. \ar.c.v \. viz: 151 \ J? \ . .%\1 \v v 2-==I-==5=1=5;:=-=,_-=-;===_-------_=-:== =====_w-ml-=1:-=_===_-==-=-:--_-E-_ .-.:...-....-.-.:....€3.--.2....1;.2 .5... :. ................... I _=.a_..._.....__wr|" . .5 - Tv.|41Ia.r.I- . .\r )1 v“/. K,’ -\‘}.~\'/ A‘) ' iliffilifiiiifl AMOENA _l i|iflFfifiu'fifiFfiF|flifiififiinifi' quAER|5'PENmULAM' .\!L\!) U! ).,\!l.\l7,\!I,\S!A IllllllllllllllIllIIIIIIIIIIIIIIIIIIIIIIKTB """'iiuifiififlfi flfiihifiifiififfluifl _.k "llhlI'IlillIl'II J : _.. ._.__.=_._E_.. _.__._m_-=.=._ _._=._ ,.__._m___._.__.__._._ . _ pAMp§L§T$,E0m0€Gp.,Ey€ and Ear,v0l.1. i. to "Q . O) 0'!‘ |l> OD. l-‘*v ;4 FA A3¥A CO H P‘- UT LI>~ Q 93113 F-a‘_..' . @A |\¢ ;. . O) Oooper,P; Eelatiom 05 Lewis,F.P. éutowtcxis _ %ilson,T.P.‘@L ;‘fiilso VJ W -_ ‘ - .- . hOOQy&bL,W.J. QEWFWWQ. U !\ ./‘i .__.~ 1., \ " H , U Q fig Ct’ ' eyes. 1896. ~.- - _ 7;; 7* v ‘ Y _. " 7 4. -'3 1 : J-\' &lflQ,%.P. Osélcalar m5S@3§€...G€&iL€ES...1§U}, " -- * —m --r P E LF~ w - .. K1;nencal3,J.€. impciiancc CL bit €&LlY correc - ,-3, _’ __ _~ ___A n_ ('\/‘€r':' Z102 OI E€fl£dCLl§~‘..iChw. — , I». / A7 Q 0 ——.— :_ 7 la ‘-\._ I ‘_ _v_ _._~ ' _ _ M N J ., §crmcn,&.b. ACQLE %L&UCCHU- luwo. Cf (I F‘ ‘ > /‘ K ‘J ""7 ___ ' ,!_ __I_ ‘ 1} I ‘it ,_J_._ __ '1 ._: .__- ‘Y bp€H'€L,%. ELtd M@HL Oi giwfiula L ¢U4. C (1 W- _ ,_ . ?L5U Omd. _ . "I M ' -— '— A ,-A-, 14- __ ,~ I; 3 /~_. -'>--—.-/_3~. " ' 1-’ ‘.s~‘_3‘:'2i1§1b , O . Z‘-fl . OD. Mi _ l1'i1QC_. L:<".\;1T_’}\_3 Q k./Si L &.L1{.-lh;Q .' A , ,_._.- __ ‘ ' ; ‘ _ ,3-_‘, _:_ _\ __ ‘ H , Zia 1ias,u.B. _L§L:C“l&¢ CiuL€bfii. 1876. ’ ' 9.", _ . .1- ‘ _ _ _ . ll$On,u. Raip 10 1n acuie Miaucomfi, n_d_ e ({ ‘ - __ 1 \_ ‘ 1’-\’- 19» S5 @9€s... 3%?‘ (C 1—\ 2‘ My -7“ V__ _ VI -.::'7;:__, V ‘ V T V _ I .4 _,_v___,_____‘_~__'_M ‘.‘ n,f.€. Rout " v4;: ~ - g. ~ Compliments of the Author. ASTHENOPIA -»)>a‘%,,<<\/*——" I N’; wow ’IL\‘ A MONOGRAPH ‘I Q‘! \'I =%~'éWe2\i$ =/Ré Egq,s*A‘\=' __BY__ E. T.~ ALLEN, A. M., M. 1). OMAHA. cr-use & EDDY, PRINTERS, 1887. f. w ~£-‘-::-k-41’.-I-;'.‘-,:.~.: n>..:.-“:w “ 0 I 4 . L -~-.. 7 U H _ I ,-2" ".~-.';m\-> ;~ -' ' (';®n1‘erzi‘§ ' PAGE I. GENERAL C01\'SIDERA'I‘IONS..... 3 - II. MYOPIA, Near-Siglztcdmss ................ .. 6 III. HYPERMETROPIA, Far-Svlghtedness .................. .. 12 IV. PRESBYOBIA, Old-Sightedness ................... .. 19' V. ASTIQLIATISM, Irregular Refraction ............ .. 21 VI. INSUFFICIENCY, Muscular Weakness ............. .. 25 ‘ VII. RETINAL ASTHENOPIA, Over-Se1zsit'z'-veness ............... .. 28 VIII. SPECTACLES ......................... .. 29 IX. TEST TYPE ............... ........ .. . 32 E v {I ‘THE HOMCEOPATHIG PRQFEQSION OMAHA, NEBRASKA. J. AHMANSON, M. D. Frenzer’s Block, Room 6, (Opposite Post Office). MARY J. BRECKENRIDGE, M. D. Oflice and Residence, 1809 Farnam Street. S. M. CAMPBELL, M. D. 1237 Park Avenue, Hours: 10 to 12 A.M., 3 to 5 and 7 to 8 P.M. EMMA J. DAVIES, M. D. Residence and Ofiice, 322 North Sixteenth Street. W. H. HANCHETT, M. D. 323 South Fifteenth Street, Tel. 526, Hours: 11 to 12 A.M., 2 to 4 and 7 to 8 RM. GEO. H. PARSELL, M. D. Ofiice, Cor. Capitol Ave. and Fifteenth St. Charity Patients, 11 to 12 A.M. C. G. SPRAGUE, M. D. Office and Res., 1704 Capitol Avc., Tel. 393, Hours: 8 to 9 A.M., 1 to 3 and 7 to 8 RM. O. S. WOOD, M. D. N .W. Cor. Fifteenth dz Capitol Ave., Tel. 239, Hours: 8 to 10 A.M., 1 to 4 and 6 to 8 P.M. CQUNOIL BLUFFS, IOWA. W. A. EDMONDS, M. D. 533 Broadway, Hours: 9 to 11 A.M., 1 to 3 and 7 to 8 RM. A. P. HANCHETT, M. D. 12 Pearl Street, Hours: 10 to 12 A.M., 2 to 4 P.M. Tel. 10. W. L. PATTON, M. D. Cor. Main Street and First Avenue, Telephone 14. E. T. ALLEN, A. M.; M. D. William’s Block, Room 9, .Cor. Fifteenth and Dodge Streets. AMELIA BURROUGHS, M. D. 1617 Dodge Street, Hours: 2 to 5 P.M. Tel. 144. B. W. CONNELL, M. D. 313 South Fourteenth Street, Telephone 589. 0. M. DINSMOOR, A. M.; M. D. 111 North Fifteenth>Street, Hours: 8 to 10 A-.M“-., 2 to 4 and 7 to 8 RM. A. W. HARTUPEE, M. D. Residence, 1812 Chicago Street. W. H. PARSONS, M. D. Cor. Fifteenth and Harney Streets, Hours: 10 to 12 A.M., 3 to 5 and 7 to 8 RM. G. W. WILLIAMS, M. D. Ofiice and Res., Arlington Block, Room 17, Telephone 838. H. A. WORLEY, M. D. 1419 Dodge Street, Telephone 482, Res. 1712 Capitol Ave. 0. W. GORDON, M. D. 556 Broadway. J. P. MONTGOMERY, M. D. 9 North Main Street, Hours: 9 to 12 A.M., 2 to 4 and 7 to 8 RM. W. D. STILLMAN, M. D. 615 Willard Avenue. Tel. 145. Hours: 8 to 9 A.M., 12 to 2 and 6 to 8 P.M. TO MY PROFESSIONAL BRETHREN OF THE CITIES OF OMAHA AND COUNCIL BLUFFS, IN GRATEFUL ACKNOWLEDGMENT OF THEIR MANY KIND FAVORS, THIS LITTLE EFFORT IS INSCRIBED B Y THE AUTHOR . EYE AND MUSCLES. (FROM GRAY), Page 244. THE EYE IN , SECTIONS. (AFTER ANGELL). ~ M " " ,.RE1'|NA ' ; <:/cannula \‘.ORNEA~-,_ A‘-';' .' \ ' . . _..scu:aoflc l7RYSEE.L§\N5..,, " |ms....v- ~~..OF‘flO NERVE X TENBQN or mzvrus ASTHENOPIA; A MONOGRAPH ON WEAK EYES, BY E. T. ALLEN, A. M., M. 1)., SPECIALIST IN DISEASES OF THE EYE, EAR, NOSE AND THROAT- I.——GENERAL CONSIDERATIONS. Asthenopia, from the two Greek words ’asthenes and ’0ps. signifies “weak eyes,” or weakness of sight.* As such it is to be differentiated from dimness of vision, due to ulcers of the cornea, or their cicatrices (scars), from cataract,T from cloudi- ness of the aqueous or vitreous humors and from disease of the optic nerve, all of which render sight difficult or poor. The complaints of an asthenopic patient are various and significant. He says: “My eyes are weak; they burn and feel badly on reading, sewing or writing,” or, “they blurr, water and give out; have a strained feeling;” or, “they become trouble- some on use and make my head ache." The patient may be dizzy or nauseated, or have constant pain in the back whenever the eyes are employed. There may be “sensitiveness to light,” or “floating specks.” The lids are “dry, become red and stick together in the morning,” and vision is worse by artificial light.I In former times, for such troubles as these, blisters and bleed- ings, emetics and setons were used until the poor sufferer pre- '< Dunglison. T An affection of the crystalline Lense. l Vida “1,060 Cases of Asthenopia,” C. R. Agnew, M. D., New York. 4 ' ASTHENOPIA ; ferred, in silence, to “ endure the ills he had, than fly to others that he knew not of.” But thanks to those great reformers, Helmholtz, Donders, Von Graefe and Hahnemann, the blind may see by the gentler methods of our modern day. Yet, alas! too many, ignorant of the meaning of these symptoms, con- tinue to abuse their eyes, or neglect the remedy, until the mis- chief done is almost irremediable. To successfully deal with these affections it is necessary that not only the specialist, but the regular physician as well, should be able to form a correct diagnosis in each case. Thus he can advise his patients wisely, treat them understandingly, and, when necessary, send them to the expert oculist. \ To prevent the onset of these troubles, or to delay their coming, to maintain the eye in health and aid the physician in his efforts to cure, it is necessary that the laity, also, be well in- formed as to the strength and weakness of their organs of sight: for a defect in the eye greatly influences the system at large, and in turn the visioih is affected by remote troubles, such as indi- gestion, general debility and uterine dieaseq; and to know and re- move the cause of the trouble is half the battle. IThe causes of Asthenopia are principally six:* Myopia (near-sightedness). asses? Hypermetropia (far-sightedness). Presbyopia (old-sightedness). Astigmatism (irregular refraction). Insufficiency (weakness of the internal recti muscles). Hyperaesthesia (over-sensitiveness) of the retina. In the earlier stages of some of these forms of Asthenopia, medicine and hygiene may accomplish a great deal—indeed, es- tablish a cure. In other forms the defect is congenital, and can only be remedied by applying a lens in front of the eye to aid in focusing images exactly upon the retina, instead of allowing them to fall behind or in front of it. I The normal eye is emmetropicT—i. e., it is so shaped as to bring rays of light which come from distant obj‘ects to a focus exactly on the retina. *A and F are sometimes called apparent Asthenopia; B, C and 1), true or ac- commoelatvlve Asthenopza ; E, muscular Asthenopia.—[Dunglison.] 1‘ Diseases of the Eye. Wells’; p. 621. A MONOGRAPH on WEAK rnzns. 5 FIG. 1. W 1% EMMETBOPIO Ern.—(After Hartwldge.) It also possesses the power of “accommodation,” by which is meant the ability to adjust itself unconsciously for different distances,—at one moment regarding a book but a few inches from the eye; at the next, clearly beholding distant objects, and sweeping the horizon at a glance. The power of accommodation lies in the ciliary muscle (see Plate 1), which controls the shape of the crystalline lens, mak- ing it more or less convex as is needed. How marvelously ac- curate is this little muscle in the performance of its functions, which regulate to such a nicety the most important of our special senses! And what wonder that it is deranged and embarrassed in its action by improper and excessive use! But it may be asked, are not all eyes shaped alike‘? No. A large percentage are too flat from before backward, making the possessor far-sighted; others are too long, causing near-sighted- ness. According to the best authorities, not over ten per cent. of all eyes are really emmetropic, and Hartridge remarks* that emmetropia, in a strictly mathematical sense, is exceedingly rare."f '~ The Refraction of the Eye. p. 24. Jr “ Optical Defects of the Normal E3/e.—The eye is sometimes spoken of as if it were a perfect optical instrument. This, however, is very far from being the case. It is, it is true, wonderfully adapted for its purpose, . . . but the perfect adaption to all the requirements of vision does not so much depend upon its perfection as an optical instrument as on its free mobility, the great sensibility of the retina, and the readiness with which the mind interprets the impressions conveyed to it. “Spherical aberration is, to a great extent, but not entirely, obviated by the iris, and chromatic aberration is considerable. The cornea is not aperfectly spherical surface. Strictly speaking, it is not a portion of a spherical surface at all, but forms the extremity of an elipse. . . . The media of the eye, moreover, are not perfectly clear, for in the lens are numerous striae and spots, . . . and in the vitreous are a large number of floating cells and fibers. 6 ASTHENOPIA ; Still ninety per cent. of the people do not have trouble with their sight, because the defects are overcome or completely hid- den by a strong ciliary muscle; and Dr. Agnew, of New York, says: “Many subjects unconsciously overcome physical defects in their visual organs without experiencing any abnormal sensa- tion, . . . and are carried along in the daily use of their eyes without pain or inconvenience, even though they have a decided error of refraction or accommodation.“< n Let us now briefly consider the conditions above referred to sem'atz'm—their etiology, diagnosis, treatment and prognosis. II.—MYOPIA. (N ear-Sightedness.) Myopia is that condition of the eye in which it is impossible to see distant objects clearly. An eye so affected is sometimes called a microscopic eye, 'us., the telescopic, as in Hypermetropia. Myopia is classed under false or accomodative Asthenopia, since the weakness is due to inability to relax the accommodation for distance. Hence parallel rays (those coming from a distance,) are focused in front of the retina, thus causing the image formed to be confused and indistinct. The optical defect is that the eye-ball is too long from before back, due to its bulging posteriorly, at the entrance of the optic nerve.T FIG. 2. MYOPIO EYE. The retina, too, has a large hiatus, formed by the entrance of the optic nerve—~ the ‘blind spot.’ Most people are quite unaware that they have in each field of vision a gap sufiicient to include a man’s head at a distance of seven feet.”-— Opthalmic Science and Practice. J uler, p. 342. "< Ten Hundred and Sixty Cases of Asthenopia. T Diseases of the Eye, Angell, p. 58, Id. Wells, p. 629. Ophthalmic Science and Practice, J uler, p. 346. Refraction of the Eye, Hartridge, p. 22. A MONOGBAPH on WEAK EYES. 7 This anomaly requires especial and considerate attention as “its tendency is constantly to progress, until it may terminate in complete blindness.”* The myopic eye is always a diseased eye. [Hartridge.] Myopia occurs but rarely in infancy or among the illiterate, while with savage races it is almost unknown. The disease develops in childhood and adolescence. as the eyes are used for near work, and, according to German ophthalmologists, in- creases in direct proportion to the amount of education.]L ETIoLoGY.—(Gauses.)—1. Myopia is sometimes congenital and often hereditaryi 2. It is generally due to the prolonged and close application of an individual to work of a very fine character, which causes congestion of the tuhics of the eye-ball and their consequent weakness. 3. The strong convergence of the eyes for near work. and the resulting pressure upon them by the tendons of the eocternal recti muscles cause the eye-ball to bulge posteriorly.—(Wells.)§ 4. The stooping position which myopes assume, increases the chronic congestion of the eye tunics, resulting in further softening and still further bulging of the posterior pole.h 5. Work in a defective light, which necessitates the holding of the book close to the eyes.>l<>’-< 6. Overuse of the eyes in youth when the tissues are yielding. 7. Spasm of the ciliary muscle in Hypermetropia.—[See p. 19.] 8. Gloudiness of the cornea. 9. Lamellar cataract. 10. Too strong glasses. Draenosrs.——The patient finds it diflicult to see well at a dis- tance; he may be unable to recognize people across the street; \ Ophthalmic Therapeutics. Norton, p. 311. T Vida Dr. Cohn’s “ Untersuchung der Augen von zehn Tousen u11d sechzig Schulkindern.” I Diseases of the Eye. Wells, p. 629. Q Watchmakers and jewelers, who use but one eye, are seldom affected, proving that close work without convergence does not produce Myopia.-— [I-Iartridge.] ll Diseases of the Eye. Angell, p. 63. ““ Ophthalmic Science and Practice. J uler, p. 349. 