protective is thus diminished. He has accord-
ingly prepared a vaccine from fresh cultures of the B,
pestis on agar. These are emulsified in broth or salt-
solution, and sterilised at 65' C. The reiiction produced
by injection of Pfeiffer's preparations is more intense than
that seen after HafFkine's prophylactic. No statistics are
available for forming a judgment as to the value of this
vaccine as compared with HafFkine's.
TERXI AND BANDI'S VACCINE.
Terni and Bandi ~ prepare a special material for use as
a vaccine against plague, by injecting guinea-pigs intraperi-
toneally with plague-bacilli and collecting the inflammator}-
fluid which is secreted into the peritoneal cavities of the
animals. This fluid is sterilised by heating for a short
period of time, on each of several consecutive days, to 50°
C, and is preserved by the addition of a small proportion
of carbolic acid. The inventors claim that by means of
this vaccine immunity may be produced in eight to ten
hours, and that the blood of j^ersons so treated possesses
bactericidal powers.
Havelburg^ records that this vaccine was used with
good effects in Brazil. Pinto ^ also records good results
with anti-plague vaccinations (with this remedy 1) : out of
1,803 persons vaccinated only two contracted plague, and one
1 Quoted by Marx," Diag-nostik,Serumtlierapie ii. Prophy]axe,"p.81.
2 Deutsch. mcd. Woch., loOl.
» Berlin, klin. Woch., 1901.
■* Abstr. iu. Journ. of the American Mcd. Assoc, 1902, i., 681. The
nature of the vaccine used is not stated in the abstiact. I have been
unable to obtain the original article [Tidsskrift f. d. Xon^ke Laer/eforen.,
Feb. 1, 1902).
168 SERUMS, VACCINES, AND TOXINES.
of these cases occurred immediately after the vaccination.
He considers the results of the treatment to be brilliant,
but it must also be remembered that the plague in Brazil
was apparently of a mild type. Kolle and Otto ^ regard
Terni and Bandi's vaccine as quite inert.
LUSTIG AND GALEOTTI'S VACCINE.
The material used for the preparation of Lustig's serum
(p. 171) may be employed for the purpose of vaccinating
against the disease. It is prepared by growing the bacilli
in broth and then on agar. The bacteria are then washed
off and dissolved in a 1 -per cent, solution of caustic potash,
and the fluid is neutralised with 1-per-cent. acetic acid. A
precipitate is thus formed, which is highly toxic, containing
as it does the intracellular poisons of the bacilli. It is
dried iii vacuo, and can be readily preserved in this form.
For use as a vaccine, it is dissolved in a weak solution (1 or
2 per cent.) of sodium carbonate. The dose for an adult is
0'0133 grm. of solid substance. Two grammes of the solid
dissolved in 1 litre of solution will afford material for 143
vaccinations.- Statistics as to the use of this vaccine are
not available.
YERSIN'S SERU^I.
Preparation of the Serum.— The original method of
Yersin, Calmette, and Borel '^ for the preparation of anti-
plague serum was by inoculation of horses with fresh agar-
cultures of the bacilli. It was subsequently found by
Eoux and Wladiniiroff that as effective a serum could be
obtained by injection of cultures sterilised by heat, by
which proceeding the danger attending the use of living
organisms could be avoided. The serum is diflicult to
prepare of adequate strength, and attempts at its manu-
facture are at times unsuccessful. Krumbein, Tavel, and
Glucksraann ^ took a year and a half in attaining a suffi-
1 Deutsch. mecl. Woch., July 9, 1903.
- Deutsch. u. Feistraantel, " Impstoffe u. Sera," Leipzig, 1903.
3 Ann. de VInst. Fasiem; 1895, p. 590.
^ Centralbl.f. BakterioL, 1901, p. 742.
YERSIN'S SERUM. 169
ciently active serum. Six months is the time usually found
necessary for the preparation of the serum at the Pai-is
' institution. Be^re the serum is tinally drawn off for use,
the blood of the horse is tested on mice to ascertain that no
living bacilli are contained in it. One-tenth of a cubic
centimetre of serum should protect a mouse from a dose of
living bacilli which kills a control-mouse in two or three
days.
Value of Yersin's Serum.— Yersin gives the follow-
ing account of his experiences in Amoy.^ Twenty- three
cases were treated in all. Of these : —
Six cases treated on the first day, all recovered within
24 hours. Dose, 20-30 cc. ^o suppuration
occurred.
Six cases treated on the second day, all recovered within
3-4 days. No suppuration. Dose, 30-50 cc.
M Four cases treated on the third day, all recovered within
m 4-5 days. Dose, 40-60 cc. Two suppurated.
B Three cases treated on the fourth day, all recovered
K within 5-6 days. Dose, 20-50 cc. One suppurated.
W Four cases treated on the fifth day, two died. Dose,
60-90 cc.
