id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_kzg7dwuxyfhatn3bus46erdaue	Laura E. Hays	A Molecular, Genetic, and Diagnostic Spotlight on Fanconi Anemia	2012	3	.pdf	application/pdf	1472	115	56	Hays,1 Stefan Meyer,2 and Henri J. variable clinical presentation, is based on increased chromosomal breakage in response to DNA crosslinking agents. heterodimer then functionally interacts with downstream FA proteins (FANCD1/BRCA2, FANCJ/BRIP1, patients, ERCC1 interacts with FANCP/SLX4 and functions in interstrand crosslink repair [5], the critical type of DNA molecular aspects of this disease, new model systems, and stalling is detected by the FANCM sensor complex and triggers the activation of the Rad3-related protein kinase ATR, resulting in the reviews aspects of protein interactions and function in the FA the United States-based Fanconi Anemia Research Fund (FARF: http://www.fanconi.org.uk/) has been responsible Anemia Aid Association: http://www.fanconi.de/), has been P. Alter, "Cancer incidence in persons with Fanconi anemia," Blood, vol. C. West, "DNA interstrand crosslink repair FA Patient Support Groups by Bob Dalgleish, Fanconi Hope, UK. FA Patient Support Groups by Bob Dalgleish, Fanconi Hope, UK.	./cache/work_kzg7dwuxyfhatn3bus46erdaue.pdf	./txt/work_kzg7dwuxyfhatn3bus46erdaue.txt