8 ASTHENOPIA. ; the child at school cannot read what is written on the black- board; yet both see well enough if the object is near at hand. There is liable to be pain and fatigue, with intolerance of light, and after prolonged use, especially in the evening, the eyes become irritable, red and hot. Vision, too, is apt to be interfered with by “mustece eolttanes” (floating specks before the eyes), or by fixed scotomata (dark spots). Objective ,S't'gns.——The lids, which are held nearly closed, to di- minish the circles of diffusion, (whence the name muo, I close, and ’0ps, the eye,) are generally found congested. The eyes themselves are usually prominent, and the pupils, especially in young people, quite large. The patient leans over his work, and holds his book close to the eyes. With the ophthalmoscope, of which, in this enlightened age, every scientific physician should be master,>'~< the appearances are quite characteristic. The fundus is hyperaemic, and white spots of choroidal atrophy are seen, one of which is crescentic-shaped and lies on the outer side of the optic nerve disc. ' Tests.—With the Opthalmoscope we can see the details of the fundus at some distance from the eye, if the degree of Myopia be at all great. When regarding the optic disc, if we move our head slightly to one side we notice that the image moves in the contrary direction.T If we bring the instrument close to the eye, the weakest concave glass with which we see the fundus clearly is the measure of the myopia,I the accommodation being at rest. *’ “In the whole history of medicine there is no more beautiful episodethan the invention of the ophthalmoscope, and physiology has few greater triumphs. With it, it is like walking into Nature’s laboratory and ‘seeing the infinite in action,’ since by its means we are enabled to look upon the only nerve in the whole body, which can ever lie open to our inspection under physiological con- ditions, and to follow in a transparent membrane, an isolated circulation from its entrance into the eye through the arteries, to its exit in the veins. We are further enabled to watch and study daily, morbid processes in every phase, from simple hyperaemia to absolute stasis; while oftentimes through its agency also we get the first intimation of disease in remote and seemingly unconnected organs, so as to read, as if in a book, ‘the written troubles of the brain,’ the heart, the spleen, the kidneys, and the spine. . . Nor is it surprising that the student, once entered upon its investigation, finds the study as fascinating as it can be made profound.”—Loring’s Ophthalmeseopy, p. 1. D. Appleton & ‘o. L T Diseases of the Eye. Wells’; p. 633. 3 Refraction of the Eye. Hartridgc; p. 133. A MONOGBAPH ON WEAK EYES. 9 By Retinoscopy, or “the Shadow Test,” the image moves with the mirror. The great advantage of this method over all others is that by it the refraction of little children and the illiterate can be tested most accurately and defects detected which no other test reveals. It is so simple, too, that anyone can master it in a very short time.>’-< Placing the patient with his back to the light, which is two inches higher than his head. and taking your station four feet in front of him, have him look slightly to your right as’ you examine his right eye, and vice tersa. Looking through the hole in your ophthalmoscope, as you reflect the light into his eye, you will perceive the red fundus reflex. As you now rotate the in- strument very slightly you will see a shadow come out from be- hind the iris and partly cross the pupillary space. If this shadow moves with the mirror, or in the same direction that the reflected light on the face moves, the case is one of Myopia. If the shadow is well defined, and moves slowly against the mirror, Hyperme- tropia is indicated. If the shadow moves obliquely, or if, instead of a solid shadow, dark and light lines cross the pupillary space, Astigmatism is proved to exist. With the Test Type (see p. 33) each eye should be examined separately, the other being covered. as often a great difierence exists between the two eyes. The Myopia Eye can never read N0. XX. at twenty feet, bat, as a rule, can read even diamond type if held close to the eye. Distant vision is improved by concave lenses. Myopia is very often simulated by spasm of the ciliary muscles in a hypermetropic eye; and unless atropine has been used, the tests above given may not detect it; but, as the treat- ment depends entirely upon the cause of the asthenopia, an er- ror in diagnosis might lead to grave results, so that it often be- comes necessary in children and young people to thoroughly par- alyze the ciliary muscle with atropine, when the fault can no longer be hidden. (See p. 16.) TBEATMENT.——Th6 aim in the treatment of Myopia is five-fold. 1. To prevent its onset. 2. To prevent its increase. < See Hartridge’s admirable article on Retinoscopy (Chap. iv.; Refraction of the Eye). Also one by Dr. Jackson, in the American Journal of the Zlfedical Sciences (Philadelphia), for April, 1885. ~\Ivu L4 0 Jbuu 'I '\l -I J 10 ASTHENOPIA ; 3. By suitable glasses to enable the patient to see at a greater distance. 4. To remove any muscular asthenopia. 5. To combat secondary disturbances. First. We cannot too forcibly impress upon all who have to do with the education of the young, the importance of prevent- ing Myopia, by removing all conditions which are likely to cause it; and it is the duty of physicians everywhere to enlighten par- ents and teachers as to the fatal defects in school-room furniture. The seats and desks should be so arranged that no stooping is necessary, or even possible, but the children be compelled to sit erect and hold their books high. The light should be good, but never facing the 1ou1o't'ls.* Children should not be allowed to read too much in the evening, especially by a weak light. When in poor health, after passing through the measles or scarlet fever, or when affected with any cloudiness of the cornea, increased caution is necessary. Second. To carry out the second indication, the prevention of an increase of Myopia, patients should be instructed never to read in a moving train or carriage; to avoid all fine work, such as crocheting——at least temporarily; to use books printed in good, bold type; to write in a large hand, and sit erect when so engaged; to sit so that the light may come in over the left shoulder, and to use artificial light as little as possible; never to hold the book nearer than twelve inches to the face, and always rest the eyes as soon as they become tired. “It is desirable that patients discontinue work and rest their eyes from two to five minutes every half hour.”T Patients should exercise daily in the open air and practice looking at distant objects. Third. The correct prescription of glasses for Myopia is a delicate matter, and requires an exact knowledge of the intra- ocular conditions present. Patients, if left to choose for them- selves, always select glasses which are to strong; and there is nothing which so rapidly increases an existing near-sightedness as the use of such improper lenses.1j Patients, therefore, should / * Vide Juler’s Ophthalmic Science and Practice, p. 351, and Augell’s Dis- eases of the Eye, p. 77. T Ophthalmic Therapeutics. Norton, p. 311. I Norton, p. 312. Wells, p. 635. Angel], p. 74. ‘J L A ,- r c I ( I l A MONOGBAPH ON WEAK EYES. 11 never buy their spectacles at random of opticians or itinerant venders, but should always have them accurately prescribed by a competent physician or oculist. The evil result of random spectacle-buying may be seen in Dr. Oohn’s report. Of 1,004; near-sighted children in the German gymnasia, only 107 wore glasses at all. Of these, 99 had selected their own “and 11, only, wore those which were not injurious to the eye.” To-day, alady came to me suffering with inflammation of the optic nerve and retina, induced by glasses which she had chosen for herself and which were two dioptrics too strong. She also had astigmatism, which, of course, her glasses failed to correct. Sometimes no glasses at all are needed, but if found necessary we give the weakest concave lenses, which removes unpleasant symptoms and renders distant vision easy, though not usually perfect. Glasses are often prescribed for certain distances, as two feet for reading music. In high degrees of Myopia, two pairs may be employed, one for distance and a weaker pair for near work. The patient should be impressed with the fact that the glasses given him for reading are not to enable him to see better, but to “increase the distance at which near objects may be seen," and thus relieve the eye of the necessity of strong convergence. Fourth. For the treatment of the muscular asthenopia, if any, see Sec. VI. Fifth. To combat the secondary disturbances, great atten- tion must be given to the general health. Regular out-of-door exercise should be taken, and the system built up by suitable diet, tonics and medicines. Sa1isbury’s beef and hot water regimen is excellent. Iron is often useful, and also jaborandi, physostigma, ruta and sulphur. For pain and inflamation com- pare belladonna, phosphorus, prunus spin, spigelia and thuja* cactus and macrotin.T In case the disease progresses steadily we must give the eye entire rest by the installation of a one grain solution of Atropine three times a day for some weeks. We may also apply dry cup- ping to the temples with decided benefit. Often a wash of tepid milk and water is grateful to the eye. If there is much conges- * Norton’s Ophthalmic Therapeutics, p. 311. T Ange11’s Diseases of the Eye, p. 70. 12 ASTHENOPIA ; tion, add a few drops of hamamelis and administer the same in- ternally. Pace-Nosrs—-(Expected Results.)——-Myopia may (exceptionally) remain stationary from childhood to old age; or it may temporarily progress to the age of thirty and then stop, if the general health be good; or, again, it may progress steadily and constantly. It never decreases—a'. e., the globe never resumes its normal shape, and the “far point” can never recede from the eye; but, due to the influence of age, the near point does recede, as that we oftentimes find old people who at sixty are able to read without glasses. This is proof positive that they have Myopia,* though you would probably be unable to convince them of the fact. If the near-sightedness is of high degree, amblyopia (poor vision, due to nervous defect or injury,) is the invariable result. The patient is greatly troubled with floating specks and black spots in the field of vision. When Myopia is progressive from the first, changes take place in the back of the eye, causing in- fiammation, and sometimes resulting in the complete disorgan- ization of the part.T In youth almost all Myopia is progress- ive, and “prog¢~esstoe Myopia threatens bZz'ndness.”I It does not always result in blindness by any means, but it may do so. Hence we see the necessity of giving early attention to this affection, and making every effort to check its progress. III.-—HYPERMETROPIA. (Far-Stghtedness.) Hypermetropia (hg/per, in excess; metron, measure; ’ops, eye;) is that condition of the eye in which rays of light from distant objects are brought to a focus, behind the retina, the eye being at rest. This condition gives rise to true or accommodative Asthenopta, ‘ Hartridge Refraction of the Eye, p. 124. T These changes are, choroidal atrophy, opacities and disorganization of the vitreous, intraocular haemorrhage, detachment of the retina, and cata- ractous condition of the lens. I Angel], p. 68. A MONOGBAPH on WEAK runs. 13 since the eyes cannot be used continuously for near vision even in the best light without pain and confusion of sight, although for the first moments the patient can see easily and distinctly. The optical defect is that the antero-posterior diameter of the ball is too short, or the refractive power is too low, so that, O Fre. 3. HYPERMETROPIO EYE. instead of getting clear images upon the retina, the patient per- ceives everything as confused.* The hypermetrope, therefore, has to use some of his accommo- dation to see distant objects distinctly, and hence starts with a deficit for all other requirements. Hence to see print at the or- dinary distance requires all the eifort of the emmetropie eye, plus enough more to overcome the amount of his Hypermetro- pia. The defect is a congenital one, and due to an imperfect de- velopment of the eye-ball, but is seldom discovered until the child begins to read. In youth the hypermetropie eye is strong, since the constant efiort put forth to see, gives the ciliary muscle increased growth and power. But some overuse of the eye, some excessive strain, especially in a period of ill health, so weakens it that thenceforth it is unequal to the task it has to perform. For this reason we find nearly twice as many cases of weak-sight in hypermetropie eyes among women as among men, not because more of them are hypermetropie, but because they are weaker physically, their hours of work are more, and their occupation, especially sewing by artificial light, requires greater exertion of their power of accommodation. Men naturally endowed with great power of * Hartridge, p. 103. Juler, p. 351. Wells, p. 640. Etc., etc. 14 ASTHENOPIA ; accommodation may escape Asthenopia altogether. Thus, the American Indians are almost universally hypermetropic and yet they are possessed of the keenest sight and strongest eyes. A physician of this city, well along in years, having a hypermetro- pia of high degree, is yet able to see very fine print close to the face. Among men, we find the most cases;of true asthenopia in students, clerks, book-keepers, and professional gentlemen, who use their eyes constantly for near work. While Far-sightedness resembles Old-sightedness (Presbyopia) in many of its symptoms, the two must not be confounded. The former is due to the shape of the globe, the latter to a gradual loss of power of the accommodation. rThe former exists in child- hood and continues through life. The latter only exists as the result of age. Both, however, are relieved by convex glasses. E'rIoLoGr.———The causes of Hypermetropia are : 1. Heredity. Several members of the family being usually affected. 2. The result of disease, the cornea having become flattened. 8. Absence of the lens, as after cataract operations. 4. Detachment of the retina, from tumor or exudation be- hind it. 5. Reduction in the power of refraction of the lens. The defect is as a rule, congenital, and an over-strain brings it to light; but general debility, fevers, dissipation, worriment, etc., may do the same. Hrsronr AND Drxeuosrs.