In Nhatrang (Annam),^ out of 23 cases treated with
the serum, 19 recovered and 14 died (mortality, 42 per
cent.) ; of 39 cases not treated, all died (100 per cent.).
Calmette and Salimbeni ^ used the serum in Oporto.
They report that, of 142 cases injected with the serum, 24
died, a mortality of 14*78 per cent; among 72 patients not
so treated, 46 died, a death-rate of 63*72 per cent. They
find that the serum reduces the pain in the bubo, and limits
the inflammation ; suppuration is often aborted by its
early use.
Cairns,^ as the result of experience of the remedy in
cases at Glasgow, concludes that : — -
1. Yersin's serum is a remedy of the greatest value.
1 Ann. de VInst. Pasteur, 1897, p. 81. 3 /^j^^ i899, p. 865.
2 Ibid, 1899, p. 2.51. ^ Lancet, 1,903, i., 1,287.
170 SERUMS, VACCINES, AND TOXINES.
2. Its action is bactericidal — as shown by the degenera-
tion induced in the bacilli — as well as antitoxic.
3. Good results are best secured by the early adminis-
tration of large doses, subcutaneously, into the area
from which lymph drains towards the bubo, and also
intravenously.
4. In mild cases the subcutaneous method alone is
sufficient, but in severe attacks combined subcutaneous
and intravenous administration is advisable. The total
combined dose in the latter condition should be 150 to
300 cc, the proportion given intravenously varying with
the severity of the attack.
Dose and Administration of the Serum.— From
what has just been said it may be seen that large doses of
the serum are to be employed, if the amount is available.
Yersin appears to give doses of 20-90 cc. according to the
date at which the case comes under treatment. He
administers the remedy subcutaneously. Cairns uses still
larger amounts (150 to 300 cc), and gives the serum both
subcutaneously and intravenously ; and the advantages of
employing large doses are also insisted upon by Duprat.^
Brownlee ~ insists on the intravenous use of the serum, and
advises doses of 60 cc. ; Lia^nieres ^ ofives the same advice.
There is no reason to fear the use of the larger amounts.
The only ill effects recorded have been pains in the joints
and erythema, noted by Calmette and Salimbeni, analogous
to those met with after diphtherial and other antitoxines.
Denys and Tartakovsky * insist on the importance of
local injections of the serum into the neighbourhood of the
buboes. Thus, in cases of inguinal buboes, the remedy
should be injected into the leg. They found that if guinea-
pigs were inoculated intraperitoneally with plague-bacilli?
0-1 cc. of serum injected into the peritoneal cavity would
1 Ann. de Vlnst. Pasteur, Sept. 25, 1903.
■■^ Lancet, Aug. 17, 1901.
3 Ann. de Vlnst. Pasteur, 1901, p. 808.
^ Semaine Mid., 1900, p. 40.
LUSTIG'S SERUM. ITI
act as a protective close ; whereas 10 cc. administered
subcutaneously were of no avail in saving the li\'es of the
animals.
Prophylactic Use of the Serum.— Yersin records
that in Xhatrang no cases of plague occurred among those
who had received prophylactic injections of the serum.
Calmette and Salimbeni also used the serum as a protective,
giving doses of 5 cc. injected under the skin of the abdomen.
According to these observers, the protection only lasts
fifteen days, so that it is advisable to repeat the injections
at the end of this time. Calmette recommends the injection
of some of the serum along with the use of Haffkine's
prophvlactic, in order to counteract the first depressing
effects of the latter. This suggestion seems worthy of
serious consideration, when the prophylactic is used in
the actual presence of an epidemic.
LUSTIG'S SERUM.
Antitoxic Serum. — Yersin's serum appears to be
bactericidal in nature, though it ma}^ possess some antitoxic
power. Lustig considers that a curative serum for plague
should be mainly antitoxic, and he therefore proceeds to
obtain such a preparation by immunising horses with the
vaccine-material already described (p. 168), which consists
of a poisonous bacterio-proteid. The immunising process
lasts two or three weeks.
Lustig and Galleotti i record that among 475 cases of
plague treated with the serum the recovery-rate was 39-36
per cent., whereas among 5,952 patients not so treated the
recoveries were only 20-6 per cent. Choksy ~ puts the rate
of recovery after use of the serum at 38-2 per cent., while
in other patients not so treated it was only 19*5 pev cent.
In another series of cases, 480 patients were treated with
the serum, and the same number without it. Eliminating
^"arious sources of error, he found that the recoveries among
the serum-cases amounted to 39*62 per cent., whereas
1 Brit. Med. Journ., Jan. 16, 1901. - Lancet, 1900, ii., 291.
172 SERUMS, VACCINES, AND TOXINES.
among the non-injecteJ cases they were only 20 "21 per
cent. The following table shows the results obtained in
India with this remedy (Choksy) : —
Table showixg Results or Theatmext ov Plague with Lustig's
Serum. Bombay, 1898—1902.1
Period.
