id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_icsipe3itza57oc7ahgvt4fxwi	György Csordás	MICU1-dependent Threshold and Cooperativity of Mitochondrial Ca2+ Uptake in the Liver	2013	1	.pdf	application/pdf	1581	129	52	The voltage dependent anion channel (VDAC) is involved in regulation of metabolite flux across the mitochondrial outer membrane (MOM). G6P, which is known to cause HXK2 detachment from the MOM, fully reverses the inhibition of tubulin-VDAC binding. Thus we propose a novel mechanism of mitochondrial respiration control in cancer cells through the competition between HXK2 and tubulin for VDAC binding. matrix [Ca2þ] response in permeabilized shMICU1 cells. MICU1-dependent Threshold and Cooperativity of Mitochondrial Ca of MICU1 in HeLa cells causes lower threshold and decreased cooperativity of Silencing of MICU1 caused a leftward-shifted dose response and decreased cooperativity of mitochondrial Novel Mechanism of Mitochondrial Respiration Control through Competition between Hexokinase-2 and Tubulin for VDAC Binding Reprogramming of Mitochondrial Ca2+ Handling in MICU1-Deficient HeLa Cells MICU1-dependent Threshold and Cooperativity of Mitochondrial Ca2+ Uptake in the Liver MICU1-dependent Threshold and Cooperativity of Mitochondrial Ca2+ Uptake in the Liver	./cache/work_icsipe3itza57oc7ahgvt4fxwi.pdf	./txt/work_icsipe3itza57oc7ahgvt4fxwi.txt