id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-322728-10m3xscs	Severance, Emily G.	Chapter 29 Role of Immune and Autoimmune Dysfunction in Schizophrenia	2016-12-31		.txt	text/plain	8336	379	34	Because schizophrenia is thought to originate as a result of aberrant neurodevelopment, it is important to note that for a number of these classic immune factors, including complement, MHC, Toll-like receptors, and pentraxins, additional functions in the developing brain are continuously being identified (Benoit & Tenner, 2011; Bialas & Stevens, 2013; Boulanger, 2009; Fourgeaud & Boulanger, 2007; Frodl & Amico, 2014; Garate et al., 2013; Nagyoszi et al., 2010; Pribiag & Stellwagen, 2014; Stephan et al., 2013; Stevens et al., 2007; Trotta, Porro, Calvello, & Panaro, 2014) . Here, we present the case of complement C1q as an example of an immune molecule that is highly active in the developing brain and that is also implicated in schizophrenia-associated gene and environmental studies.	./cache/cord-322728-10m3xscs.txt	./txt/cord-322728-10m3xscs.txt