Serum-treated
Patients.
Patients under
Ordinary Treatment.
Ditfer-
ence in
favour
of the
Serum-
patients
percent.
No.
! Case-
cent.
No.
Deaths
595
Case-
mortali-
ty, per
cent.
May to October, 1898
257
145 56-4
752
79-1
22-7
January to April and
June, 1899
189
124 65-60
884
734
83-03
17-4
May, 1899, and July.
1899, to Aug-., 1900
484
329 68 00
484
385
79-5
11-5
xVug-ust, 1900, to Feb-
ruary, 1901 (3 ex-
tra cases)
55
36 65-45
184
144-
78-26
12-81
March, April, and
May, 1901
104
81 77-82
102
81
79-42
1-53
Mayr- gives an account of 361 cases treated with the
serum, among whom the recov^ery-rate was 33-8 per cent. ;
while among cases treated by other methods only 21-3 per
cent, survived. He says that the general recovery-rate in
hospitals where the serum was used was 4-5 per cent,
higher than in those where it was not employed. He
considers that the curative properties of the serum are
definitely established.
1 Choksv, " The Treatment of Plague with Professor Lustig's
Serum," Bombay, 1903, p. 110.
2 Lancet, 190*0, ii., p. 461.
AGGLUTINATION OF PLAGUE-BACILLI. 173
A mere study of the above records does not produce a
very favourable impression of the value of the remedy.
The results obtained do not seem so striking as those seen
with Yersin's preparation. More experience is, however,
needed to enable us to form a judgment. The opinions of
those who have used Lustig's serum, as quoted above,
appear to be favourable.
AGGLUTINATION OF BACILLUS PESTIS.
Plague-bacilli, like those of enteric fever and many
others, are agglutinated by tho serum of patients who have
just suffered from the disease, or of animals which have
been inoculated with the bacilli or their products. There is
some difficulty in performing the test owing to the normal
occurrence of the bacilli on nutrient media in closely-
adherent masses. Klein ^ advises that they should be
grown on gelatine, on which a drier and less sticky culture
is formed, and that they should then be suspended in salt-
solution. If it be found that the bacilli are still present in
clumps and not distributed singly, it is better to make a
thick emulsion of them, and to filter it through a double
thickness of filter-paper. The microscopic method of ex-
amination must be applied, as the naked-eye or " sediment-
ation test '^ is unreliable. Emulsions of the bacilli in broth
are also to be avoided for this test, as they tend to sponta-
neous agoiutination without the aid of immune serum.
Klein recommends a dilution of 1 : 20 for use, and a
time-limit of half-an-hour. He finds that the blood of
immunised animals, though strongly agglutinative, is not
bactericidal.
In human patients the agglutinative power of the blood
does not develop until late in the disease, often not till
convalescence is established. The test is therefore useless
clinically. It may, however, be valuable as a proof that a
specimen of bacillus under examination is B, pestis.
^ Slst Annual Report of the L. G. B., 1901-2. Supplement con-
taining the Eeport of the Medical Officer. Appendix B, No. 1, p. 361.
174 SERUMS, VACCINES, AND TOXINES.
CONCLUSIONS.
1. Haffkine^s 2'>i^ophylactic is a valuable means of pro-
tection against plague. There is some doubt as to whether its
use in the presence of an epidemic is advisable, owing to the
possibility of an increased susceptibility being at first pro-
duced. The employment of some of Yersin's serum along
with the vaccine seems to ofter a means of counteracting
this depressing effect, if it really exist.
2. Sufhcient evidence is not yet available to enable us
to decide as to the efficacy of Lustig's or of Terni and
Bandis vaccines.
3. Yersins serum is of value as a remedy for the
disease. It should be given early in the case and in large
quantities. Some of the serum should be injected intra-
venously, the rest subcutaneously into the area of skin
which is drained by the lymphatics leading to the bubo.
The dose may be from 60 to 150 or even 300cc,
4. The claims of Lustiy^s serum as a remedy are less
w^ell established than those of Yersin's serum, but some
evidence has been adduced in its favour.
5. Yersin's serum may also be used ioroi:)hylacticalhj
(dose 5 to lOcc), but the protection gained is transitory, so
that repeated injections are necessary in presence of an
epidemic of plague.
6. The agglutination-reaction occurs wdth B. pestis, but
it is of no use for purposes of clinical diagnosis, as it occurs
too late in the disease.
175
CHAPTER XT.
ENTERIC FEVER.
Causal Organism. — The Bacillus tyj)homs was dis-
covered by El)ei"tli in 1881, and is consequently often called
Eberth's bacillus. It is a short, thick bacillus, provided with
a large number (8-12) of Hagella. It is vigorously motile
in young cultures on suitable media, but in older growths it
loses some of its power of movement, and often tends to
form chains of two or three organisms united end to end.
