id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_u3qvh3es3nbipgsrz6dsngq5bi	Soumya Sinha Roy	Bak/bax-dependent Apoptotic Signaling In Vdac2-/- Cells	2009	1	.pdf	application/pdf	1502	103	48	Regulation of Mitochondrial Motility by Milton-like Proteins, OIP106 and Regulation of Mitochondrial Motility by Milton-like Proteins, OIP106 and GRIF1 on mitochondrial motility and its inhibition by agonist-induced cytoplasmic [Ca2þ] ([Ca2þ]c) signals in H9c2 cells. In OIP106 and GRIF1 overexpressing cells, elongated mitochondria with varied degree of aggregation were Mitochondrial contribution to cell signaling and function relies on the association and local communication of mitochondria with the ER and the plasma the kinetics and short-term effects of the linkage formation, we have now devised fluorescent protein pairs targeted to the OMM and ER or PM containing formation between the ER and mitochondria was followed by a decrease in mitochondrial motility (>40% in 15 min) and by sensitization of mitochondria to MEFs lack tBid induced complete cyto c release and loss of MEFs and also supported the tBid-induced cyto c release. Regulation of Mitochondrial Motility by Milton-like Proteins, OIP106 and GRIF1	./cache/work_u3qvh3es3nbipgsrz6dsngq5bi.pdf	./txt/work_u3qvh3es3nbipgsrz6dsngq5bi.txt