id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-295051-upyar7en	Ahmadian, Elham	Covid‐19 and kidney injury: Pathophysiology and molecular mechanisms	2020-10-06		.txt	text/plain	4859	321	45	The SARS‐CoV‐2‐induced kidney damage is expected to be multifactorial; directly it can infect the kidney podocytes and proximal tubular cells and based on an angiotensin‐converting enzyme 2 (ACE2) pathway it can lead to acute tubular necrosis, protein leakage in Bowman's capsule, collapsing glomerulopathy and mitochondrial impairment. 6, 7 The initial impact might be the direct role of the virus on the renal parenchyma mediated by activating the angiotensin-converting enzyme 2 (ACE2), which functions as a SARS-CoV-2 receptor. 22 Altogether, these reports clarify that kidney cells are targeted by SARS-CoV-2 and new strategies are needed to treat Covid-19 to prevent organ infection and dysfunction. Human kidney is a target for novel severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. Coronavirus disease 19 infection does not result in acute kidney injury: an analysis of 116 hospitalized patients from Wuhan, China Acute kidney injury in SARS-CoV-2 infection: direct effect of virus on kidney proximal tubule cells	./cache/cord-295051-upyar7en.txt	./txt/cord-295051-upyar7en.txt