id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
cord-331927-b7pfm3i0	Winn, Soe P	Diabetic Ketoacidosis in Coronavirus Disease Patients With Type 2 Diabetes Mellitus	2020-08-14		.txt	text/plain	1772	97	53	Since the emergence of the coronavirus disease (COVID-19) pandemic, we have seen many cases and studies on the underlying pathophysiology of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pneumonia with or without respiratory failure. We have also learned that the angiotensin-converting enzyme receptor is one of the major entry sites of SARS-CoV-2 infection, and it might be one of the causes that predispose patients to DKA. also stated that the human pancreas also expresses ACE2, and therefore, patients with diabetes are more vulnerable to SARS-CoV-2 infection than the general population [9] . In our cases, the transient damage of pancreatic beta-cell function leading to reduced levels of serum C peptide may be the reason for our patients experiencing acute insulin-dependent DKA for a brief period during the course of COVID-19. COVID-19 may cause DKA by increasing insulin requirement induced by ACE2-mediated destruction of pancreatic beta cells, as evidenced by reversible decreased serum C peptide levels or other unexplored mechanisms.	./cache/cord-331927-b7pfm3i0.txt	./txt/cord-331927-b7pfm3i0.txt