id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-006034-xfyavk3m	Gil-Cruz, Cristina	Fibroblastic reticular cells regulate intestinal inflammation via IL-15-mediated control of group 1 ILCs	2016-10-31		.txt	text/plain	5437	280	47	Moreover, we found that the lack of innate immunological sensing in Myd88-cKO mice did not affect expression of the Ccl19-Cre transgene in CD31 − PDPN + stromal cells of PPs or mLNs (Fig. 1c,d) . Likewise, the expression of other canonical FRC markers was not altered by the absence of MyD88 in EYFP + cells of PPs or mLNs (Fig. 1g) MyD88 signaling in FRCs controls antiviral ILC1 responses To assess whether an invasive enteric pathogen would substantially alter the activity of FRCs, we infected MyD88-sufficient mice and mice with FRC-specific MyD88 deficiency with mouse hepatitis virus (MHV). The accelerated viral control in Myd88-cKO mice as early as on day 3 after infection suggested that innate antiviral immune cells had been activated by the FRC-specific MyD88 deficiency.	./cache/cord-006034-xfyavk3m.txt	./txt/cord-006034-xfyavk3m.txt