—The onset of the trouble is at first gradual. The eyes become tired more and more easily. The lids are irritable. Headaches are common. The remedy is at flrst neglected, as such people object to beirig laughed at, by the ignorant, for “wearing glasses so young!” Hence the patient continues to overuse and strain his eyes, determined to accom- plish given amounts of work in certain times, till there comes_ a day in which his eyes suddenly give out. He “cannot see !” the letters are blurred. He has moments of blindness, and in alarm he bethinks himself of the oculist he should have consulted long ago.* The patient states to you that he‘ can see distant objects very well, but that, after reading a little while, the print becomes in- =‘~ The author’s own experience. A MONOGRAPH ou WEAK turns. 15 distinct and the letters run together; the eyes ache and fill with tears, and there is such a feeling of discomfort that he must rest them for a time. The symptoms then quickly subside, but reappear when work is resumed. If relief is not given, the eyes become bloodshot, and upon the least service are fatigued and painful, so that they can scarcely be kept open, and the lids ad- here in the morning. In other cases reading may invariably be followed by‘such headaches that cerebral trouble is apprehended, and the patient is condemned to spend her time in idleness, when the whole trouble might be removed by correcting the hyperme- tropia with suitable glasses. [Juler.] If young, the patient’s friends may think him short-sighted because he holds the book close to his face. This is owing to a spasm of the ciliary muscle, which produces a temporary myopia. This condition is exceedingly common, and physicians must always be on their guard against it. Such a patient selecting glasses for himself would choose concaves, and thus bring on a confirmed myopia, whereas he ought to have the spasm relaxed, and wear convex lenses. Objective Sz'gns.—Frequently a fiat-looking face, with eyes small, and seemingly far apart, are indications of hypermetropia. [Hartridge] The lids look irritable, and may even become granular from this cause alone. Hence, in all chronic ophthal- mic troubles, the refraction should be tested, as “frequently com- plaints which have been intractable are easily and quickly cured when the proper glasses have been prescribed.”< The pupils are large and sluggish,’[ in cases accompanied with irritation or dis- turbances of the uterine or digestive functions. Convergent squint in a child is an almost certain sign of hypermetropia. Test Tr/pe.——The manner in which the patient reads the type at twenty feet is often an indication of the trouble. It takes some time for him to make out each line, but, if not hurried, he reads it all correctly. If, however, the degree of error be high, or there is much spasm of the ciliary muscle, he may fail to read No. XX at twenty feet. Except in this last condition, a weak convex glass before the eye aids his vision, and the glass with which he sees best is the measure of his manifest hypermetropia. > Refraction of the Eye; p. 111. T Diseases of the Eye. Angel]; p. 88. 16 ’ ASTHENOPIA ; Owing to the tonic contraction of the ciliary muscle, some hy- permetropia is usually concealed. As age advances, however, this also becomes manifest. To discover the entire error of refraction in young people and children, it is necessary to relax the ciliary muscle with atro- pine. By this, also, we are able to diagnose a spasm of the mus- cle in hypermetropia from the simulated myopia. Under 35 years of age no harm need be feared at the hands of an expe- rienced physician, a 4-grain solution being used three times a day for three days. Above the age mentioned it is seldom necessary to use a mydriaticfi< Pin-Hole Test.-—This simple test may be used when the proper convex and concave trial lenses are not at hand. The patient is to look through a large pin-hole in a blackened card at a light twenty feet away. Vision is at once improved. When the card is moved rapidly before the eye the light seems to dance. If two holes are made an eighth of an inch apart he sees two lights, “the second at the left of the other.”T Opthatmoscopic Test.-—The patient’s accommodation having been paralyzed, and your own entirely relaxed, find the strong- est convex glass in your ophthalmoscope, with which you can clearly see the vessels and nerve. This is approximately the measure of his total hypermetropia. By Retinoscopy the shadow moves against the mirror. The greater the error of refraction, the slower the movement and the less distinct the outline. As it is often important to find the exact error in cross-eyed children too young to read, this method gives it to us very easily and perfectly. TREATMEN'r.——The primary and most important indication in the treatment of hypermetropia is the selection of proper convex lenses, which should be prescribed on the first appear- ance of asthenopic symptoms. These obviously relieve the strain upon the ciliary muscle, since they produce the same effect upon the eye as increased accommodation—i. e., they adapt the eye for nearer vision, allowing it easily to bring diver- gent rays of light to a focus upon the retina. >" The pupils may be contracted again with eserine sulph. 4 gr. to the ounce water, applied every three hours. 1- Dr. Thompson, in the American Journal of the Medical Sciences. A MONOGRAPH on WEAK nrns. 17 One might, at first, suppose that the proper glasses would be those_which would exactly correct the error present; but this is far from the case, since the eyes are unable at once to accustom themselves to so great a change of refraction. People selecting spectacles for themselves generally choose those which are too strong. Even the most experienced oculists sometimes have “ considerable trouble in finding glasses which can be worn with comfort; and yet, if the eyes are to be used at all, the help of glasses is absolutely required.” [Angell.] Again, so many cases are complicated with astigmatism that it is unsafe for anyone except an occulist to risk a prescription. If the hypermetropia is of high degree, it may be necessary to begin with weak glasses and increase the strength every few weeks, in order to effect a perfect cure. For young people it is best to prescribe for reading only, since, if glasses are worn constantly, they become indispensible. Nevertheless, there are cases in very young children, possessed of marked hypermetropia, where we should insist upon their con- stant use in order to avert the change to myopia, which often occurs as the child begins to readfl< And again, if the eyes are crossed, the patient should wear glasses all of the time. Aged people, with much hypermetropia, and those who have endured the operation for cataract, must have two pairs of glasses—one for distance, and the other a stronger pair, for near vision. In uncomplicated cases, eye-glasses may be used. Regimen.-—Certain cases, in which the error of refraction is slight, and the trouble has been brought on by muscular debility, may be relieved without the use of spectacles by restoring the patient to vigorous health through a change of air, a generous diet and the proper internal medication. In all cases great benefit may be derived from graduated exercise of the eyes, to be pursued as follows: Abstaining from all other near work let the patient practice the reading of some, not-too-fascinating, book printed in clear, large type, and held in a good light. The first day he should read till his eyes tire; noting the time, let him add one minute to its length daily, per- forming the exercise ter die. In all cases of Asthenopia work should be interrupted every * liqtde J uler’s Opthalmic Science and Practice, p. 407. 18 ASTHENOPIA ; half hour to rest the eye and enable it to resume with vigor and case. In obstinate cases “it may be necessary to completely paralyze the accommodation for some weeks by the instila- tion of atropine, simply for the purpose of resting the eyes.” * If, under the use of the glasses prescribed, all symptoms of Asthenopia do not disappear, the fault probably lies in the power of convergence, (Muscular Asthenopia). Jldedtctnallg/, we may often greatly benefit our patients by the use of macrotin, nux vomica, spigelia, or gelseminum. If re- tinal hyperaemia coexist, cactus or belladonna may be called for. Irritability from over-use is relieved by joborandi; and ruta may be of service in restoring the strained musculus ctltow~is to its normal tone. P:aoeuosrs.—By the proper treatment, attended to early, we may hope for the most favorable results; otherwise the follow- ing sequelae often occur: Convergent Strabismus. Spasm of the ciliary muscle. Myopia. Premature Presbyopia. Disease. First. Convergent Staabismus (Gross-eye) is very frequently due to hypermetropia, at least 80 per cent. of all cases being traceable to it.']' The trouble may, however, be checked in its very beginning, at the age of three or four years, by paralyzing the accommodation for a length of time, and the using of proper spectacles. Second. Spasm of the Ctltary Muscle is more often found in slight Hypermetropia than in that of high degree. It causes dread of light (photophobia), watering at the eyes (lachryma- tion), pain, contracted pupil and hyperaemia of the retina. Its treatment consists in paralyzing the muscle by “ the methodical use of atropine (gr. iv. to the ounce) applied two or three times daily, until the accommodation is quite relaxed. It sometimes yields in a few hours, in other cases not for several days.”I Glasses must immediately be applied that the patient may be- ¢"':“E"~"E°!" Angell, Diseases of the Eye, p. 92; Wells, Diseases of the Eye, p. 645, etc. 1‘ Refraction of the Eye, Hartridge, p. 181. l Wells, Diseases of the Eye, p. 630. A nronoenarn on WEAK EYES. 19 come accustomed to them while the effect of the atropine is wearing off, as otherwise the spasm will return. If the refrac- tion is not corrected this spasm of the ciliary muscle will change a hypermetropic to a myopic eye, or cause myopic astigmatism. Third. Myopia.—-Many children, who are hypermetropic at birth, become myopic when they begin to use their eyes contin- uously for reading, sewing or fine work. [Angell.] This is especially apt to be the case if the child is not robust. Fifth. Disease.—Hypermetropia, when combined with astig- matism tends to produce certain morbid changes in the fundus like those which occur in myopia. Nearly three-fourths of all cases of that dreaded affection, Glaucoma, which so often result in blindness, occur in hypermetropic eyes. IV.-—— PRESBYOPIA. (Old-Sightedness.) [.Presbus, old; ’0ps, eye.] Several changes take place in the eye as the result of increas- ing age. The acuteness of vision diminishes as the media lose their transparency, and the retina its sensitiveness. The power of accommodation gradually diminishes, and hence the near point of vision steadily recedes. This is due to the lens becom- ing firmer, and so losing its elasticity, and also to a loss of power in the ciliary muscle. The condition is entirely physiolog- ical, no anomaly of refraction being present. When nine inches is the nearest point at which we can see to read No. 1 type, we consider Presbyopia to have begun. In the emmetropic eye this occurs at 45 years of age: with myopia later, and hypermetropia earlier. Since, however, all of the accommodation cannot be used continuously for reading, when the near point is nine inches, the book is always held at a much greater distance from the eye. Erronoc+r.-——The chief cause, as already mentioned, is Old Age; but Broken Health, Prostrating Disease, 20 ASTHENOPIA ; Hypermetropia, and Incipient Cataract may precipitate the trouble. , Drseuosrs.——-The presbyope enjoys good sight for distance, but finds it difficult or impossible to maintain clear vision for near objects, especially in the evening, Print is indistinct and confused. The eyes become tired and perhaps painful. In reading he places the lamp between his eye and the page so as to have the strongest possible light upon what he is reading, and at the same time to make his pupils contract, and thus shut off the circles of difiusion. Objective Signs.——The patient’s distant vision may be §-E}, i. e., he reads No. XX at twenty feet, but he cannot read small print as near the eye as he should. Convex glasses are not needed for distance, but they greatly improve near vision. THE TREATMENT consists in furnishing artificial accommo- dation——i. e., in adjusting such convex lenses as bring the near point back to the normal distance. Tall people naturally work at longer range than those short of stature, and the glasses should be adjusted to the distance at which the patient’s occu- pation requires him to work. For reading they should render the type clear without magnifying it. Patients selecting glasses for themselves almost always choose those which do magnify, and are hence too strong. These not only fail to give relief, but cause increased weariness of the eye. It is not best as many imagine, to defer to the latest the wearing of proper lenses. Discomfort and fatigue of the eyes are sufficient indications that the time has come when they need relief. Suitable glasses, then prescribed, instead of increasing the Presbyopia, tend to prevent it. Where Presbyopia exists in an eye strongly hypermetropic, or myopic, the patient will re- quire two pairs of glasses, one pair for reading and another for distance. Pnoeuosrs.——In the emmetropic eye, Presbyopia increases about one dioptric every five years from the age of 45 to that of 70. At 75, as a rule, all accommodation is lost. If the glasses have to be changed much oftener than every three or four years, it is not a favorable symptom, as rapidly in- creasing Presbyopia is a precursor of Glaucoma. When this A MONOGBAPH ON WEAK mans. , 21 disease exists no glasses at all should be used, as it is necessary to avoid all tention in the eye. Second Sight, is a popular term to indicate that one who has worn glasses, no longer needs them. This condition is due to a contracted pupil, admitting only the central rays of light, which do not need much focusing. Or it may be due to cataract begin- ning at the edge of the lens, (cortical,) by which the refractive power is increased. Elderly people who never seem to need glasses give, thereby, positive evidence that they were near- sighted, unless the conditions just mentioned are present. V.