id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_ncb3aud2g5ebjlpiiax275hz7e	Nima Niknejad	Targeting Mcl-1 and Bak as a Therapeutic Tool to Selectively Induce Apoptosis in Hepaptocellualr Carcinoma	2013	2	.pdf	application/pdf	3020	237	49	The voltage dependent anion channel (VDAC) is involved in regulation of metabolite flux across the mitochondrial outer membrane (MOM). Thus we propose a novel mechanism of mitochondrial respiration control in cancer cells through the competition between HXK2 and tubulin for VDAC binding. found that specifically the VDAC2 isoform is needed for tBid-induced cytochrome c release, due to its role in supporting Bak insertion to the OMM Thus, OMM function depends on mitochondrial fusion Here we applied mitochondria matrix-targeted DsRed and photoactivable GFP to study mitochondrial fusion in rat FDB SM fibers. Novel Mechanism of Mitochondrial Respiration Control through Competition between Hexokinase-2 and Tubulin for VDAC Binding Reprogramming of Mitochondrial Ca2+ Handling in MICU1-Deficient HeLa Cells MICU1-dependent Threshold and Cooperativity of Mitochondrial Ca2+ Uptake in the Liver Which Domain of VDAC2 is Necessary for Bak Insertion to the Outer Mitochondrial Membrane and tBid Induced Cytochrome C Re ...	./cache/work_ncb3aud2g5ebjlpiiax275hz7e.pdf	./txt/work_ncb3aud2g5ebjlpiiax275hz7e.txt