id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_agpvyzmp7fcjvjbrnx366n3wky	J G Mulle	Dense SNP association study for bipolar I disorder on chromosome 18p11 suggests two loci with excess paternal transmission	2006	9	.pdf	application/pdf	6567	617	58	Dense SNP association study for bipolar I disorder on number of complex disease phenotypes, including bipolar disorder. linkage on chromosome 18 is modified when allelic parent-of-origin is accommodated in the chromosome 18p11 and investigate this parent-of-origin hypothesis in an association context. test analysis revealed no statistically significant association to SNPs or haplotypes within this Keywords: bipolar disorder; genetic association; 18p11; psychiatric genetics; parent of origin; informative SNPs, and further investigate the parentof-origin effect observed in our linkage study. Table 1 Candidate genes studied for bipolar disorder on chromosome 18p11 peak linkage region of paternal allele sharing previously reported in this population9 (Figure 2). GNAL has previously been investigated for parent-oforigin effects in bipolar disorder, and no association Evidence for linkage of bipolar disorder to chromosome 18 with Dense SNP association study for bipolar I disorder on chromosome 18p11 suggests two loci with excess paternal transmission	./cache/work_agpvyzmp7fcjvjbrnx366n3wky.pdf	./txt/work_agpvyzmp7fcjvjbrnx366n3wky.txt