—ASTIGMATISM. (Inregiilar Refraction.) Astigmatism is a condition of the eye in which the rays of light emanating from a point are not brought to a focus at any place within the eye, so that a dot appears as a line, an oval or a circle, but never as a point; hence the name: ’a, privative, (with- out,) stigma, a point. The optical defect lies in the cornea, which, instead of being round, is egg-shaped, or curved like the bowl of a spoon, having different degrees of curvature in its two principal meridians, which latter are always at right angles to each other. This un- equal curvature produces unequal refraction, so that rays are brought to a focus by one meridian in front or behind the place where they are focused by its conjugate. In this way we account for the letters of a printed page seeming to “ overlap each other” and their outlines appearing so indistinct. Astigmatism is the most common of all the refractive errors of the eye, as it complicates very many cases of Hypermetmpia, and still more of Myopia. Indeed, all eyes are very slightly astigmatic, but the asymetry is usually so little that it occasions no annoyance. The forms of Astigmatism are principally two,-—the regular and the irregular. Of the second but little need here be said, as it is almost irremediable with glasses, though it may be improved by various other devices. 22 ASTHBNOPIA ; Of the regular Astigmatism there are five varieties: Simple hypermetropic Astigmatism. Compound hypermetropic Astigmatism. Simple myopic Astigmatism. Compound myopic Astigmatism. Mixed Astigmatism. In the simple forms one meridian is emmetropic, and that at right angles to it, hypermetropic or myopic. In the compound varieties both principal meridians are hypermetropic or myopic, but unequally so. In mixed Astigmatism one meridian is hy- permetropic and the other myopic; this form is quite rare, how- ever. Usually both eyes are affected symmetrically, but this does not always hold true. In most of the marked cases of Astig- matism vision is somewhat impaired by an associated defective retina or nerve (amblyopia). , E'rroLoGx.-—Regular Astigmatism is congenital, and often her- editary. Occasionally it may be caused by ulcers, or wounds of the cornea, as the cataract operation. The irregular variety when acquired is due to ulcers, injuries, maculae, conical cornea, or a displaced lens; when congenital, to irregularities in the refractive power of different sectors of the lens.* DIAGNOSIS-—Whi16 the patient is young but few symptoms are manifest, as he is able largely to overcome the defect by un- equal contractions of the ciliary muscle. With increasing years the lens becomes less plastic, the eyes are more constantly used and vision is noticed to be defective. w'Qbiects appear differ- ently as he tips his head from side to side,‘ and the eyes become wearied if he use them too long for near work. The letters run together, and distant objects are indistinct in outline. The pa- tient has probably tried many pairs of spectacles, but never found one to suit. Headache is a very prominent symptom. It may be frontal or occipital; is often nervous, attended with nausea; a headache for which the patient has “tried everything!” Nothing perma- nently relieves it, because nothing has removed the cause. Some refer their symptoms to the spine, and complain of $"'."‘S”E°£"‘ * Wells’ Diseases of the Eye, p. 150. Hartridge, Refraction of the Eye, p. 139. A MONOGRAPH ON WEAK EYES. 23 severe aching in the neck and down the back, with great weari- ness on slight exertion, as knitting, crocheting or reading. Objective Signs.—In the astigmatie subject we frequently note a lack of symmetry in the two sides of the face. To see better the patient often tips the head to one side, and whenever we observe him so doing we may be sure Astigmatism is present. Tests.—Having failed to bring the vision up to normal with convex or concave lenses, and suspecting Astigmatism, we direct the patient to look at a disc of radiating lines in groups of threes. FIG. 4. ~~\\\ 90 got Placing it first at a distance of 30 feet, we gradually approach the patient and find out whether at any point he is able to see some of the lines clearly while others are indistinct. If this be the case the presence of Astigmatism is proved, and the direction of the most distinct line is also the direction of one of the 24 ASTHENOPIA ; principal meridians.“< Of course each eye is to be tested seper- ately. In some cases the above method is not reliable. Hence, it is best to resort to Retinoscopy, which immediately reveals the nature of the error and the direction of the princi- pal meridians, with the approximate amount of error of each. I cannot too strongly urge upon all physicians to make them- selves master of a method of diagnosis at once so simple, so easy, so accurate. In Astigmatism the red fundus reflex is crossed by bright and dark lines, which indicate the direction of the principal axis. Opthalmoscopic Test.—By this the optic nerve disc appears oval, instead of round,]L the long axis corresponding to the mer- idian of greatest refraction, when we pursue the indirect method, and vice rversa when the direct.I As all are not supplied with the requisite appliances for trying the tests already described, we will give another and very simple one: Have the patient look through a large pin-hole in a blackened card at a light thirty feet away. If, on moving the card back and forth in one way, the light seems to dance, while it does not do so when the card is moved in the contrary direction, Astigmatism is proved to exist. Various other methods are in use among specialists for the detection and estimation of astigmatism by means of the sten- opaeic slit, the prisoptometer, Tweedy’s optometer, and the ophthal- mometer of Javal and Schiotz. TREATMENT.-—NO one but an oculist should attempt to pre- scribe glasses for an astigmatic eye, since it requires great nicety in locating the angle of the principal axis and a thor- ough understanding of the theory of irregular refraction. The glasses required are cylindricals—i. e., those which are per- fectly straight in one direction, but ground convex or con- cave in the opposite. When the astigmatism is combined with hypermetropia or myopia, a lens which is cylin- drical on one side and spherical on the other is used, while for the correction of mixed astigmatism, crossed cylinders, or combinations equivalent to the same, are to be prescribed. Eye- * For measuring the amount of error, see the large works on this subject. 1- I t may have this shape from other causes. I For the estimation of refraction by this method see Ophthalmic Science and Practice Juler, p. 393, or Loreng’s Ophthalmoscopy, p. 118. A morzocnxrn on WEAK mus. 25 glasses should never be used, as with them it is impossible to preserve the proper axis, but spectacles should be ordered for constant use. Medicinally, agaracus, jaborandi and physostigma are beneficial where unequal contraction of the ciliary muscle exists. Atropine will relieve the headaches temporarily. Pno~ Hartridge, Refraction of the Eye, p. 194. A MOEIOGBAPH on WEAK EYES. 27 to again fix the object.* The reason of this is that FIG- 5- when one eye is covered the stimuleus to binocular vision is wanting, and the covered eye is left to the control of the stronger muscle. Another and better test is to place a prism of 10 °, base down, before the right eye, and have the patient look at a dot on a vertical line (fig. 5). He will see two dots, one above the other. If both are on the line, there is no Insufficiency present; but if the upper dot is to the right, the internal recti are deficient; if to the left, the external recti have been overcome. TREATMENT.-—Th6 treatment must be directed at the cause of the trouble. When this is general debility, we must build up the system; if an error of refraction, that must be corrected; if excessive tea-drinking, that must be stopped. Weak prisms, with the bases toward the insufficient muscles, give speedy re- lief, as a rule. We must, however, be on our guard, lest, in wear- ing them, our patient’s internal recti deteriorate from want of sufficient exercise. To prevent this we have him practice sys- tematically looking at the finger or a pencil as it is carried back and forth, away from and near to the eye, and far to the right and left. In Myopia the simple correction of the refraction often re- lieves the entire trouble. Sometimes, however, it is necessary to combine with the correcting lens a weak prism, in order to give entire satisfaction. Where the health of the patient is good and a 2° or 4° prism does not effect a cure, it becomes neces- sary to divide one of the external recti, as in squint operation. Internally the following remedies have proved riseful, accord- iug to indications: Iron, salt, arnica, ruta, conium, jaborandi, phosphorous, calcarea and argentum nitricum.T The judi- cious use of the galvanic current is also beneficial. We should encourage only a moderate use of the eyes until the weakened muscles have regained their normal tone. * Refraction of the Eye, p. 194. T Ophthalmic Therapeutics. Norton. 28 ASTHENOPIA ; VII.—RETINAL ASTHENOPIA. (Over-Sensitiveness of the ‘Retina.) An irritability of the retina, accompanied by hyperaemia (congestion) of the optic nerve and retina, frequently exists in young persons, especially young ladies of delicate, nervous tem- perament, in ill-health. The sensitiveness of the visual nerves is increased to a painful degree, and the condition thus caused closely resembles accommodative Asthenopia, although no error of refraction is present. ETIOLOGY.—A slight blow upon the eye, a violent shock to the nervous system, a bright flash of lightning, exposure of the eyes to strong light, and especially prolonged use of the eyes by arti- finial light, as reading when in bed, are fruitful causes of the dis- order. It also occurs as a complication of hypermetropia, or is. consequent upon a severe inflammatory attack. Frequently, however, it is a reflex in nature, and arises from spinal afections, indigestion, decayed teeth, uterine troubles, etc. If, in young subjects, leucorrhoea co-exists, masturbation is to suspected (Hartridge). Retinal Asthenopia may also arise with- out any apparent causes except general debility. Dmenosrs.—The most marked symptom is aversion to light. This is sometimes intense and accompanied by ciliary neuralgia, the pain shooting into the face and head. Usually some soreness of the eyes is present, with itching and an increased secretion of tears. Vision is perfect, although sight is easier in a dim light. If any object be intently regarded for a few moments the impress- ion is retained upon the retina for some time, and by slightly pressing the eyeballs the patient sees flashes of light or bright rings (photopsies). Objective Signs.—The pupils are enlarged; the field of vision is diminished, yet the presence of the photopsies prevents our mistaking this for incipient amourosis. [Von Graefe.] To the ophthalmoscope the eye appears natural, though the retina may be injected and the edges of the disc hazy. TBEATMEN'1‘.——-All over-use of the eyes is to be avoided. If the trouble is due to an error of refraction, that must be cor- rected by the proper glasses. A MONOGBAPH on wnxx nrns. 29 As the condition of the retina is a good index of the condi- tion of the general nervous system, treatment must be directed accordingly. The patient needs encouragement, and must be kept cheerful. The general health also must be built up and any local trouble relieved. Where photophohia is excessive, it may be necessary to con- fine the patient in the dark for some days, and then gradually accustom their eyes to the light. [Von Graefe and Norton.] But, as a rule, the patient needs the air and sunshine; and, if the eyes are closely bandaged, or protected by blue glass, he may be allowed out of doors much of the time. Even enforced exposure to the light is recommended by certain high authori- ties {Agnew and Webster.] Remedially, depletions are to be avoided, but dry cuppings on the temples are beneficial. Tonics are often indicated, especially zinc lactate, beginning with gr. ss. and increasing to gr. iv., twice daily; also, steel and quinine. Macrotin has proved useful, es- pecially with women, as have also jaborandi and lilium. Gel- seminum, ignatia, nux vomica, conium, belladonna, lactic acid, mercurious, pulsatilla and scutillaria are all highly esteemed by some authorities. G-alvanism, a mild current, with the — pole on the eye, and the + at the back of the neck, is very benefi- cial. The Prognosis is usually favorable. VIII.-—SPECTACLES. The use of spectacles to correct optical defects is one of the greatest achievements of the present age. No organ in the body is so precious as the eye, and it seems strange that so many centuries should have passed before its anomalies of refraction and accommodation were understood, and their remedy scientifi- cally prescribed. Until very recently few physicians, and still fewer of the laity had any true conception of the nature of Asthenopia, of the great benefit to the eyes of accurately fitting glasses, and of the evils resulting from those unsuited to the case. At the first, prejudice, detering the needy from their use, and later fashion, 30 ASTHENOPIA ; luring the thoughtless to their random purchase, have each pro- duced disastrous consequences. How many, many eyes the oculist examines, which, by the timely use of glasses, might have been reserved! And if this little work convinces some of the profession that delay is danger- ous, it will not have been prepared in vain. It will have been successful, if it has further revealed the danger of the hazardous practice of buying spectacles of any jeweler or street vender, _who, though perfectly ignorant of all the laws of optics, claims, for the sake of a bargain, that he can fit any eyes, that his goods are “genuine Scotch and French pebble,” that his glasses “will draw the eye out to its right shape,” or that his “blue glass is a panacea.” In eight cases out of ten, the person choosing selects the wrong glasses, not knowing that each eye should be fitted separately, or ignorant of the amount of Astigmatism present. On this point the great Dr. Wells, of England, speaks as fol- lows: “I have no hesitation in saying that the empyrical, haz- ardous plan of selection generally employed by opticians, is but too frequently attended by the worst consequences, and that eyes are often permanently injured which might, by skillful treatment, have been preserved for years. For this reason I most strongly urge upon medical men the necessity of examining the state of the eye, and ascertaining the exact nature of the affection of re- fraction and accommodation.”