It grows readily on all ordinary laboratory culture-
media, but the appearances of the growths are not very
characteristic. It is decolorised by Gram's method. It
does not liquefy gelatine, does not coagulate milk, does not
give rise to gas-formation in solid media, and does not form
indol. In the three last particulars it differs from a closely-
allied bacillus, the Bacillus coli cortimunis. The latter is
also more feebly motile, and possesses a smaller number of
flagella (6-8).
The Bacillus typhosus is capable of existing outside the
body of a living host for considerable periods of time. The
recent experience of an outbreak of enteric fever, due to the
use of infected blankets which had been used by troops
suffering from enteric fever in South Africa and had been
sold without disinfection, appears to prove that the organisms
may remain alive and virulent for at least six months.
Epidemics of the disease are most commonly due to infected
drinking-water. It seems j^robable that infection may also
be conveyed by dust, and in the South African war flies
were largely blamed as carriers of the disease. Oysters
contaminated with sewage have also caused outbreaks of the
disease.
176 SERUMS, VACCINES, AND TOXINES.
The lower animals do not suffer spontaneously from
enteric fever ; but they may be artificially infected, and
succumb to the toxines of the organism.
Occurrence in the Body, — The bacilli are found in
greatest numbers in the alimentary canal, but they also exist
in the blood of the infected individual, in the spleen, and in
the lymphatic glands of the abdomen. They may usually be
found in the " rose-spots " on the skin, which form the typical
rash of the malady. They are excreted in considerable
quantities in the urine, as well as in the faeces, and appear
in the sputum of cases complicated by lesions of the lungs
or larynx. It appears, therefore, that the disease cannot be
considered to be a local infection only, but is of the nature
of a septicaemia or general infection. (Wright.)
Complications. — As in other infective diseases, the
complications met with in the course of enteric fever, or
during convalescence from it, are largely due to secondary
invasion by other organisms, which effect a lodgment in
tissues worn out Ijy conflict with a primary illness. The
hectic temperature met with in the fourth week in severe
cases of enteric fever is probably due to the action of
pyogenic bacteria ; while to these, or, in some instances, to
the Bacillus coli, are to be ascribed most of the suppurative
lesions (periostitis, perichondritis, otitis, epididymitis, &c.)
which are seen in the later weeks. Venous thrombosis, so
often met with in convalescence, is also to be attributed to
pyogenic bacteria. In some cases, however, typhoid bacilli
are found in local suppurative lesions, though it cannot be
regarded as certain whether they are the primary cause, or
only find a favourable soil in lesions caused by other]
bacteria. Possibly the B. typhosus, when its virulence is!
somewhat reduced, becomes a pyogenic organism, asj
Donzello^ maintains. The cystitis which sometimes occurs,
though it is rarely met with apart from catheterisation, mayj
be due to the bacilli contained in the urine.
1 Zo Sperimeutnle, 1901, Iv., p. 670.
CHANTEMESSE'S SERUM. 177
Toxines of B. Typhosus. — Cultures of typlioid-^acilli
do not as a rule contain any considerable quantity of free
toxic matter, but the bodies of dead bacteria are themselves
poisonous. Hence the toxines of the B. typhosus are
generally spoken of as " intracellular " (see p. 9). Chante-
messe, however, claims to have succeeded in growing the
l)acilli in a special medium, containing spleen-pulp and
bone-marrow, and from this to have obtained a toxine of
considerable potency, which he has used for the preparation
of an antitoxine.
The effects of the poison as seen in disease are primarily
a destruction of lymphoid tissue in the Peyer's patches of
the intestine, which slough away, leaving ulcerated surfaces.
The number of leucocytes present in the blood falls consider-
ably. This may be due to a destruction of these cells, similar
to that of the closely-allied lymphoid tissue. Welsh^ speaks
of the poison as "lymphocytotoxic." The poison has a
profound effect on the nervous system, drowsiness and
delirium being early features, and the so-called " typhoid
state "being conmion in severe attacks; while the heart and
voluntary muscles are found degenerated in fatal cases.
ANTITOXIC SERUM.
Chant emesse's Serum. — By means of the toxines
prepared as above mentioned, Chantemesse^ has produced
a serum for the cure of enteric fever. It is prepared by
inoculation of horses with the toxine in the usual manner.
The process of inoculation is a long and tedious one, as
very small doses must be employed at first ; otherwise the
horses may be killed by the toxine, Chantemesse speaks
of losing several in the course of his investigations. The
immunisation of the animals was begun in 1896, whereas
the experiments with the serum on patients suffering from
enteric fever w^ere apparently carried out in 1900 (?). It
may be concluded that at least two years were consumed
1 Huxley Lecture, Lancet, 1902, ii., 977.
'- La Presse Med., 1901. No. 93, p. 285.
