Rotenone-induced energy stress decompensated in ventral mesocerebrum is associated with Parkinsonism progression in rats. | Semantic Scholar Skip to search formSkip to main content> Semantic Scholar's Logo Search Sign InCreate Free Account You are currently offline. Some features of the site may not work correctly. DOI:10.3892/ETM.2016.3352 Corpus ID: 40324033Rotenone-induced energy stress decompensated in ventral mesocerebrum is associated with Parkinsonism progression in rats. @article{Bai2016RotenoneinducedES, title={Rotenone-induced energy stress decompensated in ventral mesocerebrum is associated with Parkinsonism progression in rats.}, author={Qunhua Bai and Junlin He and Yong Tang and S. Wang and Jing-fu Qiu and Yang Wang and C. Yu}, journal={Experimental and therapeutic medicine}, year={2016}, volume={12 2}, pages={ 1060-1066 } } Qunhua Bai, Junlin He, +4 authors C. Yu Published 2016 Biology, Medicine Experimental and therapeutic medicine Parkinson's disease (PD) is the second most common neurodegenerative disorder, which is characterized by the hallmark feature of loss of dopaminergic neurons in the substantia nigra. Energy metabolic disorder is associated with the pathogenesis of PD; however, the development of this disorder is yet to be elucidated. PD-like characteristics have been demonstrated in a rotenone rat model. In the present study, energy metabolism status was investigated in a rat model following intraperitoneal… Expand View on PubMed spandidos-publications.com Save to Library Create Alert Cite Launch Research Feed Share This Paper 3 Citations View All Figures and Topics from this paper figure 1 figure 2 figure 3 figure 4 figure 5 View All 5 Figures & Tables Rotenone Parkinsonian Disorders Lactic acid Parkinson Disease Neurodegenerative Disorders Adenosine Triphosphate Metabolic Diseases Substantia nigra structure Dopamine Hydrochloride Dopaminergic Neurons NADH Tyrosine 3-Monooxygenase Lactate Dehydrogenase 3 Citations Citation Type Citation Type All Types Cites Results Cites Methods Cites Background Has PDF Publication Type Author More Filters More Filters Filters Sort by Relevance Sort by Most Influenced Papers Sort by Citation Count Sort by Recency Docosahexaenoic acid protection in a rotenone induced Parkinson's model: Prevention of tubulin and synaptophysin loss, but no association with mitochondrial function Norma Serrano-García, F. Fernández-Valverde, E. Luis-García, L. Granados-Rojas, A. Jiménez-Anguiano Chemistry, Medicine Neurochemistry International 2018 6 Save Alert Research Feed Bougainvillea spectabilis flowers extract protects against the rotenone-induced toxicity. O. Abdel-Salam, E. Youness, +4 authors D. Abouelfadl Biology, Medicine Asian Pacific journal of tropical medicine 2017 9 Save Alert Research Feed Proteomic changes driven by urban pollution suggest particulate matter as a deregulator of energy metabolism, mitochondrial activity, and oxidative pathways in the rat brain. J. E. Araujo, S. Jorge, +7 authors J. L. Capelo Chemistry, Medicine The Science of the total environment 2019 6 Save Alert Research Feed References SHOWING 1-10 OF 35 REFERENCES SORT BYRelevance Most Influenced Papers Recency A highly reproducible rotenone model of Parkinson's disease J. Cannon, Víctor Tapias, H. Na, Anthony S. Honick, J. T. 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Greene, A. Noorian, S. Srinivasan Medicine Experimental Neurology 2009 132 Save Alert Research Feed ... 1 2 3 4 ... Related Papers Abstract Figures and Topics 3 Citations 35 References Related Papers Stay Connected With Semantic Scholar Sign Up About Semantic Scholar Semantic Scholar is a free, AI-powered research tool for scientific literature, based at the Allen Institute for AI. Learn More → Resources DatasetsSupp.aiAPIOpen Corpus Organization About UsResearchPublishing PartnersData Partners   FAQContact Proudly built by AI2 with the help of our Collaborators Terms of Service•Privacy Policy The Allen Institute for AI By clicking accept or continuing to use the site, you agree to the terms outlined in our Privacy Policy, Terms of Service, and Dataset License ACCEPT & CONTINUE