>l< This being done, they can de- termine whether the patient really needs glasses, and, if so, send him to the scientific oculist, who will prescribe them accurately. The proper prescription having been given, a few other points need attention. Care should be taken that the frames fit well, that the glasses are on the same level, and that the centre of each lens is exactly opposite the pupil. This last point is of great importance, as otherwise the glasses act as prisms and give rise to annoying diplopia, or muscular Asthenopia. Concave glasses should be worn close to the eyes, while with convexes this is not at all necessary. The Ben Franklin glasses are used in Myopia combined with Presbyopia, as the lower half is convex. for reading, and the up- per concave, for distance. Painters and lecturers often find this 1’ Diseases of the Eye; p. 667. A MONOGRAPH ON WEAK EYES. 31 form very useful. Some, however, prefer to use spectacles for reading, and folders for distance. Folders (pince-nez) may be used where there is no Astigma- tism, provided we are sure that the lenses are well centered. Where, however, constant use of the eyes for a long time is re- quired, it is better to have spectacles, as they are more firm and steady. Single eye-glasses are not to be recommended. Stenopaeic glasses (those in which one sees through a narrow slit only) are of use in cloudy co'rnea or high degrees of Myopia. Blue-tinted glasses are useful when the retina is sensitive, as it is the orange rays in sunlight which hurt the eye, aud blue ab- sorbs these. Smoked glasses are not good, as they simply reduce the whole amount of light received- into the eye, and thus ren- der all objects indistinct. Goggles heat the eye, and therefore should yield to large curved glasses, which are better. To shield eyes exposed to fly- ing splinters, chips or bits of stone, clear mica protectors are the best. The physician is often asked by the patient whether he should order lenses made of pebble or glass. The only advantages of the former are that they are a little lighter and cooler, while, on the other hand, being cut the wrong way of the crystal, they are very seldom free from flaws, which, though invisible to the eye, still irritate it by causing irregular refraction)‘< Beside, pebble is harder than glass, and hence more diflicult to cut accurately, cylindrical lenses never being made of it. It is possessed of a double refraction, which is a decided objection; it does not re- fract regularly, and is much more expensive. On these accounts experienced oculists no longer advise the use of pebble, though it is still hawked about the country by wandering charlatans. The respectable manufacturers of the country use nothing but the best crown plate glass, whose cost accrues from the care- ful grinding to make it accurately conform to the prescription -Of the scientific oculist. ' " Opthalmic Science and Practice. J uler, p. 409. 32 ASTHENOPIA ; TEST TYPES, CORRESPONDING TO THE SOHBIFT-SOALEN OF EDWARD JAEGEB, OF VIENNA. No. 1, PEARL. In using the reading tests, the patient should endeavor, with each eye, separately, to read the smallest type that he can, at the nearest and farthest pomts possible. This type should be read easily at 9 to 12 inches. If1t is necessary to hold it farther away, the eye needs attention, as presby- opia is probably present. No. 2, he ATE. The perfectly normal eye is able to read this type at 8 to 16 mches from the face for some time, without tiring. If reunal asthenopm be present, it will be seen best in a dim hght. No. 4, NONPARIEL. A person with normal vision can read this type without difiiculty at 7 and also 25 inches from the eye. If unable to read it at both these distances, a physician should be consulted. No. 6, BREVIER. This type should be read at 6 and also at 40 inches. In near-sightedness the latter is generally difficult, if not impossi- ble, while in Hypermetropia the first will severely test the accom- modation. No. 8, LONG PRIMER. Although a person may be able to read this print at 48 inches, if the eyes become tired and seem weak when used for a short time for near work, the internal recti muscles may be at fault. No. Io, PICA. When marked Astigmatism is present it will be difficult to read this print at 7 feet, by each eye, separately. A MONOGBAPH ON WEAK runs 33 No. 12, GREAT PRIMER. * This type should be read 9 feet from the face, though, if able to do so, the patient may be hyper- - metropic. VZYAFEGILPN NPRRUDBZ RXPXXNCB F Z~B E 34 ASTHENOPIA ; A monocnnn on WEAK mzns. LO LXX. r 'l' J 0 z L ’ . \ \- u \ '>‘2i% VERNON HEIGHTS... ->=>->:>-$;,'»,s~e¢<-~sw- Beautiful tor ~Situation. ~»’9-*»>&>>",',\-i<~€<-G<- The Most Desirable Loc'at~io~-n In or About Omaha $4 ég M .03.... V); ea H 0 Ni E S as WW -e->:>$%>>->>§sr7‘4 Near Grounds Lately Purchased for St. llery’~s College. %%%% PRICES or LOTS MODERATE. TERMS EASY. *»®§@§@ EDSON & CO. ARCADE OFFICES No. 16, 1509 FARNAM ST. The Geneva @pticat Ciompang, Manufacturers of Spectacles, Eye-Glasses, Trial Lenses, Etc. PRESCRIPTIONS FILLED PROM PTLY. THE PRISOPTOMETER, .';g'g,=,r I An Instrument for the quick 1. detection and correction of My- ( cpia, Hypernmtropia and Astig- ' matism in all its forms. Its operation will be readily seen by the following illustra- tions. The Instrument is fixed to a table. A board fifteen in. square, with black background, on which is a white disc about five inches in diameter, is placed at a distance of say fifteen feet. IMPROVED TRIAL FRAME WITH SCREW ADJUST- MENT EOR PUPILLARY DISTANCE, ADAPTATION FOR GIVING HEIGHT OF NOSE PIECE AND INCL1NA- TION FRONT OR BACK OF PLANE OF LENSES- The patient is directed to look at the white disc through the in- strument. There will appar- ently be two discs visible to the patient, just touching if the eye , _ is normal. The Hyper metropic eye will see them separated and the Myopic eye will see two discs apping. The great val- ue oftheiustru- meut is in diag- nosing quickly the most com- plex cases, as ' each meridian of the eye is examined separately .. ' ' . ., " “ ‘ . “ . . , y , by rotating the index finger C. M ' V ., '- ._ ,,, . - -- - it The Prisoptomeler isundoubt- .1.-rt -P.‘.‘,,m.,,§,r-,,.....-W],_“_‘__,_ ' - - “ edly a great addition to the cou- ‘""' " " i"' " . ' ‘ . lists’ instruments for examining Tnun Lnnsns rs Snrs. Lower pmces and better 10 rel~,.,,c,i0ns_ It does not 5upe,._ quality than can be obtained elsewhere. cede buts“ p]ement,,t,.,,,11enses Send for Illustrated Price List. and is use with them’ and with a good set of trial lenses and test types, makes the ideal outfit... Price, $25 with table; $21 without table.—Prescriptions from Oculists or 0 ticians filled promptly, correctly and at lowest rates. Lenses ground to order at our actory or Chicago Oifice. ..S;:.5‘.:£.;€.a“‘§::.:“.%‘.?.:;?‘* GENEVA PTIBAL 60., Geneva, N. Y. ht". PATHOLOGY, DIAGNOSIS, AND TREATMENT. OCULAR DISEASES, E.~T. ALLEN, A. M., M. 1)., Diseases of the Bethe and Me Nerve. OMAHA: wssrsau mmrme COMPANYI 1888- COl\l'l‘I—IN'1‘S.. ; PAGE 5 H yTDQ1‘zl’ll1l2l Actha .lleI';. ct. N. O . . . . . .. 3 Hyneraeinia Passiva Re-t. ct. N. O. . . . _. I -A'p-npiexia Retina‘ . . . . . . . . . . . . . . . . . . . . .. 5 ,' IsclniriniaZltetinug . . . . . . . . . . . H lietinitis Scrosa... . . . . . . . . . . . . . . . . . . . .. 7 1 Betinitis lEx11dat-iva . . . . . . . . . . . .. V . . . . . R ! lictinitis Apoplectica. . . ...... .. u { R_etinitis.AlbumiI1urica . . . . . . . . . . . .. _. . . 10 , Rctinitis Syphilitica . . . . . . . . . _ . . . . . . ll ltetinitis Pigmentosa . . . . . . . . . , . _ . . . . . . . . . . 12 } Rot-init-is Leuoaeinic-a . . . . . . . . . . . . . .. 13 ‘f N.euro-Iietinitis . . . . . . . . . . . . . . . . . . . . . . . .. 14 { Ncuritis Optica . . . . . . . . . . . _ . . . . . . . . ._ . . . . 1:3 T . . Ncuritis Optica Orbitalis . . . . . . . . . . . . . . . .. 16 ‘ Atrophia Rot. ct N. O . . . . . . . . . . . . . . . . .. 1'7 Embolia Arteria Cont. nee . . . . . . . . . . . . . . . . 18 \ Amotio Retin: ~_ ................ ....... .. 19 l Glioma R(~.‘t-llliD. .', . . . . . . . . . . . . . . . . . . . . . . 20 Cyst-icorcus . . . . . . . . . . . . . . . . . . . . . . . . . . . .. 21 A Commotio Rotimr‘. . . . . . . . . . . . . . . . . . . .. 22 ‘ l{y_pe1':est-hcsia Re-tin-.1>.. .. . . . . . . . . . . . . 23 g Aneestliesia Rotiiue. . . . . . . . . . . . . . . . . .. 24 Am-blyopia . . . . . . . ..‘. . . . . . . . . . . . . . . . . . . . . . .. Aclironiatopsia . . . . . . . . . . . . . . . . . . . . . . . . . .. :26, I-lomeralopia . . . . . . . . . . . . . . . . . . . . _ . . . . . . . . . 27 z Hemiopia . . . . . . . . . . . . . . .. . . . . . . . . . . . . . . . .. 28 'Malingering../ . . . . . . . . . . . . . .. .* . . . . .. :29 ‘ Index . . . . . . . . . . . . . . . . . . . . ..; . _ . . . . . . . . .. 30 T0 SPECTACLE ANDEYE-GLAS3wEARE~RS.~ It is just as escential to have the frames of your Specs and Eye“-_ Glasses fitted properly as to have the proper glasses. , - @ ' ’ AL0E’§ PERFECT, TCOMFQRT @ ~ WE CALL , \. '3'-‘.';' , ?,~'32;..>- i ATTENTION ~ ‘THE BEST ——TO—- - in the world. ' fit @*~*’ ~ ' They sit, straight on the face, do not twist or turn, and the eye is always in centre of lense; do not press the ,nose,_and, as the name states, perfectly comfortable. ~ ALOE’S PERFECTED LENSES ~ are unequaled. Our Brazilian Pebbles-are cut on their proper aXes_. K _ . Aross CORK Nose SPECTACLES--q are unequalled -in comfort,‘ in steel or gold. Send for ourlPrice " List. We have the-only first-cla/ss factory inthe West. ~ A. s. AL()E~€5-CO., PRACTICA_L ()P~Tr_crANs,i‘[Q ~ Corner 4th and Olive Streets. ~~ g~ ~ST. L~OUIS,"E_MO.~ C()IP~RESPOI“Rest of eyes, especially 3. Disorders of the uterus. by ,,,.,,fiC,a1l,g.ht_ - Q Moderate exercise in open 4. Intracramal tumors. air with eyes protected 5. Glaucoma. . Homueopathic. 6. Errors of refraction. Bell., 3, 4, 6, 9, 10, 13, 15. 7. Alcoholism. Bry., 2, 5. 15. '1 C€tCt.., 1 D|AGNOS|S_ 8. %N0 I)lf;)OlSl&, 6, 8, 9, 10, 11, 13, 9. N0 lachrymation. Lept..3- _ _ _ , Macrot., 8. IO. Vision impaired. Merc..2,13. . . Nux v., 2, 7, 10, 11. 0/J/zz‘/2. II. Dull red disc with hazy border. *P,,1s_,3,6,10,13, . r 1.2, 3. [Alcohohsm.] bu 1 I2. Larger vessels seem to be fringed. (1/. s. Allonathic. Hypertrophy.) I 3. *Veins dilated, dark, tortuous. 14. Venous pulsations. I 5. Oedematous condition of the retina. PROGNOSIS. Favorable. SEQUELA. Retinitis Serosa. 5 APOPLEXIA RETINE. [Hwmorrhage into the Retz'na.] PATHOLOGY. Non-in-flamatory extravisations of TREATMENT- I blood into the Retina. Genera‘ Measures- _ _ , Rest. ETl0LOGY- I. 'X‘Haemorrhag1c diathesis. Avoid light. . . . Wea s oked glasses. 2. Pern1c1ous an&m1a. Remf,,;‘,,u,e_ 3. *Disorders of menstruation. , . Homceopathw. 4. *Degenerat1on of blood-vessels. Be1L,3,12, M -><- - h *Crot., 1, 4. 5. Cardiac hypertrop y. G1On_ 6. Valvular disease. *L*kll@h--11~2:1 9 10 1 1 *P OS" 1 5 7 1 2') 41 7. Pulmonary emphysema. 15,16. 8. Severe coughmg. A"0pa,h,c_ 9. Retinitis. Digitalis. - Leeches. IO. Choroidal atrophy. TOnics_ . T raumatism; results of iridectomy and Di“1‘eti<>S- H )-1 reduced intra-ocular tention. DIAGNOSIS. 12. Everything seems red. 13. Injury to vision depends upon the location of the effusion; if at macula there is a dense central scotoma. Op/22‘/2. I4. Vitreous clouded if haemorrhage is abundant. 15. *Hmmorrhagic spots smooth, uniform, under vessels. [In deep layers] 16. *Haemorrhagic spots striated, edges irreg- ular, lie over vessels, and occur near disc. [In superficial layers] 17. Metamorphopsia, late. PROGNOSIS. Extravisation absorbed, leaving no trace, if small. Leaves pigmented spots or causes z'nflammatory c/zcmges, if large. Vis- ion rarely regained, if at macula. SEQUEL/E. Retinitis. May be forerunner of cerebral disease. 6 ISCHZEMIA RETINZE. PATHOLOGY. Sudden, total arrest of the retinal cir- TREATMENT. Culation. General Measures. ' Invigorate the general No tissue changes. health _ Paracentisis. ETIOLOGY. I. Epilepsy. Iridectomy. 2. Obstruction to circulation. [Ernbolism.] Homoeopathic. DIAGNOSIS. 3. Rare occurrence. Asar-.1- V_ , Calc. c. 4. "‘Bl11’ld1'l6SS complete, sudden, both eyes. Ch,,,,,,8,9_10,11,12_ . 0 - Fer.,4. 5. Tent1on normal, (6) later Increased. Phosq 4’ 5, 10_ 7. Balls very white. Puls' 8. Pupils dilated. Allopathic. Op/zz‘/z. 9. Disc white, transparent.(r/.s.Atrophy.) Stimulants. IO. Arteries reduced to threads, bloodless. II. Veins reduced, or irregularly hypereemic. I2. *PuZse 2/cry rapia’. PROGNOSIS. Favorable. Generally recover entirely in a few days. 7 RETINITIS SEROSA. PATHOLOGY. An aedematous infiamation, sometimes TREATMENT- accompanied by slight haemorrhages. General Measures. The poor vision is caused by compression of Absolute rest of the eyes until cured. nerve elements. . Dark blue glasses ETIOLOGY. 1. *Prolonged exposure to very bright light Homoeopathic. - - *Bell., 6. 7, 9, 11.12.12, 14, 2. Neglected passive hyperaemia. 16’ 18, 21, 22, 23, 25, 26. Diseases of (3) heart, (4) liver, (5) kidneys, Bry.,12. *Duboisia, 2, 9, 11, 16, 21, (6) uterus, (7) brain. 22, 23_ 26_ 8. Pregnancy. Merc., 4. 12. 13, 16,18, 20, _,(_ - 22, 24, 25, 26. DIAGNOSIS. 9. N0 lachrymatmn. PhOs“12, 13_ 15‘ 18, 22 23 1o. *No pain. 25.26. -><- T - Plumb. II. ./to photophob1a. I2. Sees a thin gray film before eyes. Allopathic. 13. Vision mich impaired, especially central. Local depletion. 14. May become blind. 15. Eye looks well externally. 16. Pupil dilated and sluggish at times. Op/at/l. 17. No marked changes. 18. *Under dim illumination a delicate uni- form bluish veil over fundus (z/. 5. R. exu- dativa), shading off toward the periphery. 19. Retina swollen; not granulor (2/. .s R. ex- udativa). Fundus reflex reduced. 20. 21. Macula reddened. 22. *Disc swellen; sometimes reddened. 23. Disc outlines indistinct. 24. *Arteries normal or enlarged; slightly veiled. 25. *Veins large, dark, tortuous ; at points covered by effusion. 26. Small extravisations of blood along the vessels. PROGNOSIS. Guarded. Often bad results. May last a long time. SEOUEL/E. Atrophy. R. exudativa. Disease of choroid or vitreous. Detached retina. 8 \ RETINITIS EXUDATIVA. PATHOLOGY. Paranchematous inflammation, with hypertrophy; blood stasis; proliferation of cells; fatty degeneration; sclerosis; extra- visations; derangement of rods and cones; involvement of charoid. ETIOLOGY. I. Neglected hyperaemia. 