178 SERUMS, VACCINES, AND TOXINES.
in tlie preparation of the remedy. The results of this
method of treatment, as recorded by its author, are very
encouraging. It is difficult to ascertain the average mor-
tality of the disease, as it varies much in severity in different
years, the reasons for this variability not being known. In
Paris in the years 1899 and 1901 the death-rate was 18-5
per cent. In 1901, from January to October, it was 29 per
cent, among 371 patients treated in nine hospitals. Chante-
messe treated 100 patients by his method, with six deaths.
All those who were treated before the tenth day recovered.
Of those treated later three cases died of perforation of the
intestine ; one (injected on the twenty-first day of the
illness) of pneumonia ; one of hyperpyrexia (injected on
the twenty-fifth day) ; and one of a sacral bedsore, acquired
before admission to hospital. Two subsequent cases, which
were injected on admission to hospital in a moribund
condition, are not included in the statistics.
Chantemesse gives charts of some of the cases treated
by the serum, showing that the injections are followed by
a rapid fall of temperature and improvement in the pulse.
The earlier the serum is administered, the more marked is
the eflfect. If the remedy be given before the eighth day
in cases of ordinary severity, the disease may be cut short
within a period of a few^ days. Sometimes the first improve-
ment is not maintained, and the temperature rises again
on a later day ; in such instances a second injection should
l)e given, and may be followed by rapid recovery. The
accompanying charts, modified from those given in Cliante-
messe's article, show the results obtained in some of his
cases (Charts 1 and 2).
Besides the eff'ects on the pulse and temperature, the
serum has a beneficial influence on the excretion of urine,
which increases in quantity as the pulse and temperature
fall. Albuminuria is not caused by the serum itself — a
point in which it appears to differ from diphtherial anti-
toxine, which is accused of causing the appearance of
albumen — indeed, in cases in which there is already
CHANTEMESSE'S SERUM.
179
albuminuria, this may decrease as the result of serum -
treatment. A liyper-leucocjtosis is produced in the blood,
in opposition to the leuco})enia (defective number of leuco-
cytes) which is characteiistic of enterica. The leucocytosis
is exactly similar to that which is normally seen in conva-
lescents from enteric fever. The myelocytes which are pre-
sent during the disease disappear, while the other varieties
(lymphocytes, eosinoj)hile cells, and multinuclear leucocytes)
increase to their normal amount : they may even be in
excess at first.
Complications are rare in cases treated with serum,
but are not entirely absent. Chantemesse noted in
180 SERUMS, VACCINES, AND TOXINES.
his series one case of perforation and one of pneumonia,
])otli fatal ; and others of otitis media, ]ia?morr]iage, and
CHANTEMESSE'S SERUM. 181
phleljitis, all of which recovered. Probably the serum has
no direct influence on the occurrence of complications ;
these are due to other organisms, which are unaffected by
it ; but if it control and cut short the typhoid infection, it
must indirectly diminish the risk of secondary lesions.
The serum is injected under the skin of the forearm in
the neighbourhood of the usual point for bleeding. The
skin and the syriuge are carefully sterilised, and the veins
of the part must not be wounded. The ordinary dose of
serum is 10 or 12 cc. A second dose may be given at the
end of eight or ten days, if the temperature has risen
again, or if there is any other indication ; the second dose
may be smaller than the first — 4 or 5 cc. There are two
indications for giving a smaller dose than the above-men-
tioned as a first injection, viz. (1) when the patient comes
under treatment in quite the early days of the disease
(fifth or sixth) ; and (2) when the disease has already
lasted for a considerable time, and the general intoxication
is profound. In these cases 5 to 8 cc. are sufiicient.
The injections are followed by a reaction, shown by a
rise of temperature, which quickly falls again. Chante-
messe attributes this reaction to the great destruction of
the bacilli which is induced by the serum, and the conse-
quent absorption of a large dose of their toxines. He con-
siders his serum to be bactericidal as well as antitoxic. It
is difficult to accept this explanation in view of the nature
of the serum, which, from its manner of preparation, should
be purely antitoxic, and not bactericidal. An additional
argument against this assumption may be derived from the
fact that the injection of this serum does not increase the
agglutinative power of the blood. ^
Along with the serum, other treatment should not be
omitted, especially reduction of temperature by baths, and
the supply of plenty of liquid nourishment to the j^atient.
It may be necessary to stop milk-feeding for a time after
^ Josias and Tollemer, Congress of Madrid, 1903. La Fn-sse
Med., Juno 24, 1903, p. 468.
182 SERUMS, VACCINES, AND TOXINES.
the injection, as milk often appears to be ill-digested ;
it may be resumed again as the temperature falls.
No bad effects are produced by the serum, with the
exception of slight erythema, which only appeared in two
out of Chantemesse's one hundred cases.
Besides Chantemesse himself, Boutleux^ treated 15
cases wdth the serum ; all of these recovered. Josias - reports
50 cases in children in which he used the remedy. Among
them there were two deaths, a mortality of 4 per cent.
