id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_3phhi5olf5ehbnyeoksqmsdscu	Laurent M. Dejean	Bcl-xL Increases Bax Mitochondrial Localization and Activation in Non-Apoptotic Cells	2012	1	.pdf	application/pdf	1498	112	49	We have recently found that specifically the VDAC2 isoform is needed for tBid-induced cytochrome c release, In this study, we evaluated and compared the biophysical properties of mitochondrial Ca2þ uptake in different mouse tissues. from mouse heart, skeletal muscle, liver, kidney, spleen and brown adipose tissue were patch clamped in the whole-mitoplast configuration. Bcl-xL Increases Bax Mitochondrial Localization and Activation in NonApoptotic Cells We report that, in non-apoptotic pro-lymphocyte cells, Bcl-xL induced an increase of the mitochondrial localization of Bax up to the level found in apoptotic cells. mutant of Bcl-xL, that lost its capacity to inhibit Bax-induced release of cytochrome c, still increased the mitochondrial localization of wild-type Bax, thus The Isoform Specific N Terminus of VDAC2 is Dispensable for tBid Induced Cytochrome C Release Patch-Clamp Analysis of Mitochondrial Ca2+ Uniporter in Different Tissues Bcl-xL Increases Bax Mitochondrial Localization and Activation in Non-Apoptotic Cells Bcl-xL Increases Bax Mitochondrial Localization and Activation in Non-Apoptotic Cells	./cache/work_3phhi5olf5ehbnyeoksqmsdscu.pdf	./txt/work_3phhi5olf5ehbnyeoksqmsdscu.txt