2. Bright’s disease, or, (3.) diabetes. 4. Syphilis. 5. Cerebral affections. DIAGNOSIS. 6. Vision good until macula becomes involved. 7. Micropsia [late] 8. Field generally normal; possibly, (9.) sco- toma, and (10.) dimness at the periphery. Op/zz‘/z. 11. Vitreous sometimes hazy. 12. Disc swollen; (I3.) red. I4. Disc gray, if there is serous infiltration. 15. Disc outlines irregular; (16.) indistinct. I 7. Arteries diminished in size; <18.) later like white threads, with thick walls. 1/. s. Embolia Art. Cent. Ret). 19. Red blood current visible, if light is thrown near one side of artery. (7/. s. Em- bolia.) 20. Veins swollen, dark, tortuous. 21. Vessels hidden in spots, by exudation. 22. *Exudation white; or, <23.) gray dots or patches. 24. Blood extravisations. PROGNOSIS. Depends on the cause and severity; often unfavorable. Sight may be restored and the exudations absorbed. SEQUEL/E. *Atrophy. Detached retina. Sclerosis of arteries. TREATMENT. General Measures. Blue glasses. Hygienic t/reatment. Hommpathic. Ars., 2, 22, 24. *Bell., 5, 11, 12, 13, 15, 16, 20, 21, 22, 24. G1on., 7. *Merc., 4, 9, 10, 11, 12, 15, 20, 21, 22, 24. Phos., 2, 9, 10, 11, 12, 13, 15, 17,_24. Puls., 2, 12, 13, 15, 16, 17, 20, 21. Zinc, 5, 10. Allopathic. 9 RETINITIS APOPLECTICA. " [Syn Retinitis Haemorrhayz'm.] PATHLOLOGY. Fatty or atheromatous degeneration TREATMENT- of the blood vessels. Genera‘ Measures- . . . - gu -. 1 1. Slight serous infiltration. §§1?,‘j,.;1,.‘,),y ({,1:e]:a1,%,it]s(?a1t 1 Extravisations of blood. Dark glasses. . , *Absolu.te rest of the eyes ETIOLOGY. 1. Disturbances of the general circula- and brain. tion. Change of air, etc. 2. Embolic obstruction of a retinal artery. If hgemorrhages Ire h.'e' . . . quent, bandage eyes 3. Tumors in orbit or cranium. and confine patient to 4. Degeneration of coats of vessels. bed- 5. Alterations in quality of blood. *1‘“lf)1‘11°:i‘1 use of Stimu- 6. Syphilitic; 7. rheumatic ; or 8. gouty an S‘ _ diathesis. Homueonathm 9_ Injuries. A1‘I1., -L, 5, 9, 13, 16, 23. 13611., 1, 13, 15, 18,19, 20, DIAGNOSIS. IO. Adults and old peopie. 22, 23» $- 11. Often only one eye. ](;1;?,§;i:,’a241'9 20 22 12. Tendency to relapses. * L,,.,;,__ 5: 16: 20,’23_' I 3. *Attacks very sudden. *Mer. cor., 4, 6, 12. 18.17. I4. Vision good, unless, 15. macula involved. Pigs 4 10 16 17 N 20 16. Black spots in field of vision. 211%, ’ ’ ’ " ’ 0/§/ll‘/Z. I7. Dense opacities, if blood has es- Pu1s.,12,19, 20, 21, 22. caped into vitreons. S12-» 12, 16, 17, 19» 2°» 22- 18. ""'Retina looks watery. [Serous effusion] M3‘ _ 19. Disc faintly hyperaemic or deep red. _ Allopathm 20. *Disc swollen and edges indistinct. B1.°.m1d.°S ’ O D1Q1t€l11S. 21 Arteries normal or reduced. Alkalies. 22. *Veins dilated, dark, tortuous. Artificial leech. 23. *Haemorrhages, especially at division of vessels. 24. ""‘Many small heemorrhages parallel to vessels. 25. Heemorrhagic spots round, sharply de- fined, under vessels. [Lie deep] 26. Heemorrhagic spots, striated, cover ves- sels. [Superfiicial.] 27. Heemorrhagic spots retain color for a long time. PROGNOSIS. Depends on cause. Time, three to twelve months. Tendency to relapses. Effusion ‘ generally absorbed, but leaves its traces in dark lines. Unfavorable, if involve macula, or frequently recur. SEQUEL/E. Glaucoma. Atrophy of nerve. Pigmentary deposits. Total blindness. IO RETINITIS ALBUMINURICA. [lnflammatz'on of the Retzfla, from B7’Z1f/76278 Dz'smse.] PATHOLOGY. Bright’s disease. Serous infiltration of nerve and retina. Patches of fatty degeneration. Hypertrophy and sclerosis of nerve fibres and connective tissue. Arterial walls thickened. Capillary haemorrhages. ETl0LOGY- I. “Contracted granular kidney. 2. Pregnancy. 3. Scarlatina. 4. Heart hypertrophy. 5. Atheromatous condition of vessels. DIAGNOSIS. 6. *Both eyes. 7. *Impairment of vision; but it does not correspond with the opthalmoscopic ap- pearances. 8. Vision slowly deteriorates. (Suddenly in uraemia.) 9. Field not limited. 1o. Scotoma sometimes. II. Hypermetropia developed (?) Op/22%. I2. Early, over fundus a dull, gray haze; I 3. striated. I4. Disc bluish-gray. (2/. s. red, in Neuro- Retinitis). 15. *Disc swollen slightly. (2/. 5. greatly, in Neuro-Retinitis). 16. *Disc outlines indistinct. I7. Arteries narrowed; hidden in places. 18. Arteries as white lines [perivascularitis] 19. *Veins full, tortuous; 20. hidden in places. 21. *Hmmorrhages small; or, 22. large; 23. yellowish-red. 24. .rzzda2‘z'07zs,- a broad, 10/22%, gZz'sz‘¢’1zz'/lg, irregular ring around disc, at little distance. 25. *Pzzz‘r/265 white, soft-edged, covering ves- sels. 26. *Small, sic//ate, w/1220, gZz'sz‘mz'l2g 579015 az‘ 1/zacula. (2/. 5. near disc in Neuro-Ret.) 27. Later, general exudation. PROGNOSIS. Seldom leads to blindness. Seldom regain perfect vision. Favorable in fevers, pregnancy, and if exud- ations lie behind blood vessels. SEQUEL/E. Atrophy of nerve. Detached retina. TREATMENT. General Measures. Remedy cause. Best to eyes. Protect from light. Skim-milk diet. Avoid all exposures. \ Homoeopathic. Apis., 2, 3, 12. *Ars., 1. 4, 7, 8, 24. Bell., 2, 3, 7. 12, 15, 16, 1!), 20, 21, 25. *Gels., 2, 8, 12, 14, 16. 21, 24, 25. Hep. Kali. acet. Kalmia, 2, 24. *Me/re. 001:, 1, 2, 5, 7', 21, 24, 25, 26, 27. Pbos., 1, 5, 7, 8, 10, 12, 15, 16, 17, 21. Plumb. Zinc. Allopathic. Tonics. Diaphoretics. Purgatives. Muriate of iron. Citrate of iron and steel. Artificial leech. Dry cup [in anaemia]. II RETINITIS SYPHILITICA. [Sg/n. (]horio-Retinitia] PATH0l-0GY- Serous infiltration of retina. TREATMENT- Hypertrophy or sclerosis of connective tissue. General Measures Inflammation and degeneration of choroid. Cure syphilis. ' O _ Build up system. ETIOLOGY- I. Secondary or hereditary syphilis. (Jod-liver 011. . Dilate pupils. DIAGNOSIS. I. History of case. .. H ur . 3. XTendency to relapses. °"“B°pa ‘° . A‘g. ‘t. . One eye at a time. A;af_m . Scintillations and *phosphenes. *Aurum-i 14- *Kali jod, 1, 3, 9. 13, 14, 15, 4 5 6. Muscm. 7. Metamorphopsia, 8. Micrpsia. 18, 27, 28, 9 . *Vision poor, even in good light. ('2'. 5. *Me1*- e0r-.1T. 28- . Sul.,1,3. 6, 9,13, 14, 20. R. pigmentosa.) IO. Vision improves and retrogrades. Allopaihi<=- I I. Scotomata appear and disappear. Bi-chloride of mercury- I2. Field not limited. Iodlde Of Potash- I3. Traces of old iritis. Op/zz‘/z. I4. Vitreous hazy, with black specks. I5. Retina and disc hyperaemic, at first. I6. Fundus striated, especially near disc. 17. Faint haze at center of fundus. I8. *Stellate spots in macula appear and dis- appear, but do not glisten. (11. s. R. album.) I9. Sometimes macula is red-brown. 20. *Disc slightly swollen, ill-defined. 2 I. Bluish-gray film at disc and along vessels. 22. Arteries diminished in size. 2 3. Sometimes vessels appear as white lines, but do not glisten. (21. 5. R. albumin.) 24. Haemorrliages, very mre. 25. White or hazy patches, not glistening. (21. s. R. albumin.) 26. Gray dots, intermixed with white dots. 27. *Gray patches, fringed with black zone. 28. Gray patches, showing choroidal vessels. PROGNOSIS. Generally favorable. *Great tendency to relapses. Vision may be permanently impaired. Unfavorable, if macula is involved. SEQUELIE. Atrophy of nerve. Blindness. Forerunner of brain disease. I2 RETINITIS PIGMENTOSA. [Syn Pelr/mentary Degenerafion of the Rete'na.] PATHOLOGY. Proliferation of connective tissue. TREATMENT- Atrophy of nerve elements. General Measures. Deposit of pigment around vessels. Sustain general health. Hyaline thickening of arterial walls. nfy3S1:ig_ht T orpid condition of retina. light. Degeneration of choroid. BO W"-U g'““"d"d in the , , . use of mercury or Vitreous may be filled with fioatmg specks. iodide of potash. . Galvanism. ETIOLO Y. 1. Heredity. . . H ih . 2. Consangumeous marriages. °'"°e°pa ‘° . hilis. *Lycop.,11, 13. 3 . . _ Nux.v. 4. Long residence 1n hot chmates. Plms,13,14.19,22. 5. May be congenital. A"opa,h;c_ DIAGNOSIS. 6. Witl1 deaf-mutism: 7. microphthal P,,1h,,t,,,e_ mus; and, 8. nystagamus. “U11i1\'ai1iI1s‘-” 9. Begins at 15-20; complete after 35. 10. Both eyes affected. 11. 'X'[Vz;g/zz‘-é/2'/M72653, but central vision is good in bright light. (2/. s. R. Syphilitica.) 12. Later, total blindness. I3. "V"F2'c/ti c07zccnz‘7'z'ca//y (0/zz‘maz‘ra’, both for white and colors, so that it is difficult to walk without stumbling, and patient must turn to see surrounding objects. 14. Pupil small. Anterior chamber shallow. 0/2/1272. 15. Opacity at posterior pole of lens. 16. *DOZs and nrz‘zc/07'/e of pig?/zczzz‘ at /)(’7'z'/9/zcry of f2//zdus, and along 1242'/ea! 11655515. 17. *Large, irregular patches of pigment. 18. "“"Black lines in place of vessels. (21. s. Black circles in R. syphilitica.) 19. Disc red; or, 20. gray; later, 21. yellow, waxy. 22. Arteries reduced; small ones obliterated. 23. Veins enlarged; later, 24. reduced. 25. Choroidal vessels visible. 26. Yellow or buff, marbled appearance of fundus. PROGNOSIS. Blindness at age of fifty or before. Rarely arrested. SEQUEL/E. Atrophy of nerve. Irido-choroiditis. Cataract I 3 RETINITIS LEUCEMICA. PATHOLOGY. Diffuse inflammation, with haemorrh- TREATMENT- ages. General Measures. Hypertrophied and sclerosed nerve fibres, Treat the general disease. [white spots] Masses of fat granules. Homceopathic. Orot., 2, 3, 5, 9, 10, 13. - A‘ . ETIOLOGY. 1. Leucocytheemia. Ip1::e"' l 2. Previous haemorrhagic effusions. Nat. mur. Nat. phos. (‘?) DIAGNOSIS. 3. Bilateral. II‘)I;;Xa‘;' 4. Scotomata in visual field, corresponding Thuja. J to retinal patches. Op/at/z. 5. 'X‘Fnna’zzs pale yellow. 6. Haziness near disc, radiating in fine lines near vessels. 7. Striate opacity near disc. 8. Disc pale; outlines indistinct, hidden by serous effusion. 9. Vessels clouded and pale. IO. Veins pale; 11. dilated ; I2. tortuous. I3. *Haemorrhagic effusions, pale yellow. 14. Small irregular pale patches near macula. 15. *Round white spots, with red areola, pro- ject into vitreous, at periphery. Allopathic. “ General.” PROGNOSIS. Unfavorable. I4 PATHOLOGY. ETIOLOGY. I , 2. 0 J- 4. rl D0 DIAGNOSIS. NEURO-RETINITIS. [ Syn. Nearitis Optica Descenclena] Inflammatory proliferation of connec- tive tissue elements, with serous infiltra- tion. *Intra-cranial tumors. Spinal irritation. Neurosis of the symphathetic. Catamenial disorders. Heredity in males. 6. Both eyes. 7. Photopsies, and chromopsies. 8. Letters look red. 9. *Vision greatly suddenly. IO. II. Op/zz‘/z. 13. 15. impaired, sometimes *Field greatly contracted, [late.] Pupil dilated, and (12,) sluggish. albuminurica.) *Disc swollen. (1 6.) oedematous. *Retina hazy; (14) swolen (21. s. R. (1/. s. R. albuminurica.) 17. Disc red early, later (18) dull gray, (i9.) 20. 2I. 22. 23. 24. 25. 26. Exudations close to disc. 27. PROGNOSIS. striated. *Disc outlines indistinct, “woolly.” In macula symetrical group of highly refractive globules. *Arteries small, hardly visible. *Veins large, dark, tortuous. albuminurica.) Heemorrhages. *Exudations, as white or yellow dots or (7/. 5. R. patches, cover vessels in places. minurica.) Headaches. V er guarded. (1/. s. R. albu- Favorable if from uterine derangement, or of rapid development. Unfavorable in children; or with greatly SEQUELIE. contracted field. ~ Atrophy. *Total blindness. Death. TREATMENT. General Measures. Remedy the cause. Galvanism of the sympa- thetic. Dry cupping. Incision of the o p ti 0 nerve. Homoeopathic. *Ars., 9, 11, 25, 26, 27. *Aurum, 9. *Bell., 2, 4, 7, 9, 11, 12,14, 15, 17, 20, 23, 24, 25. Cact., 23. Con., 4, 9, 11. Crocus, 9. Gels., 11, 13, 14, 24, 25, 27. *Kali¢jod, 9, 23. Lach,, 9, 24. Macrot., 4. *Merc., 6, 9, 11, 13, 15, 23, 24, 25, 27. Nux v., 2, 3, 9. *Ph0s., 3, 7, 8, 9, 10, 15, 17, 20, 22, 24. Sang., 9. *Zino., 1, 7, 9. Allopathic. Seton in temple. Leeches. Ice applications. Bichloride [inunction]. Diaphoretics. Bromide of potash. I5 NEURITIS OPTICA. [Syn Engorged Papilla, Pepillitis, Oholced Disc, Ste./,uung‘.s Przpillc//.] PATHOLOGY. Inflammation begins in papilla and is TREATMENT- generally confined to it. Serous infiltra- General Measures. tion; haemorrhage. Nerve strangled by Remedy the cause. scleral ring. Connective tissue increased; Resteyes' . Smoked glasses. nerve fibers atrophied. Hygienic regulations. ETIOLOGY. 1. *Intra-cranial disease, in 80 per cent. Homeopathic. 2. Renal disease. 8. Acute myelitis. A,.S,,2,3,18,38, 3. Corneal ulcer. 9. Amenorrhoea. Aumm- . _ _ Bell., 1, 6, 12,17, 18, 19, 25, 4. Alcholism. IO. Orbital lesion. 31, 33, 36, 37, 38, ' ' ' >-*Nutritious but light diet. Plenty of sleep. *Avoid injurious amuse- ments, stimulants and tobacco. Galvanism. Homceopathic. *Arg. nit., 16, 23. *Nux. v., 2, 13, 14, 15, 16, 23, 24., 27, 32, 37. Strych. Verat vir., 8, 14, 16, 23, 24, 26, 31, 32, 37. Zinc phos. Allopathic. “ N 0 benefit." Aperionts. Tonics, especially iron. Steel and quinine. Strychnia. Zinc lactate or sulphate. *Leeches to temple. *Seton in back of neck. Bichloridc of mercury. Iodide of potash. Bromide of potash. * H ypodermic injections of strychnia. 18 EMBOLIA ARTERIA CENTRALIS RETINZE. [Embolison of the Central Artery of the Retina] PATHOLOGY. The blood clot generally lies just be- TREATMENT- hind the lamina cribrosa, but may occupy General Measures. one of the branches. A serous exudate occurs at the macula. . _ Homceopaihic. ETIOLOGY. 1. Cardiac disease. Am. (2) 2. Haemorrhages in the optic nerve sheath. Cr0ta1us,4_ 21_ 3. Anurism of the aorta. ](;‘i’*)§11:f:_iS£?)12’ 21- 4. Atheroma of large vessels. Prunus. 5. *Bright’s disease. A",,pa,|,,c_ 6' Fevers“ “Little can be done.’ 7. *Pregnancy. “Dietetic advice.” DIAGNOSIS. A rare affection, 8, One eye. 9. *V2'si0n Zest szuidenly/, often totally. IO. Size of scotoma depends on location of embolus. II. Impossible to excite phosphenes. Op/zt/z. I2. *A bluish or gray-white opacity around macula, [gd to 18th day.] 13. *A bright red spot at fovea centralis. 14. *Disc pale, transparent, may be surround- ed with haze; later ( 15,) atrophic. 16. *Vessels, especially arteries, very small; ( 17,) at points look like w/zite threads. 18. Veins irregularly filled with blood, com- pletely empty in places. 19. Irregular movements of column of blood. 2o. *Pressure does not cause pulsations. 21. Sometimes a few small haemorrhages. PROGNOSIS. Very bad. May recover so as to read. Generally become totally blind. SEQUEL/E. *Atrophy of the optic nerve. Glaucoma. I9 AMOTIO RETINE. [Detachment of the Retz‘na.] PATHOLOGY. Partial or total displacement of the retina by fluid effused between it and the choroid. The fluid, blood, pus or serum. Atrophy of the nerve elements. ETIOLOGY. 