Simultaneously, in other children's hospitals in Paris the
mortality was 14*2 per cent. He confirms the benefit de-
rived from early administration of the serum, and states
that though relapses occurred in four cases, they were mild
in type. The doses given amounted to 1 cc. for each 30
kilogrammes of body-weight. In a certain proportion of
cases in young children pain in the bowels was complained
of, some days after the injections ; and this was at times so
severe as to create a suspicion of peritonitis. No ill effects,
however, actually ensued. Josias gives further statistics of
cases treated by Chantemesse, amounting to 507 in all,
among which the mortality remained at 6 per cent., as in
the first 100 cases. Osteitis and periostitis occurring during
convalescence from enteric fever are said to be benefited by
the serum.
Before we can pronounce a definite verdict on the value
of this serum it will be necessary to wait until a larger
number of physicians have used it in the treatment of
enteric fever and recorded their results. At present
adequate confirmatory experiences are wanting. Chante-
messe's results, however, are very encouraging, although 100
cases (or even 500) is far too small a material on which to
base an opinion, since the disease is so variable in its
severity at different times and different places.
^ Quoted by Chantemesse, he. cit.
- International Medical Cong-ress of Madrid. See Med. Press and
Circular, July 29, 1903, p. 109. A?m. de Med. et Chir. Infantiles
1903, No. 11, p. 367.
i
ANTITYPHOID SERUM. 183
Other Serums. — W. Y. Shaw i obtained toxic material
by " digesting " B. typhosus in normal blood-serum. He in-
jected this into a horse and obtained a serum which had
some protective power, while the injections were at first
followed by a fall in the bactericidal property of the horse's
serum — "negative phase." From this it would appear that
the serum is antibacterial rather than antitoxic, and hence
comparable with that mentioned in the following section
rather than with Chantemesse's preparation.
JNIacFadyen and Rowland have produced a serum by ino-
culating horses with the substance obtained by triturating the
bacilli ; this, if dissolved in salt-solution, is highly toxic to
laboratory animals. The serum obtained from the inoculated
horse is said to be both antitoxic and bactericidal. No
records of the trial of this serum on human patients are
available.
BACTERICIDAL SERUM.
Antityphoid Serum.-— The serum of convalescents from
enteric fever is bactericidal and not antitoxic in its action ;
and most of the serums on the market, which are professedly
"antityphoid," are of similar nature. They are prepared
by immunising a horse with the actual bacilli of enteric fever.
Chantemesse ^ alludes to experiments made by Widal and
himself in order to produce a serum of this nature, but he
speaks of the results obtained as unsatisfactory. In 1898
Bokenham ^ prepared an antityphoid serum by inoculating
a horse with filtered c^^ltures of the bacilli and then with the
dead bodies of the organisms themselves ; he found that the
serum acted as a protective to rabbits.
Walker ^ describes the method adopted by Krumbein,
who uses first filtered cultures, then bacteria killed by car-
bolic acid. The bacilli are grown for fourteen days in broth,
1 Lancet, Oct. 3, 1903, p. 948.
'-' An antitvplioid serum is inanufacturel by Messrs. Burroughs,
Wellcome & Co. " fur trial."
3 Loc. cit. •* Trans. Path. Soc. Loud., 1898, p. 373.
5 Journ. of Fathol. and Bacteriol , 1901, p. 251.
184 SERUMS, VACCINES, AND TOXINES.
to which ^ per cent, of phenol is then added. The cultures
are injected subcutaneously, and considerable constitutional
disturbance may be produced. Abscesses may also form at
the seat of injection. After a point had been reached at
which 150 cc. were given for a dose, the serum of the horse
was drawn off and used. In subsequent experiments the
living bacilli were injected during the later periods of the
immunising process.
It appears that the B. typhosus is modified to some extent
by its surroundings, and that different strains of bacteria
thus produced, taken from different cases, may act variably
towards a particular serum. In other words, a serum is
found to have a more marked effect on the strain of bacilli
from which it was prepared. It is, therefore, advisable to make
use of several varieties in the immunisation of the horse.
The serum of the horse becomes highly agglutinative towards
the bacilli as the result of the treatment. Walker considers
that the agglutinative power increases practically ^9a?'i passu
with the protective j)roperty, but that the two are not directly
proportional to one another. He finds that the serum pre-
pared as above is antitoxic as well as antibacterial. He
also makes the suggestion that the horse should be immun-
ised against the B. coli communis as well as against the
B. typhosus, or that some " anti-coli " serum should be addeS
to the antityphoid serum for therapeutic use.
Experience at the present day is not very favourable to
the use of an antibacterial serum in the treatment of enteric
fever. The present writer has seen the ordinary serum
which is on the market tried in several cases of the disease,
but in none of them was it possible to be sure of any definite
benefit accruing to the patient. Kelapse was not prevented
by the use of the serum. Walker concludes that " most
antityphoid sera which have been prepared have given no
marked assistance in the treatment of the disease in man."
