id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-297132-lhfa9fl5	Aghagoli, Ghazal	Neurological Involvement in COVID-19 and Potential Mechanisms: A Review	2020-07-13		.txt	text/plain	5940	280	36	In this review, we synthesize a range of clinical observations and initial case series describing potential neurologic manifestations of COVID-19 and place these observations in the context of coronavirus neuro-pathophysiology as it may relate to SARS-CoV-2 infection. The novel 2019 coronavirus disease (COVID-19) caused by Severe Acute Respiratory Syndrome coronavirus 2 (SARS-CoV-2) results in a variety of symptoms including fever, cough, and fatigue [1] . The Kawasaki-like syndrome that is now described in young patients following COVID-19 infection and associated with a hyper-inflammatory state is further suggestive of a vascular inflammatory potential of SARS-CoV-2 [48, 49] . Once established in the CNS, SARS-CoV, the virus responsible for Severe Acute Respiratory Syndrome (SARS), has been shown to be capable of inducing rapid transneuronal spread and death of infected neurons in transgenic mice models expressing human ACE2 receptors [63] .	./cache/cord-297132-lhfa9fl5.txt	./txt/cord-297132-lhfa9fl5.txt