id	author	title	date	pages	extension	mime	words	sentences	flesch	summary	cache	txt
work_f7ccqu64lvgn3nc4ayevvondua	John M McPartland	Travell trigger points--molecular and osteopathic perspectives	2004	6	.pdf	application/pdf	4374	515	57	The proposed etiology of Travell trigger points (TrPs) has and reporting the results of new approaches using capsaicin, a vanilloid-receptor agonist, and ACh antagonists the etiology of TrPs and the way in which this new hypothesis changes our treatment of these points. The 1999 edition of Travell and Simons' Myofascial Pain and Dysfunction: The Trigger Point Manual5 proposes an "integrated would then activate those receptors in motor end plates, potentially causing TrPs. Single genes may also become dysregulated should they When a motor end plate becomes dysfunctional, several perverse mechanisms cause it to persist as a TrP. Travell and Simons attributed motor end plate dysfunction to an excessive release of acetylcholine (ACh) from the Travell and Simons' revised edition of their manual advocates muscle energy, counterstrain, myofascial release, and inhibits end plate activity by reducing ACh release (a presynaptic mechanism) and by modifing nAChRs (a postsynaptic	./cache/work_f7ccqu64lvgn3nc4ayevvondua.pdf	./txt/work_f7ccqu64lvgn3nc4ayevvondua.txt