Beasons for this have been already suggested. In the first
place, it is not definitely proved that the disease is a sej^ti-
cfEmia, in which condition an antil^acterial seruui might be
ANTITYPHOID EXTRACT OF JEZ. 185
supposed to be the most useful. It may be that the bacilli
are for the most part localised iu the aliiuentary canal, and
that the toxines are absorbed, as in cholera, without any
considerable escape of the organisms into the general circu-
lation. If so, it may be ditHcult for the antibacterial serum
to reach them ; and attention should be turned to the pre-
paration rather of an antitoxic serum than of one that is
germicidal. In the second place, it may be that the copula or
immune l)ody present in horse-serum is not capal)le of unit-
ing with the alexine or complement found in human blood,
in which case no bacteriolysis would be produced. It has
further to be remembered that enteric fever is characterised
by a gradual onset, so that it is seldom recognised until it
has lasted five or six days at least. Hence the first re-
quisite in the administration of any kind of serum — early
injection — is generally impossible, and it is unreasonable to
exj)ect as good results to occur as can be obtained in diph-
theria. It is possible that by the time the serum is used
there may be a deficiency of alexine in the patient's Ijluod,
and that bacteriolysis may not occur, even if the copula
supplied be suitable.
ANTITYPHOID EXTRACT OF JEZ.
Jez 1 starts with the assumption that the serum ob-
tained from immunised animals is bactericidal, and not anti-
toxic, and that such a serum is of no value for the treat-
ment of enteric fever. Some other method of conferring
immunity must be tried. Now, Wassermann found that
the spleen, bone-marrow, and lymphatic glands of an
immunised animal had protective properties ; and Jez has
made use of this discovery to prepare a substance which
he considers to be curative of enteric fever. He makes his
antityphoid extract by rubbing up in a mortar the brain,
spinal cord, spleen, marrow, &c., of immunised rabbits,
and adding to the pulp thus obtained saline solution, to
make an emulsion, along with a small amount of alcohol and
1 JFew. med. JFoch., Feb. 18, 1899, p. 346.
186 SERUMS, VACCINES, AND TOXINES.
of carbolic acid. The fluid is filtered after it has stood for
a time, to ensure solution of the protective bodies. In
later experiments Jez added also a certain proportion of
pepsine, presumably in order to facilitate solution.
The filtered fluid is antitoxic, but not agglutinative or
bacteriolytic. As a remedy for enteric fever, it is given by
the mouth ; but if for any reason this is impossible, it
can be administered subcutaneously. A tablespoonful
constitutes a dose, which may be given every two hours or
more frequently. Considerable quantities are needed for
each case, reaching a pint or more.
Jez finds that, as the result of treatment with his ex-
tract, the temperature falls, the pulse improves, and the
general condition of the patient is ameliorated. Diarrhoea
is usually checked. Sometimes sweating is produced by the
action of the remedy. Jez records the trial of the extract
in eighteen cases, all of which recovered.
These results are confirmed by Kluk-Kluczycki,i who
finds that the fever-reducing effect is manifested within
twenty-four hours ; the duration of pyrexia is considerably
reduced, an ap3^rexial condition being often reached within
three weeks. The pulse falls rapidly from, say, 100 to 76,
and loses its dicrotic character. He concludes that the
(extract is a specific remedy for enteric fever; that it is
harmless ; and that it neutralises the toxines and shortens
the disease. It acts so regularly that it constitutes a
diagnostic agent, since its eflfects are not manifested in other
conditions than enterica. The cost per patient works out
at forty to fifty marks (shillings). Eichhorst ~ has also tried
the extract in a small number of cases (twelve), and is
favourably impressed with the results produced ; and a
similar verdict is pronounced by du Mesnil de Rochemont ■
and by Einhorn,^ who observed a reduction of fever and
1 JFein. klin. Jf'och., li)01, Xo. 4, p. Si.
- Therap. Monaish., 1900, p, 115.
3 Thcraj). Monatsh , Jan., 190i, p. 13 (7 cases).
^ Med. Record, Jan. 16, 1904, p. 81 (3 cases).
TYPHOiN. 187
some mental improvement. On the other hand, Pometta'
found Jez's preparation quite useless.
The use of Jez's extract does not seem to have become
at all general, so that there is not sufficient information
available upon the subject to enable us to form a satis-
factory judgment as to its efficacy. The idea underlying it
is not to be neglected, as Wassermann's experiments, con-
firmed by Jez, seem to point to the existence of a protec-
tive pnnciple in the organs of immunised animals. This
does not, however, necessarily involve its value as a cure
for the disease.
TYPHOIN.