1. *Elongation of the optic axis in jkrogressh/e myopia. 2. Posterior sclero-choroiditis. 3. Irido-cyclo-choroiditis. 4. Retinitis albuminurica. 5. Retinitis serosa. 6. Traumatism. 7. Tumors of orbit or eye. 8. Cysticercus. 9. Haemorrhage. DIAGNOSIS. Premonitory; (1o.) muscm; ( II.) phosphenes; <12.) fluid vitreous. I3. Occurs generally near equator at lower half. 14. Suddenly. 15. One eye. 16. No pain as a rule. 17. *Decreased tention. (71. .1‘. Tumor.) 18. Vision impaired; at first a, ( I9.) cloud or dark spot surrounded with a lighter halo. 2o. *Wavy outlines of objects. 21. Objects fringed with red, violet or blue. 22. Sometimes photopsies. 23. *Field z'mpaz'1'ed 2'72 one pan‘ of eye; (24.) later, actually contracted. Op/'zt/z. Use dz'recZ method, at ten or twelve inches after atropine. Vitreous sometimes clouded. Part of fundus red, the rest dark. *Gray-blue waving cloud. [detached part.] Detached part reflects light strongly. Shadow of detached part on fundus. If entire retina detached, appears as opaque bluish mass behind lens. 31. Fovea bright red, (as in embolism.) 32. Vessels day’/é, crooked, undulating; no light streak in arteries; disappear abrupt- ly at fold of detachment. PROGNOSIS. Unfavorable. Better if limitations of field exactly correspond to detachment. Generally leads to total blindness. 25. 26. 27. 28. 29. 30. TREATMENT. General Measures. *If recent, confine to bed, chiefly on back, with eyes bandaged. Abstain from all use of eyes. Protect from light. Proper glasses. Atropine. Keep up general health. Out-of-door life. bimple puncture through sclera, at seat of de- tachment. (?) Homtnopathic. Apis., 5. *Arn., 1, 6, 14, 18. Ars., 4. *Aurum, 5, 10, 23, 25. Bry. *Dig., 23. *Ge1s., 1, 3, 6, 25. Hep. Kali jod. Merc., 10, 12. Rhus. Allopathlc. Mercury. Artificial leech. Injections of hydrochlo- rate of phylocarpine. Sequelce. Cataract. lrido-cyclitis, Phthysis bulbi. Atrophy of nerve. Metamorphopsia. Other eye is in danger of same. 2O GLIOMA RETINZE. [Syn Fungus Hcematodea] PATHOLOGY. Round-celled sarcoma which has a tendency to metastases. A very vascular encephaloid. ETIOLOGY. Obscure. I. *Heredity. 2. Traumatism. DIAGNOSIS. 3. *Children under twelve. 4. Several of the family. 5. No history of previous inflammation of brain or cord. (7/. 5. Choroiditis metast.) 6. At first no pain ; later intense pain. 7. *Vision lost early. 8. *At first eye looks healthy, except a ewe: yellow or pin/2 reflex, (11. 5. dull yel- low of Suppurative Hyalitis.) 9. Ball may appear atrophied for a time, with insensibility of cornea, (7/. s. Atrophy of Irido-Cyclitis.) IO. Later exophthalmus; movements curtail- ed ; palpebral fissure obliterated. ll. *Tention increased. ('0. 5. Detached Re- tina. and Suppurative Hyalitis.) 12. *Eye bursts; (13.) tumor dark and en- crusted; (14.) saneous foetid discharges. I 5. *Pupil dilated and sluggish. 16. Lens opaque. 17. Anterior chamber shallow. 0;?/ez‘/2. 18. Circumscribed part of retina dark and thickened ; later protrudes into the vit- reous, as a yellow-white mass; grows rap- idly. 19. White patches lying partly behind retin- al vessels ; ez'1’e2zZa7', s/zazp/y defined, rzoz‘ striate nor punctate. (e. s. Retinitis ;) me- talic white. (v. s. Cream tint of Retinitis.) 20. Retina may become detached. 21. Cachexia late. PROGNOSIS. Gear/e. Favorable if removedvery early. Fatal unless removed early. Certainly fatal if it returns after removal. Death fro1n exhaustion or cancer of brain? Time three years. SEQUEL/E Sarcoma of brain. Encephalitis. Gen- eralization. TREATMENT. General Measures. *Enucl,eate at the very be- gtrtrttng, clear back to the optic foramen, and apply chloride of zinc paste. ' Homeopathic. Garb .ac. Tx., in water af- ter enucleatrlon. Allopathie. 21 CYCTICERCUS. PATHOLOGY. This entozoon is considered to be the “TREATMENT, , offspring of _the tape-worm in one stage Genera, Measmes_ of lts (zieveloprnent _ Remove by an incision Deposited from the blood under the retina over the seat of the 0 O - e . t 1 . it gives rise to effusion and inflammatory La,§§lf1be‘:1uc1eatiOn may €:lCl'.lO11. be demanded. DIAGNOSIS. I. Vision steadily diminishes. 2. Visual field interrupted where the para- cite is or has been. Op/at/2. 3. Vitreous becomes turbid. 4. At first the spot affected is transparent and the animal may be seen ; later. opaque. 5. Violent inflammation. PROGNOSIS. Unfavorable. SEQUEL/E. Detachment of the retina. Destruction of the eye. NOTE. Tumors of the optic nerve are very rare. They gen- erally cause neuro-retinitis and exophthalinus, Enuclea tion is generally demanded. 22 COMMOTIO RETINPE. ' [ Concussion of the Retz'na.] PATHOLOGY. Shock. TREATMENT. General Measures. ETIOLOGY- I. Blows upon the eye-ball or neigh- . At 0 ‘ e. bormg parts, Re:t_p1n ‘ ' 2. Stroke of lrghtenrng. Bandage- . . . Homaeopathic. DIAGNOSIS. 3*Sudden loss of vision; entire, or A ., 1, 3. only central. . Oglum, 5_ 4. Vision gradually rmproves. B11118- Verat. alb., 5. 5. *Pupil contracted. 6. Pupil resists atropine. Opkt/z. 7. Gray dots appear in the retina. coalesce change to white and disappear. PROGNOSIS. Generally favorable. SEQUELE. Rupture of the retina. Panophthalmitis. 23 HYPERPESTHESIA RETINZE. [0cersensiticeness of the retina] Syn. Retinal Asthenopia. PATHOLOGY. Irritation of the ciliary plexus. TREATMENT- ACt1V€ hYP€I‘£I'IIiEl.. General Measures. . . . Atrop'ne. ETIOLOGY. I. Hysteria. 2. Hypochondriasis. Blue gasses. 3. Error of refraction. C°1‘PectYef1'a<>fi0n- . . Improve general health. 4. Catamemal disorders. Asswre pat/wntof f/M1,], -,-6- covery! 5' Confinrnent _ , _ _ Moderate light, but note. 6. General nervous irritability. dark room. ‘X’ ' Accustom eyes gradually 7. Prolonged use of the eyes. especially on to daylight bright Ob_]€CtS. I-lommopathlc. 8. Flash of lightening. 12 16 18 .ACOD.., , s ' 9- Accldent Ars.,12,16,17,18,21. . . *Bell., 3, 6,10, 12, ,16,17, DIAGNOSIS. IO. Occurs in young delicate females. 18,21. * . . . . . China, 4‘ 5. 6, 171 I I. Retinal impressions retained a long time. won” 12,13’18, I2. *Ser/ere photop/zohza. *Gels..3,5,10, 21. 0 . ° Ht‘/p., 79 141- I3. Photopsies, stars, (14.) colored rings. HYOS_ 15. Can produce ph'otopsies by pressing on *Isn-.1.10.12.18.16- J abor., 7, 11. H 09 as the eye. (2/. s. Amaurosis.) ,,Mam,Ot_, L 4, 6, 10,12, 16, I6. 'X'C1liary neuralgia. 17.22- ., - Merc., 2, 18, 17, 18. 17. ' Lachrymation. Nah mm.“ ,,,10,12_ 18. Spasmodic closure of eye. *I\]181x v-.1.2.6.12. 16. 17. 19. *Vision good. Can read the smallest phO'S., 6. 7, 10, 18, 19, 20, rint in dim li ht r ith . 23- p g 0 W blue glasses Puls.,1, 2,3, 4, 8, 10, 12,14. 2o. *Field usually contracted concentrically. 16,17. (6. f. Amaurosis.) gglts-.89.12.17.18. 2 I. Pupils dilated. Scut. 22. *No ophthalmoscopic changes. Se1I,’7'* 1’ 3’ 4’ 6* 7’ 1°’ 13’ 14’ Suh, 7, 12,16, 17. PROGNOSIS. Favorable. Zinc., 12,13,14. Allopathic. Tonics. Quinia. Zinc and steel. Dry cupping. Blisters. Nora. Snow-blindness and Nyctalopia or day-blindness are forms of Hyperaestlmsia. The former is caused by pro- longed exposure to the bright reflection from the snow. It is characterized by great irritability of the eyes with conjunctival hyperwmia, pain and photophobia. Dura- tion, a few hours to several months. Treatment as above. 4 24_ ANZESTHESIA RETINE. [See Amblg/opz'a.] PATHOLOGY. Defective inervation and hence di- minished supply of visual purple. ETIOLOGY. 1. Exposure of eyes to extremely bright light for long time. 2. Continued application of eyes under strong artificial light. 3. Rariflcation of air in high altitudes. 4. Smoking. (P) 5. Anaemia. DIAGNOSIS. 6. Phosphenes in those parts of the retina which are insensitive. (1/. 5. Am- aurosis.) 7. *Vision diminished greatly. 8. Haze or cloud before eyes. 9. Impossible to see except in bright light. Io. No ophthalmoscopic changes. PROGNOSIS. Favorable. TREATMENT. General Measures. Rest. Avoid cause. Homuepathw. Agaric. [low], 5, 6, 7, 8. Ars., 5, '7, 8. China, 5, 6, 7, 9. Fer., 5. >*Hep. \high], 1, 6, 7. Ign., 7. J abor. [low], 2, 7, 8, 10. Lyc. [high], 6, 7, 9. Zinc. Allopathic. “ Blisters. Artificial leech. Injections of strychnia. 25 AMBLYOPIA. [(/trllecl ArImm'0sz's, when totally bZe'nd.] PATHOLOGY Often entirely functional. Diffuse in_ TREATMENT- terstitial sclerosis of neuroglia. General Measures. - - Stop tobacco and liquors, ETIOLOGY. I. High degrees of ametropia. short! Nutritious food. 2. Non-use from cicatrix or squint. Plenty Of sleep 3. Passive congestion of the nerve, brain, Exercise body and eye, - Tinted glasses. llver OT uterus; Operate for squint or cat- 4. Disturbance of nervous function. aract in children. - Turkish or sea baths. 5. Exposure to wet or cold. 6. Xf£I§1?. Galvanism. 7. *Tobacco. 8. co 0. _ Homrnonalhrc. 9. Lead. IO. Mercury. . . Arg. 11. 11. Quinine. 12. Si ver._ ,,Ars, 7, 23, 24, 27, 28, 31, I 3. Neuralgia. 14. Hysteria. $341,137,182. )9 31 38 . e.,,,2, ,8"',. I5. Gestation. 16. Confinment. B,.y_,5131_3,,. ‘ I7. Lactation. I8. Haemorrhage *C31;in2e;~8-17,18-23.38.24. I9. Heredity. 20. Sexual excesses. 1.-e,.f,2?'_ 21. Fright. 22. Shock. §iI;—84~20-2‘3~31- . 1‘., . 23. Anaemia. 24. Fevers. Nux Va 3, 4, 75 8, 20‘ 26, 2 5. Amenorrhoea. 26. Indigestion. §77r4129r 31» 32» 34» 34, 35- . *Phos., 3, 4, 7, 18, 2O, 2*, DIAGNOSIS. 27. Men over thirty-five who have 28,31’;-,4,,40_ ‘ been great smokers and drinkers. Puls-~ 1» 3-4414-23-22 26- . 31, 34, 37. 28. Onset slow, or (29.) sudden, according to Scut"22_ cause. S113l~.,Z,)19, 26, 28,31, 64, 36, . . 1, . 30. Both eyes. [One if from squint] 31. Vision impaired or suspended. . . - - ~ Opium. Morph. 32. Central vision poor even in bright light. Strychnine[injectiom] (2/. 5. Glaucoma and R. pigment.) Bromide of potash. .X. - - - - Tonics. Wine. 33. Pinhole -test does not improve vision. Stmycllnine and iron. 34. Vision misty and hazy. [Alcoholism] Artifieialleech. 35. ""Central scotoma for colors. [Tobacco.] 36. Weakness of accommodation. [Alcohol.] 37. Dry irritable conjunctina. [Tobacco] 38. Pupil moderately dilated, (contracted in uraemia.) 39. Pupil reacts to light. [Intermittents.] Allopathic. Op/zz‘/2. Nothing important. 40. Disc hyperaemic, or (4i.) pale or normal. PROGNOSIS. Extremely guarded if from optic neuritis. Better if onset was sudden, and pupil reacts to light. May recover after years of blindness. 26 . ACHROMATOPSIA. [S3/n. (blor Blimlness.] PATHOLOGY. Unknown. TREATMENT. General Measures. ETIULOGY. I. Over-use of color sense. 2. Myelitis. 6, Syphilis‘ Education. (ll) 2. Neuritis. 7. Detached retina. “°"‘°e°Pa"“°- 4. Retinitis. 8. Pregnancy. Con., 16. (i) 5- Atrophy 9. Heredity. Sant'(?)A 1 llopa hic. DIAGN08lS- IO. Occurs more often in males than “ T _ females. l\Othmg' II. In lower classes of society, and in the Socity of Friends. 12. Congenital, or (13) acquired. 14. Both eyes. 15. Inability to distinguish red from green. 16. Inability to distinguish any color, rare. I7. No disturbance of other functions of the eye. I8. Central scotoma for red. [Tobacco] PROGNOSIS. Unfavorable. 27 HEMERALOPIA. [Syn Nright Blz'ndness.] mruoroev. '><'FunctiOna1_ TREATMENT. Torpor of the retina. General Measures. lnsufficient blood supply. *Build up the general Impoverished blood. health- "‘N0urishing diet. ETIDLOGY. 1. Prolonged exposure to glaring Protect eyes against . bright light. llght. Dark smoked glasses. ‘ - Rest. 2‘ DebI11tY' Change of climate. 3. Sclerosis of lrver. *Cod-liver oil. 4- SCuTVY- Homuaopaihlc. - - - Bell. DIAGNOSIS. 5. Frequently occurs with 1nterm1t- China”, 4‘ 5‘ 6‘ 71 10_ 1,-,_ tent fever in ill-fed, ill-housed peasants. Hyos. 6. Sailors with scurvy, in tropics. ‘i;’§’(;:2' 9‘1°'13‘ 7. Both eyes always affected. Tobac. 8. After-images, but no phosphenes. Allopalhlc. 9. *Vision perfectly good in strong light. Tonics_ 10. *Can see scarcely at all in dim light, as a Quinine. . . . . s 1. cloud seems to render Ob_]€CtS 1nd1st1nct tee II. Time of day makes no difference. I2. *No limitations of field. (7/. s. R. pigment.) I 3. There may be scotomata. 14. Color-blind for red, blue and violet in dim light. 15. *Impairment of accommodation. 16. lnsufficiency. I7. Conjunctivitis. I8. *Silver-gray scaly I patch of epithelium, at outer portion of ocular conjunctiva. Op/12‘/1. Nothing. PROGNOSIS. Favorable under good circumstances. Tendency to recurrence. Unfavorable if with hepatic cirrhosis. 28 HEMIOPIA. [Half-sight] Syn. Hernianopsia. PATHOLOGY. Lesion at or beyond the commissure, TREATMENT- [both eyes affected.] General Measures. Lesion in front of commissure, [one eye Discover cause, and rem- afr'ected.] edy “- Lesion at commissure, [temporal half of Homwopathia each field lost.] *Aurum, 2,10. Lesion behind corpora geniculata, [nasal g?1g°'§b’9' half of each field lost.] Ge1s., 7,10. . . . *L'th. . ' hth lf t- Lesion on opposite side of head from want- i:1g_]° [mg *1 wan ing field. *Lyc0p, [di_tto.] . M h. 1.9. Lesion unknown when upper or lower M§),I§ao,s_11,,_ half is lost. Plumb-. 9. . Rhus.,9. Functional. (P) Sepqgv _ _ _ Stram., 9. ETIOLOGY. 1. Distubance of circulation. Bov.Lob. Nat. mur. 2. Syphilitic or tubercular disease of brain. Qulma Vloaodor 3. Intra-cranial tumors. 4. Apoplectic clots. Allopalhic. DIAGNOSIS. 5. One, or both eyes. 7. Onset sudden, [apoplexy,] or (8.) slow, [tumor.] ’ 9. *Loss of lateral half of field. 10. Upper, or (11.) lower half very rarely lost. Op/ah. Nothing unless it be, (12.) contraction of arteries, or <13.) hyperaemia of disc. 14. Later atrophic appearance of disc. PROGNOSlS- Grave, especially if both eyes are af- fected. SEQUEL/E. Atrophy of nerve. Death. 29 MALINGERING. [S3/n. Simzelcetion of Blz'ndness.] occumzuce. Nervous hysterical people. Conscripts. Prisoners. Beggars. That one eye, generally the right one, or both eyes, are “defective.” CLAIM. oanzcnow IF omz EYE. 1. Hold narrow ruler midway between eye and page, and it will not interrupt vision. 2. Place a—2o. D. lens before the good eye, and he can still read. 3. Can see better with stereoscope. 4. Prism base down, causes double images. 5. Prism base in, or out, causes change of axes when removed. IF BOTH EYES. 6. Pupil responds to light. (7/. s. Amaurosis, unless from intermittents.) 7. Pupils may be excessively dilated by atropine. (Amaurosis moderately.) 8. Pupils may not be dilated at all. Amaurosis.) 9. Lids close to protect from pretended blow. (7/. 5. TREATMENT. Do not let patient suspect your motive in making the examination, 10 ut cause him to think that the desire is only to de- termine the cause of his misfortune. 30 INDEX. A _ Con 0estion assiv 4 ‘ D , p‘ I- 0, - . . . . - - . . . . . . . . . - . . . . . . . . . . . .. .6 ‘ Accolnodatlon impaired, . . . . . . . 8 25 27 CgE§I~iInIIeI‘IiI1IiIIIII I I I I I I I I I I I I I I I IIIIIIII Accomodation, Spasnis of, . . . . . . .8I CornIea.IIIfoiI(3I3>~Ii1IIbIilIdié;IIII .2E311111I1‘;);Il2l,l'.‘().p-Slit, . . . . . . . . . . . . . . . . . . gorneai irisenbsible, . . . . . .I.I .I . I . After images, ................ I I. .............. . .I.27 I CIvIfIcI’IiiIcIIIIel'IcIu(SIIII I I I I I I I I I I I I I I I I II ‘I I‘I>5I Alcoholism, . . . . . . . . . . . . .4, 15, 16, 17 25 “ I I I I I I ' ' I' ' ' ' ' ' ' ' ‘III III‘ Amblyopia, . . . . . . . . . . . . . . . . . . . . . . . .I.25. D Amaurosis, . . . . . . . . . . . . . . . . . . . . . . . .25. Day "blindness \ - - - - - - - ‘)3 Amotio retinae,..... . . . . . . . . . . . . ..19. Deblmys ~ - - - - - - - - - -. Anaem1a,._L . . . . . . . . . . . . . . . . . . . . .5, 24, 25. Dgaf-Inulisln, - - - - - - - - - - . - - I12I Anaesthesia retinae . . . . . . . . . . . . . . . . . .24. Dental I16111“(l-lgiil. - ~ - -- - - - - - -I -I ignterlor Clprmber, $11-,1]1()w‘, _ _ _ 20, 12_ Degeneration of coats of vessels,. . . . . .9. A§101l1;1]SeI(1:1ti<(); Ct1l(1)psaorta., . . . . . . . . . . . . . . . Ilgetilcltlment of the retina, . . . . . .19, 21, 26. ., _ , , _ , _ _ , _ _ __ , _ raaees, . . . . . . . . . . . . . . . . . . . . . . . . .. Apoplcxia retinae . . . . . . . . . . . . . . . . . . . . .5. Disc a$1'OP11iC, - - - - - - - - - - - - - - - - 1% Arteries as white lines , . . . . .. ...8, 10, 18. Disc, bluisll-.s‘1'ay,~ - --- - ... . . . I I10I 11I Arteries diminished in size, 6, 8, 9, 10 11 Disc, bluish green . . . . . I I I l'7I A _ [12,14,15,18j<,>,8i Disc choked,. . . . . . . I. I.I. I. I. .I .I I I I .Ii5.I Iellélraglgpd, _ , , _ _ _ _ _ _ _ _ _ _ , _ qoncave saucer-lil\c.. . . . . . . . . . . .17. _ , . . . . . . . . . . . . . . »c,‘l'a, . . . . . . . . . . . . . . .. s 2 " ArteI'}6s bloodless, . . . . . . . . . . . . . . . . .6 15.‘ Disc §‘1'e:sI’IiS11-Wllile ---- - - I I I III Arteries veiled, , _ . , , _ _ , _ _ _ _ _ _ _ _ , _ _ _I_ _7_ Disc hyperazmic, . . . . . . . I.9I I1I1I I I2 I Arteries without light streak.. . . . . . 19. Disc 1@V61, - - - - - - - . - . . . I I I l7I Astigmatism, . . . . . . . . . . . . . . . . . . . . . . .8. DiS0, 116W VGSSP-18 OH. . . . .. . I I I I I I I 8 Atheroma of large vessels, _ _ _ _ _1g, Disc ccdematous, . . . . . . . . . . . I I I Atrophy of the optic nerve, . . . . . . .17, 26. Disc opaque, - - - - - - - - - - - - - - - - - - - I -I1I5 I17: Attacks very sudden, _ _ _ _ _ _ _ , _ _ _ _ _ _ _9. Disc, outer half Wlllt6.. . . .. . . .. . . .. . .I.l'7. B Disc outlines indistinct, .13, 4, 7, 8, 9, 10. ‘ _ ' _ _ 1,18, 14, 15,17. ]B3il.E}‘i:];l’l‘(é%Tr(2:l.1]€)3IS)‘(3‘€)..l.8LlfI:C1hlll6(1, . . . . . . . . ougllines li‘11'€g‘lll211‘, . _ . . . . . . . . . . . .8, 1'7. Black lines in place of vessels. . . . . Disii IdiitlIiIl.dI;I:l S“vIlIrI(>IZ>IlIlIrII’I’IIIIIeIII I I I I I I I IIZI Black spots in field of vision, .... .. Disc pale II IIII III I Bhndness, . .' . . . . . . . . . . . . . . . .6, T, 12, 17. Disc pa1'iIIcolIoIrIeIdI I I I I I I I I I I I I I? Bhndness, Simulation of, . . . . .29. DiscIred I I ITI I1llI ’”I Bhnd spot enlarged, . . . . . . . 15. Disc striIaIted I I I I I I I I I II I I 14:I 15I Blood. altel'atio1ls in quality of, . . . .9. Disc swollcnI I I I I I I I I I I I I 9I 11IlI 1'”I Blood, irregular movements of, . . . . . .18. Disc swollenI(I2I'eatIl I I I I I I I I I I I I II Bram, diseases of. . . . . . . .7 8 25 28 Disc sw ll I/l'O'l I I I I I I I I I I I I I I I I II5I Brights disease , 8, IO, 18. _ . 0 en s 1,7 it y,... . . . . . . . . . . .10, 11. ,.... . . . . .., , . Dlseswollenatmargm,.... O Disc waxy, . . . . . . . . . . . . . . . . u ' . . . . . . . .12. Cachexia, ‘ _ . _ . . I I . . . 90 Disc white opaque, . . . . . . . . . . . . . . .15, 17. Cellulitis, _ . _ . . ' ‘ . . . _ ‘ _ I . _ _ - - - - - - - -1~r;- Disc ulnte, transparent, . . . . . . . . . . . . . .6. Children undm twelve, - _ . _ i ' D 0- - - - 20- Disc yellow. . . . . . . . . 1 . . . . . . . . . . . . . . .12. Choked Disc, . _ _ _ - - -15- Discharge saneous lcetlcl. . . . . . . . . . . .20. Chromopsies, . . . . . . . . . . . . . . I142 E 8€g;i)(;g;itl;ltl1l\):).S1; . . . . . . ll). 1li11rr,b01Zz'swt of the centml (m5e7',?/, . . .6, 9, 18. Choroidial ring distinct, . . . . . . . . El1I(,'I;]I)Il:t(IlI(§IlI(IlI IIIIIIIIOIIIIIIIIYI I I I I I I I I I I -I I I IOIIIII Choroldal vessels visible, . . . . . . . .12. Epilepsy I I I I I I I I I I I I I I I I I I I I I I I Ciliary neuralgia, . . . . . . . . . . . . . . . . . Erysipel:isI I I I I I I I I I I I I I I I I I I I I I I I I T I IIII Cloud surrounded with halo,. . . . . . . .19. Errors of lI'eIflI'actIiIoIllI I I I I I I I I ‘III Color‘-blindness . . . . . . . . . . . .15, 16, 26, 27. Exophthalmus II I II I I I II I ’ 98’ 35 . - . ‘ . .27. s , . . . . . . . . . . . . . . . . - . .~ , I15. 0 orec I‘1Il"'-9, sees,. . . . . . . . .23. E: . ' ‘I I I I‘ I I I I I I I I I I I I I I II I II Color field Feclucecl concentrically, . . . . .15. I I I I I I I I I I I I I I I g((.:l;)1:I3)(‘i;(1'):.')O1I‘l(:'i\i,l]ti)GDtl'%ll, . . . . . . . . . . . . . Exudations (‘Z05-eu to disc, . . . . . . . . :14: Concussion of H38. . . . . %XLl(1l)‘clllllOllS at distance from (lisC, . . . . . .10. Congenital . . . . . . . . . . . .I.I . I I I I I5§fIL:.IbILITl‘:t;IIIIy WIIIICI I I I I I I I I I I I I I I I I.>I6I Conj_uncti_va., irritable,. . . . . . . .25. Eye looks IcIxI[IcII'ilIqll I I I I I I I I I I I IIIOI Congunctlva, silver gray patch on ‘>7 E T: 0 9 HI - I -3’ ' ' ' ' ' ',' L ' ‘ ' ' ' '7' . .,...T.). . .~‘. _JT’., 1,... , v, I, , , ,17,2III Congestion, actiic, . . . . . . .3 [26, 27) 28, 31 F Females, . . . . . . . . . . . . . . . . . . . . . . . . . . . . .23. Fevers . . . . . . . . . . . . . . . . . . . .16, 17, 18, 25. Field contracted . . . . . . . . . . . . . . . . . .14, 19. Field contracted concentrically. .12, 1'7, Field t'mpai"recl in one part. . . . .19, 21, 28. Field limitations begin on temporal s1 e . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 7 Field not limited . . . . . . . . . . . . . .10, 11, 27. Fright . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . Fovea bright red . . . . . . . . . . . . . . . . . .18, 19. Fundus marbled . . . . . . . . . . . . . . . . . . . . .12. Fundus hazy . . . . . . . . . . . . . . . . . . . . . . . . .10. Fundus reflex reduced . . . . . . . . . . . . . . . . .7. Funrlus, shadow of detached part on.. .19. Fundus striated . . . . . . . . . . . . . . . . . . .10, 11. F undus uniformly reddened . . . . . . . . . . . Fundus veiled . . . . . . . . . . . . . . . . . . . . . . . .7. Funclue yellow . . . . . . . . . . . . . .. . . . . .12, 13. Fzlngztsliremrttode-<: . . . . . . . . _ . . . . . . . . . . . .20. . . D . ' G Gestation. ; . . . . . . . . . . . . . . . . . . . . . . . .25. Glaucoma . . . . . . . . . . . . . . . .‘ . . . . . . . . .4. Glioma reti_n_ze . . . . . .. p . . . . . . . . 20. Gray dot-s_ appear in 1'elin-a. . . . . . . .22. Gray dots inteI'iiiixed with white dots . .11. Gray ,patch'es'_'fring'ed with black zone. .11, Gray patcliessliowing choroidal vessels, 11. ~ H Iltemorrhage into the 7’6Z"l7Z(t, .5.. 7 . 8, 9, 10, ‘[11, 13,14,115, 16, 18, 19 Haemorrliagic di-atliesis,. . . : . . . . .' . . . . . . .5. Hznmorrhagie effusions, pale yellow, . . . I-Iaemorrliage, general, . . . . . . . . . . '. . . Haemorrhages, small, . . . . . . . . . . . . . . . .18.‘ Half-sight, . . . . . Q . . . . . . . . . . . . . . . . . . . . .28.‘ Haze or cloud before eyes, . . . . . . . .24. Haziness of retina, . . . . . . . . . . . . .10, 13, 16. Haze at center of fundus . . . . . . . . . . . . . .11. Headaches, . . . . . . . . . . . . . . . . . . . . . . .14, 16. Heart, disease of, . . . . . . . . . . . . . .4, 5, 7, 10. Iiemeralopia, . . . . . . . . . . . . . . . . . . . . . . . .27. Hemianopsia, . . . . . . . . . . . . . . . . . . . . .15, 28. Hemiopia, . . . . . . . . . . . . . . . . . . . . . . . . . . . .28. Heredity. . - .. . . . . . . .12, 14, P7, 20, 26. I-lyperzemia, . . . . . . . . . . . . . . . . . . . . . . . . . .8. Hyperaemia activa, . . . . . . . . . . . . . . . . .3, Hypercemia passiva, . . . . . . .4, 7. Hyperaesthesia retinas, . . . . . . ' ' ‘3, 23. Hypermetropia, . . . . . . . . . . . . . . . . .3, 10. Hypocliondriasis, . . . . . . . . . . . . . . . . . . . . Hysteria, . . . . . . . . . . . . . . . . . . . . . . . . .23, 25. N‘ Nervous irritability . . . . . . . . . . . . . . .23, 25. N euralgia . . . . . . . . . . . . . . . . . . . . . . . . . . . Neuritis optica . . . . . . . . . . . . . . . . . . . .15, 26. Neuritis optiea descenclens . . . . . . . . . . . .14. N euritis optica orbitalis . . . . . . . . . . . . . .16. Neuro-retinitis . . . . . . . . . . . . . . . . . . . . . . .14. N eurosisof the sympathetic . . . . . . . . . . .14. Night-blindness . . . . . . . . . . . . . . . . . . .12, 27 . ~Nyctalopia . . . . . . . . . . . . . . . . . . . . . . . . . Nystagamus . . . . . . . . . . . . . . . . . . . . . . . . .12. Q Objects fringed with red, violet or blue, 19. Occurs in young delicate females . . . . . .23. Oedema of retina,. . . . . . . . . . . . . . .4, 7. Onset insidious . . . . . . . . . . . . . . . . . . . . . . .16. Onset slow . . . . . . . . . . . . . . . . . . . . . . .25, 28. Onset sudden . . . . . . . . . . . . . .16, 19, 25, 28. Opacity on lens . . . . . . . . . . . . . . . . . . . . . .12. Opaque bluish mass behind lens . . . . . .19. Ophthalmoscopic charges, No. 23, 24, 25, [26, 27, 28, 29. Optic neuritis . . . . . . . . . . . . . . . . . . . . . . . .17. Orbital lesion . . . . . . . . . . . . . . . . . . . . . . , .15. Oversensitiveness of the retina. . .\.. . . .23. P Pain, intense . . . . . . . . . . . . . . . . . . . . . . . . .20. Pain, no . . . . . . . . . . . . . . . . . . . . . ..7, 16, 19. Palpebral fissure obliti-1-alcrl . . . . . . . . . . .20. Papilitis . . . . . . . . . . . . . . . . . . . . . . . . . . . . .15. Panophthalmitis . . . . . . . . . . . . . . .21, 22. Patches, gray, . . . . . . . . . . . . . . . . . . . . . .8. Patches of pigment . . . . . . .' . . . . . . . . . . . .12. Patches, white, . . . . . . . . . . . . . . . . . . .10, 20. Peasants, ill—fed . . . . . . . . . . . . . . . . . . . . . .27. Perivascularit-is . . . . . . . . . . . . . . . . . . . . . .10. Phosphenes . . . . . . . . . 11, 15, 19, 24. Phosphenes no . . . . . . . . . . . . . . . . . . . . . . . .27. Phosphenes impossible to excite . . . . . . .18. Photophobia, . . . . . . . . . . . . . . . . . . . . . .3, 23. Photophobia, no . . . . . . . . . . . . . . . . . . . .4, 7. Photopsies . . . . . . . . . . . . . . . . . . . .14, 19, 23. Pigmented spots. -. . . . . '. . . . .‘ . . . . . .5, 9, 12. Pregnancy .. . . .‘ . . . . . . . . . . . ..7,-10, 18, 26-. Prolonged use of the eyes. . . . . . . . .23. Pupil contracted . . . . . . . . . . . . . .12, 22, 29. Pupils dilated . . . . . .\.- .. .16,23, 25,29. Pupils dilated and sluggish .. . . .7, 14, 20. Pupil reacts to light . . . . . . . . . . . . . . . . . .25. Pupil resists atropine . . . . . . . . . . . . . . . . .22. Pulsations venous . . . . . . . . . . . . . . . ' . . . . . .4. Pressure does not cause pulsations. . . . .18. Pulse -very rapid . . . . . . . . . . . . . . . . . . . . . .6 Q Quinine . . . . . . . . . . . . . . . . . . . . . . . . . .25. R Rarification of air . . . . . . . . . . . . . . . . . . . Red. everything seems . . . . . . . . . . . . . . .5.‘ Red letters look. . . . . . . . . . . . . . . . . . . . .14. Red-green blindness . . . . . . . .15, 17, 26, 27. Redness, subconjunctival . . . . . . . . . . . . Reflex bright yellow... . . .. . . . . . . . .20. Reflex reduced . . . . . . . . . . . . . . . . . . . . . .7. Retina part of dark . . . . . . . . . . . . . . . . . . .20. Retina, dark lines in, . . . . . . . . . . . . . . . . . .9. Retina detached . . . . . . . . . . . . . . . . 20. Retina, gray dots in, . . . . . . . . . . . . Retina hazy . . . . . . . . . . . . . . . . . . . . . .14 ,16. Retinahyperaemic.......... . .. ...11. Retina oedernatous . . . . . . . . . . . . . . . . . 4. Retina, opaque patches in,. . . . . .21. Retina, rupture of, . . . . . . . . . . . . . . . . . .22. Retina swollen . . . . . . . . . . . . . . . . . . . . .7, 14. Retina torpid . . . . . . . . . . . . . . . . . . . . . . . .12. Retina watery . . . . . . . . . . . . . . . . . . . . . . . . .9. Retinal asthenopia . . . . . . . . . . . . . . . . . . .23. Retinal impressions retained . . . . . . . . . . .23. Retinitis . . . . . . . . . . . . . . . . . . . . . 17, 26. Retinitis albuminuriea . . . . . . . . . . . . . . . .19. Retinitis apoplectiea . . . . . . . . . . . . . . . . . . .9. Retinitis exudativa . . . . . . . . . . . . . . . . . . . . .8. Retinitis lcucaemica . . . . . . . . . . . . . . . . . . .13. Retinitis pigmentosa . . . . . . . . . . . . . . . . . .12. Retinitis scrosa . . . . . . . . . . . . . . . . . . . . . . .19. Retinitis syphilitica . . . . . . . . . . . . . . . . . . .11 Retro-bulbar optic neuritis . . . . . . . . . . . .16. Rheumatism . . . . . . . . . . . . . . . . . . . . ..16, 17. 32 S - ‘U’ Sarcoma . . . . . . . . . . . . . . . . . . . . . . . . . . . . .20. Urzemia, . . . . . . . . . . . . . . . . . . . . . . . . . . . . .25. Scarlatina . . . . . . . . . . . .‘ . . . . . . . . . . . . . . .10. Uterus, disease of,. . . . . . . . . . . . . . .4. '7, 25. Scintillations . . . . . . . . . . . . . . . . . . . . . . . . .11. V ;, Sclero-choroiditis . . . . . . . . . . . . . . . . . . . . .19. _ ‘ Scotoma, _ _ _ , _ _ _ _ _ , . _ _ _8, 9) 13, 18, 21, 27_ Venous pulsations, . . . . . . . . . . . . . . . . . . . .4. Scotoma central . . . . . . . . . . . . . . ..5, 16, 22. Vessels a»tteI111_a-ted_, - - - - - - - - - - - --16, 17, 18- Scotolna; central for C0101-S _ _ _ _ _ . _ _ _ . _ Vessels as Whlte 1111.68, . . ... . . .' . . . . . . . . Scotoma, centra1 for red _ _ _ _ _ _ _ _ _ _ _ . _ _ _26_ Vessels bordered With wlnte l1nes,. . . ...1'7. Scotomata appear and disappear . . . . . . .11. Vessels da/"I6, Crooked, 11I1d\11a't1n8‘,- -19- Scurvy . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .27. Vessels, degenemtloll Ofw - - - - - - - -5. 10- Serous efiusion . . . . . . . . . . . . . . . .10, 13, 14. %GSSQ]S