Petrushky ~ has made experiments with a prepara_
tion which he calls typhoin, consisting of dead bacilli.
He reports good results in cases of uncomplicated
enteric fever, if the remedy is given early in the course of
the illness. It is not suitable for patients in -whom the
disease is advanced and in whom there is already a tendency
to heart-failure or general intoxication. Small, gradually-
increasing doses are given, and the first injections are accom-
panied by some antityphoid serum, to prevent ill eifects.
It is difficult to believe that this method of treatment will
prove advantageous. It is practically a treatment by
toxines, analogous to the tuberculin-treatment of phthisis ;
but the diseases are not the same in nature, enteric fever
being an acute malad}", whereas tuberculosis is a very
chronic one. Even in the latter the toxine-treatment has
not yet won its way to general acceptance. Until further
experience of the working of Petrushky's preparation is
obtainable, its curative action must be held not only un-
proved, but improbable.
ANTITYPHOID INOCULATION.
Wright S Vaccine. — Experiments were made by Pfeiffer
and Kolle-5 in 1896 as to the effect of inoculating patients
1 Wein. med. TFoch., 1901. No. 4G.
- Ueut. med. Woch., 1D02, p.212 . ^ j[)cut. wed. Woch., 1896.
183 SERUMS, VACCINES, AND TOXINES.
with cultures of typboid-)3acilli ; but although it was shown
that the blood of those so treated had a protective
influence on guinea-pigs, no practical use seems to have
been made of the method. It is to Wright that the prac-
tical introduction of vaccination as a means of prophylaxis
against enteric fever is entirely due.
The vaccine used by Wright ^ consists of cultures of B.
tyiDhosus in broth, grown for four weeks, and then sterilised
by heating for ten to fifteen minutes at 60° C. A small
amount of carbolic acid or lysol is subsequently added to
ensure sterility and the preservation of the vaccine. A
large number of separate cultures are mixed together, so as
to obtain a fluid of the standard strength. The virulence
of the material can be roughly gauged by its opacity to
light, for the measurement of which Wright has de\'ised
an ingenious arrangement. Special flasks also are used for
the preparation of the cultures, in order to facilitate the
subsequent mixing.
The dose used for an injection on man is the minimal
lethal dose for a guinea-pig weighing 100 grammes, or
rather the proportional fraction of the dose which proves
fatal to one of the ordinary weight (250 to 300 gr.).
A virulent culture will contain the requisite quantity in
0*5 cc, but with weaker vaccine it is necessary to give
sometimes as much /as 1*5 cc. Wright also used a vaccine
consisting of agar-cultures of the bacilli, grown for twenty-
four hours, and sterilised at 60^ C. : these are less toxic
than the broth-cultures.
The injections are followed by redness and pain at the
site of inoculation, with some lymphangitis and enlargement
of neighbouring glands. There may be nausea and even
vomiting, and there is considerable feeling of illness, with
some rise of temperature. Occasionally a condition
approaching collapse is observed. These symptoms pass
off rapidly without leaving any permanent ill effects, but
they are severe enough to act as a very real deterrent.
1 Lancet, 1901, i., p. 150.
ANTITYPHOID VACCINATION. 189
Wrigliti now advises the employment of a weaker
vaccine, given in two divided doses, as causing less con-
stitutional disturbances, while it affords equal or greater
protection.
The immediate result of the vaccination is to produce a
lowering of the resistance offered by the individual to in-
fection by enteric bacilli. If large doses of the vaccine are
given, this fall in immunity may be very marked, and may
last for some weeks. If small doses are given, the fall in
resistance is very slight and transitory. For these reasons
it is advisable to make use of small doses, repeated if
necessary, rather than one large dose. It is also im-
portant not to vaccinate in the presence of an epidemic, as
such a procedure would tend to make the subject more
liable to contract the infection.
Almost all the statistics as to the efficacy of Wright's
vaccination are derived from observations on different units
of the British Army, in South Africa during the recent war
and in India. On the following page are some of the
figures given by Wright himself.
Cayley ~ also gives favourable figures with regard to
the use of inoculation in the members of the Scottish
National Red Cross Hospital. Among fifty-seven in-
oculated persons in the 1st Section no attacks occurred ;
among eighty-two of the 2nd Section, the greater number
were inoculated with old vaccine, and five orderlies developed
enteric fever ; one nurse refused inoculation, and she also
suffered. Among the 3rd Section (20) all were inoculated,
and no cases of the disease occurred. Cayley considers that
cases which do occur in inoculated persons are milder and
run a shorter course than in the uninoculated.
Birt-^ quotes his experience in an epidemic at Har-
rismith. Among 947 unvaccinated patients the mortality
was 14-25 per cent., while of 283 who had been inoculated,
1 Practitioner, March, 1904, p. 361.
•- Brit. Med. Jonrn., Feb. 9, 1901.
3 Ibid., Jsiu. 11, 1902.
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